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Mairpady Shambat S, Gómez-Mejia A, Schweizer TA, et al. Hyperinflammatory environment drives dysfunctional myeloid cell effector response to bacterial challenge in COVID-19. PLoS Pathog. 2022;18(1):e1010176doi: 10.1371/journal.ppat.1010176.
Mairpady Shambat, S., Gómez-Mejia, A., Schweizer, T. A., Huemer, M., Chang, C. C., Acevedo, C., Bergada-Pijuan, J., Vulin, C., Hofmaenner, D. A., Scheier, T. C., Hertegonne, S., Parietti, E., Miroshnikova, N., Wendel Garcia, P. D., Hilty, M. P., Buehler, P. K., Schuepbach, R. A., Brugger, S. D., & Zinkernagel, A. S. (2022). Hyperinflammatory environment drives dysfunctional myeloid cell effector response to bacterial challenge in COVID-19. PLoS pathogens, 18(1), e1010176. https://doi.org/10.1371/journal.ppat.1010176
Mairpady Shambat, Srikanth, et al. "Hyperinflammatory environment drives dysfunctional myeloid cell effector response to bacterial challenge in COVID-19." PLoS pathogens vol. 18,1 (2022): e1010176. doi: https://doi.org/10.1371/journal.ppat.1010176
Mairpady Shambat S, Gómez-Mejia A, Schweizer TA, Huemer M, Chang CC, Acevedo C, Bergada-Pijuan J, Vulin C, Hofmaenner DA, Scheier TC, Hertegonne S, Parietti E, Miroshnikova N, Wendel Garcia PD, Hilty MP, Buehler PK, Schuepbach RA, Brugger SD, Zinkernagel AS. Hyperinflammatory environment drives dysfunctional myeloid cell effector response to bacterial challenge in COVID-19. PLoS Pathog. 2022 Jan 10;18(1):e1010176. doi: 10.1371/journal.ppat.1010176. Epub 2022 Jan 10. PMID: 35007290.
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