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Wu Z, Liao F, Luo G, et al. NR1D1 Deletion Induces Rupture-Prone Vulnerable Plaques by Regulating Macrophage Pyroptosis via the NF-. Oxid Med Cell Longev. 2021;2021:5217572doi: 10.1155/2021/5217572.
Wu, Z., Liao, F., Luo, G., Qian, Y., He, X., Xu, W., Ding, S., & Pu, J. (2021). NR1D1 Deletion Induces Rupture-Prone Vulnerable Plaques by Regulating Macrophage Pyroptosis via the NF-. Oxidative medicine and cellular longevity, 20215217572. https://doi.org/10.1155/2021/5217572
Wu, Zhinan, et al. "NR1D1 Deletion Induces Rupture-Prone Vulnerable Plaques by Regulating Macrophage Pyroptosis via the NF-." Oxidative medicine and cellular longevity vol. 2021 (2021): 5217572. doi: https://doi.org/10.1155/2021/5217572
Wu Z, Liao F, Luo G, Qian Y, He X, Xu W, Ding S, Pu J. NR1D1 Deletion Induces Rupture-Prone Vulnerable Plaques by Regulating Macrophage Pyroptosis via the NF-. Oxid Med Cell Longev. 2021 Dec 16;2021:5217572. doi: 10.1155/2021/5217572. eCollection 2021. PMID: 34956438; PMCID: PMC8702349.
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Oxid Med Cell Longev. 2021 Dec 16;2021:5217572. doi: 10.1155/2021/5217572. eCollection 2021.

NR1D1 Deletion Induces Rupture-Prone Vulnerable Plaques by Regulating Macrophage Pyroptosis via the NF-.

Oxidative medicine and cellular longevity

Zhinan Wu, Fei Liao, Guqing Luo, Yuxuan Qian, Xinjie He, Wenyi Xu, Song Ding, Jun Pu

Affiliations

  1. Department of Cardiology, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.

PMID: 34956438 PMCID: PMC8702349 DOI: 10.1155/2021/5217572

Abstract

Vulnerable plaque rupture is the main trigger of most acute cardiovascular events. But the underlying mechanisms responsible for the transition from stable to vulnerable plaque remain largely unknown. Nuclear receptor subfamily 1 group D member 1 (NR1D1), also known as REV-ERB

Copyright © 2021 Zhinan Wu et al.

Conflict of interest statement

The authors declare that they have no conflicts of interest.

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