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J Biochem Pharmacol Res. 2014 Jun;2(2):54-56.

A new mouse model to study compensatory mechanisms that support normal motor function in Parkinson's disease.

Journal of biochemical and pharmacological research

Xiang Bai, Stacy A Hussong

Affiliations

  1. Department of Pharmacology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA ; Barshop Institute for Longevity and Aging Studies, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA.
  2. Barshop Institute for Longevity and Aging Studies, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA ; Department of Physiology, University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA.

PMID: 26405617 PMCID: PMC4578241

[No abstract available.]

Keywords: Parkinson's disease; compensatory mechanisms; dopamine depletion; motor function; mouse model

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