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Kloner RA, Przyklenk K, Whittaker P, et al. Preconditioning stimuli and inadvertent preconditioning. J Mol Cell Cardiol. 1995;27(2):743-7doi: 10.1016/0022-2828(95)90079-9.
Kloner, R. A., Przyklenk, K., Whittaker, P., & Hale, S. (1995). Preconditioning stimuli and inadvertent preconditioning. Journal of molecular and cellular cardiology, 27(2), 743-7. https://doi.org/10.1016/0022-2828(95)90079-9
Kloner, R A, et al. "Preconditioning stimuli and inadvertent preconditioning." Journal of molecular and cellular cardiology vol. 27,2 (1995): 743-7. doi: https://doi.org/10.1016/0022-2828(95)90079-9
Kloner RA, Przyklenk K, Whittaker P, Hale S. Preconditioning stimuli and inadvertent preconditioning. J Mol Cell Cardiol. 1995 Feb;27(2):743-7. doi: 10.1016/0022-2828(95)90079-9. PMID: 7776379.
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Elsevier Science

J Mol Cell Cardiol. 1995 Feb;27(2):743-7. doi: 10.1016/0022-2828(95)90079-9.

Preconditioning stimuli and inadvertent preconditioning.

Journal of molecular and cellular cardiology

R A Kloner, K Przyklenk, P Whittaker, S Hale

PMID: 7776379 DOI: 10.1016/0022-2828(95)90079-9

Abstract

There are several factors besides brief episodes of total coronary occlusion which can provide sufficient stress to result in a preconditioning-like effect on the size of a myocardial infarction. Partial coronary artery stenosis, hypoxia, stretch, catecholamines, rapid pacing, and certain pharmacologic therapies may provide preconditioning stimuli. These same factors as well as mechanical complications in which a coronary artery is briefly occluded or stenosed prior to a subsequent coronary occlusion may lead to inadvertent preconditioning and confound the results of experimental cardiology studies.

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