Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 241 to 248 of 248 entries
Sorted by: Best Match Show Resources per page
Exosomes derived from cochlear spiral ganglion progenitor cells prevent cochlea damage from ischemia-reperfusion injury via inhibiting the inflammatory process.

Cell and tissue research

Yang T, Cai C, Peng A, Liu J, Wang Q.
PMID: 34155579
Cell Tissue Res. 2021 Nov;386(2):239-247. doi: 10.1007/s00441-021-03468-x. Epub 2021 Jun 22.

Ischemia-reperfusion injury (I/R)-induced inflammatory process can mediate cochlea damage-related hearing loss; whether cochlear spiral ganglion progenitor cell-derived exosomes (CSGPC-exos) play a protective role by carrying functional microRNAs into recipient cells is unknown. Different doses of CSGPC-exos (0.1 μg/μl, 0.2...

Cite
Yang T, Cai C, Peng A, et al. Exosomes derived from cochlear spiral ganglion progenitor cells prevent cochlea damage from ischemia-reperfusion injury via inhibiting the inflammatory process. Cell Tissue Res. 2021;386(2):239-247doi: 10.1007/s00441-021-03468-x.
Yang, T., Cai, C., Peng, A., Liu, J., & Wang, Q. (2021). Exosomes derived from cochlear spiral ganglion progenitor cells prevent cochlea damage from ischemia-reperfusion injury via inhibiting the inflammatory process. Cell and tissue research, 386(2), 239-247. https://doi.org/10.1007/s00441-021-03468-x
Yang, Tao, et al. "Exosomes derived from cochlear spiral ganglion progenitor cells prevent cochlea damage from ischemia-reperfusion injury via inhibiting the inflammatory process." Cell and tissue research vol. 386,2 (2021): 239-247. doi: https://doi.org/10.1007/s00441-021-03468-x
Yang T, Cai C, Peng A, Liu J, Wang Q. Exosomes derived from cochlear spiral ganglion progenitor cells prevent cochlea damage from ischemia-reperfusion injury via inhibiting the inflammatory process. Cell Tissue Res. 2021 Nov;386(2):239-247. doi: 10.1007/s00441-021-03468-x. Epub 2021 Jun 22. PMID: 34155579.
Copy Download .nbib Format: NLM
Isosteviol improves cardiac function and promotes angiogenesis after myocardial infarction in rats.

Cell and tissue research

Liu F, Song L, Lu Z, Sun T, Lun J, Zhou C, Sun X, Tan W, Zhao H.
PMID: 34820705
Cell Tissue Res. 2021 Nov 25; doi: 10.1007/s00441-021-03559-9. Epub 2021 Nov 25.

Isosteviol has been indicated as a cardiomyocyte protector. However, the underlying mechanism remains unclear. Thus, we sought to confirm the protective effect of isosteviol after myocardial infarction in a model of permanent coronary artery occlusion and investigate the potential...

Cite
Liu F, Song L, Lu Z, et al. Isosteviol improves cardiac function and promotes angiogenesis after myocardial infarction in rats. Cell Tissue Res. 2021;doi: 10.1007/s00441-021-03559-9.
Liu, F., Song, L., Lu, Z., Sun, T., Lun, J., Zhou, C., Sun, X., Tan, W., & Zhao, H. (2021). Isosteviol improves cardiac function and promotes angiogenesis after myocardial infarction in rats. Cell and tissue research, . https://doi.org/10.1007/s00441-021-03559-9
Liu, Fei, et al. "Isosteviol improves cardiac function and promotes angiogenesis after myocardial infarction in rats." Cell and tissue research vol. (2021). doi: https://doi.org/10.1007/s00441-021-03559-9
Liu F, Song L, Lu Z, Sun T, Lun J, Zhou C, Sun X, Tan W, Zhao H. Isosteviol improves cardiac function and promotes angiogenesis after myocardial infarction in rats. Cell Tissue Res. 2021 Nov 25; doi: 10.1007/s00441-021-03559-9. Epub 2021 Nov 25. PMID: 34820705.
Copy Download .nbib Format: NLM
Retraction Note to: CiRS-126 inhibits proliferation of ovarian granulosa cells through targeting the miR-21-PDCD4-ROS axis in a polycystic ovarian syndrome model.

Cell and tissue research

Lu J, Xue Y, Wang Y, Ding Y, Zou Q, Pan M, Qiao L, Zhang C, Ge Q, Wang T, Li H.
PMID: 34213639
Cell Tissue Res. 2021 Oct;386(1):213. doi: 10.1007/s00441-021-03484-x.

No abstract available.

Cite
Lu J, Xue Y, Wang Y, et al. Retraction Note to: CiRS-126 inhibits proliferation of ovarian granulosa cells through targeting the miR-21-PDCD4-ROS axis in a polycystic ovarian syndrome model. Cell Tissue Res. 2021;386(1):213doi: 10.1007/s00441-021-03484-x.
Lu, J., Xue, Y., Wang, Y., Ding, Y., Zou, Q., Pan, M., Qiao, L., Zhang, C., Ge, Q., Wang, T., & Li, H. (2021). Retraction Note to: CiRS-126 inhibits proliferation of ovarian granulosa cells through targeting the miR-21-PDCD4-ROS axis in a polycystic ovarian syndrome model. Cell and tissue research, 386(1), 213. https://doi.org/10.1007/s00441-021-03484-x
Lu, Jiafeng, et al. "Retraction Note to: CiRS-126 inhibits proliferation of ovarian granulosa cells through targeting the miR-21-PDCD4-ROS axis in a polycystic ovarian syndrome model." Cell and tissue research vol. 386,1 (2021): 213. doi: https://doi.org/10.1007/s00441-021-03484-x
Lu J, Xue Y, Wang Y, Ding Y, Zou Q, Pan M, Qiao L, Zhang C, Ge Q, Wang T, Li H. Retraction Note to: CiRS-126 inhibits proliferation of ovarian granulosa cells through targeting the miR-21-PDCD4-ROS axis in a polycystic ovarian syndrome model. Cell Tissue Res. 2021 Oct;386(1):213. doi: 10.1007/s00441-021-03484-x. PMID: 34213639.
Copy Download .nbib Format: NLM
Aloin antagonizes stimulated ischemia/reperfusion-induced damage and inflammatory response in cardiomyocytes by activating the Nrf2/HO-1 defense pathway.

Cell and tissue research

Sun W, Wang Z, Sun M, Huang W, Wang Y, Wang Y.
PMID: 33502605
Cell Tissue Res. 2021 Jun;384(3):735-744. doi: 10.1007/s00441-020-03345-z. Epub 2021 Jan 27.

Myocardial ischemia/reperfusion injury (I/RI) frequently incurs in acute myocardial infarction with high morbidity and mortality worldwide and is characterized with cardiomyocyte apoptosis and inflammatory response. Aloin is a major anthraquinone from Aloe species and fulfills pleiotropic protective functions in...

Cite
Sun W, Wang Z, Sun M, et al. Aloin antagonizes stimulated ischemia/reperfusion-induced damage and inflammatory response in cardiomyocytes by activating the Nrf2/HO-1 defense pathway. Cell Tissue Res. 2021;384(3):735-744doi: 10.1007/s00441-020-03345-z.
Sun, W., Wang, Z., Sun, M., Huang, W., Wang, Y., & Wang, Y. (2021). Aloin antagonizes stimulated ischemia/reperfusion-induced damage and inflammatory response in cardiomyocytes by activating the Nrf2/HO-1 defense pathway. Cell and tissue research, 384(3), 735-744. https://doi.org/10.1007/s00441-020-03345-z
Sun, Wei, et al. "Aloin antagonizes stimulated ischemia/reperfusion-induced damage and inflammatory response in cardiomyocytes by activating the Nrf2/HO-1 defense pathway." Cell and tissue research vol. 384,3 (2021): 735-744. doi: https://doi.org/10.1007/s00441-020-03345-z
Sun W, Wang Z, Sun M, Huang W, Wang Y, Wang Y. Aloin antagonizes stimulated ischemia/reperfusion-induced damage and inflammatory response in cardiomyocytes by activating the Nrf2/HO-1 defense pathway. Cell Tissue Res. 2021 Jun;384(3):735-744. doi: 10.1007/s00441-020-03345-z. Epub 2021 Jan 27. PMID: 33502605.
Copy Download .nbib Format: NLM
Retraction Note to: Mesenchymal stem cells seeded onto tissue-engineered osteoinductive scaffolds enhance the healing process of critical-sized radial bone defects in rat.

Cell and tissue research

Oryan A, Eslaminejad MB, Kamali A, Hosseini S, Moshiri A, Baharvand H.
PMID: 34424398
Cell Tissue Res. 2021 Oct;386(1):211. doi: 10.1007/s00441-021-03520-w.

No abstract available.

Cite
Oryan A, Eslaminejad MB, Kamali A, et al. Retraction Note to: Mesenchymal stem cells seeded onto tissue-engineered osteoinductive scaffolds enhance the healing process of critical-sized radial bone defects in rat. Cell Tissue Res. 2021;386(1):211doi: 10.1007/s00441-021-03520-w.
Oryan, A., Eslaminejad, M. B., Kamali, A., Hosseini, S., Moshiri, A., & Baharvand, H. (2021). Retraction Note to: Mesenchymal stem cells seeded onto tissue-engineered osteoinductive scaffolds enhance the healing process of critical-sized radial bone defects in rat. Cell and tissue research, 386(1), 211. https://doi.org/10.1007/s00441-021-03520-w
Oryan, Ahmad, et al. "Retraction Note to: Mesenchymal stem cells seeded onto tissue-engineered osteoinductive scaffolds enhance the healing process of critical-sized radial bone defects in rat." Cell and tissue research vol. 386,1 (2021): 211. doi: https://doi.org/10.1007/s00441-021-03520-w
Oryan A, Eslaminejad MB, Kamali A, Hosseini S, Moshiri A, Baharvand H. Retraction Note to: Mesenchymal stem cells seeded onto tissue-engineered osteoinductive scaffolds enhance the healing process of critical-sized radial bone defects in rat. Cell Tissue Res. 2021 Oct;386(1):211. doi: 10.1007/s00441-021-03520-w. PMID: 34424398.
Copy Download .nbib Format: NLM
Id proteins: emerging roles in CNS disease and targets for modifying neural stemcell behavior.

Cell and tissue research

Chu YH, Lin JD, Nath S, Schachtrup C.
PMID: 34302526
Cell Tissue Res. 2021 Jul 24; doi: 10.1007/s00441-021-03490-z. Epub 2021 Jul 24.

Neural stem/progenitor cells (NSPCs) are found in the adult brain and spinal cord, and endogenous or transplanted NSPCs contribute to repair processes and regulate immune responses in the CNS. However, the molecular mechanisms of NSPC survival and integration as...

Cite
Chu YH, Lin JD, Nath S, et al. Id proteins: emerging roles in CNS disease and targets for modifying neural stemcell behavior. Cell Tissue Res. 2021;doi: 10.1007/s00441-021-03490-z.
Chu, Y. H., Lin, J. D., Nath, S., & Schachtrup, C. (2021). Id proteins: emerging roles in CNS disease and targets for modifying neural stemcell behavior. Cell and tissue research, . https://doi.org/10.1007/s00441-021-03490-z
Chu, Yu-Hsuan, et al. "Id proteins: emerging roles in CNS disease and targets for modifying neural stemcell behavior." Cell and tissue research vol. (2021). doi: https://doi.org/10.1007/s00441-021-03490-z
Chu YH, Lin JD, Nath S, Schachtrup C. Id proteins: emerging roles in CNS disease and targets for modifying neural stemcell behavior. Cell Tissue Res. 2021 Jul 24; doi: 10.1007/s00441-021-03490-z. Epub 2021 Jul 24. PMID: 34302526.
Copy Download .nbib Format: NLM
LncRNA FENDRR promotes apoptosis of Leydig cells in late-onset hypogonadism by facilitating the degradation of Nrf2.

Cell and tissue research

Liu Y, Liu Y, Wang J, Huang F, Du P, Wu L, Guo F, Song Y, Qin G.
PMID: 34278519
Cell Tissue Res. 2021 Nov;386(2):379-389. doi: 10.1007/s00441-021-03497-6. Epub 2021 Jul 19.

This study aimed to investigate the role of lncRNA FENDRR in apoptosis of Leydig cells and the further mechanism. The apoptosis of Leydig cells (TM3 cell line) was induced by H

Cite
Liu Y, Liu Y, Wang J, et al. LncRNA FENDRR promotes apoptosis of Leydig cells in late-onset hypogonadism by facilitating the degradation of Nrf2. Cell Tissue Res. 2021;386(2):379-389doi: 10.1007/s00441-021-03497-6.
Liu, Y., Liu, Y., Wang, J., Huang, F., Du, P., Wu, L., Guo, F., Song, Y., & Qin, G. (2021). LncRNA FENDRR promotes apoptosis of Leydig cells in late-onset hypogonadism by facilitating the degradation of Nrf2. Cell and tissue research, 386(2), 379-389. https://doi.org/10.1007/s00441-021-03497-6
Liu, Yanling, et al. "LncRNA FENDRR promotes apoptosis of Leydig cells in late-onset hypogonadism by facilitating the degradation of Nrf2." Cell and tissue research vol. 386,2 (2021): 379-389. doi: https://doi.org/10.1007/s00441-021-03497-6
Liu Y, Liu Y, Wang J, Huang F, Du P, Wu L, Guo F, Song Y, Qin G. LncRNA FENDRR promotes apoptosis of Leydig cells in late-onset hypogonadism by facilitating the degradation of Nrf2. Cell Tissue Res. 2021 Nov;386(2):379-389. doi: 10.1007/s00441-021-03497-6. Epub 2021 Jul 19. PMID: 34278519.
Copy Download .nbib Format: NLM
Loss of Nucleobindin-2/Nesfatin-1 increases lipopolysaccharide-induced murine acute lung inflammation.

Cell and tissue research

Hui J, Aulakh GK, Unniappan S, Singh B.
PMID: 33783610
Cell Tissue Res. 2021 Jul;385(1):87-103. doi: 10.1007/s00441-021-03435-6. Epub 2021 Mar 30.

NUCB2/nesfatin-1 is expressed in variety of tissues. Treatment with nesfatin-1 reduces inflammation in rat models of subarachnoid hemorrhage-induced oxidative brain damage and traumatic brain injury as well as myocardial injury. There is only one study showing anti-inflammatory actions of...

Cite
Hui J, Aulakh GK, Unniappan S, et al. Loss of Nucleobindin-2/Nesfatin-1 increases lipopolysaccharide-induced murine acute lung inflammation. Cell Tissue Res. 2021;385(1):87-103doi: 10.1007/s00441-021-03435-6.
Hui, J., Aulakh, G. K., Unniappan, S., & Singh, B. (2021). Loss of Nucleobindin-2/Nesfatin-1 increases lipopolysaccharide-induced murine acute lung inflammation. Cell and tissue research, 385(1), 87-103. https://doi.org/10.1007/s00441-021-03435-6
Hui, Jasmine, et al. "Loss of Nucleobindin-2/Nesfatin-1 increases lipopolysaccharide-induced murine acute lung inflammation." Cell and tissue research vol. 385,1 (2021): 87-103. doi: https://doi.org/10.1007/s00441-021-03435-6
Hui J, Aulakh GK, Unniappan S, Singh B. Loss of Nucleobindin-2/Nesfatin-1 increases lipopolysaccharide-induced murine acute lung inflammation. Cell Tissue Res. 2021 Jul;385(1):87-103. doi: 10.1007/s00441-021-03435-6. Epub 2021 Mar 30. PMID: 33783610.
Copy Download .nbib Format: NLM
Showing 241 to 248 of 248 entries