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Testosterone improves erectile function through inhibition of reactive oxygen species generation in castrated rats.

PeerJ

Li R, Meng X, Zhang Y, Wang T, Yang J, Niu Y, Cui K, Wang S, Liu J, Rao K.
PMID: 27168996
PeerJ. 2016 May 03;4:e2000. doi: 10.7717/peerj.2000. eCollection 2016.

Testosterone is overwhelmingly important in regulating erectile physiology. However, the associated molecular mechanisms are poorly understood. The purpose of this study was to explore the effects and mechanisms of testosterone in erectile dysfunction (ED) in castrated rats. Forty male...

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Li R, Meng X, Zhang Y, et al. Testosterone improves erectile function through inhibition of reactive oxygen species generation in castrated rats. PeerJ. 2016;4:e2000doi: 10.7717/peerj.2000.
Li, R., Meng, X., Zhang, Y., Wang, T., Yang, J., Niu, Y., Cui, K., Wang, S., Liu, J., & Rao, K. (2016). Testosterone improves erectile function through inhibition of reactive oxygen species generation in castrated rats. PeerJ, 4e2000. https://doi.org/10.7717/peerj.2000
Li, Rui, et al. "Testosterone improves erectile function through inhibition of reactive oxygen species generation in castrated rats." PeerJ vol. 4 (2016): e2000. doi: https://doi.org/10.7717/peerj.2000
Li R, Meng X, Zhang Y, Wang T, Yang J, Niu Y, Cui K, Wang S, Liu J, Rao K. Testosterone improves erectile function through inhibition of reactive oxygen species generation in castrated rats. PeerJ. 2016 May 03;4:e2000. doi: 10.7717/peerj.2000. eCollection 2016. PMID: 27168996; PMCID: PMC4860316.
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Effect of Saw Palmetto Supplements on Androgen-Sensitive LNCaP Human Prostate Cancer Cell Number and Syrian Hamster Flank Organ Growth.

Evidence-based complementary and alternative medicine : eCAM

Opoku-Acheampong AB, Penugonda K, Lindshield BL.
PMID: 27272436
Evid Based Complement Alternat Med. 2016;2016:8135135. doi: 10.1155/2016/8135135. Epub 2016 May 04.

Saw palmetto supplements (SPS) are commonly consumed by men with prostate cancer. We investigated whether SPS fatty acids and phytosterols concentrations determine their growth-inhibitory action in androgen-sensitive LNCaP cells and hamster flank organs. High long-chain fatty acids-low phytosterols (HLLP)...

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Opoku-Acheampong AB, Penugonda K, Lindshield BL. Effect of Saw Palmetto Supplements on Androgen-Sensitive LNCaP Human Prostate Cancer Cell Number and Syrian Hamster Flank Organ Growth. Evid Based Complement Alternat Med. 2016;2016:8135135doi: 10.1155/2016/8135135.
Opoku-Acheampong, A. B., Penugonda, K., & Lindshield, B. L. (2016). Effect of Saw Palmetto Supplements on Androgen-Sensitive LNCaP Human Prostate Cancer Cell Number and Syrian Hamster Flank Organ Growth. Evidence-based complementary and alternative medicine : eCAM, 20168135135. https://doi.org/10.1155/2016/8135135
Opoku-Acheampong, Alexander B, et al. "Effect of Saw Palmetto Supplements on Androgen-Sensitive LNCaP Human Prostate Cancer Cell Number and Syrian Hamster Flank Organ Growth." Evidence-based complementary and alternative medicine : eCAM vol. 2016 (2016): 8135135. doi: https://doi.org/10.1155/2016/8135135
Opoku-Acheampong AB, Penugonda K, Lindshield BL. Effect of Saw Palmetto Supplements on Androgen-Sensitive LNCaP Human Prostate Cancer Cell Number and Syrian Hamster Flank Organ Growth. Evid Based Complement Alternat Med. 2016;2016:8135135. doi: 10.1155/2016/8135135. Epub 2016 May 04. PMID: 27272436; PMCID: PMC4870347.
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Assessment of steroidogenic pathways that do not require testosterone as intermediate.

Hormone molecular biology and clinical investigation

Luu-The V.
PMID: 25961251
Horm Mol Biol Clin Investig. 2011 Mar 01;5(3):161-5. doi: 10.1515/HMBCI.2011.007.

Traditional literature and textbooks generally describe that estradiol (E2) and dihydrotestosterone (DHT) are synthesized from the aromatization and 5α-reduction of testosterone (T), respectively, following a pathway in which T is an essential intermediate (Tpath). This pathway implies that the...

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Luu-The V. Assessment of steroidogenic pathways that do not require testosterone as intermediate. Horm Mol Biol Clin Investig. 2011;5(3):161-5doi: 10.1515/HMBCI.2011.007.
Luu-The, V. (2011). Assessment of steroidogenic pathways that do not require testosterone as intermediate. Hormone molecular biology and clinical investigation, 5(3), 161-5. https://doi.org/10.1515/HMBCI.2011.007
Luu-The, Van. "Assessment of steroidogenic pathways that do not require testosterone as intermediate." Hormone molecular biology and clinical investigation vol. 5,3 (2011): 161-5. doi: https://doi.org/10.1515/HMBCI.2011.007
Luu-The V. Assessment of steroidogenic pathways that do not require testosterone as intermediate. Horm Mol Biol Clin Investig. 2011 Mar 01;5(3):161-5. doi: 10.1515/HMBCI.2011.007. PMID: 25961251.
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Androgens, body fat Distribution and Adipogenesis.

Current obesity reports

Zerradi M, Dereumetz J, Boulet MM, Tchernof A.
PMID: 26626916
Curr Obes Rep. 2014 Dec;3(4):396-403. doi: 10.1007/s13679-014-0119-6.

Androgens are regulators of important adipocyte functions such as adipogenesis, lipid storage, and lipolysis. Through depot-specific impact on the cells of each fat compartment, androgens could modulate body fat distribution patterns in humans. Testosterone and dihydrotestosterone have been shown...

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Zerradi M, Dereumetz J, Boulet MM, et al. Androgens, body fat Distribution and Adipogenesis. Curr Obes Rep. 2014;3(4):396-403doi: 10.1007/s13679-014-0119-6.
Zerradi, M., Dereumetz, J., Boulet, M. M., & Tchernof, A. (2014). Androgens, body fat Distribution and Adipogenesis. Current obesity reports, 3(4), 396-403. https://doi.org/10.1007/s13679-014-0119-6
Zerradi, Mouna, et al. "Androgens, body fat Distribution and Adipogenesis." Current obesity reports vol. 3,4 (2014): 396-403. doi: https://doi.org/10.1007/s13679-014-0119-6
Zerradi M, Dereumetz J, Boulet MM, Tchernof A. Androgens, body fat Distribution and Adipogenesis. Curr Obes Rep. 2014 Dec;3(4):396-403. doi: 10.1007/s13679-014-0119-6. PMID: 26626916.
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Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells.

Annals of dermatology

Shin S, Kim K, Lee MJ, Lee J, Choi S, Kim KS, Ko JM, Han H, Kim SY, Youn HJ, Ahn KJ, An IS, An S, Cha HJ.
PMID: 27274631
Ann Dermatol. 2016 Jun;28(3):327-34. doi: 10.5021/ad.2016.28.3.327. Epub 2016 May 25.

BACKGROUND: Dihydrotestosterone (DHT) induces androgenic alopecia by shortening the hair follicle growth phase, resulting in hair loss. We previously demonstrated how changes in the microRNA (miRNA) expression profile influenced DHT-mediated cell death, cell cycle arrest, cell viability, the generation...

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Shin S, Kim K, Lee MJ, et al. Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells. Ann Dermatol. 2016;28(3):327-34doi: 10.5021/ad.2016.28.3.327.
Shin, S., Kim, K., Lee, M. J., Lee, J., Choi, S., Kim, K. S., Ko, J. M., Han, H., Kim, S. Y., Youn, H. J., Ahn, K. J., An, I. S., An, S., & Cha, H. J. (2016). Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells. Annals of dermatology, 28(3), 327-34. https://doi.org/10.5021/ad.2016.28.3.327
Shin, Shanghun, et al. "Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells." Annals of dermatology vol. 28,3 (2016): 327-34. doi: https://doi.org/10.5021/ad.2016.28.3.327
Shin S, Kim K, Lee MJ, Lee J, Choi S, Kim KS, Ko JM, Han H, Kim SY, Youn HJ, Ahn KJ, An IS, An S, Cha HJ. Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells. Ann Dermatol. 2016 Jun;28(3):327-34. doi: 10.5021/ad.2016.28.3.327. Epub 2016 May 25. PMID: 27274631; PMCID: PMC4884709.
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The effect of epitestosterone on spermatogenesis in rats.

Endocrine regulations

Behre HM, Hampl R, Lapcik O, Hill M, Starka L.
PMID: 10993981
Endocr Regul. 1995 Dec;29(4):195-199.

The effect of 3-week treatment with increasing doses of endogenous steroid with antiandrogenic properties, epitestosterone (ET), on the spermatogenesis was studied in adult male rats by flow cytometry and correlated with the effect on gonadotropin gene expression and secretion,...

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Behre HM, Hampl R, Lapcik O, et al. The effect of epitestosterone on spermatogenesis in rats. Endocr Regul. 1995;29(4):195-199
Behre, H. M., Hampl, R., Lapcik, O., Hill, M., & Starka, L. (1995). The effect of epitestosterone on spermatogenesis in rats. Endocrine regulations, 29(4), 195-199.
Behre, et al. "The effect of epitestosterone on spermatogenesis in rats." Endocrine regulations vol. 29,4 (1995): 195-199.
Behre HM, Hampl R, Lapcik O, Hill M, Starka L. The effect of epitestosterone on spermatogenesis in rats. Endocr Regul. 1995 Dec;29(4):195-199. PMID: 10993981.
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Xenobiotic and steroid biotransformation activities in rainbow trout gill epithelial cells in culture.

Aquatic toxicology (Amsterdam, Netherlands)

Leguen I, Carlsson C, Perdu-Durand E, Prunet P, Pärt P, Cravedi JP.
PMID: 10686323
Aquat Toxicol. 2000 Mar 01;48(2):165-176. doi: 10.1016/s0166-445x(99)00043-0.

The biotransformation of xenobiotics and steroids was investigated in cultured respiratory epithelial cells from rainbow trout (Oncorhynchus mykiss) gills. As a first approach, ethoxyresorufin-O-deethylase (EROD), chosen as a marker of CYP1A activity, was measured in monolayers of adherent cells....

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Leguen I, Carlsson C, Perdu-Durand E, et al. Xenobiotic and steroid biotransformation activities in rainbow trout gill epithelial cells in culture. Aquat Toxicol. 2000;48(2):165-176doi: 10.1016/s0166-445x(99)00043-0.
Leguen, I., Carlsson, C., Perdu-Durand, E., Prunet, P., Pärt, P., & Cravedi, J. P. (2000). Xenobiotic and steroid biotransformation activities in rainbow trout gill epithelial cells in culture. Aquatic toxicology (Amsterdam, Netherlands), 48(2), 165-176. https://doi.org/10.1016/s0166-445x(99)00043-0
Leguen, et al. "Xenobiotic and steroid biotransformation activities in rainbow trout gill epithelial cells in culture." Aquatic toxicology (Amsterdam, Netherlands) vol. 48,2 (2000): 165-176. doi: https://doi.org/10.1016/s0166-445x(99)00043-0
Leguen I, Carlsson C, Perdu-Durand E, Prunet P, Pärt P, Cravedi JP. Xenobiotic and steroid biotransformation activities in rainbow trout gill epithelial cells in culture. Aquat Toxicol. 2000 Mar 01;48(2):165-176. doi: 10.1016/s0166-445x(99)00043-0. PMID: 10686323.
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Effects of Estradiol and Testosterone on the Synthesis, Expression and Degradation of Androgen Receptor in Human Uterine Endometrial Fibroblasts.

Journal of biomedical science

Fujimoto J, Nishigaki M, Hori M, Ichigo S, Morishita S, Tamaya T.
PMID: 11725052
J Biomed Sci. 1995 Apr;2(2):160-165. doi: 10.1007/BF02253067.

The mechanism of known receptor-mediated androgen effects on the endometrial stroma was studied in endometrial fibroblasts derived from human uterus. 17beta-Estradiol (E) induced the expressions of androgen receptor (AR) mRNA, and predominantly increased the level of testosterone-binding sites (TBS)...

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Fujimoto J, Nishigaki M, Hori M, et al. Effects of Estradiol and Testosterone on the Synthesis, Expression and Degradation of Androgen Receptor in Human Uterine Endometrial Fibroblasts. J Biomed Sci. 1995;2(2):160-165doi: 10.1007/BF02253067.
Fujimoto, J., Nishigaki, M., Hori, M., Ichigo, S., Morishita, S., & Tamaya, T. (1995). Effects of Estradiol and Testosterone on the Synthesis, Expression and Degradation of Androgen Receptor in Human Uterine Endometrial Fibroblasts. Journal of biomedical science, 2(2), 160-165. https://doi.org/10.1007/BF02253067
Fujimoto, J., et al. "Effects of Estradiol and Testosterone on the Synthesis, Expression and Degradation of Androgen Receptor in Human Uterine Endometrial Fibroblasts." Journal of biomedical science vol. 2,2 (1995): 160-165. doi: https://doi.org/10.1007/BF02253067
Fujimoto J, Nishigaki M, Hori M, Ichigo S, Morishita S, Tamaya T. Effects of Estradiol and Testosterone on the Synthesis, Expression and Degradation of Androgen Receptor in Human Uterine Endometrial Fibroblasts. J Biomed Sci. 1995 Apr;2(2):160-165. doi: 10.1007/BF02253067. PMID: 11725052.
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Single-step purification of full-length human androgen receptor.

Nuclear receptor signaling

Juzumiene D, Chang CY, Fan D, Hartney T, Norris JD, McDonnell DP.
PMID: 16604169
Nucl Recept Signal. 2005;3:e001. doi: 10.1621/nrs.03001. Epub 2005 Oct 21.

The full-length human androgen receptor with an N-terminal biotin acceptor peptide tag was overexpressed in Spodoptera frugiperda cells in the presence of 1 microM dihydrotestosterone. Site-specific biotinylation of BAP was achieved in vivo by co-expression of E. coli biotin...

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Juzumiene D, Chang CY, Fan D, et al. Single-step purification of full-length human androgen receptor. Nucl Recept Signal. 2005;3:e001doi: 10.1621/nrs.03001.
Juzumiene, D., Chang, C. Y., Fan, D., Hartney, T., Norris, J. D., & McDonnell, D. P. (2005). Single-step purification of full-length human androgen receptor. Nuclear receptor signaling, 3e001. https://doi.org/10.1621/nrs.03001
Juzumiene, Dalia, et al. "Single-step purification of full-length human androgen receptor." Nuclear receptor signaling vol. 3 (2005): e001. doi: https://doi.org/10.1621/nrs.03001
Juzumiene D, Chang CY, Fan D, Hartney T, Norris JD, McDonnell DP. Single-step purification of full-length human androgen receptor. Nucl Recept Signal. 2005;3:e001. doi: 10.1621/nrs.03001. Epub 2005 Oct 21. PMID: 16604169; PMCID: PMC1402216.
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5alpha-reductase: history and clinical importance.

Reviews in urology

Marks LS.
PMID: 16985920
Rev Urol. 2004;6:S11-21.

The treatment of men with symptomatic benign prostatic hyperplasia (BPH) has shifted dramatically from surgery to drug therapy over the past decade. The revolution in BPH treatment began with the discovery of congenital 5alpha-reductase (5AR) deficiency, leading to the...

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Marks LS. 5alpha-reductase: history and clinical importance. Rev Urol. 2004;6:S11-21
Marks, L. S. (2004). 5alpha-reductase: history and clinical importance. Reviews in urology, 6S11-21.
Marks, Leonard S. "5alpha-reductase: history and clinical importance." Reviews in urology vol. 6 (2004): S11-21.
Marks LS. 5alpha-reductase: history and clinical importance. Rev Urol. 2004;6:S11-21. PMID: 16985920; PMCID: PMC1472916.
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Transdifferentiation of cultured human prostate cancer cells to a neuroendocrine cell phenotype in a hormone-depleted medium.

Urologic oncology

Shen R, Dorai T, Szaboles M, Katz AE, Olsson CA, Buttyan R.
PMID: 21227062
Urol Oncol. 1997 Mar-Apr;3(2):67-75. doi: 10.1016/s1078-1439(97)00039-2.

Neuroendocrine (NE) cells are enigmatically found in association with human prostate cancers and their numbers are reported to increase in advanced and hormoneresistant tumors. The origin of this cell type and the reason for their appearance in prostate tumors...

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Shen R, Dorai T, Szaboles M, et al. Transdifferentiation of cultured human prostate cancer cells to a neuroendocrine cell phenotype in a hormone-depleted medium. Urol Oncol. 1997;3(2):67-75doi: 10.1016/s1078-1439(97)00039-2.
Shen, R., Dorai, T., Szaboles, M., Katz, A. E., Olsson, C. A., & Buttyan, R. (1997). Transdifferentiation of cultured human prostate cancer cells to a neuroendocrine cell phenotype in a hormone-depleted medium. Urologic oncology, 3(2), 67-75. https://doi.org/10.1016/s1078-1439(97)00039-2
Shen, R, et al. "Transdifferentiation of cultured human prostate cancer cells to a neuroendocrine cell phenotype in a hormone-depleted medium." Urologic oncology vol. 3,2 (1997): 67-75. doi: https://doi.org/10.1016/s1078-1439(97)00039-2
Shen R, Dorai T, Szaboles M, Katz AE, Olsson CA, Buttyan R. Transdifferentiation of cultured human prostate cancer cells to a neuroendocrine cell phenotype in a hormone-depleted medium. Urol Oncol. 1997 Mar-Apr;3(2):67-75. doi: 10.1016/s1078-1439(97)00039-2. PMID: 21227062.
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5α-Reductase Isozymes in the Prostate.

Journal of medical sciences (Taipei, Taiwan)

Zhu YS, Sun GH.
PMID: 18483578
J Med Sci. 2005;25(1):1-12.

5α-reductases convert testosterone to dihydrotestosterone (DHT). There are two 5α-reductase isozymes, type 1 and type 2 in humans and animals. Mutations in type 2 isozyme with decreased enzymatic activity cause male pseudohermaphroditism. The affected 46XY individuals have high normal...

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Zhu YS, Sun GH. 5α-Reductase Isozymes in the Prostate. J Med Sci. 2005;25(1):1-12
Zhu, Y. S., & Sun, G. H. (2005). 5α-Reductase Isozymes in the Prostate. Journal of medical sciences (Taipei, Taiwan), 25(1), 1-12.
Zhu, Yuan-Shan, and Sun, Guang-Huan. "5α-Reductase Isozymes in the Prostate." Journal of medical sciences (Taipei, Taiwan) vol. 25,1 (2005): 1-12.
Zhu YS, Sun GH. 5α-Reductase Isozymes in the Prostate. J Med Sci. 2005;25(1):1-12. PMID: 18483578; PMCID: PMC2386416.
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Showing 13 to 24 of 576 entries