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Oikonomaki M, Bady P, Hegi ME. Ubiquitin Specific Peptidase 15 (USP15) suppresses glioblastoma cell growth via stabilization of HECTD1 E3 ligase attenuating WNT pathway activity. Oncotarget. 2017;8(66):110490-110502doi: 10.18632/oncotarget.22798.
Oikonomaki, M., Bady, P., & Hegi, M. E. (2017). Ubiquitin Specific Peptidase 15 (USP15) suppresses glioblastoma cell growth via stabilization of HECTD1 E3 ligase attenuating WNT pathway activity. Oncotarget, 8(66), 110490-110502. https://doi.org/10.18632/oncotarget.22798
Oikonomaki, Maria, et al. "Ubiquitin Specific Peptidase 15 (USP15) suppresses glioblastoma cell growth via stabilization of HECTD1 E3 ligase attenuating WNT pathway activity." Oncotarget vol. 8,66 (2017): 110490-110502. doi: https://doi.org/10.18632/oncotarget.22798
Oikonomaki M, Bady P, Hegi ME. Ubiquitin Specific Peptidase 15 (USP15) suppresses glioblastoma cell growth via stabilization of HECTD1 E3 ligase attenuating WNT pathway activity. Oncotarget. 2017 Nov 30;8(66):110490-110502. doi: 10.18632/oncotarget.22798. eCollection 2017 Dec 15. PMID: 29299163; PMCID: PMC5746398.
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Oncotarget. 2017 Nov 30;8(66):110490-110502. doi: 10.18632/oncotarget.22798. eCollection 2017 Dec 15.

Ubiquitin Specific Peptidase 15 (USP15) suppresses glioblastoma cell growth via stabilization of HECTD1 E3 ligase attenuating WNT pathway activity.

Oncotarget

Maria Oikonomaki, Pierre Bady, Monika E Hegi

Affiliations

  1. Department of Clinical Neurosciences, University Hospital Lausanne, Lausanne, Switzerland.
  2. Department of Research and Education, University Hospital Lausanne, Lausanne, Switzerland.
  3. Bioinformatics Core Facility, SIB Swiss Institute of Bioinformatics, Lausanne, Switzerland.

PMID: 29299163 PMCID: PMC5746398 DOI: 10.18632/oncotarget.22798

Abstract

Expression based prediction of new genomic alterations in glioblastoma identified the de-ubiquitinase Ubiquitin Specific Peptidase 15 (

Keywords: HECTD1; USP15; WNT pathway; glioblastoma; tumor suppressor

Conflict of interest statement

CONFLICTS OF INTEREST The authors declare no conflicts of interest.

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