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Multifocal calcific periarthritis with distinctive clinical and radiological features in patients with CD73 deficiency.

Rheumatology (Oxford, England)

Cudrici CD, Newman KA, Ferrante EA, Huffstutler R, Carney K, Betancourt B, Miettinen M, Siegel R, Katz JD, Nesti LJ, St Hilaire C, Lakshmipathy D, Wen H, Bagheri MH, Boehm M, Brofferio A.
PMID: 33744914
Rheumatology (Oxford). 2021 Dec 24;61(1):163-173. doi: 10.1093/rheumatology/keab270.

OBJECTIVES: Arterial calcification due to deficiency of CD73 (ACDC) is a hereditary autosomal recessive ectopic mineralization syndrome caused by loss-of-function mutations in the ecto-5'-nucleotidase gene. Periarticular calcification has been reported but the clinical characterization of arthritis as well as...

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Cudrici CD, Newman KA, Ferrante EA, et al. Multifocal calcific periarthritis with distinctive clinical and radiological features in patients with CD73 deficiency. Rheumatology (Oxford). 2021;61(1):163-173doi: 10.1093/rheumatology/keab270.
Cudrici, C. D., Newman, K. A., Ferrante, E. A., Huffstutler, R., Carney, K., Betancourt, B., Miettinen, M., Siegel, R., Katz, J. D., Nesti, L. J., St Hilaire, C., Lakshmipathy, D., Wen, H., Bagheri, M. H., Boehm, M., & Brofferio, A. (2021). Multifocal calcific periarthritis with distinctive clinical and radiological features in patients with CD73 deficiency. Rheumatology (Oxford, England), 61(1), 163-173. https://doi.org/10.1093/rheumatology/keab270
Cudrici, Cornelia D, et al. "Multifocal calcific periarthritis with distinctive clinical and radiological features in patients with CD73 deficiency." Rheumatology (Oxford, England) vol. 61,1 (2021): 163-173. doi: https://doi.org/10.1093/rheumatology/keab270
Cudrici CD, Newman KA, Ferrante EA, Huffstutler R, Carney K, Betancourt B, Miettinen M, Siegel R, Katz JD, Nesti LJ, St Hilaire C, Lakshmipathy D, Wen H, Bagheri MH, Boehm M, Brofferio A. Multifocal calcific periarthritis with distinctive clinical and radiological features in patients with CD73 deficiency. Rheumatology (Oxford). 2021 Dec 24;61(1):163-173. doi: 10.1093/rheumatology/keab270. PMID: 33744914.
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Ecto-5'-Nucleotidase (CD73) Regulates the Survival of CD8+ T Cells.

Frontiers in cell and developmental biology

Rosemblatt MV, Parra-Tello B, Briceño P, Rivas-Yáñez E, Tucer S, Saavedra-Almarza J, Hörmann P, Martínez BA, Lladser Á, Rosemblatt M, Cekic C, Bono MR, Sauma D.
PMID: 33928082
Front Cell Dev Biol. 2021 Apr 13;9:647058. doi: 10.3389/fcell.2021.647058. eCollection 2021.

Ecto-5'-nucleotidase (CD73) is an enzyme present on the surface of tumor cells whose primary described function is the production of extracellular adenosine. Due to the immunosuppressive properties of adenosine, CD73 is being investigated as a target for new antitumor...

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Rosemblatt MV, Parra-Tello B, Briceño P, et al. Ecto-5'-Nucleotidase (CD73) Regulates the Survival of CD8+ T Cells. Front Cell Dev Biol. 2021;9:647058doi: 10.3389/fcell.2021.647058.
Rosemblatt, M. V., Parra-Tello, B., Briceño, P., Rivas-Yáñez, E., Tucer, S., Saavedra-Almarza, J., Hörmann, P., Martínez, B. A., Lladser, �. �., Rosemblatt, M., Cekic, C., Bono, M. R., & Sauma, D. (2021). Ecto-5'-Nucleotidase (CD73) Regulates the Survival of CD8+ T Cells. Frontiers in cell and developmental biology, 9647058. https://doi.org/10.3389/fcell.2021.647058
Rosemblatt, Mariana V, et al. "Ecto-5'-Nucleotidase (CD73) Regulates the Survival of CD8+ T Cells." Frontiers in cell and developmental biology vol. 9 (2021): 647058. doi: https://doi.org/10.3389/fcell.2021.647058
Rosemblatt MV, Parra-Tello B, Briceño P, Rivas-Yáñez E, Tucer S, Saavedra-Almarza J, Hörmann P, Martínez BA, Lladser Á, Rosemblatt M, Cekic C, Bono MR, Sauma D. Ecto-5'-Nucleotidase (CD73) Regulates the Survival of CD8+ T Cells. Front Cell Dev Biol. 2021 Apr 13;9:647058. doi: 10.3389/fcell.2021.647058. eCollection 2021. PMID: 33928082; PMCID: PMC8076893.
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CD73 induces gemcitabine resistance in pancreatic ductal adenocarcinoma: A promising target with non-canonical mechanisms.

Cancer letters

Yu X, Liu W, Wang Z, Wang H, Liu J, Huang C, Zhao T, Wang X, Gao S, Ma Y, Wu L, Li X, Yang S, Hao J.
PMID: 34302921
Cancer Lett. 2021 Oct 28;519:289-303. doi: 10.1016/j.canlet.2021.07.024. Epub 2021 Jul 21.

CD73, a cell surface-localized ecto-5'-nucleotidase, is the major enzymatic source of extracellular adenosine. Canonically, it plays multiple roles in cancer-related processes via its metabolite. As a druggable target, clinical trials targeting CD73 in various malignant diseases are currently ongoing....

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Yu X, Liu W, Wang Z, et al. CD73 induces gemcitabine resistance in pancreatic ductal adenocarcinoma: A promising target with non-canonical mechanisms. Cancer Lett. 2021;519:289-303doi: 10.1016/j.canlet.2021.07.024.
Yu, X., Liu, W., Wang, Z., Wang, H., Liu, J., Huang, C., Zhao, T., Wang, X., Gao, S., Ma, Y., Wu, L., Li, X., Yang, S., & Hao, J. (2021). CD73 induces gemcitabine resistance in pancreatic ductal adenocarcinoma: A promising target with non-canonical mechanisms. Cancer letters, 519289-303. https://doi.org/10.1016/j.canlet.2021.07.024
Yu, Xiaozhou, et al. "CD73 induces gemcitabine resistance in pancreatic ductal adenocarcinoma: A promising target with non-canonical mechanisms." Cancer letters vol. 519 (2021): 289-303. doi: https://doi.org/10.1016/j.canlet.2021.07.024
Yu X, Liu W, Wang Z, Wang H, Liu J, Huang C, Zhao T, Wang X, Gao S, Ma Y, Wu L, Li X, Yang S, Hao J. CD73 induces gemcitabine resistance in pancreatic ductal adenocarcinoma: A promising target with non-canonical mechanisms. Cancer Lett. 2021 Oct 28;519:289-303. doi: 10.1016/j.canlet.2021.07.024. Epub 2021 Jul 21. PMID: 34302921.
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Substrate binding modes of purine and pyrimidine nucleotides to human ecto-5'-nucleotidase (CD73) and inhibition by their bisphosphonic acid derivatives.

Purinergic signalling

Scaletti E, Huschmann FU, Mueller U, Weiss MS, Sträter N.
PMID: 34403084
Purinergic Signal. 2021 Dec;17(4):693-704. doi: 10.1007/s11302-021-09802-w. Epub 2021 Aug 17.

Human ecto-5-nucleotidase (CD73) is involved in purinergic signalling, which influences a diverse range of biological processes. CD73 hydrolyses AMP and is the major control point for the levels of extracellular adenosine. Inhibitors of CD73 thus block the immunosuppressive action...

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Scaletti E, Huschmann FU, Mueller U, et al. Substrate binding modes of purine and pyrimidine nucleotides to human ecto-5'-nucleotidase (CD73) and inhibition by their bisphosphonic acid derivatives. Purinergic Signal. 2021;17(4):693-704doi: 10.1007/s11302-021-09802-w.
Scaletti, E., Huschmann, F. U., Mueller, U., Weiss, M. S., & Sträter, N. (2021). Substrate binding modes of purine and pyrimidine nucleotides to human ecto-5'-nucleotidase (CD73) and inhibition by their bisphosphonic acid derivatives. Purinergic signalling, 17(4), 693-704. https://doi.org/10.1007/s11302-021-09802-w
Scaletti, Emma, et al. "Substrate binding modes of purine and pyrimidine nucleotides to human ecto-5'-nucleotidase (CD73) and inhibition by their bisphosphonic acid derivatives." Purinergic signalling vol. 17,4 (2021): 693-704. doi: https://doi.org/10.1007/s11302-021-09802-w
Scaletti E, Huschmann FU, Mueller U, Weiss MS, Sträter N. Substrate binding modes of purine and pyrimidine nucleotides to human ecto-5'-nucleotidase (CD73) and inhibition by their bisphosphonic acid derivatives. Purinergic Signal. 2021 Dec;17(4):693-704. doi: 10.1007/s11302-021-09802-w. Epub 2021 Aug 17. PMID: 34403084.
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Vanillin modulates activities linked to dysmetabolism in psoas muscle of diabetic rats.

Scientific reports

Salau VF, Erukainure OL, Olofinsan KA, Ijomone OM, Msomi NZ, Islam MS.
PMID: 34548565
Sci Rep. 2021 Sep 21;11(1):18724. doi: 10.1038/s41598-021-98158-7.

Skeletal muscles are important in glucose metabolism and are affected in type 2 diabetes (T2D) and its complications. This study investigated the effect of vanillin on redox imbalance, cholinergic and purinergic dysfunction, and glucose-lipid dysmetabolism in muscles of rats...

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Salau VF, Erukainure OL, Olofinsan KA, et al. Vanillin modulates activities linked to dysmetabolism in psoas muscle of diabetic rats. Sci Rep. 2021;11(1):18724doi: 10.1038/s41598-021-98158-7.
Salau, V. F., Erukainure, O. L., Olofinsan, K. A., Ijomone, O. M., Msomi, N. Z., & Islam, M. S. (2021). Vanillin modulates activities linked to dysmetabolism in psoas muscle of diabetic rats. Scientific reports, 11(1), 18724. https://doi.org/10.1038/s41598-021-98158-7
Salau, Veronica F, et al. "Vanillin modulates activities linked to dysmetabolism in psoas muscle of diabetic rats." Scientific reports vol. 11,1 (2021): 18724. doi: https://doi.org/10.1038/s41598-021-98158-7
Salau VF, Erukainure OL, Olofinsan KA, Ijomone OM, Msomi NZ, Islam MS. Vanillin modulates activities linked to dysmetabolism in psoas muscle of diabetic rats. Sci Rep. 2021 Sep 21;11(1):18724. doi: 10.1038/s41598-021-98158-7. PMID: 34548565; PMCID: PMC8455626.
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Showing 193 to 197 of 197 entries