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Mmp-9, a potential target for cerebral ischemic treatment.

Current neuropharmacology

Dong X, Song YN, Liu WG, Guo XL.
PMID: 20514206
Curr Neuropharmacol. 2009 Dec;7(4):269-75. doi: 10.2174/157015909790031157.

Matrix metalloproteinase-9 (MMP-9) which is a member of matrix metalloproteinases family that normally remodel the extracellular matrix, has been shown to play an important role in both animal models of cerebral ischemia and human stroke. The expression of MMP-9...

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Dong X, Song YN, Liu WG, et al. Mmp-9, a potential target for cerebral ischemic treatment. Curr Neuropharmacol. 2009;7(4):269-75doi: 10.2174/157015909790031157.
Dong, X., Song, Y. N., Liu, W. G., & Guo, X. L. (2009). Mmp-9, a potential target for cerebral ischemic treatment. Current neuropharmacology, 7(4), 269-75. https://doi.org/10.2174/157015909790031157
Dong, Xue, et al. "Mmp-9, a potential target for cerebral ischemic treatment." Current neuropharmacology vol. 7,4 (2009): 269-75. doi: https://doi.org/10.2174/157015909790031157
Dong X, Song YN, Liu WG, Guo XL. Mmp-9, a potential target for cerebral ischemic treatment. Curr Neuropharmacol. 2009 Dec;7(4):269-75. doi: 10.2174/157015909790031157. PMID: 20514206; PMCID: PMC2811860.
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Arachidonate Metabolism in the Anterior Pituitary: Effect of Arachidonate Inhibitors on Basal and Stimulated Secretion of Prolactin, Growth Hormone and Luteinizing Hormone. II. Hormone Release from Dispersed Pituitary Cells.

Journal of neuroendocrinology

Bihoreau C, Rasolonjanahary R, Gerozissis K, Clauser H, Kordon C.
PMID: 19215372
J Neuroendocrinol. 1990 Aug 01;2(4):445-52. doi: 10.1111/j.1365-2826.1990.tb00431.x.

Abstract In the accompanying study, we reported the effects of inhibitors of arachidonic acid metabolism on the regulation of prolactin, growth hormone (GH) and luteinizing hormone secretion by male hemipituitaries. The present work extends these investigations to primary cell...

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Bihoreau C, Rasolonjanahary R, Gerozissis K, et al. Arachidonate Metabolism in the Anterior Pituitary: Effect of Arachidonate Inhibitors on Basal and Stimulated Secretion of Prolactin, Growth Hormone and Luteinizing Hormone. II. Hormone Release from Dispersed Pituitary Cells. J Neuroendocrinol. 1990;2(4):445-52doi: 10.1111/j.1365-2826.1990.tb00431.x.
Bihoreau, C., Rasolonjanahary, R., Gerozissis, K., Clauser, H., & Kordon, C. (1990). Arachidonate Metabolism in the Anterior Pituitary: Effect of Arachidonate Inhibitors on Basal and Stimulated Secretion of Prolactin, Growth Hormone and Luteinizing Hormone. II. Hormone Release from Dispersed Pituitary Cells. Journal of neuroendocrinology, 2(4), 445-52. https://doi.org/10.1111/j.1365-2826.1990.tb00431.x
Bihoreau, C, et al. "Arachidonate Metabolism in the Anterior Pituitary: Effect of Arachidonate Inhibitors on Basal and Stimulated Secretion of Prolactin, Growth Hormone and Luteinizing Hormone. II. Hormone Release from Dispersed Pituitary Cells." Journal of neuroendocrinology vol. 2,4 (1990): 445-52. doi: https://doi.org/10.1111/j.1365-2826.1990.tb00431.x
Bihoreau C, Rasolonjanahary R, Gerozissis K, Clauser H, Kordon C. Arachidonate Metabolism in the Anterior Pituitary: Effect of Arachidonate Inhibitors on Basal and Stimulated Secretion of Prolactin, Growth Hormone and Luteinizing Hormone. II. Hormone Release from Dispersed Pituitary Cells. J Neuroendocrinol. 1990 Aug 01;2(4):445-52. doi: 10.1111/j.1365-2826.1990.tb00431.x. PMID: 19215372.
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Pharmacoeconomics of Surgical Interventions vs. Cyclooxygenase Inhibitors for the Treatment of Patent Ductus Arteriosus.

The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG

Turck CJ, Marsh W, Stevenson JG, York JM, Miller H, Patel S.
PMID: 23055853
J Pediatr Pharmacol Ther. 2007 Jul;12(3):183-93. doi: 10.5863/1551-6776-12.3.183.

Management of neonatal patent ductus arteriosus (PDA) often is resource-intensive and costly. Therefore, it is in hospitals' best interests to ensure the most cost-efficient use of associated resources. Clinical status, comorbidities, and response to prior therapy are considered in...

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Turck CJ, Marsh W, Stevenson JG, et al. Pharmacoeconomics of Surgical Interventions vs. Cyclooxygenase Inhibitors for the Treatment of Patent Ductus Arteriosus. J Pediatr Pharmacol Ther. 2007;12(3):183-93doi: 10.5863/1551-6776-12.3.183.
Turck, C. J., Marsh, W., Stevenson, J. G., York, J. M., Miller, H., & Patel, S. (2007). Pharmacoeconomics of Surgical Interventions vs. Cyclooxygenase Inhibitors for the Treatment of Patent Ductus Arteriosus. The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG, 12(3), 183-93. https://doi.org/10.5863/1551-6776-12.3.183
Turck, Charles J, et al. "Pharmacoeconomics of Surgical Interventions vs. Cyclooxygenase Inhibitors for the Treatment of Patent Ductus Arteriosus." The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG vol. 12,3 (2007): 183-93. doi: https://doi.org/10.5863/1551-6776-12.3.183
Turck CJ, Marsh W, Stevenson JG, York JM, Miller H, Patel S. Pharmacoeconomics of Surgical Interventions vs. Cyclooxygenase Inhibitors for the Treatment of Patent Ductus Arteriosus. J Pediatr Pharmacol Ther. 2007 Jul;12(3):183-93. doi: 10.5863/1551-6776-12.3.183. PMID: 23055853; PMCID: PMC3462100.
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Short-term perioperative parecoxib is not detrimental to shaft fracture healing in a rat model.

Bone & joint research

Hjorthaug GA, Søreide E, Nordsletten L, Madsen JE, Reinholt FP, Niratisairak S, Dimmen S.
PMID: 31728186
Bone Joint Res. 2019 Nov 02;8(10):472-480. doi: 10.1302/2046-3758.810.BJR-2018-0341.R1. eCollection 2019 Oct.

OBJECTIVES: Experimental studies indicate that non-steroidal anti-inflammatory drugs (NSAIDs) may have negative effects on fracture healing. This study aimed to assess the effect of immediate and delayed short-term administration of clinically relevant parecoxib doses and timing on fracture healing...

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Hjorthaug GA, Søreide E, Nordsletten L, et al. Short-term perioperative parecoxib is not detrimental to shaft fracture healing in a rat model. Bone Joint Res. 2019;8(10):472-480doi: 10.1302/2046-3758.810.BJR-2018-0341.R1.
Hjorthaug, G. A., Søreide, E., Nordsletten, L., Madsen, J. E., Reinholt, F. P., Niratisairak, S., & Dimmen, S. (2019). Short-term perioperative parecoxib is not detrimental to shaft fracture healing in a rat model. Bone & joint research, 8(10), 472-480. https://doi.org/10.1302/2046-3758.810.BJR-2018-0341.R1
Hjorthaug, G A, et al. "Short-term perioperative parecoxib is not detrimental to shaft fracture healing in a rat model." Bone & joint research vol. 8,10 (2019): 472-480. doi: https://doi.org/10.1302/2046-3758.810.BJR-2018-0341.R1
Hjorthaug GA, Søreide E, Nordsletten L, Madsen JE, Reinholt FP, Niratisairak S, Dimmen S. Short-term perioperative parecoxib is not detrimental to shaft fracture healing in a rat model. Bone Joint Res. 2019 Nov 02;8(10):472-480. doi: 10.1302/2046-3758.810.BJR-2018-0341.R1. eCollection 2019 Oct. PMID: 31728186; PMCID: PMC6825043.
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Ductus Arteriosus of Extremely Preterm Twins is More Resistant to Cyclooxygenase Inhibitors Than Those of Singletons.

Pediatric cardiology

Ono T, Miura Y, Kaga M, Sato T, Sanjo M.
PMID: 34716772
Pediatr Cardiol. 2021 Oct 30; doi: 10.1007/s00246-021-02765-0. Epub 2021 Oct 30.

Symptomatic patent ductus arteriosus (sPDA) is common among preterm infants, and can lead to several complications. This is particularly true for extremely preterm infants, as closure of the ductus arteriosus using cyclooxygenase inhibitors is often difficult. A recent study...

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Ono T, Miura Y, Kaga M, et al. Ductus Arteriosus of Extremely Preterm Twins is More Resistant to Cyclooxygenase Inhibitors Than Those of Singletons. Pediatr Cardiol. 2021;doi: 10.1007/s00246-021-02765-0.
Ono, T., Miura, Y., Kaga, M., Sato, T., & Sanjo, M. (2021). Ductus Arteriosus of Extremely Preterm Twins is More Resistant to Cyclooxygenase Inhibitors Than Those of Singletons. Pediatric cardiology, . https://doi.org/10.1007/s00246-021-02765-0
Ono, Toshikazu, et al. "Ductus Arteriosus of Extremely Preterm Twins is More Resistant to Cyclooxygenase Inhibitors Than Those of Singletons." Pediatric cardiology vol. (2021). doi: https://doi.org/10.1007/s00246-021-02765-0
Ono T, Miura Y, Kaga M, Sato T, Sanjo M. Ductus Arteriosus of Extremely Preterm Twins is More Resistant to Cyclooxygenase Inhibitors Than Those of Singletons. Pediatr Cardiol. 2021 Oct 30; doi: 10.1007/s00246-021-02765-0. Epub 2021 Oct 30. PMID: 34716772; PMCID: PMC8556772.
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A systematic review of the evidence supporting post-operative antithrombotic use following cardiopulmonary bypass in children with CHD.

Cardiology in the young

Thompson EJ, Foote HP, Li JS, Rotta AT, Goldenberg NA, Hornik CP.
PMID: 34986908
Cardiol Young. 2022 Jan;32(1):10-20. doi: 10.1017/S1047951121005205. Epub 2022 Jan 06.

OBJECTIVES: To determine the optimal antithrombotic agent choice, timing of initiation, dosing and duration of therapy for paediatric patients undergoing cardiac surgery with cardiopulmonary bypass.METHODS: We used PubMed and EMBASE to systematically review the existing literature of clinical trials...

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Thompson EJ, Foote HP, Li JS, et al. A systematic review of the evidence supporting post-operative antithrombotic use following cardiopulmonary bypass in children with CHD. Cardiol Young. 2022;32(1):10-20doi: 10.1017/S1047951121005205.
Thompson, E. J., Foote, H. P., Li, J. S., Rotta, A. T., Goldenberg, N. A., & Hornik, C. P. (2022). A systematic review of the evidence supporting post-operative antithrombotic use following cardiopulmonary bypass in children with CHD. Cardiology in the young, 32(1), 10-20. https://doi.org/10.1017/S1047951121005205
Thompson, Elizabeth J, et al. "A systematic review of the evidence supporting post-operative antithrombotic use following cardiopulmonary bypass in children with CHD." Cardiology in the young vol. 32,1 (2022): 10-20. doi: https://doi.org/10.1017/S1047951121005205
Thompson EJ, Foote HP, Li JS, Rotta AT, Goldenberg NA, Hornik CP. A systematic review of the evidence supporting post-operative antithrombotic use following cardiopulmonary bypass in children with CHD. Cardiol Young. 2022 Jan;32(1):10-20. doi: 10.1017/S1047951121005205. Epub 2022 Jan 06. PMID: 34986908.
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Differential effects of cyclooxygenase-2 (COX-2) inhibitors on endoplasmic reticulum (ER) stress in human coronary artery endothelial cells.

Vascular pharmacology

Haas MJ, Warda F, Bikkina P, Landicho MA, Kapadia P, Parekh S, Mooradian AD.
PMID: 34843981
Vascul Pharmacol. 2021 Nov 27;106948. doi: 10.1016/j.vph.2021.106948. Epub 2021 Nov 27.

Selective cyclooxygenase-2 (COX-2) inhibitor rofecoxib was pulled off the market because of its association with increased risk of adverse cardiovascular effects. The precise underlying mechanism for the differential effects of COX-2 inhibitors on cardiovascular risk is not known. Since...

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Haas MJ, Warda F, Bikkina P, et al. Differential effects of cyclooxygenase-2 (COX-2) inhibitors on endoplasmic reticulum (ER) stress in human coronary artery endothelial cells. Vascul Pharmacol. 2021;106948doi: 10.1016/j.vph.2021.106948.
Haas, M. J., Warda, F., Bikkina, P., Landicho, M. A., Kapadia, P., Parekh, S., & Mooradian, A. D. (2021). Differential effects of cyclooxygenase-2 (COX-2) inhibitors on endoplasmic reticulum (ER) stress in human coronary artery endothelial cells. Vascular pharmacology, 106948. https://doi.org/10.1016/j.vph.2021.106948
Haas, Michael J, et al. "Differential effects of cyclooxygenase-2 (COX-2) inhibitors on endoplasmic reticulum (ER) stress in human coronary artery endothelial cells." Vascular pharmacology vol. (2021): 106948. doi: https://doi.org/10.1016/j.vph.2021.106948
Haas MJ, Warda F, Bikkina P, Landicho MA, Kapadia P, Parekh S, Mooradian AD. Differential effects of cyclooxygenase-2 (COX-2) inhibitors on endoplasmic reticulum (ER) stress in human coronary artery endothelial cells. Vascul Pharmacol. 2021 Nov 27;106948. doi: 10.1016/j.vph.2021.106948. Epub 2021 Nov 27. PMID: 34843981.
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NSAID use and clinical outcomes in COVID-19 patients: A 38-center retrospective cohort study.

medRxiv : the preprint server for health sciences

Reese JT, Coleman B, Chan L, Blau H, Callahan TJ, Cappelletti L, Fontana T, Bradwell KR, Harris NL, Casiraghi E, Valentini G, Karlebach G, Deer R, McMurry JA, Haendel MA, Chute CG, Pfaff E, Moffitt R, Spratt H, Singh J, Mungall CJ, Williams AE, Robinson PN.
PMID: 33907758
medRxiv. 2021 Dec 22; doi: 10.1101/2021.04.13.21255438.

BACKGROUND: Non-steroidal anti-inflammatory drugs (NSAIDs) are commonly used to reduce pain, fever, and inflammation but have been associated with complications in community-acquired pneumonia. Observations shortly after the start of the COVID-19 pandemic in 2020 suggested that ibuprofen was associated...

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Reese JT, Coleman B, Chan L, et al. NSAID use and clinical outcomes in COVID-19 patients: A 38-center retrospective cohort study. medRxiv. 2021;doi: 10.1101/2021.04.13.21255438.
Reese, J. T., Coleman, B., Chan, L., Blau, H., Callahan, T. J., Cappelletti, L., Fontana, T., Bradwell, K. R., Harris, N. L., Casiraghi, E., Valentini, G., Karlebach, G., Deer, R., McMurry, J. A., Haendel, M. A., Chute, C. G., Pfaff, E., Moffitt, R., Spratt, H., Singh, J., Mungall, C. J., Williams, A. E., & Robinson, P. N. (2021). NSAID use and clinical outcomes in COVID-19 patients: A 38-center retrospective cohort study. medRxiv : the preprint server for health sciences, . https://doi.org/10.1101/2021.04.13.21255438
Reese, Justin T, et al. "NSAID use and clinical outcomes in COVID-19 patients: A 38-center retrospective cohort study." medRxiv : the preprint server for health sciences vol. (2021). doi: https://doi.org/10.1101/2021.04.13.21255438
Reese JT, Coleman B, Chan L, Blau H, Callahan TJ, Cappelletti L, Fontana T, Bradwell KR, Harris NL, Casiraghi E, Valentini G, Karlebach G, Deer R, McMurry JA, Haendel MA, Chute CG, Pfaff E, Moffitt R, Spratt H, Singh J, Mungall CJ, Williams AE, Robinson PN. NSAID use and clinical outcomes in COVID-19 patients: A 38-center retrospective cohort study. medRxiv. 2021 Dec 22; doi: 10.1101/2021.04.13.21255438. PMID: 33907758; PMCID: PMC8077581.
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Eicosanoids mediate microaggregation and nodulation responses to bacterial infections in black cutworms, Agrotis ipsilon, and true armyworms, Pseudaletia unipuncta.

Journal of insect physiology

Stanley-Samuelson D, Rana RL, Pedibhotla VK, Miller JS, Jurenka RA.
PMID: 12769916
J Insect Physiol. 1997 Feb 21;43(2):125-133. doi: 10.1016/s0022-1910(96)00102-3.

Nodulation is the first, and quantitatively predominant, cellular defense reaction to bacterial infection in insects and other invertebrates. Inhibition of eicosanoid biosynthesis in true armyworms, Pseudaletia unipuncta, and black cutworms, Agrotis ipsilon, immediately prior to intrahemocoelic injections with heat-killed...

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Stanley-Samuelson D, Rana RL, Pedibhotla VK, et al. Eicosanoids mediate microaggregation and nodulation responses to bacterial infections in black cutworms, Agrotis ipsilon, and true armyworms, Pseudaletia unipuncta. J Insect Physiol. 1997;43(2):125-133doi: 10.1016/s0022-1910(96)00102-3.
Stanley-Samuelson, D., Rana, R. L., Pedibhotla, V. K., Miller, J. S., & Jurenka, R. A. (1997). Eicosanoids mediate microaggregation and nodulation responses to bacterial infections in black cutworms, Agrotis ipsilon, and true armyworms, Pseudaletia unipuncta. Journal of insect physiology, 43(2), 125-133. https://doi.org/10.1016/s0022-1910(96)00102-3
Stanley-Samuelson, D, et al. "Eicosanoids mediate microaggregation and nodulation responses to bacterial infections in black cutworms, Agrotis ipsilon, and true armyworms, Pseudaletia unipuncta." Journal of insect physiology vol. 43,2 (1997): 125-133. doi: https://doi.org/10.1016/s0022-1910(96)00102-3
Stanley-Samuelson D, Rana RL, Pedibhotla VK, Miller JS, Jurenka RA. Eicosanoids mediate microaggregation and nodulation responses to bacterial infections in black cutworms, Agrotis ipsilon, and true armyworms, Pseudaletia unipuncta. J Insect Physiol. 1997 Feb 21;43(2):125-133. doi: 10.1016/s0022-1910(96)00102-3. PMID: 12769916.
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Evidence for the participation of arachidonic acid metabolites in trehalose efflux from the hormone activated fat body of the cockroach (Periplaneta americana).

Journal of insect physiology

Ali I, Finley C, Steele JE.
PMID: 12770411
J Insect Physiol. 1998 Nov;44(11):1119-1126. doi: 10.1016/s0022-1910(97)00076-0.

The hypertrehalosemic hormones, HTH-I and HTH-II, activate trehalose synthesis and increase the rate of sugar efflux from Periplaneta americana fat body in vitro. These processes are unaffected by the diacylglycerol, 1-oleyl-2-acetyl-sn-glycerol, an activator of protein kinase C. Similarly, H-7...

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Ali I, Finley C, Steele JE. Evidence for the participation of arachidonic acid metabolites in trehalose efflux from the hormone activated fat body of the cockroach (Periplaneta americana). J Insect Physiol. 1998;44(11):1119-1126doi: 10.1016/s0022-1910(97)00076-0.
Ali, I., Finley, C., & Steele, J. E. (1998). Evidence for the participation of arachidonic acid metabolites in trehalose efflux from the hormone activated fat body of the cockroach (Periplaneta americana). Journal of insect physiology, 44(11), 1119-1126. https://doi.org/10.1016/s0022-1910(97)00076-0
Ali, I, et al. "Evidence for the participation of arachidonic acid metabolites in trehalose efflux from the hormone activated fat body of the cockroach (Periplaneta americana)." Journal of insect physiology vol. 44,11 (1998): 1119-1126. doi: https://doi.org/10.1016/s0022-1910(97)00076-0
Ali I, Finley C, Steele JE. Evidence for the participation of arachidonic acid metabolites in trehalose efflux from the hormone activated fat body of the cockroach (Periplaneta americana). J Insect Physiol. 1998 Nov;44(11):1119-1126. doi: 10.1016/s0022-1910(97)00076-0. PMID: 12770411.
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Prediction of the potency of mammalian cyclooxygenase inhibitors with ensemble proteochemometric modeling.

Journal of cheminformatics

Cortes-Ciriano I, Murrell DS, van Westen GJ, Bender A, Malliavin TE.
PMID: 25705261
J Cheminform. 2015 Jan 16;7:1. doi: 10.1186/s13321-014-0049-z. eCollection 2015.

Cyclooxygenases (COX) are present in the body in two isoforms, namely: COX-1, constitutively expressed, and COX-2, induced in physiopathological conditions such as cancer or chronic inflammation. The inhibition of COX with non-steroideal anti-inflammatory drugs (NSAIDs) is the most widely...

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Cortes-Ciriano I, Murrell DS, van Westen GJ, et al. Prediction of the potency of mammalian cyclooxygenase inhibitors with ensemble proteochemometric modeling. J Cheminform. 2015;7:1doi: 10.1186/s13321-014-0049-z.
Cortes-Ciriano, I., Murrell, D. S., van Westen, G. J., Bender, A., & Malliavin, T. E. (2015). Prediction of the potency of mammalian cyclooxygenase inhibitors with ensemble proteochemometric modeling. Journal of cheminformatics, 71. https://doi.org/10.1186/s13321-014-0049-z
Cortes-Ciriano, Isidro, et al. "Prediction of the potency of mammalian cyclooxygenase inhibitors with ensemble proteochemometric modeling." Journal of cheminformatics vol. 7 (2015): 1. doi: https://doi.org/10.1186/s13321-014-0049-z
Cortes-Ciriano I, Murrell DS, van Westen GJ, Bender A, Malliavin TE. Prediction of the potency of mammalian cyclooxygenase inhibitors with ensemble proteochemometric modeling. J Cheminform. 2015 Jan 16;7:1. doi: 10.1186/s13321-014-0049-z. eCollection 2015. PMID: 25705261; PMCID: PMC4335128.
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Secretion of β-thromboglobulin and Serotonin from Human Platelets Induced by Microbubbles Differs from Secretion Induced by Collagen and Polystyrene Microspheres.

Platelets

Stain-Malmgren R, Thorsen T, Nordvik A, Holmsen H.
PMID: 21043721
Platelets. 1994;5(4):209-13. doi: 10.3109/09537109409006048.

The effect of nitrogen (N(2))-microbubbles on human platelets resembles that of common agonists in terms of aggregation, but displays one unusual feature in that cyclooxygenase-inhibitors (e.g. aspirin) poorly inhibit the reaction. In the present study, we have investigated the...

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Stain-Malmgren R, Thorsen T, Nordvik A, et al. Secretion of β-thromboglobulin and Serotonin from Human Platelets Induced by Microbubbles Differs from Secretion Induced by Collagen and Polystyrene Microspheres. Platelets. 1994;5(4):209-13doi: 10.3109/09537109409006048.
Stain-Malmgren, R., Thorsen, T., Nordvik, A., & Holmsen, H. (1994). Secretion of β-thromboglobulin and Serotonin from Human Platelets Induced by Microbubbles Differs from Secretion Induced by Collagen and Polystyrene Microspheres. Platelets, 5(4), 209-13. https://doi.org/10.3109/09537109409006048
Stain-Malmgren, R, et al. "Secretion of β-thromboglobulin and Serotonin from Human Platelets Induced by Microbubbles Differs from Secretion Induced by Collagen and Polystyrene Microspheres." Platelets vol. 5,4 (1994): 209-13. doi: https://doi.org/10.3109/09537109409006048
Stain-Malmgren R, Thorsen T, Nordvik A, Holmsen H. Secretion of β-thromboglobulin and Serotonin from Human Platelets Induced by Microbubbles Differs from Secretion Induced by Collagen and Polystyrene Microspheres. Platelets. 1994;5(4):209-13. doi: 10.3109/09537109409006048. PMID: 21043721.
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