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Solana C, Tarazona R, Solana R. Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer's Disease. Int J Alzheimers Dis. 2018;2018:3128758doi: 10.1155/2018/3128758.
Solana, C., Tarazona, R., & Solana, R. (2018). Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer's Disease. International journal of Alzheimer's disease, 20183128758. https://doi.org/10.1155/2018/3128758
Solana, Corona, et al. "Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer's Disease." International journal of Alzheimer's disease vol. 2018 (2018): 3128758. doi: https://doi.org/10.1155/2018/3128758
Solana C, Tarazona R, Solana R. Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer's Disease. Int J Alzheimers Dis. 2018 Nov 01;2018:3128758. doi: 10.1155/2018/3128758. eCollection 2018. PMID: 30515321; PMCID: PMC6236558.
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Int J Alzheimers Dis. 2018 Nov 01;2018:3128758. doi: 10.1155/2018/3128758. eCollection 2018.

Immunosenescence of Natural Killer Cells, Inflammation, and Alzheimer's Disease.

International journal of Alzheimer's disease

Corona Solana, Raquel Tarazona, Rafael Solana

Affiliations

  1. Centro Hospitalar Psiquiátrico, Lisbon, Portugal.
  2. Immunology Unit, University of Extremadura, Caceres, Spain.
  3. Instituto Maimónides de Investigación Biomédica (IMIBIC), Córdoba, Spain.
  4. Reina Sofia University Hospital, Córdoba, Spain.
  5. University of Cordoba, Córdoba, Spain.

PMID: 30515321 PMCID: PMC6236558 DOI: 10.1155/2018/3128758

Abstract

Alzheimer's disease (AD) represents the most common cause of dementia in the elderly. AD is a neurodegenerative disorder characterized by progressive memory loss and cognitive decline. Although the aetiology of AD is not clear, both environmental factors and heritable predisposition may contribute to disease occurrence. In addition, inflammation and immune system alterations have been linked to AD. The prevailing hypothesis as cause of AD is the deposition in the brain of amyloid beta peptides (A

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