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Showing 25 to 36 of 790 entries
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Recent Advances in Molecular Genetics of Cardiovascular Disorders - Implications for Atherosclerosis and Diseases of Cellular Lipid Metabolism.

Pathology oncology research : POR

Schmitz G, Aslanidis C, Lackner KJ.
PMID: 11173654
Pathol Oncol Res. 1998;4(2):153-161.

Two developments in molecular genetics will profoundly influence our understanding and the diagnosis of cardiovascular disorders. First, the identification of genes responsible for monogenic and polygenic traits by analysis of e.g. large pedigrees and affected sib pairs provides invaluable...

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Schmitz G, Aslanidis C, Lackner KJ. Recent Advances in Molecular Genetics of Cardiovascular Disorders - Implications for Atherosclerosis and Diseases of Cellular Lipid Metabolism. Pathol Oncol Res. 1998;4(2):153-161
Schmitz, G., Aslanidis, C., & Lackner, K. J. (1998). Recent Advances in Molecular Genetics of Cardiovascular Disorders - Implications for Atherosclerosis and Diseases of Cellular Lipid Metabolism. Pathology oncology research : POR, 4(2), 153-161.
Schmitz, Gerd, et al. "Recent Advances in Molecular Genetics of Cardiovascular Disorders - Implications for Atherosclerosis and Diseases of Cellular Lipid Metabolism." Pathology oncology research : POR vol. 4,2 (1998): 153-161.
Schmitz G, Aslanidis C, Lackner KJ. Recent Advances in Molecular Genetics of Cardiovascular Disorders - Implications for Atherosclerosis and Diseases of Cellular Lipid Metabolism. Pathol Oncol Res. 1998;4(2):153-161. PMID: 11173654.
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Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus.

European journal of internal medicine

Jude EB, Douglas JT, Anderson SG, Young MJ, Boulton AJ.
PMID: 12020626
Eur J Intern Med. 2002 May;13(3):185-189. doi: 10.1016/s0953-6205(02)00014-6.

BACKGROUND: Macrovascular disease in diabetes mellitus is a multifactorial process resulting in part from accelerated atherosclerosis and increased thrombosis. The resultant cardiovascular mortality is up to five times that of an age-matched non-diabetic population. Recently, cell adhesion molecules (CAMs)...

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Jude EB, Douglas JT, Anderson SG, et al. Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. Eur J Intern Med. 2002;13(3):185-189doi: 10.1016/s0953-6205(02)00014-6.
Jude, E. B., Douglas, J. T., Anderson, S. G., Young, M. J., & Boulton, A. J. (2002). Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. European journal of internal medicine, 13(3), 185-189. https://doi.org/10.1016/s0953-6205(02)00014-6
Jude, Edward B., et al. "Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus." European journal of internal medicine vol. 13,3 (2002): 185-189. doi: https://doi.org/10.1016/s0953-6205(02)00014-6
Jude EB, Douglas JT, Anderson SG, Young MJ, Boulton AJ. Circulating cellular adhesion molecules ICAM-1, VCAM-1, P- and E-selectin in the prediction of cardiovascular disease in diabetes mellitus. Eur J Intern Med. 2002 May;13(3):185-189. doi: 10.1016/s0953-6205(02)00014-6. PMID: 12020626.
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Radiation-Mediated Gene Expression in the Pathogenesis of the Clinical Radiation Response.

Seminars in radiation oncology

Hallahan DE.
PMID: 10717183
Semin Radiat Oncol. 1996 Oct;6(4):250-267. doi: 10.1053/SRAO00600250.

Cells and tissues respond to reactive oxygen intermediates during physiological processes such as inflammation, ischemia, and reperfusion. Ionizing radiation mimics naturally occurring free radicals to produce similar responses. Radiation induces the production of reactive oxygen intermediates and subsequent oxidation...

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Hallahan DE. Radiation-Mediated Gene Expression in the Pathogenesis of the Clinical Radiation Response. Semin Radiat Oncol. 1996;6(4):250-267doi: 10.1053/SRAO00600250.
Hallahan, D. E. (1996). Radiation-Mediated Gene Expression in the Pathogenesis of the Clinical Radiation Response. Seminars in radiation oncology, 6(4), 250-267. https://doi.org/10.1053/SRAO00600250
Hallahan. "Radiation-Mediated Gene Expression in the Pathogenesis of the Clinical Radiation Response." Seminars in radiation oncology vol. 6,4 (1996): 250-267. doi: https://doi.org/10.1053/SRAO00600250
Hallahan DE. Radiation-Mediated Gene Expression in the Pathogenesis of the Clinical Radiation Response. Semin Radiat Oncol. 1996 Oct;6(4):250-267. doi: 10.1053/SRAO00600250. PMID: 10717183.
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A role for interferon-beta in Guillain-Barré Syndrome?.

BioDrugs : clinical immunotherapeutics, biopharmaceuticals and gene therapy

Créange A.
PMID: 18034551
BioDrugs. 2000 Jul;14(1):1-11. doi: 10.2165/00063030-200014010-00001.

Guillain-Barré syndrome (GBS) is an acute inflammatory demyelinating neuropathy that is associated with long-lasting morbidity and a substantial risk of mortality. The 2 reference treatments, plasma exchange and intravenous immunoglobulins (IVIg), do not change the functional prognosis for the...

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Créange A. A role for interferon-beta in Guillain-Barré Syndrome?. BioDrugs. 2000;14(1):1-11doi: 10.2165/00063030-200014010-00001.
Créange, A. (2000). A role for interferon-beta in Guillain-Barré Syndrome?. BioDrugs : clinical immunotherapeutics, biopharmaceuticals and gene therapy, 14(1), 1-11. https://doi.org/10.2165/00063030-200014010-00001
Créange, A. "A role for interferon-beta in Guillain-Barré Syndrome?." BioDrugs : clinical immunotherapeutics, biopharmaceuticals and gene therapy vol. 14,1 (2000): 1-11. doi: https://doi.org/10.2165/00063030-200014010-00001
Créange A. A role for interferon-beta in Guillain-Barré Syndrome?. BioDrugs. 2000 Jul;14(1):1-11. doi: 10.2165/00063030-200014010-00001. PMID: 18034551.
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Immunology of inflammatory bowel disease.

Current opinion in gastroenterology

Levine AD, Fiocchi C.
PMID: 17031093
Curr Opin Gastroenterol. 2000 Jul;16(4):306-9. doi: 10.1097/00001574-200007000-00003.

The immunology of inflammatory bowel disease continues to be an intense area of investigation for clues to the pathogenesis of Crohn disease and ulcerative colitis. As typical with complex diseases, inflammatory bowel disease research is continuously evolving. Without abandoning...

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Levine AD, Fiocchi C. Immunology of inflammatory bowel disease. Curr Opin Gastroenterol. 2000;16(4):306-9doi: 10.1097/00001574-200007000-00003.
Levine, A. D., & Fiocchi, C. (2000). Immunology of inflammatory bowel disease. Current opinion in gastroenterology, 16(4), 306-9. https://doi.org/10.1097/00001574-200007000-00003
Levine, A D, and Fiocchi, C. "Immunology of inflammatory bowel disease." Current opinion in gastroenterology vol. 16,4 (2000): 306-9. doi: https://doi.org/10.1097/00001574-200007000-00003
Levine AD, Fiocchi C. Immunology of inflammatory bowel disease. Curr Opin Gastroenterol. 2000 Jul;16(4):306-9. doi: 10.1097/00001574-200007000-00003. PMID: 17031093.
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Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils.

Clinical molecular pathology

Leite RP, Carmo-Fonseca M, Cabeçadas J, Parreira A, Parreira L.
PMID: 16695989
Clin Mol Pathol. 1995 Apr;48(2):M93-M100. doi: 10.1136/mp.48.2.m93.

Aims-To analyse the topographical distribution of adhesion molecules involved in lymphocyte recirculation in human lymph nodes and tonsils. The study focused on the expression of LECAM-1 (CD62L), VLA-alpha4 (CD49d), VLA-beta1 (CD29), LFA-1 alphaL (CD11a), LFA-beta2 (CD18), VCAM-1 (CD106), ICAM-1...

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Leite RP, Carmo-Fonseca M, Cabeçadas J, et al. Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils. Clin Mol Pathol. 1995;48(2):M93-M100doi: 10.1136/mp.48.2.m93.
Leite, R. P., Carmo-Fonseca, M., Cabeçadas, J., Parreira, A., & Parreira, L. (1995). Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils. Clinical molecular pathology, 48(2), M93-M100. https://doi.org/10.1136/mp.48.2.m93
Leite, R P, et al. "Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils." Clinical molecular pathology vol. 48,2 (1995): M93-M100. doi: https://doi.org/10.1136/mp.48.2.m93
Leite RP, Carmo-Fonseca M, Cabeçadas J, Parreira A, Parreira L. Differential expression of cell adhesion molecules in the functional compartments of lymph nodes and tonsils. Clin Mol Pathol. 1995 Apr;48(2):M93-M100. doi: 10.1136/mp.48.2.m93. PMID: 16695989; PMCID: PMC407932.
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Prostate cancer: serum and tissue markers.

Reviews in urology

Miller GJ, Brawer MK, Sakr WA, Thrasher JB, Townsend R.
PMID: 16985995
Rev Urol. 2001;3:S11-9.

The detection of prostate cancer, its clinical staging, and the prediction of its prognosis remain topics of paramount importance in clinical management. The digital rectal exam, although once the "gold standard," has been largely supplanted by a variety of...

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Miller GJ, Brawer MK, Sakr WA, et al. Prostate cancer: serum and tissue markers. Rev Urol. 2001;3:S11-9
Miller, G. J., Brawer, M. K., Sakr, W. A., Thrasher, J. B., & Townsend, R. (2001). Prostate cancer: serum and tissue markers. Reviews in urology, 3S11-9.
Miller, G J, et al. "Prostate cancer: serum and tissue markers." Reviews in urology vol. 3 (2001): S11-9.
Miller GJ, Brawer MK, Sakr WA, Thrasher JB, Townsend R. Prostate cancer: serum and tissue markers. Rev Urol. 2001;3:S11-9. PMID: 16985995; PMCID: PMC1476078.
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Memory beyond the synapse.

Neuron glia biology

Rose SP.
PMID: 18634598
Neuron Glia Biol. 2004 Aug;1(3):211-7. doi: 10.1017/S1740925X05000116.

Based on studies of the molecular and cellular cascades that occur during memory consolidation for a one-trial passive-avoidance learning task in the young chick, I review the evidence that memory is encoded in permanent changes in synaptic connectivity ina...

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Rose SP. Memory beyond the synapse. Neuron Glia Biol. 2004;1(3):211-7doi: 10.1017/S1740925X05000116.
Rose, S. P. (2004). Memory beyond the synapse. Neuron glia biology, 1(3), 211-7. https://doi.org/10.1017/S1740925X05000116
Rose, Steven P R. "Memory beyond the synapse." Neuron glia biology vol. 1,3 (2004): 211-7. doi: https://doi.org/10.1017/S1740925X05000116
Rose SP. Memory beyond the synapse. Neuron Glia Biol. 2004 Aug;1(3):211-7. doi: 10.1017/S1740925X05000116. PMID: 18634598.
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Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Lee JH, Kim CH, Seo GH, Lee J, Kim JH, Kim DG, Ahn YS.
PMID: 19967061
Korean J Physiol Pharmacol. 2008 Oct;12(5):231-6. doi: 10.4196/kjpp.2008.12.5.231. Epub 2008 Oct 31.

Heparin is a well-known anticoagulant widely used in various clinical settings. Interestingly, recent studies have indicated that heparin also has anti-inflammatory effects on neuroinflammation-related diseases, such as Alzheimer's disease and meningitis. However, the underlying mechanism of its actions remains...

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Lee JH, Kim CH, Seo GH, et al. Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. Korean J Physiol Pharmacol. 2008;12(5):231-6doi: 10.4196/kjpp.2008.12.5.231.
Lee, J. H., Kim, C. H., Seo, G. H., Lee, J., Kim, J. H., Kim, D. G., & Ahn, Y. S. (2008). Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 12(5), 231-6. https://doi.org/10.4196/kjpp.2008.12.5.231
Lee, Jeong Ho, et al. "Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 12,5 (2008): 231-6. doi: https://doi.org/10.4196/kjpp.2008.12.5.231
Lee JH, Kim CH, Seo GH, Lee J, Kim JH, Kim DG, Ahn YS. Heparin Attenuates the Expression of TNFalpha-induced Cerebral Endothelial Cell Adhesion Molecule. Korean J Physiol Pharmacol. 2008 Oct;12(5):231-6. doi: 10.4196/kjpp.2008.12.5.231. Epub 2008 Oct 31. PMID: 19967061; PMCID: PMC2788641.
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A novel Mutein of TNFalpha Containing the Arg-Gly-Asp Sequence Shows Reduced Toxicity in Intestine.

Mediators of inflammation

Shikama H, Miyata K, Sakae N, Kuroda K, Nishimura K, Yotsuya S, Kato M.
PMID: 18472928
Mediators Inflamm. 1994;3(2):111-6. doi: 10.1155/S096293519400013X.

The effects of human tumour necrosis factor-alpha (TNFalpha), or its mutein (F4168) having the cell adhesive Arg-Gly-Asp sequence at the N-terminus, on intestinal injury, were examined. Histopathological examination revealed that an intravenous injection of TNFalpha resulted in marked haemorrhage...

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Shikama H, Miyata K, Sakae N, et al. A novel Mutein of TNFalpha Containing the Arg-Gly-Asp Sequence Shows Reduced Toxicity in Intestine. Mediators Inflamm. 1994;3(2):111-6doi: 10.1155/S096293519400013X.
Shikama, H., Miyata, K., Sakae, N., Kuroda, K., Nishimura, K., Yotsuya, S., & Kato, M. (1994). A novel Mutein of TNFalpha Containing the Arg-Gly-Asp Sequence Shows Reduced Toxicity in Intestine. Mediators of inflammation, 3(2), 111-6. https://doi.org/10.1155/S096293519400013X
Shikama, H, et al. "A novel Mutein of TNFalpha Containing the Arg-Gly-Asp Sequence Shows Reduced Toxicity in Intestine." Mediators of inflammation vol. 3,2 (1994): 111-6. doi: https://doi.org/10.1155/S096293519400013X
Shikama H, Miyata K, Sakae N, Kuroda K, Nishimura K, Yotsuya S, Kato M. A novel Mutein of TNFalpha Containing the Arg-Gly-Asp Sequence Shows Reduced Toxicity in Intestine. Mediators Inflamm. 1994;3(2):111-6. doi: 10.1155/S096293519400013X. PMID: 18472928; PMCID: PMC2367030.
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Evaluation of the expression of integrins and cell adhesion molecules through tissue microarray in lymph node metastases of prostate cancer.

Journal of carcinogenesis

Pontes-Junior J, Reis ST, Dall'Oglio M, Neves de Oliveira LC, Cury J, Carvalho PA, Ribeiro-Filho LA, Moreira Leite KR, Srougi M.
PMID: 19240373
J Carcinog. 2009;8:3. doi: 10.4103/1477-3163.48453.

BACKGROUND: Integrins and adhesion molecules are responsible for the maintenance of the epithelial phenotype. Cell culture studies have reported the correlation between adhesion molecule expression and prostate carcinoma, but their role in the metastatic process is not yet known....

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Pontes-Junior J, Reis ST, Dall'Oglio M, et al. Evaluation of the expression of integrins and cell adhesion molecules through tissue microarray in lymph node metastases of prostate cancer. J Carcinog. 2009;8:3doi: 10.4103/1477-3163.48453.
Pontes-Junior, J., Reis, S. T., Dall'Oglio, M., Neves de Oliveira, L. C., Cury, J., Carvalho, P. A., Ribeiro-Filho, L. A., Moreira Leite, K. R., & Srougi, M. (2009). Evaluation of the expression of integrins and cell adhesion molecules through tissue microarray in lymph node metastases of prostate cancer. Journal of carcinogenesis, 83. https://doi.org/10.4103/1477-3163.48453
Pontes-Junior, Jose, et al. "Evaluation of the expression of integrins and cell adhesion molecules through tissue microarray in lymph node metastases of prostate cancer." Journal of carcinogenesis vol. 8 (2009): 3. doi: https://doi.org/10.4103/1477-3163.48453
Pontes-Junior J, Reis ST, Dall'Oglio M, Neves de Oliveira LC, Cury J, Carvalho PA, Ribeiro-Filho LA, Moreira Leite KR, Srougi M. Evaluation of the expression of integrins and cell adhesion molecules through tissue microarray in lymph node metastases of prostate cancer. J Carcinog. 2009;8:3. doi: 10.4103/1477-3163.48453. PMID: 19240373; PMCID: PMC2678866.
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The circumventricular organs participate in the immunopathogenesis of experimental autoimmune encephalomyelitis.

Cerebrospinal fluid research

Schulz M, Engelhardt B.
PMID: 16197544
Cerebrospinal Fluid Res. 2005 Sep 30;2:8. doi: 10.1186/1743-8454-2-8.

BACKGROUND: During inflammatory conditions of the central nervous system (CNS), such as in multiple sclerosis or in its animal model, experimental autoimmune encephalomyelitis (EAE), immune cells migrate from the blood stream into the CNS parenchyma and into the cerebrospinal...

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Schulz M, Engelhardt B. The circumventricular organs participate in the immunopathogenesis of experimental autoimmune encephalomyelitis. Cerebrospinal Fluid Res. 2005;2:8doi: 10.1186/1743-8454-2-8.
Schulz, M., & Engelhardt, B. (2005). The circumventricular organs participate in the immunopathogenesis of experimental autoimmune encephalomyelitis. Cerebrospinal fluid research, 28. https://doi.org/10.1186/1743-8454-2-8
Schulz, Martina, and Engelhardt, Britta. "The circumventricular organs participate in the immunopathogenesis of experimental autoimmune encephalomyelitis." Cerebrospinal fluid research vol. 2 (2005): 8. doi: https://doi.org/10.1186/1743-8454-2-8
Schulz M, Engelhardt B. The circumventricular organs participate in the immunopathogenesis of experimental autoimmune encephalomyelitis. Cerebrospinal Fluid Res. 2005 Sep 30;2:8. doi: 10.1186/1743-8454-2-8. PMID: 16197544; PMCID: PMC1262737.
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Showing 25 to 36 of 790 entries