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The effects of morphine, methadone, and fentanyl on mitochondria: A live cell imaging study.

Brain research bulletin

Nylander E, Zelleroth S, Nyberg F, Grönbladh A, Hallberg M.
PMID: 33741459
Brain Res Bull. 2021 Jun;171:126-134. doi: 10.1016/j.brainresbull.2021.03.009. Epub 2021 Mar 16.

The important role of mitochondria in maintaining normal brain cell function has been demonstrated in several neurodegenerative diseases where mitochondrial dysfunction is a prominent feature. Accumulating evidence indicates that opioids may induce neuronal cell death and inhibit neurogenesis, two...

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Nylander E, Zelleroth S, Nyberg F, et al. The effects of morphine, methadone, and fentanyl on mitochondria: A live cell imaging study. Brain Res Bull. 2021;171:126-134doi: 10.1016/j.brainresbull.2021.03.009.
Nylander, E., Zelleroth, S., Nyberg, F., Grönbladh, A., & Hallberg, M. (2021). The effects of morphine, methadone, and fentanyl on mitochondria: A live cell imaging study. Brain research bulletin, 171126-134. https://doi.org/10.1016/j.brainresbull.2021.03.009
Nylander, Erik, et al. "The effects of morphine, methadone, and fentanyl on mitochondria: A live cell imaging study." Brain research bulletin vol. 171 (2021): 126-134. doi: https://doi.org/10.1016/j.brainresbull.2021.03.009
Nylander E, Zelleroth S, Nyberg F, Grönbladh A, Hallberg M. The effects of morphine, methadone, and fentanyl on mitochondria: A live cell imaging study. Brain Res Bull. 2021 Jun;171:126-134. doi: 10.1016/j.brainresbull.2021.03.009. Epub 2021 Mar 16. PMID: 33741459.
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APP palmitoylation is involved in the increase in Aβ.

Brain research

Song J, Yuan C, Li W, Gao T, Lu X, Wang L.
PMID: 34758347
Brain Res. 2022 Jan 01;1774:147709. doi: 10.1016/j.brainres.2021.147709. Epub 2021 Nov 07.

The increase in Aβ

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Song J, Yuan C, Li W, et al. APP palmitoylation is involved in the increase in Aβ. Brain Res. 2021;1774:147709doi: 10.1016/j.brainres.2021.147709.
Song, J., Yuan, C., Li, W., Gao, T., Lu, X., & Wang, L. (2022). APP palmitoylation is involved in the increase in Aβ. Brain research, 1774147709. https://doi.org/10.1016/j.brainres.2021.147709
Song, Jing, et al. "APP palmitoylation is involved in the increase in Aβ." Brain research vol. 1774 (2022): 147709. doi: https://doi.org/10.1016/j.brainres.2021.147709
Song J, Yuan C, Li W, Gao T, Lu X, Wang L. APP palmitoylation is involved in the increase in Aβ. Brain Res. 2022 Jan 01;1774:147709. doi: 10.1016/j.brainres.2021.147709. Epub 2021 Nov 07. PMID: 34758347.
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Peripherally administered cannabinoid receptor 2 (CB.

Brain research

Wilkerson JL, Alberti LB, Thakur GA, Makriyannis A, Milligan ED.
PMID: 34774500
Brain Res. 2022 Jan 01;1774:147721. doi: 10.1016/j.brainres.2021.147721. Epub 2021 Nov 10.

The transient receptor potential (TRP) superfamily of cation channels, of which the TRP vanilloid type 1 (TRPV1) receptor plays a critical role in inflammatory and neuropathic pain, is expressed on nociceptors and spinal cord dorsal horn neurons. TRPV1 is...

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Wilkerson JL, Alberti LB, Thakur GA, et al. Peripherally administered cannabinoid receptor 2 (CB. Brain Res. 2021;1774:147721doi: 10.1016/j.brainres.2021.147721.
Wilkerson, J. L., Alberti, L. B., Thakur, G. A., Makriyannis, A., & Milligan, E. D. (2022). Peripherally administered cannabinoid receptor 2 (CB. Brain research, 1774147721. https://doi.org/10.1016/j.brainres.2021.147721
Wilkerson, Jenny L, et al. "Peripherally administered cannabinoid receptor 2 (CB." Brain research vol. 1774 (2022): 147721. doi: https://doi.org/10.1016/j.brainres.2021.147721
Wilkerson JL, Alberti LB, Thakur GA, Makriyannis A, Milligan ED. Peripherally administered cannabinoid receptor 2 (CB. Brain Res. 2022 Jan 01;1774:147721. doi: 10.1016/j.brainres.2021.147721. Epub 2021 Nov 10. PMID: 34774500.
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Docosahexaenoic acid reduces hypoglycemia-induced neuronal necroptosis via the peroxisome proliferator-activated receptor γ/nuclear factor-κB pathway.

Brain research

Huang L, Zhou Y, Gou ZX, Zhang F, Lu LQ.
PMID: 34785255
Brain Res. 2022 Jan 01;1774:147708. doi: 10.1016/j.brainres.2021.147708. Epub 2021 Nov 14.

DHA has been shown to be neuroprotective and important to neurogenesis, but its role in HG-induced brain injury and the underlying mechanisms remain unknown. To elucidate the therapeutic effect of DHA, we established a mouse model with insulin-induced hypoglycemic...

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Huang L, Zhou Y, Gou ZX, et al. Docosahexaenoic acid reduces hypoglycemia-induced neuronal necroptosis via the peroxisome proliferator-activated receptor γ/nuclear factor-κB pathway. Brain Res. 2021;1774:147708doi: 10.1016/j.brainres.2021.147708.
Huang, L., Zhou, Y., Gou, Z. X., Zhang, F., & Lu, L. Q. (2022). Docosahexaenoic acid reduces hypoglycemia-induced neuronal necroptosis via the peroxisome proliferator-activated receptor γ/nuclear factor-κB pathway. Brain research, 1774147708. https://doi.org/10.1016/j.brainres.2021.147708
Huang, Lin, et al. "Docosahexaenoic acid reduces hypoglycemia-induced neuronal necroptosis via the peroxisome proliferator-activated receptor γ/nuclear factor-κB pathway." Brain research vol. 1774 (2022): 147708. doi: https://doi.org/10.1016/j.brainres.2021.147708
Huang L, Zhou Y, Gou ZX, Zhang F, Lu LQ. Docosahexaenoic acid reduces hypoglycemia-induced neuronal necroptosis via the peroxisome proliferator-activated receptor γ/nuclear factor-κB pathway. Brain Res. 2022 Jan 01;1774:147708. doi: 10.1016/j.brainres.2021.147708. Epub 2021 Nov 14. PMID: 34785255.
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Protective effect of Myrcia pubipetala Miq. against the alterations in oxidative stress parameters in an animal model of depression induced by corticosterone.

Brain research

Shuster AL, Rocha FE, Wayszceyk S, de Lima DD, Barauna SC, Lopes BG, Alberton MD, Magro DDD.
PMID: 34785258
Brain Res. 2022 Jan 01;1774:147725. doi: 10.1016/j.brainres.2021.147725. Epub 2021 Nov 13.

Depression is a debilitating disorder in humans that significantly affects quality of life. As such, alternative therapies are highly sought after by patients seeking treatment for depression. Experimentally, the chronic administration of corticosterone (CORT) in rodents has been reported...

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Shuster AL, Rocha FE, Wayszceyk S, et al. Protective effect of Myrcia pubipetala Miq. against the alterations in oxidative stress parameters in an animal model of depression induced by corticosterone. Brain Res. 2021;1774:147725doi: 10.1016/j.brainres.2021.147725.
Shuster, A. L., Rocha, F. E., Wayszceyk, S., de Lima, D. D., Barauna, S. C., Lopes, B. G., Alberton, M. D., & Magro, D. D. D. (2022). Protective effect of Myrcia pubipetala Miq. against the alterations in oxidative stress parameters in an animal model of depression induced by corticosterone. Brain research, 1774147725. https://doi.org/10.1016/j.brainres.2021.147725
Shuster, Anna Lydia, et al. "Protective effect of Myrcia pubipetala Miq. against the alterations in oxidative stress parameters in an animal model of depression induced by corticosterone." Brain research vol. 1774 (2022): 147725. doi: https://doi.org/10.1016/j.brainres.2021.147725
Shuster AL, Rocha FE, Wayszceyk S, de Lima DD, Barauna SC, Lopes BG, Alberton MD, Magro DDD. Protective effect of Myrcia pubipetala Miq. against the alterations in oxidative stress parameters in an animal model of depression induced by corticosterone. Brain Res. 2022 Jan 01;1774:147725. doi: 10.1016/j.brainres.2021.147725. Epub 2021 Nov 13. PMID: 34785258.
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Inhibition of ROCK2 kinase activity improved behavioral deficits and reduced neuron damage in a DEACMP rat model.

Brain research bulletin

Xu L, Liu X, Guo C, Wang C, Zhao J, Zhang X, Li W, Zhang P, Gu R.
PMID: 34990732
Brain Res Bull. 2022 Jan 03;180:24-30. doi: 10.1016/j.brainresbull.2021.12.018. Epub 2022 Jan 03.

The main pathological changes that occur in delayed encephalopathy after acute carbon monoxide poisoning (DEACMP) are extensive demyelination of brain white matter and neuron damage. Previous studies suggested that demyelination and neuron injury are related to activating the Rho/ROCK...

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Xu L, Liu X, Guo C, et al. Inhibition of ROCK2 kinase activity improved behavioral deficits and reduced neuron damage in a DEACMP rat model. Brain Res Bull. 2022;180:24-30doi: 10.1016/j.brainresbull.2021.12.018.
Xu, L., Liu, X., Guo, C., Wang, C., Zhao, J., Zhang, X., Li, W., Zhang, P., & Gu, R. (2022). Inhibition of ROCK2 kinase activity improved behavioral deficits and reduced neuron damage in a DEACMP rat model. Brain research bulletin, 18024-30. https://doi.org/10.1016/j.brainresbull.2021.12.018
Xu, Linlin, et al. "Inhibition of ROCK2 kinase activity improved behavioral deficits and reduced neuron damage in a DEACMP rat model." Brain research bulletin vol. 180 (2022): 24-30. doi: https://doi.org/10.1016/j.brainresbull.2021.12.018
Xu L, Liu X, Guo C, Wang C, Zhao J, Zhang X, Li W, Zhang P, Gu R. Inhibition of ROCK2 kinase activity improved behavioral deficits and reduced neuron damage in a DEACMP rat model. Brain Res Bull. 2022 Jan 03;180:24-30. doi: 10.1016/j.brainresbull.2021.12.018. Epub 2022 Jan 03. PMID: 34990732.
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Fifteen years of urea cycle disorders brain research: Looking back, looking forward.

Analytical biochemistry

Sen K, Whitehead M, Castillo Pinto C, Caldovic L, Gropman A.
PMID: 34637785
Anal Biochem. 2022 Jan 01;636:114343. doi: 10.1016/j.ab.2021.114343. Epub 2021 Oct 09.

Urea cycle disorders (UCD) are inherited diseases resulting from deficiency in one of six enzymes or two carriers that are required to remove ammonia from the body. UCD may be associated with neurological damage encompassing a spectrum from asymptomatic/mild...

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Sen K, Whitehead M, Castillo Pinto C, et al. Fifteen years of urea cycle disorders brain research: Looking back, looking forward. Anal Biochem. 2021;636:114343doi: 10.1016/j.ab.2021.114343.
Sen, K., Whitehead, M., Castillo Pinto, C., Caldovic, L., & Gropman, A. (2022). Fifteen years of urea cycle disorders brain research: Looking back, looking forward. Analytical biochemistry, 636114343. https://doi.org/10.1016/j.ab.2021.114343
Sen, Kuntal, et al. "Fifteen years of urea cycle disorders brain research: Looking back, looking forward." Analytical biochemistry vol. 636 (2022): 114343. doi: https://doi.org/10.1016/j.ab.2021.114343
Sen K, Whitehead M, Castillo Pinto C, Caldovic L, Gropman A. Fifteen years of urea cycle disorders brain research: Looking back, looking forward. Anal Biochem. 2022 Jan 01;636:114343. doi: 10.1016/j.ab.2021.114343. Epub 2021 Oct 09. PMID: 34637785.
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Repetitive transcranial magnetic stimulation during a spatial memory task leads to a decrease in brain metabolic activity.

Brain research

Zorzo C, Méndez M, Pernía AM, Arias JL.
PMID: 34380023
Brain Res. 2021 Oct 15;1769:147610. doi: 10.1016/j.brainres.2021.147610. Epub 2021 Aug 08.

Repetitive transcranial magnetic stimulation (rTMS) is a non-invasive neuromodulation technique that is able to generate causal-based interferences between brain networks and cognitive or behavioral responses. It has been used to improve cognition in several disease models. However, although its...

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Zorzo C, Méndez M, Pernía AM, et al. Repetitive transcranial magnetic stimulation during a spatial memory task leads to a decrease in brain metabolic activity. Brain Res. 2021;1769:147610doi: 10.1016/j.brainres.2021.147610.
Zorzo, C., Méndez, M., Pernía, A. M., & Arias, J. L. (2021). Repetitive transcranial magnetic stimulation during a spatial memory task leads to a decrease in brain metabolic activity. Brain research, 1769147610. https://doi.org/10.1016/j.brainres.2021.147610
Zorzo, Candela, et al. "Repetitive transcranial magnetic stimulation during a spatial memory task leads to a decrease in brain metabolic activity." Brain research vol. 1769 (2021): 147610. doi: https://doi.org/10.1016/j.brainres.2021.147610
Zorzo C, Méndez M, Pernía AM, Arias JL. Repetitive transcranial magnetic stimulation during a spatial memory task leads to a decrease in brain metabolic activity. Brain Res. 2021 Oct 15;1769:147610. doi: 10.1016/j.brainres.2021.147610. Epub 2021 Aug 08. PMID: 34380023.
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Injectable biomaterial shuttles for cell therapy in stroke.

Brain research bulletin

Samal J, Segura T.
PMID: 34391821
Brain Res Bull. 2021 Nov;176:25-42. doi: 10.1016/j.brainresbull.2021.08.002. Epub 2021 Aug 12.

Ischemic stroke (IS) is the leading cause of disability and contributes to a significant socio-economic cost in the western world. Brain repair strategies investigated in the pre-clinical models include the delivery of drug or cell-based therapeutics; which is hindered...

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Samal J, Segura T. Injectable biomaterial shuttles for cell therapy in stroke. Brain Res Bull. 2021;176:25-42doi: 10.1016/j.brainresbull.2021.08.002.
Samal, J., & Segura, T. (2021). Injectable biomaterial shuttles for cell therapy in stroke. Brain research bulletin, 17625-42. https://doi.org/10.1016/j.brainresbull.2021.08.002
Samal, Juhi, and Segura, Tatiana. "Injectable biomaterial shuttles for cell therapy in stroke." Brain research bulletin vol. 176 (2021): 25-42. doi: https://doi.org/10.1016/j.brainresbull.2021.08.002
Samal J, Segura T. Injectable biomaterial shuttles for cell therapy in stroke. Brain Res Bull. 2021 Nov;176:25-42. doi: 10.1016/j.brainresbull.2021.08.002. Epub 2021 Aug 12. PMID: 34391821; PMCID: PMC8524625.
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Showing 2245 to 2253 of 2253 entries