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Alnahas S, Hagner S, Raifer H, et al. IL-17 and TNF-α Are Key Mediators of . Front Immunol. 2017;8:1562doi: 10.3389/fimmu.2017.01562.
Alnahas, S., Hagner, S., Raifer, H., Kilic, A., Gasteiger, G., Mutters, R., Hellhund, A., Prinz, I., Pinkenburg, O., Visekruna, A., Garn, H., & Steinhoff, U. (2017). IL-17 and TNF-α Are Key Mediators of . Frontiers in immunology, 81562. https://doi.org/10.3389/fimmu.2017.01562
Alnahas, Safa, et al. "IL-17 and TNF-α Are Key Mediators of ." Frontiers in immunology vol. 8 (2017): 1562. doi: https://doi.org/10.3389/fimmu.2017.01562
Alnahas S, Hagner S, Raifer H, Kilic A, Gasteiger G, Mutters R, Hellhund A, Prinz I, Pinkenburg O, Visekruna A, Garn H, Steinhoff U. IL-17 and TNF-α Are Key Mediators of . Front Immunol. 2017 Nov 14;8:1562. doi: 10.3389/fimmu.2017.01562. eCollection 2017. PMID: 29184554; PMCID: PMC5694487.
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Front Immunol. 2017 Nov 14;8:1562. doi: 10.3389/fimmu.2017.01562. eCollection 2017.

IL-17 and TNF-α Are Key Mediators of .

Frontiers in immunology

Safa Alnahas, Stefanie Hagner, Hartmann Raifer, Ayse Kilic, Georg Gasteiger, Reinier Mutters, Anne Hellhund, Immo Prinz, Olaf Pinkenburg, Alexander Visekruna, Holger Garn, Ulrich Steinhoff

Affiliations

  1. Institute of Medical Microbiology and Hospital Hygiene, University of Marburg, Marburg, Germany.
  2. Institute of Laboratory Medicine and Pathobiochemistry, Molecular Diagnostics, Member of the German Center for Lung Research, University of Marburg, Marburg, Germany.
  3. Institute of Medical Microbiology and Hygiene, FZI Research Center for Immunotherapy, University of Mainz Medical Center, Mainz, Germany.
  4. Institute of Immunology, Hannover Medical School, Hannover, Germany.

PMID: 29184554 PMCID: PMC5694487 DOI: 10.3389/fimmu.2017.01562

Abstract

Alterations of the airway microbiome are often associated with pulmonary diseases. For example, detection of the bacterial pathogen

Keywords: IL-17; Moraxellaceae infections; TNF-α; exacerbation of allergic reactions; exacerbation of pulmonary inflammation; infection and allergy; microbial exacerbation of pulmonary inflammation; pulmonary inflammation

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