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Binding Sites for Vasopressin in the Human Pituitary are Associated with Corticotrophs and may Differ from Other Known Vasopressin Receptors.

Journal of neuroendocrinology

Pasquier D, Loup F, Dubois-Dauphin M, Dreifuss JJ, Tribollet E.
PMID: 19215459
J Neuroendocrinol. 1991 Jun 01;3(3):237-47. doi: 10.1111/j.1365-2826.1991.tb00270.x.

Abstract In view of the fact that Vasopressin can induce pituitary adrenocorticotrophin release, we performed an autoradiographical study of [(3)H]arginine vasopressin binding in human pituitary tissue obtained post-mortem from adults and foetuses. Sites of specific, high affinity binding (IC(50)...

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Pasquier D, Loup F, Dubois-Dauphin M, et al. Binding Sites for Vasopressin in the Human Pituitary are Associated with Corticotrophs and may Differ from Other Known Vasopressin Receptors. J Neuroendocrinol. 1991;3(3):237-47doi: 10.1111/j.1365-2826.1991.tb00270.x.
Pasquier, D., Loup, F., Dubois-Dauphin, M., Dreifuss, J. J., & Tribollet, E. (1991). Binding Sites for Vasopressin in the Human Pituitary are Associated with Corticotrophs and may Differ from Other Known Vasopressin Receptors. Journal of neuroendocrinology, 3(3), 237-47. https://doi.org/10.1111/j.1365-2826.1991.tb00270.x
Pasquier, D, et al. "Binding Sites for Vasopressin in the Human Pituitary are Associated with Corticotrophs and may Differ from Other Known Vasopressin Receptors." Journal of neuroendocrinology vol. 3,3 (1991): 237-47. doi: https://doi.org/10.1111/j.1365-2826.1991.tb00270.x
Pasquier D, Loup F, Dubois-Dauphin M, Dreifuss JJ, Tribollet E. Binding Sites for Vasopressin in the Human Pituitary are Associated with Corticotrophs and may Differ from Other Known Vasopressin Receptors. J Neuroendocrinol. 1991 Jun 01;3(3):237-47. doi: 10.1111/j.1365-2826.1991.tb00270.x. PMID: 19215459.
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Vasopressinergic innervation of the bovine pineal gland: is there a local source for arginine vasopressin?.

Molecular and cellular neurosciences

Olcese J, Sinemus C, Ivell R.
PMID: 19912907
Mol Cell Neurosci. 1993 Feb;4(1):47-54. doi: 10.1006/mcne.1993.1006.

Although the presence of arginine vasopressin (AVP) in the mammalian pineal has been characterized biochemically, the source of this nonapeptide hormone remains enigmatic. Most earlier data pointed to an extrapineal origin, although some recent evidence suggests intrapineal synthesis of...

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Olcese J, Sinemus C, Ivell R. Vasopressinergic innervation of the bovine pineal gland: is there a local source for arginine vasopressin?. Mol Cell Neurosci. 1993;4(1):47-54doi: 10.1006/mcne.1993.1006.
Olcese, J., Sinemus, C., & Ivell, R. (1993). Vasopressinergic innervation of the bovine pineal gland: is there a local source for arginine vasopressin?. Molecular and cellular neurosciences, 4(1), 47-54. https://doi.org/10.1006/mcne.1993.1006
Olcese, J, et al. "Vasopressinergic innervation of the bovine pineal gland: is there a local source for arginine vasopressin?." Molecular and cellular neurosciences vol. 4,1 (1993): 47-54. doi: https://doi.org/10.1006/mcne.1993.1006
Olcese J, Sinemus C, Ivell R. Vasopressinergic innervation of the bovine pineal gland: is there a local source for arginine vasopressin?. Mol Cell Neurosci. 1993 Feb;4(1):47-54. doi: 10.1006/mcne.1993.1006. PMID: 19912907.
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Tolvaptan: the evidence for its therapeutic value in acute heart failure syndrome.

Core evidence

Aghel A, Tang WH.
PMID: 20694082
Core Evid. 2008 Jun;3(1):31-43. doi: 10.3355/ce.2008.010.

INTRODUCTION: Acute heart failure syndrome (AHFS) is one of the leading causes of hospital admission in the US. Tolvaptan is a vasopressin V(2) receptor antagonist that blocks the effect of arginine vasopressin (AVP) in reabsorbing water from the collecting...

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Aghel A, Tang WH. Tolvaptan: the evidence for its therapeutic value in acute heart failure syndrome. Core Evid. 2008;3(1):31-43doi: 10.3355/ce.2008.010.
Aghel, A., & Tang, W. H. (2008). Tolvaptan: the evidence for its therapeutic value in acute heart failure syndrome. Core evidence, 3(1), 31-43. https://doi.org/10.3355/ce.2008.010
Aghel, Arash, and Tang, W H Wilson. "Tolvaptan: the evidence for its therapeutic value in acute heart failure syndrome." Core evidence vol. 3,1 (2008): 31-43. doi: https://doi.org/10.3355/ce.2008.010
Aghel A, Tang WH. Tolvaptan: the evidence for its therapeutic value in acute heart failure syndrome. Core Evid. 2008 Jun;3(1):31-43. doi: 10.3355/ce.2008.010. PMID: 20694082; PMCID: PMC2899804.
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Basic FGF partially prevents degeneration of paraventricular vasopressin neurons after hypophysectomy.

Restorative neurology and neuroscience

Takeichi Y, Nakasu Y, Tooyama I, Kimura H.
PMID: 21551516
Restor Neurol Neurosci. 1996 Jan 01;10(3):161-6. doi: 10.3233/RNN-1996-10304.

This experiment was designed to determine if basic fibroblast growth factor (bFGF) had neurotrophic effects on vasopressin neurons after hypophysectomy. Adult male Sprague-Dawley rats received 1 μg bFGF (bFGF group) or 0.1% bovine serum albumin (BSA group) to the...

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Takeichi Y, Nakasu Y, Tooyama I, et al. Basic FGF partially prevents degeneration of paraventricular vasopressin neurons after hypophysectomy. Restor Neurol Neurosci. 1996;10(3):161-6doi: 10.3233/RNN-1996-10304.
Takeichi, Y., Nakasu, Y., Tooyama, I., & Kimura, H. (1996). Basic FGF partially prevents degeneration of paraventricular vasopressin neurons after hypophysectomy. Restorative neurology and neuroscience, 10(3), 161-6. https://doi.org/10.3233/RNN-1996-10304
Takeichi, Y, et al. "Basic FGF partially prevents degeneration of paraventricular vasopressin neurons after hypophysectomy." Restorative neurology and neuroscience vol. 10,3 (1996): 161-6. doi: https://doi.org/10.3233/RNN-1996-10304
Takeichi Y, Nakasu Y, Tooyama I, Kimura H. Basic FGF partially prevents degeneration of paraventricular vasopressin neurons after hypophysectomy. Restor Neurol Neurosci. 1996 Jan 01;10(3):161-6. doi: 10.3233/RNN-1996-10304. PMID: 21551516.
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Anatomical Localization of Corticotropin-Releasing Factor and Arginine Vasopressin in the Human Hypothalamus; the Effect of Corticosteroids on their Concentrations in Human and Rat Hypothalami.

Journal of neuroendocrinology

Pralong FP, Linton EA, Favrod-Coune CA, Lowry PJ, Muller AF, Gaillard RC.
PMID: 19215362
J Neuroendocrinol. 1990 Jun 01;2(3):369-74. doi: 10.1111/j.1365-2826.1990.tb00420.x.

Abstract In this study, we have determined the distribution of corticotropin-releasing factor and vasopressin in the human hypothalamus, and investigated the effect of glucocorticoid administration on the concentrations of both peptides. Corticotropin-releasing factor and vasopressin were measured by a...

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Pralong FP, Linton EA, Favrod-Coune CA, et al. Anatomical Localization of Corticotropin-Releasing Factor and Arginine Vasopressin in the Human Hypothalamus; the Effect of Corticosteroids on their Concentrations in Human and Rat Hypothalami. J Neuroendocrinol. 1990;2(3):369-74doi: 10.1111/j.1365-2826.1990.tb00420.x.
Pralong, F. P., Linton, E. A., Favrod-Coune, C. A., Lowry, P. J., Muller, A. F., & Gaillard, R. C. (1990). Anatomical Localization of Corticotropin-Releasing Factor and Arginine Vasopressin in the Human Hypothalamus; the Effect of Corticosteroids on their Concentrations in Human and Rat Hypothalami. Journal of neuroendocrinology, 2(3), 369-74. https://doi.org/10.1111/j.1365-2826.1990.tb00420.x
Pralong, F P, et al. "Anatomical Localization of Corticotropin-Releasing Factor and Arginine Vasopressin in the Human Hypothalamus; the Effect of Corticosteroids on their Concentrations in Human and Rat Hypothalami." Journal of neuroendocrinology vol. 2,3 (1990): 369-74. doi: https://doi.org/10.1111/j.1365-2826.1990.tb00420.x
Pralong FP, Linton EA, Favrod-Coune CA, Lowry PJ, Muller AF, Gaillard RC. Anatomical Localization of Corticotropin-Releasing Factor and Arginine Vasopressin in the Human Hypothalamus; the Effect of Corticosteroids on their Concentrations in Human and Rat Hypothalami. J Neuroendocrinol. 1990 Jun 01;2(3):369-74. doi: 10.1111/j.1365-2826.1990.tb00420.x. PMID: 19215362.
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A treatment protocol including vasopressin and hydroxyethyl starch solution is associated with increased rate of return of spontaneous circulation in blunt trauma patients with pulseless electrical activity.

International journal of emergency medicine

Grmec S, Strnad M, Cander D, Mally S.
PMID: 19384647
Int J Emerg Med. 2008 Dec;1(4):311-6. doi: 10.1007/s12245-008-0073-8. Epub 2008 Nov 12.

BACKGROUND: Survival after cardiopulmonary resuscitation (CPR) using standard vasopressor therapy is disappointing. Vasopressin is a potent vasopressor that could become a useful therapeutic alternative in the treatment of cardiac arrest.AIMS: The aim of this prehospital prospective cohort study was...

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Grmec S, Strnad M, Cander D, et al. A treatment protocol including vasopressin and hydroxyethyl starch solution is associated with increased rate of return of spontaneous circulation in blunt trauma patients with pulseless electrical activity. Int J Emerg Med. 2008;1(4):311-6doi: 10.1007/s12245-008-0073-8.
Grmec, S., Strnad, M., Cander, D., & Mally, S. (2008). A treatment protocol including vasopressin and hydroxyethyl starch solution is associated with increased rate of return of spontaneous circulation in blunt trauma patients with pulseless electrical activity. International journal of emergency medicine, 1(4), 311-6. https://doi.org/10.1007/s12245-008-0073-8
Grmec, Stefek, et al. "A treatment protocol including vasopressin and hydroxyethyl starch solution is associated with increased rate of return of spontaneous circulation in blunt trauma patients with pulseless electrical activity." International journal of emergency medicine vol. 1,4 (2008): 311-6. doi: https://doi.org/10.1007/s12245-008-0073-8
Grmec S, Strnad M, Cander D, Mally S. A treatment protocol including vasopressin and hydroxyethyl starch solution is associated with increased rate of return of spontaneous circulation in blunt trauma patients with pulseless electrical activity. Int J Emerg Med. 2008 Dec;1(4):311-6. doi: 10.1007/s12245-008-0073-8. Epub 2008 Nov 12. PMID: 19384647; PMCID: PMC2657262.
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Real-time Imaging of Ca-handling in Intact Renal Glomeruli Using Confocal Microscopy.

Medical hypotheses and research : MHR

Ghayur MN, Janssen LJ.
PMID: 22287941
Med Hypotheses Res. 2009 Jul;5(1):47-56.

Glomeruli are filtering units in the kidneys. Being multicellular and complex in structure, many aspects of glomerular function are yet to be elucidated. Most studies use glomerular cells in culture, which may exhibit altered physiology compared to native cells....

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Ghayur MN, Janssen LJ. Real-time Imaging of Ca-handling in Intact Renal Glomeruli Using Confocal Microscopy. Med Hypotheses Res. 2009;5(1):47-56
Ghayur, M. N., & Janssen, L. J. (2009). Real-time Imaging of Ca-handling in Intact Renal Glomeruli Using Confocal Microscopy. Medical hypotheses and research : MHR, 5(1), 47-56.
Ghayur, Muhammad Nabeel, and Janssen, Luke Jeffrey. "Real-time Imaging of Ca-handling in Intact Renal Glomeruli Using Confocal Microscopy." Medical hypotheses and research : MHR vol. 5,1 (2009): 47-56.
Ghayur MN, Janssen LJ. Real-time Imaging of Ca-handling in Intact Renal Glomeruli Using Confocal Microscopy. Med Hypotheses Res. 2009 Jul;5(1):47-56. PMID: 22287941; PMCID: PMC3266942.
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Analysis of a novel AVPR2 mutation in a family with nephrogenic diabetes insipidus.

International journal of clinical and experimental medicine

Moon SD, Kim JH, Shim JY, Lim DJ, Cha BY, Han JH.
PMID: 21394280
Int J Clin Exp Med. 2011;4(1):1-9. Epub 2010 Nov 30.

Congenital nephrogenic diabetes insipidus (NDI) is a rare X-linked recessive disorder associated with germ-line mutations of the arginine vasopressin (AVP) receptor type 2 (AVPR2) gene. Recent molecular studies have demonstrated that insensitivity of renal tubule cells to AVP is...

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Moon SD, Kim JH, Shim JY, et al. Analysis of a novel AVPR2 mutation in a family with nephrogenic diabetes insipidus. Int J Clin Exp Med. 2010;4(1):1-9
Moon, S. D., Kim, J. H., Shim, J. Y., Lim, D. J., Cha, B. Y., & Han, J. H. (2011). Analysis of a novel AVPR2 mutation in a family with nephrogenic diabetes insipidus. International journal of clinical and experimental medicine, 4(1), 1-9.
Moon, Sung-Dae, et al. "Analysis of a novel AVPR2 mutation in a family with nephrogenic diabetes insipidus." International journal of clinical and experimental medicine vol. 4,1 (2011): 1-9.
Moon SD, Kim JH, Shim JY, Lim DJ, Cha BY, Han JH. Analysis of a novel AVPR2 mutation in a family with nephrogenic diabetes insipidus. Int J Clin Exp Med. 2011;4(1):1-9. Epub 2010 Nov 30. PMID: 21394280; PMCID: PMC3048978.
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[Rhinoplasty in a patient with Von Willebrand disease: case report.].

Revista brasileira de anestesiologia

Matos Junior RM, Godoy Rda C, Gobbo Mda C, Lian Junior J, Duz GL.
PMID: 19462145
Rev Bras Anestesiol. 2007 Dec;57(6):678-83. doi: 10.1590/s0034-70942007000600011.

BACKGROUND AND OBJECTIVES: Patients with von Willebrand disease present abnormal bleeding after being wounded or during surgeries since it affects primary and secondary hemostasia due to changes in factor VIII. The objective of this report was to elucidate the...

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Matos Junior RM, Godoy Rda C, Gobbo Mda C, et al. Rinoplastia em paciente com doença de Von Willebrand: relato de caso. [Rhinoplasty in a patient with Von Willebrand disease: case report.]. Rev Bras Anestesiol. 2007;57(6):678-83doi: 10.1590/s0034-70942007000600011.
Matos Junior, R. M., Godoy, R. d. a. . C., Gobbo, M. d. a. . C., Lian Junior, J., & Duz, G. L. (2007). Rinoplastia em paciente com doença de Von Willebrand: relato de caso. [Rhinoplasty in a patient with Von Willebrand disease: case report.]. Revista brasileira de anestesiologia, 57(6), 678-83. https://doi.org/10.1590/s0034-70942007000600011
Matos Junior, Roberto Martins, et al. "Rinoplastia em paciente com doença de Von Willebrand: relato de caso." [Rhinoplasty in a patient with Von Willebrand disease: case report.]. Revista brasileira de anestesiologia vol. 57,6 (2007): 678-83. doi: https://doi.org/10.1590/s0034-70942007000600011
Matos Junior RM, Godoy Rda C, Gobbo Mda C, Lian Junior J, Duz GL. Rinoplastia em paciente com doença de Von Willebrand: relato de caso. [Rhinoplasty in a patient with Von Willebrand disease: case report.]. Rev Bras Anestesiol. 2007 Dec;57(6):678-83. doi: 10.1590/s0034-70942007000600011. Portuguese. PMID: 19462145.
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P2Y receptors and kidney function.

Wiley interdisciplinary reviews. Membrane transport and signaling

Vallon V, Stockand J, Rieg T.
PMID: 23145369
Wiley Interdiscip Rev Membr Transp Signal. 2012;1(6):731-742. doi: 10.1002/wmts.61. Epub 2012 Aug 09.

Cellular release of nucleotides is of physiological importance to regulate and maintain cell function and integrity. Also in the tubular and collecting duct system of the kidney, nucleotides are released in response to changes in cell volume or luminal...

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Vallon V, Stockand J, Rieg T. P2Y receptors and kidney function. Wiley Interdiscip Rev Membr Transp Signal. 2012;1(6):731-742doi: 10.1002/wmts.61.
Vallon, V., Stockand, J., & Rieg, T. (2012). P2Y receptors and kidney function. Wiley interdisciplinary reviews. Membrane transport and signaling, 1(6), 731-742. https://doi.org/10.1002/wmts.61
Vallon, Volker, et al. "P2Y receptors and kidney function." Wiley interdisciplinary reviews. Membrane transport and signaling vol. 1,6 (2012): 731-742. doi: https://doi.org/10.1002/wmts.61
Vallon V, Stockand J, Rieg T. P2Y receptors and kidney function. Wiley Interdiscip Rev Membr Transp Signal. 2012;1(6):731-742. doi: 10.1002/wmts.61. Epub 2012 Aug 09. PMID: 23145369; PMCID: PMC3490424.
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Clinical overview of nephrogenic diabetes insipidus based on a nationwide survey in Japan.

Yonago acta medica

Fujimoto M, Okada S, Kawashima Y, Nishimura R, Miyahara N, Kawaba Y, Hanaki K, Nanba E, Kondo Y, Igarashi T, Kanzaki S.
PMID: 25324589
Yonago Acta Med. 2014 Jun;57(2):85-91. Epub 2014 Jul 30.

BACKGROUND: Nephrogenic diabetes insipidus (NDI) is a rare disease whose complications include polyuria, renal dysfunction, growth disorder and mental retardation. The details of NDI's clinical course have been unclear. To address this uncertainty, we performed a large investigation of...

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Fujimoto M, Okada S, Kawashima Y, et al. Clinical overview of nephrogenic diabetes insipidus based on a nationwide survey in Japan. Yonago Acta Med. 2014;57(2):85-91
Fujimoto, M., Okada, S., Kawashima, Y., Nishimura, R., Miyahara, N., Kawaba, Y., Hanaki, K., Nanba, E., Kondo, Y., Igarashi, T., & Kanzaki, S. (2014). Clinical overview of nephrogenic diabetes insipidus based on a nationwide survey in Japan. Yonago acta medica, 57(2), 85-91.
Fujimoto, Masanobu, et al. "Clinical overview of nephrogenic diabetes insipidus based on a nationwide survey in Japan." Yonago acta medica vol. 57,2 (2014): 85-91.
Fujimoto M, Okada S, Kawashima Y, Nishimura R, Miyahara N, Kawaba Y, Hanaki K, Nanba E, Kondo Y, Igarashi T, Kanzaki S. Clinical overview of nephrogenic diabetes insipidus based on a nationwide survey in Japan. Yonago Acta Med. 2014 Jun;57(2):85-91. Epub 2014 Jul 30. PMID: 25324589; PMCID: PMC4198574.
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Salusins: potential use as a biomarker for atherosclerotic cardiovascular diseases.

International journal of hypertension

Sato K, Watanabe R, Itoh F, Shichiri M, Watanabe T.
PMID: 24251033
Int J Hypertens. 2013;2013:965140. doi: 10.1155/2013/965140. Epub 2013 Oct 22.

Human salusin- α and salusin- β are related peptides produced from prosalusin. Bolus injection of salusin- β into rats induces more profound hypotension and bradycardia than salusin- α . Central administration of salusin- β increases blood pressure via release...

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Sato K, Watanabe R, Itoh F, et al. Salusins: potential use as a biomarker for atherosclerotic cardiovascular diseases. Int J Hypertens. 2013;2013:965140doi: 10.1155/2013/965140.
Sato, K., Watanabe, R., Itoh, F., Shichiri, M., & Watanabe, T. (2013). Salusins: potential use as a biomarker for atherosclerotic cardiovascular diseases. International journal of hypertension, 2013965140. https://doi.org/10.1155/2013/965140
Sato, Kengo, et al. "Salusins: potential use as a biomarker for atherosclerotic cardiovascular diseases." International journal of hypertension vol. 2013 (2013): 965140. doi: https://doi.org/10.1155/2013/965140
Sato K, Watanabe R, Itoh F, Shichiri M, Watanabe T. Salusins: potential use as a biomarker for atherosclerotic cardiovascular diseases. Int J Hypertens. 2013;2013:965140. doi: 10.1155/2013/965140. Epub 2013 Oct 22. PMID: 24251033; PMCID: PMC3819761.
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Showing 25 to 36 of 551 entries