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Gharesouran J, Taheri M, Sayad A, et al. The Growth Arrest-Specific Transcript 5 (GAS5) and Nuclear Receptor Subfamily 3 Group C Member 1 (NR3C1): Novel Markers Involved in Multiple Sclerosis. Int J Mol Cell Med. 2018;7(2):102-110doi: 10.22088/IJMCM.BUMS.7.2.102.
Gharesouran, J., Taheri, M., Sayad, A., Ghafouri-Fard, S., Mazdeh, M., & Omrani, M. D. (2018). The Growth Arrest-Specific Transcript 5 (GAS5) and Nuclear Receptor Subfamily 3 Group C Member 1 (NR3C1): Novel Markers Involved in Multiple Sclerosis. International journal of molecular and cellular medicine, 7(2), 102-110. https://doi.org/10.22088/IJMCM.BUMS.7.2.102
Gharesouran, Jalal, et al. "The Growth Arrest-Specific Transcript 5 (GAS5) and Nuclear Receptor Subfamily 3 Group C Member 1 (NR3C1): Novel Markers Involved in Multiple Sclerosis." International journal of molecular and cellular medicine vol. 7,2 (2018): 102-110. doi: https://doi.org/10.22088/IJMCM.BUMS.7.2.102
Gharesouran J, Taheri M, Sayad A, Ghafouri-Fard S, Mazdeh M, Omrani MD. The Growth Arrest-Specific Transcript 5 (GAS5) and Nuclear Receptor Subfamily 3 Group C Member 1 (NR3C1): Novel Markers Involved in Multiple Sclerosis. Int J Mol Cell Med. 2018;7(2):102-110. doi: 10.22088/IJMCM.BUMS.7.2.102. Epub 2018 May 07. PMID: 30276165; PMCID: PMC6148504.
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Int J Mol Cell Med. 2018;7(2):102-110. doi: 10.22088/IJMCM.BUMS.7.2.102. Epub 2018 May 07.

The Growth Arrest-Specific Transcript 5 (GAS5) and Nuclear Receptor Subfamily 3 Group C Member 1 (NR3C1): Novel Markers Involved in Multiple Sclerosis.

International journal of molecular and cellular medicine

Jalal Gharesouran, Mohammad Taheri, Arezou Sayad, Soudeh Ghafouri-Fard, Mehrdokht Mazdeh, Mir Davood Omrani

Affiliations

  1. Department of Medical Genetics, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
  2. First two authors contributed equally in the writing of the paper.
  3. Urogenital Stem Cell Research Center, Shahid Beheshti University of Medical Sciences, Tehran, Iran.
  4. Neurophysiology Research Center, Hamadan University of Medical Sciences, Hamadan, Iran.

PMID: 30276165 PMCID: PMC6148504 DOI: 10.22088/IJMCM.BUMS.7.2.102

Abstract

Recent studies have revealed that long noncoding RNAs (lncRNAs) are connected with pathogenesis of neurodegenerative diseases. Additionally, glucocorticoids have fundamental regulatory roles on the immune system, and act as potent therapeutic compounds for autoimmune and inflammatory diseases. The long noncoding RNA growth arrest-specific 5 (

Keywords: GAS5; Multiple sclerosis; NR3C1

References

  1. Tumour Biol. 2016 Feb;37(2):2691-702 - PubMed
  2. J Pathol. 2010 Jan;220(2):126-39 - PubMed
  3. Curr Mol Med. 2015;15(1):94-9 - PubMed
  4. Pharmacogenomics J. 2013 Apr;13(2):121-9 - PubMed
  5. Sci Signal. 2010 Feb 02;3(107):ra8 - PubMed
  6. Oncogene. 2009 Jan 15;28(2):195-208 - PubMed
  7. Mol Cell Biol. 1998 Dec;18(12):6897-909 - PubMed
  8. Lancet Oncol. 2010 Nov;11(11):1096-106 - PubMed
  9. Ann Neurol. 2011 Feb;69(2):292-302 - PubMed
  10. Neurol Sci. 2017 Jun;38(6):1093-1099 - PubMed
  11. Biomed Res Int. 2013;2013:358015 - PubMed
  12. Hepatology. 2014 Aug;60(2):744-53 - PubMed
  13. Cytokine. 2017 Aug;96:138-143 - PubMed
  14. Oncol Lett. 2015 Oct;10(4):1953-1958 - PubMed
  15. Pharmacol Res. 2002 May;45(5):361-8 - PubMed
  16. Cell. 1988 Sep 9;54(6):787-93 - PubMed
  17. Mult Scler Relat Disord. 2018 Jan;19:15-19 - PubMed
  18. Steroids. 2005 May-Jun;70(5-7):407-17 - PubMed
  19. Nat Rev Cancer. 2012 Jan 19;12(2):84-8 - PubMed
  20. Endocr Relat Cancer. 2015 Feb;22(1):99-109 - PubMed
  21. J Mol Neurosci. 2017 Dec;63(3-4):333-341 - PubMed
  22. Genes (Basel). 2015 Jul 07;6(3):484-99 - PubMed
  23. PLoS One. 2013;8(1):e55684 - PubMed
  24. Cell Death Dis. 2014 May 22;5:e1243 - PubMed

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