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Showing 25 to 36 of 110 entries
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Nandrolone Plus Moderate Exercise Increases the Susceptibility to Lethal Arrhythmias.

Research in cardiovascular medicine

Ghorbani Baravati H, Joukar S, Fathpour H, Kordestani Z.
PMID: 26396972
Res Cardiovasc Med. 2015 May 23;4(2):e26233. doi: 10.5812/cardiovascmed.26233v2. eCollection 2015 May.

BACKGROUND: Until now, no experimental study has directly assessed the arrhythmogenesis of chronic consumption of anabolic androgenic steroids along with moderate-intensity endurance exercise.OBJECTIVES: We evaluated the influence of integration of anabolic androgenic steroids along with moderate-intensity endurance exercise on...

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Ghorbani Baravati H, Joukar S, Fathpour H, et al. Nandrolone Plus Moderate Exercise Increases the Susceptibility to Lethal Arrhythmias. Res Cardiovasc Med. 2015;4(2):e26233doi: 10.5812/cardiovascmed.26233v2.
Ghorbani Baravati, H., Joukar, S., Fathpour, H., & Kordestani, Z. (2015). Nandrolone Plus Moderate Exercise Increases the Susceptibility to Lethal Arrhythmias. Research in cardiovascular medicine, 4(2), e26233. https://doi.org/10.5812/cardiovascmed.26233v2
Ghorbani Baravati, Hamideh, et al. "Nandrolone Plus Moderate Exercise Increases the Susceptibility to Lethal Arrhythmias." Research in cardiovascular medicine vol. 4,2 (2015): e26233. doi: https://doi.org/10.5812/cardiovascmed.26233v2
Ghorbani Baravati H, Joukar S, Fathpour H, Kordestani Z. Nandrolone Plus Moderate Exercise Increases the Susceptibility to Lethal Arrhythmias. Res Cardiovasc Med. 2015 May 23;4(2):e26233. doi: 10.5812/cardiovascmed.26233v2. eCollection 2015 May. PMID: 26396972; PMCID: PMC4576214.
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3-Deoxy-aconitine from the root of Aconitum Carmichaeli Debx.

Acta crystallographica. Section E, Structure reports online

Gao F, Zhu SA, Xiong SJ.
PMID: 21579431
Acta Crystallogr Sect E Struct Rep Online. 2010 May 15;66:o1342. doi: 10.1107/S1600536810016922.

THE TITLE COMPOUND (SYSTEMATIC NAME: 8β-acet-oxy-14α-benzo-yloxy-N-ethyl-13β,15α-dihydr-oxy-1α,6α,16β-trimeth-oxy-4β-methoxy-methyl-eneaconitane), C(34)H(47)NO(10), is a typical aconitine-type C(19)-diterpenoid alkaloid, and was isolated from the roots of the Aconitum carmichaeli Debx. The mol-ecule has an aconitine carbon skeleton with four six-membered rings and two five-membered rings,...

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Gao F, Zhu SA, Xiong SJ. 3-Deoxy-aconitine from the root of Aconitum Carmichaeli Debx. Acta Crystallogr Sect E Struct Rep Online. 2010;66:o1342doi: 10.1107/S1600536810016922.
Gao, F., Zhu, S. A., & Xiong, S. J. (2010). 3-Deoxy-aconitine from the root of Aconitum Carmichaeli Debx. Acta crystallographica. Section E, Structure reports online, 66o1342. https://doi.org/10.1107/S1600536810016922
Gao, Feng, et al. "3-Deoxy-aconitine from the root of Aconitum Carmichaeli Debx." Acta crystallographica. Section E, Structure reports online vol. 66 (2010): o1342. doi: https://doi.org/10.1107/S1600536810016922
Gao F, Zhu SA, Xiong SJ. 3-Deoxy-aconitine from the root of Aconitum Carmichaeli Debx. Acta Crystallogr Sect E Struct Rep Online. 2010 May 15;66:o1342. doi: 10.1107/S1600536810016922. PMID: 21579431; PMCID: PMC2979440.
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Involvement of Nrf2-HO-1/JNK-Erk Signaling Pathways in Aconitine-Induced Developmental Toxicity, Oxidative Stress, and ROS-Mitochondrial Apoptosis in Zebrafish Embryos.

Frontiers in pharmacology

Xia Q, Gao S, Rapael Gnanamuthu SR, Zhuang K, Song Z, Zhang Y, Wang X, Tu P, Li J, Liu K.
PMID: 33967776
Front Pharmacol. 2021 Apr 21;12:642480. doi: 10.3389/fphar.2021.642480. eCollection 2021.

Aconitine (AC), one of the bioactive diterpenoid alkaloids extracted from

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Xia Q, Gao S, Rapael Gnanamuthu SR, et al. Involvement of Nrf2-HO-1/JNK-Erk Signaling Pathways in Aconitine-Induced Developmental Toxicity, Oxidative Stress, and ROS-Mitochondrial Apoptosis in Zebrafish Embryos. Front Pharmacol. 2021;12:642480doi: 10.3389/fphar.2021.642480.
Xia, Q., Gao, S., Rapael Gnanamuthu, S. R., Zhuang, K., Song, Z., Zhang, Y., Wang, X., Tu, P., Li, J., & Liu, K. (2021). Involvement of Nrf2-HO-1/JNK-Erk Signaling Pathways in Aconitine-Induced Developmental Toxicity, Oxidative Stress, and ROS-Mitochondrial Apoptosis in Zebrafish Embryos. Frontiers in pharmacology, 12642480. https://doi.org/10.3389/fphar.2021.642480
Xia, Qing, et al. "Involvement of Nrf2-HO-1/JNK-Erk Signaling Pathways in Aconitine-Induced Developmental Toxicity, Oxidative Stress, and ROS-Mitochondrial Apoptosis in Zebrafish Embryos." Frontiers in pharmacology vol. 12 (2021): 642480. doi: https://doi.org/10.3389/fphar.2021.642480
Xia Q, Gao S, Rapael Gnanamuthu SR, Zhuang K, Song Z, Zhang Y, Wang X, Tu P, Li J, Liu K. Involvement of Nrf2-HO-1/JNK-Erk Signaling Pathways in Aconitine-Induced Developmental Toxicity, Oxidative Stress, and ROS-Mitochondrial Apoptosis in Zebrafish Embryos. Front Pharmacol. 2021 Apr 21;12:642480. doi: 10.3389/fphar.2021.642480. eCollection 2021. PMID: 33967776; PMCID: PMC8097150.
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Radiation-Associated Coronary Disease in Young Cancer Survivors: The Beat Goes On; We Must Preserve It.

JACC. CardioOncology

Bates JE, Chen MH, Constine LS.
PMID: 34604799
JACC CardioOncol. 2021 Sep 21;3(3):393-396. doi: 10.1016/j.jaccao.2021.08.002. eCollection 2021 Sep.

No abstract available.

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Bates JE, Chen MH, Constine LS. Radiation-Associated Coronary Disease in Young Cancer Survivors: The Beat Goes On; We Must Preserve It. JACC CardioOncol. 2021;3(3):393-396doi: 10.1016/j.jaccao.2021.08.002.
Bates, J. E., Chen, M. H., & Constine, L. S. (2021). Radiation-Associated Coronary Disease in Young Cancer Survivors: The Beat Goes On; We Must Preserve It. JACC. CardioOncology, 3(3), 393-396. https://doi.org/10.1016/j.jaccao.2021.08.002
Bates, James E, et al. "Radiation-Associated Coronary Disease in Young Cancer Survivors: The Beat Goes On; We Must Preserve It." JACC. CardioOncology vol. 3,3 (2021): 393-396. doi: https://doi.org/10.1016/j.jaccao.2021.08.002
Bates JE, Chen MH, Constine LS. Radiation-Associated Coronary Disease in Young Cancer Survivors: The Beat Goes On; We Must Preserve It. JACC CardioOncol. 2021 Sep 21;3(3):393-396. doi: 10.1016/j.jaccao.2021.08.002. eCollection 2021 Sep. PMID: 34604799; PMCID: PMC8463725.
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Therapeutic efficacy of catalpol against apoptosis in cardiomyocytes derived from human induced pluripotent stem cells.

AMB Express

Yang L, Feng X, Li Y, Zhang S, Ying Y.
PMID: 32198560
AMB Express. 2020 Mar 20;10(1):56. doi: 10.1186/s13568-020-00986-9.

Cardiac arrhythmia is an irregular heart rhythm that can lead to serious heart conditions and various organ disorders, and may cause sudden cardiac death. Catalpol belongs to the iridoid glycoside family and is highly abundant in Rehmannia glutinosa Libosch....

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Yang L, Feng X, Li Y, et al. Therapeutic efficacy of catalpol against apoptosis in cardiomyocytes derived from human induced pluripotent stem cells. AMB Express. 2020;10(1):56doi: 10.1186/s13568-020-00986-9.
Yang, L., Feng, X., Li, Y., Zhang, S., & Ying, Y. (2020). Therapeutic efficacy of catalpol against apoptosis in cardiomyocytes derived from human induced pluripotent stem cells. AMB Express, 10(1), 56. https://doi.org/10.1186/s13568-020-00986-9
Yang, Lingchao, et al. "Therapeutic efficacy of catalpol against apoptosis in cardiomyocytes derived from human induced pluripotent stem cells." AMB Express vol. 10,1 (2020): 56. doi: https://doi.org/10.1186/s13568-020-00986-9
Yang L, Feng X, Li Y, Zhang S, Ying Y. Therapeutic efficacy of catalpol against apoptosis in cardiomyocytes derived from human induced pluripotent stem cells. AMB Express. 2020 Mar 20;10(1):56. doi: 10.1186/s13568-020-00986-9. PMID: 32198560; PMCID: PMC7083986.
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Cancer inhibition mechanism of lung cancer mouse model based on dye trace method.

Saudi journal of biological sciences

Zhang W, Shu H, Fang L, Tang N, Li Y, Guo B, Meng F.
PMID: 32256178
Saudi J Biol Sci. 2020 Apr;27(4):1155-1162. doi: 10.1016/j.sjbs.2019.12.040. Epub 2020 Jan 03.

To minimize the incidence and mortality of cancer, dye trace method was used to explore the mechanism of drug inhibition. 60 mice were selected as the research objects and randomly divided into five groups: model group, shikonin group, aconitine...

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Zhang W, Shu H, Fang L, et al. Cancer inhibition mechanism of lung cancer mouse model based on dye trace method. Saudi J Biol Sci. 2020;27(4):1155-1162doi: 10.1016/j.sjbs.2019.12.040.
Zhang, W., Shu, H., Fang, L., Tang, N., Li, Y., Guo, B., & Meng, F. (2020). Cancer inhibition mechanism of lung cancer mouse model based on dye trace method. Saudi journal of biological sciences, 27(4), 1155-1162. https://doi.org/10.1016/j.sjbs.2019.12.040
Zhang, Wei, et al. "Cancer inhibition mechanism of lung cancer mouse model based on dye trace method." Saudi journal of biological sciences vol. 27,4 (2020): 1155-1162. doi: https://doi.org/10.1016/j.sjbs.2019.12.040
Zhang W, Shu H, Fang L, Tang N, Li Y, Guo B, Meng F. Cancer inhibition mechanism of lung cancer mouse model based on dye trace method. Saudi J Biol Sci. 2020 Apr;27(4):1155-1162. doi: 10.1016/j.sjbs.2019.12.040. Epub 2020 Jan 03. PMID: 32256178; PMCID: PMC7105657.
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Quantitative LC-MS/MS analysis of seven ginsenosides and three aconitum alkaloids in Shen-Fu decoction.

Chemistry Central journal

Guo N, Liu M, Yang D, Huang Y, Niu X, Wu R, Liu Y, Ma G, Dou D.
PMID: 24107599
Chem Cent J. 2013 Oct 10;7(1):165. doi: 10.1186/1752-153X-7-165.

BACKGROUND: Shen-Fu decoction is a traditional Chinese medicine prescription with a 3:2 ratio of Radix Ginseng and Fuzi (Radix Aconiti lateralis praeparata). Ginsenosides and alkaloids are considered to be the main active components of Shen-Fu decoction. However, no analytical...

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Guo N, Liu M, Yang D, et al. Quantitative LC-MS/MS analysis of seven ginsenosides and three aconitum alkaloids in Shen-Fu decoction. Chem Cent J. 2013;7(1):165doi: 10.1186/1752-153X-7-165.
Guo, N., Liu, M., Yang, D., Huang, Y., Niu, X., Wu, R., Liu, Y., Ma, G., & Dou, D. (2013). Quantitative LC-MS/MS analysis of seven ginsenosides and three aconitum alkaloids in Shen-Fu decoction. Chemistry Central journal, 7(1), 165. https://doi.org/10.1186/1752-153X-7-165
Guo, Na, et al. "Quantitative LC-MS/MS analysis of seven ginsenosides and three aconitum alkaloids in Shen-Fu decoction." Chemistry Central journal vol. 7,1 (2013): 165. doi: https://doi.org/10.1186/1752-153X-7-165
Guo N, Liu M, Yang D, Huang Y, Niu X, Wu R, Liu Y, Ma G, Dou D. Quantitative LC-MS/MS analysis of seven ginsenosides and three aconitum alkaloids in Shen-Fu decoction. Chem Cent J. 2013 Oct 10;7(1):165. doi: 10.1186/1752-153X-7-165. PMID: 24107599; PMCID: PMC3853001.
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On the Preparation and Medicinal Employment of Aconitine.

The Medico-chirurgical review

[No authors listed]
PMID: 29918094
Med Chir Rev. 1835 Apr 01;22(44):434-436.

No abstract available.

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On the Preparation and Medicinal Employment of Aconitine. Med Chir Rev. 1835;22(44):434-436
(1835). On the Preparation and Medicinal Employment of Aconitine. The Medico-chirurgical review, 22(44), 434-436.
"On the Preparation and Medicinal Employment of Aconitine." The Medico-chirurgical review vol. 22,44 (1835): 434-436.
On the Preparation and Medicinal Employment of Aconitine. Med Chir Rev. 1835 Apr 01;22(44):434-436. PMID: 29918094; PMCID: PMC5089043.
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Aconitine inhibits the proliferation of hepatocellular carcinoma by inducing apoptosis.

International journal of clinical and experimental pathology

Qi X, Wang L, Wang H, Yang L, Li X, Wang L.
PMID: 31949608
Int J Clin Exp Pathol. 2018 Nov 01;11(11):5278-5289. eCollection 2018.

Hepatocellular carcinoma (HCC) is the primary liver cancer that occurs with a high incidence in Asia. Owing to the poor prognosis of the disease, the mortality rate remains high, making HCC the third most common cause of cancer-related mortality...

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Qi X, Wang L, Wang H, et al. Aconitine inhibits the proliferation of hepatocellular carcinoma by inducing apoptosis. Int J Clin Exp Pathol. 2018;11(11):5278-5289
Qi, X., Wang, L., Wang, H., Yang, L., Li, X., & Wang, L. (2018). Aconitine inhibits the proliferation of hepatocellular carcinoma by inducing apoptosis. International journal of clinical and experimental pathology, 11(11), 5278-5289.
Qi, Xiuzhong, et al. "Aconitine inhibits the proliferation of hepatocellular carcinoma by inducing apoptosis." International journal of clinical and experimental pathology vol. 11,11 (2018): 5278-5289.
Qi X, Wang L, Wang H, Yang L, Li X, Wang L. Aconitine inhibits the proliferation of hepatocellular carcinoma by inducing apoptosis. Int J Clin Exp Pathol. 2018 Nov 01;11(11):5278-5289. eCollection 2018. PMID: 31949608; PMCID: PMC6963040.
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Diterpenoid Alkaloids from the Aerial Parts of Aconitum flavum Hand.-Mazz.

Natural products and bioprospecting

Zhang N, Xia F, Li SY, Nian Y, Wei LX, Xu G.
PMID: 33861417
Nat Prod Bioprospect. 2021 Aug;11(4):421-429. doi: 10.1007/s13659-021-00302-3. Epub 2021 Apr 16.

Sixteen diterpenoid alkaloids (DAs), including six aconitine-type alkaloids (5 and 9 - 13), seven 7,17-seco-aconitine-type alkaloids (1 - 4, 6 - 8), two napelline-type alkaloids (14 and 15) as well as one veatchine-type alkaloid (16), were isolated from the...

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Zhang N, Xia F, Li SY, et al. Diterpenoid Alkaloids from the Aerial Parts of Aconitum flavum Hand.-Mazz. Nat Prod Bioprospect. 2021;11(4):421-429doi: 10.1007/s13659-021-00302-3.
Zhang, N., Xia, F., Li, S. Y., Nian, Y., Wei, L. X., & Xu, G. (2021). Diterpenoid Alkaloids from the Aerial Parts of Aconitum flavum Hand.-Mazz. Natural products and bioprospecting, 11(4), 421-429. https://doi.org/10.1007/s13659-021-00302-3
Zhang, Na, et al. "Diterpenoid Alkaloids from the Aerial Parts of Aconitum flavum Hand.-Mazz." Natural products and bioprospecting vol. 11,4 (2021): 421-429. doi: https://doi.org/10.1007/s13659-021-00302-3
Zhang N, Xia F, Li SY, Nian Y, Wei LX, Xu G. Diterpenoid Alkaloids from the Aerial Parts of Aconitum flavum Hand.-Mazz. Nat Prod Bioprospect. 2021 Aug;11(4):421-429. doi: 10.1007/s13659-021-00302-3. Epub 2021 Apr 16. PMID: 33861417; PMCID: PMC8275754.
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Radiation Therapy Across Pediatric Hodgkin Lymphoma Research Group Protocols: A Report From the Staging, Evaluation, and Response Criteria Harmonization (SEARCH) for Childhood, Adolescent, and Young Adult Hodgkin Lymphoma (CAYAHL) Group.

International journal of radiation oncology, biology, physics

Hall MD, Terezakis SA, Lucas JT, Gallop-Evans E, Dieckmann K, Constine LS, Hodgson D, Flerlage JE, Metzger ML, Hoppe BS.
PMID: 34390770
Int J Radiat Oncol Biol Phys. 2022 Feb 01;112(2):317-334. doi: 10.1016/j.ijrobp.2021.07.1716. Epub 2021 Aug 12.

No abstract available.

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Hall MD, Terezakis SA, Lucas JT, et al. Radiation Therapy Across Pediatric Hodgkin Lymphoma Research Group Protocols: A Report From the Staging, Evaluation, and Response Criteria Harmonization (SEARCH) for Childhood, Adolescent, and Young Adult Hodgkin Lymphoma (CAYAHL) Group. Int J Radiat Oncol Biol Phys. 2021;112(2):317-334doi: 10.1016/j.ijrobp.2021.07.1716.
Hall, M. D., Terezakis, S. A., Lucas, J. T., Gallop-Evans, E., Dieckmann, K., Constine, L. S., Hodgson, D., Flerlage, J. E., Metzger, M. L., & Hoppe, B. S. (2022). Radiation Therapy Across Pediatric Hodgkin Lymphoma Research Group Protocols: A Report From the Staging, Evaluation, and Response Criteria Harmonization (SEARCH) for Childhood, Adolescent, and Young Adult Hodgkin Lymphoma (CAYAHL) Group. International journal of radiation oncology, biology, physics, 112(2), 317-334. https://doi.org/10.1016/j.ijrobp.2021.07.1716
Hall, Matthew D, et al. "Radiation Therapy Across Pediatric Hodgkin Lymphoma Research Group Protocols: A Report From the Staging, Evaluation, and Response Criteria Harmonization (SEARCH) for Childhood, Adolescent, and Young Adult Hodgkin Lymphoma (CAYAHL) Group." International journal of radiation oncology, biology, physics vol. 112,2 (2022): 317-334. doi: https://doi.org/10.1016/j.ijrobp.2021.07.1716
Hall MD, Terezakis SA, Lucas JT, Gallop-Evans E, Dieckmann K, Constine LS, Hodgson D, Flerlage JE, Metzger ML, Hoppe BS. Radiation Therapy Across Pediatric Hodgkin Lymphoma Research Group Protocols: A Report From the Staging, Evaluation, and Response Criteria Harmonization (SEARCH) for Childhood, Adolescent, and Young Adult Hodgkin Lymphoma (CAYAHL) Group. Int J Radiat Oncol Biol Phys. 2022 Feb 01;112(2):317-334. doi: 10.1016/j.ijrobp.2021.07.1716. Epub 2021 Aug 12. PMID: 34390770.
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New dosimetric guidelines for linear Boltzmann transport equations through comparative evaluation of stereotactic body radiation therapy for lung treatment planning.

Journal of applied clinical medical physics

Webster M, Tanny S, Joyce N, Herman A, Chen Y, Milano M, Usuki K, Constine L, Singh D, Yeo I.
PMID: 34783438
J Appl Clin Med Phys. 2021 Dec;22(12):115-124. doi: 10.1002/acm2.13464. Epub 2021 Nov 16.

PURPOSE: To propose guidelines for lung stereotactic body radiation therapy (SBRT) when using Acuros XB (AXB) equivalent to the existing ones developed for convolution algorithms such as analytic anisotropic algorithm (AAA), considering the difference between the algorithms.METHODS: A retrospective...

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Webster M, Tanny S, Joyce N, et al. New dosimetric guidelines for linear Boltzmann transport equations through comparative evaluation of stereotactic body radiation therapy for lung treatment planning. J Appl Clin Med Phys. 2021;22(12):115-124doi: 10.1002/acm2.13464.
Webster, M., Tanny, S., Joyce, N., Herman, A., Chen, Y., Milano, M., Usuki, K., Constine, L., Singh, D., & Yeo, I. (2021). New dosimetric guidelines for linear Boltzmann transport equations through comparative evaluation of stereotactic body radiation therapy for lung treatment planning. Journal of applied clinical medical physics, 22(12), 115-124. https://doi.org/10.1002/acm2.13464
Webster, Matthew, et al. "New dosimetric guidelines for linear Boltzmann transport equations through comparative evaluation of stereotactic body radiation therapy for lung treatment planning." Journal of applied clinical medical physics vol. 22,12 (2021): 115-124. doi: https://doi.org/10.1002/acm2.13464
Webster M, Tanny S, Joyce N, Herman A, Chen Y, Milano M, Usuki K, Constine L, Singh D, Yeo I. New dosimetric guidelines for linear Boltzmann transport equations through comparative evaluation of stereotactic body radiation therapy for lung treatment planning. J Appl Clin Med Phys. 2021 Dec;22(12):115-124. doi: 10.1002/acm2.13464. Epub 2021 Nov 16. PMID: 34783438.
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Showing 25 to 36 of 110 entries