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Converting Men From Clomiphene Citrate to Natesto for Hypogonadism Improves Libido, Maintains Semen Parameters, and Reduces Estradiol.

Urology

Kavoussi PK, Machen GL, Gilkey MS, Chen SH, Kavoussi KM, Esqueda A, Wininger JD, Barsky M, Kavoussi SK.
PMID: 33301741
Urology. 2021 Feb;148:141-144. doi: 10.1016/j.urology.2020.11.047. Epub 2020 Dec 07.

OBJECTIVE: To evaluate outcomes including libido, semen parameters, testosterone, estradiol (E2), follicle stimulating hormone (FSH), and luteinizing hormone when converting men with low libido on Clomiphene Citrate (CC) to Natesto.METHODS: A retrospective chart review was performed. Baseline hormones prior...

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Kavoussi PK, Machen GL, Gilkey MS, et al. Converting Men From Clomiphene Citrate to Natesto for Hypogonadism Improves Libido, Maintains Semen Parameters, and Reduces Estradiol. Urology. 2020;148:141-144doi: 10.1016/j.urology.2020.11.047.
Kavoussi, P. K., Machen, G. L., Gilkey, M. S., Chen, S. H., Kavoussi, K. M., Esqueda, A., Wininger, J. D., Barsky, M., & Kavoussi, S. K. (2021). Converting Men From Clomiphene Citrate to Natesto for Hypogonadism Improves Libido, Maintains Semen Parameters, and Reduces Estradiol. Urology, 148141-144. https://doi.org/10.1016/j.urology.2020.11.047
Kavoussi, Parviz K, et al. "Converting Men From Clomiphene Citrate to Natesto for Hypogonadism Improves Libido, Maintains Semen Parameters, and Reduces Estradiol." Urology vol. 148 (2021): 141-144. doi: https://doi.org/10.1016/j.urology.2020.11.047
Kavoussi PK, Machen GL, Gilkey MS, Chen SH, Kavoussi KM, Esqueda A, Wininger JD, Barsky M, Kavoussi SK. Converting Men From Clomiphene Citrate to Natesto for Hypogonadism Improves Libido, Maintains Semen Parameters, and Reduces Estradiol. Urology. 2021 Feb;148:141-144. doi: 10.1016/j.urology.2020.11.047. Epub 2020 Dec 07. PMID: 33301741.
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GnRH-a-Induced Perimenopausal Rat Modeling and Black Cohosh Preparations' Effect on Rat's Reproductive Endocrine.

Frontiers in endocrinology

Chen J, Wang H, Dong Z, Liu J, Qin Z, Bao M, Yu H, Zhang S, Zhang W, Qi C, Wu J.
PMID: 35002948
Front Endocrinol (Lausanne). 2021 Dec 24;12:683552. doi: 10.3389/fendo.2021.683552. eCollection 2021.

BACKGROUND: Endometriosis (EMS) is an estrogen-dependent disease, which easily recurs after operation. Gonadotropin-releasing hormone agonist (GnRH-a), an estrogen-inhibiting drug, can effectively inhibit the secretion of gonadotropin by pituitary gland, so as to significantly decrease the ovarian hormone level and...

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Chen J, Wang H, Dong Z, et al. GnRH-a-Induced Perimenopausal Rat Modeling and Black Cohosh Preparations' Effect on Rat's Reproductive Endocrine. Front Endocrinol (Lausanne). 2021;12:683552doi: 10.3389/fendo.2021.683552.
Chen, J., Wang, H., Dong, Z., Liu, J., Qin, Z., Bao, M., Yu, H., Zhang, S., Zhang, W., Qi, C., & Wu, J. (2021). GnRH-a-Induced Perimenopausal Rat Modeling and Black Cohosh Preparations' Effect on Rat's Reproductive Endocrine. Frontiers in endocrinology, 12683552. https://doi.org/10.3389/fendo.2021.683552
Chen, Jiming, et al. "GnRH-a-Induced Perimenopausal Rat Modeling and Black Cohosh Preparations' Effect on Rat's Reproductive Endocrine." Frontiers in endocrinology vol. 12 (2021): 683552. doi: https://doi.org/10.3389/fendo.2021.683552
Chen J, Wang H, Dong Z, Liu J, Qin Z, Bao M, Yu H, Zhang S, Zhang W, Qi C, Wu J. GnRH-a-Induced Perimenopausal Rat Modeling and Black Cohosh Preparations' Effect on Rat's Reproductive Endocrine. Front Endocrinol (Lausanne). 2021 Dec 24;12:683552. doi: 10.3389/fendo.2021.683552. eCollection 2021. PMID: 35002948; PMCID: PMC8739485.
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17β-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner.

Molecular and biochemical parasitology

Pereira-Suárez AL, Galván-Ramírez ML, Rodríguez-Pérez LR, López-Pulido EI, Hernández-Silva CD, Ramírez-López IG, Morales Amaya GV, Lopez Cabrera LD, Muñoz-Valle JF, Ramírez-de-Arellano A.
PMID: 34822916
Mol Biochem Parasitol. 2022 Jan;247:111433. doi: 10.1016/j.molbiopara.2021.111433. Epub 2021 Nov 22.

Toxoplasma gondii (T. gondii) is a parasite common in pregnancy. Monocytes and macrophages are a significant immunologic barrier against T. gondii by boosting up inflammation. This outcome is highly regulated by signaling pathways such as MAPK (ERK1/2) and PI3K...

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Pereira-Suárez AL, Galván-Ramírez ML, Rodríguez-Pérez LR, et al. 17β-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner. Mol Biochem Parasitol. 2021;247:111433doi: 10.1016/j.molbiopara.2021.111433.
Pereira-Suárez, A. L., Galván-Ramírez, M. L., Rodríguez-Pérez, L. R., López-Pulido, E. I., Hernández-Silva, C. D., Ramírez-López, I. G., Morales Amaya, G. V., Lopez Cabrera, L. D., Muñoz-Valle, J. F., & Ramírez-de-Arellano, A. (2022). 17β-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner. Molecular and biochemical parasitology, 247111433. https://doi.org/10.1016/j.molbiopara.2021.111433
Pereira-Suárez, Ana Laura, et al. "17β-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner." Molecular and biochemical parasitology vol. 247 (2022): 111433. doi: https://doi.org/10.1016/j.molbiopara.2021.111433
Pereira-Suárez AL, Galván-Ramírez ML, Rodríguez-Pérez LR, López-Pulido EI, Hernández-Silva CD, Ramírez-López IG, Morales Amaya GV, Lopez Cabrera LD, Muñoz-Valle JF, Ramírez-de-Arellano A. 17β-estradiol modulates the expression of hormonal receptors on THP-1 T. gondii-infected macrophages and monocytes in an AKT and ERK-dependent manner. Mol Biochem Parasitol. 2022 Jan;247:111433. doi: 10.1016/j.molbiopara.2021.111433. Epub 2021 Nov 22. PMID: 34822916.
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Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction.

American journal of clinical and experimental urology

Wegner KA, Ruetten H, Girardi NM, O'Driscoll CA, Sandhu JK, Turco AE, Abler LL, Wang P, Wang Z, Bjorling DE, Malinowski R, Peterson RE, Strand DW, Marker PC, Vezina CM.
PMID: 33816700
Am J Clin Exp Urol. 2021 Feb 15;9(1):121-131. eCollection 2021.

Urinary voiding dysfunction in aging men can cause bothersome symptoms and irreparable tissue damage. Underlying mechanisms are not fully known. We previously demonstrated that subcutaneous, slow-release testosterone and estradiol implants (T+E2) drive a pattern of urinary voiding dysfunction in...

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Wegner KA, Ruetten H, Girardi NM, et al. Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction. Am J Clin Exp Urol. 2021;9(1):121-131
Wegner, K. A., Ruetten, H., Girardi, N. M., O'Driscoll, C. A., Sandhu, J. K., Turco, A. E., Abler, L. L., Wang, P., Wang, Z., Bjorling, D. E., Malinowski, R., Peterson, R. E., Strand, D. W., Marker, P. C., & Vezina, C. M. (2021). Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction. American journal of clinical and experimental urology, 9(1), 121-131.
Wegner, Kyle A, et al. "Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction." American journal of clinical and experimental urology vol. 9,1 (2021): 121-131.
Wegner KA, Ruetten H, Girardi NM, O'Driscoll CA, Sandhu JK, Turco AE, Abler LL, Wang P, Wang Z, Bjorling DE, Malinowski R, Peterson RE, Strand DW, Marker PC, Vezina CM. Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction. Am J Clin Exp Urol. 2021 Feb 15;9(1):121-131. eCollection 2021. PMID: 33816700; PMCID: PMC8012832.
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Long term exposure to tris (2-chloroethyl) phosphate (TCEP) causes alterations in reproductive hormones, vitellogenin, antioxidant enzymes, and histology of gonads in zebrafish (Danio rerio): In vivo and computational analysis.

Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Sutha J, Anila PA, Gayathri M, Ramesh M.
PMID: 35032655
Comp Biochem Physiol C Toxicol Pharmacol. 2022 Jan 12;109263. doi: 10.1016/j.cbpc.2021.109263. Epub 2022 Jan 12.

In aquatic milieus, tris (2-chloroethyl) phosphate (TCEP) was detected as an emerging environmental contaminant. In this study, in vivo experiment and in-silico docking was integrated systematically to explore the toxic mechanisms of TCEP using zebrafish (Danio rerio). Fish (mean...

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Sutha J, Anila PA, Gayathri M, et al. Long term exposure to tris (2-chloroethyl) phosphate (TCEP) causes alterations in reproductive hormones, vitellogenin, antioxidant enzymes, and histology of gonads in zebrafish (Danio rerio): In vivo and computational analysis. Comp Biochem Physiol C Toxicol Pharmacol. 2022;109263doi: 10.1016/j.cbpc.2021.109263.
Sutha, J., Anila, P. A., Gayathri, M., & Ramesh, M. (2022). Long term exposure to tris (2-chloroethyl) phosphate (TCEP) causes alterations in reproductive hormones, vitellogenin, antioxidant enzymes, and histology of gonads in zebrafish (Danio rerio): In vivo and computational analysis. Comparative biochemistry and physiology. Toxicology & pharmacology : CBP, 109263. https://doi.org/10.1016/j.cbpc.2021.109263
Sutha, Jesudass, et al. "Long term exposure to tris (2-chloroethyl) phosphate (TCEP) causes alterations in reproductive hormones, vitellogenin, antioxidant enzymes, and histology of gonads in zebrafish (Danio rerio): In vivo and computational analysis." Comparative biochemistry and physiology. Toxicology & pharmacology : CBP vol. (2022): 109263. doi: https://doi.org/10.1016/j.cbpc.2021.109263
Sutha J, Anila PA, Gayathri M, Ramesh M. Long term exposure to tris (2-chloroethyl) phosphate (TCEP) causes alterations in reproductive hormones, vitellogenin, antioxidant enzymes, and histology of gonads in zebrafish (Danio rerio): In vivo and computational analysis. Comp Biochem Physiol C Toxicol Pharmacol. 2022 Jan 12;109263. doi: 10.1016/j.cbpc.2021.109263. Epub 2022 Jan 12. PMID: 35032655.
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Improved hormonal and oxidative changes by Royal Jelly in the rat model of PCOS: An experimental study.

International journal of reproductive biomedicine

Khazaei F, Ghanbari E, Khazaei M.
PMID: 34401646
Int J Reprod Biomed. 2021 Jul 27;19(6):515-524. doi: 10.18502/ijrm.v19i6.9373. eCollection 2021 Jun.

BACKGROUND: Polycystic ovarian syndrome (PCOS) is an endocrine and complex metabolic disorder, associated with anovulation, changes in sex hormone, biochemical factors, and ovarian tissue. Royal Jelly (RJ) has antioxidant and anti-inflammatory properties.OBJECTIVE: To examine the therapeutic effect of RJ...

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Khazaei F, Ghanbari E, Khazaei M. Improved hormonal and oxidative changes by Royal Jelly in the rat model of PCOS: An experimental study. Int J Reprod Biomed. 2021;19(6):515-524doi: 10.18502/ijrm.v19i6.9373.
Khazaei, F., Ghanbari, E., & Khazaei, M. (2021). Improved hormonal and oxidative changes by Royal Jelly in the rat model of PCOS: An experimental study. International journal of reproductive biomedicine, 19(6), 515-524. https://doi.org/10.18502/ijrm.v19i6.9373
Khazaei, Fatemeh, et al. "Improved hormonal and oxidative changes by Royal Jelly in the rat model of PCOS: An experimental study." International journal of reproductive biomedicine vol. 19,6 (2021): 515-524. doi: https://doi.org/10.18502/ijrm.v19i6.9373
Khazaei F, Ghanbari E, Khazaei M. Improved hormonal and oxidative changes by Royal Jelly in the rat model of PCOS: An experimental study. Int J Reprod Biomed. 2021 Jul 27;19(6):515-524. doi: 10.18502/ijrm.v19i6.9373. eCollection 2021 Jun. PMID: 34401646; PMCID: PMC8350846.
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17β-Estradiol Attenuates Intracerebral Hemorrhage-Induced Blood-Brain Barrier Injury and Oxidative Stress Through SRC3-Mediated PI3K/Akt Signaling Pathway in a Mouse Model.

ASN neuro

Xiao H, Liu J, He J, Lan Z, Deng M, Hu Z.
PMID: 34491125
ASN Neuro. 2021 Jan-Dec;13:17590914211038443. doi: 10.1177/17590914211038443.

Estrogen is neuroprotective in brain injury models, and steroid receptor cofactor 3 (SRC3) mediates estrogen signaling. We aimed to investigate whether and how SRC3 is involved in the neuroprotective effects of 17ß-estradiol (E2) in a mouse model of intracerebral...

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Xiao H, Liu J, He J, et al. 17β-Estradiol Attenuates Intracerebral Hemorrhage-Induced Blood-Brain Barrier Injury and Oxidative Stress Through SRC3-Mediated PI3K/Akt Signaling Pathway in a Mouse Model. ASN Neuro. 2021;13:17590914211038443doi: 10.1177/17590914211038443.
Xiao, H., Liu, J., He, J., Lan, Z., Deng, M., & Hu, Z. (2021). 17β-Estradiol Attenuates Intracerebral Hemorrhage-Induced Blood-Brain Barrier Injury and Oxidative Stress Through SRC3-Mediated PI3K/Akt Signaling Pathway in a Mouse Model. ASN neuro, 1317590914211038443. https://doi.org/10.1177/17590914211038443
Xiao, Han, et al. "17β-Estradiol Attenuates Intracerebral Hemorrhage-Induced Blood-Brain Barrier Injury and Oxidative Stress Through SRC3-Mediated PI3K/Akt Signaling Pathway in a Mouse Model." ASN neuro vol. 13 (2021): 17590914211038443. doi: https://doi.org/10.1177/17590914211038443
Xiao H, Liu J, He J, Lan Z, Deng M, Hu Z. 17β-Estradiol Attenuates Intracerebral Hemorrhage-Induced Blood-Brain Barrier Injury and Oxidative Stress Through SRC3-Mediated PI3K/Akt Signaling Pathway in a Mouse Model. ASN Neuro. 2021 Jan-Dec;13:17590914211038443. doi: 10.1177/17590914211038443. PMID: 34491125; PMCID: PMC8580490.
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Protective role of 17β-estradiol treatment in renal injury on female rats submitted to brain death.

Annals of translational medicine

Armstrong-Jr R, Ricardo-da-Silva FY, Vidal-Dos-Santos M, Correia CJ, Anunciação LF, Coutinho E Silva RDS, Moreira LFP, Leuvenink HGD, Breithaupt-Faloppa AC.
PMID: 34430566
Ann Transl Med. 2021 Jul;9(14):1125. doi: 10.21037/atm-21-1408.

BACKGROUND: Clinical and experimental data highlight the consequences of brain death on the quality of organs and demonstrate the importance of donor state to the results of transplantation. Female rats show higher cardio-pulmonary injury linked to decreased concentrations of...

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Armstrong-Jr R, Ricardo-da-Silva FY, Vidal-Dos-Santos M, et al. Protective role of 17β-estradiol treatment in renal injury on female rats submitted to brain death. Ann Transl Med. 2021;9(14):1125doi: 10.21037/atm-21-1408.
Armstrong-Jr, R., Ricardo-da-Silva, F. Y., Vidal-Dos-Santos, M., Correia, C. J., Anunciação, L. F., Coutinho E Silva, R. D. S., Moreira, L. F. P., Leuvenink, H. G. D., & Breithaupt-Faloppa, A. C. (2021). Protective role of 17β-estradiol treatment in renal injury on female rats submitted to brain death. Annals of translational medicine, 9(14), 1125. https://doi.org/10.21037/atm-21-1408
Armstrong-Jr, Roberto, et al. "Protective role of 17β-estradiol treatment in renal injury on female rats submitted to brain death." Annals of translational medicine vol. 9,14 (2021): 1125. doi: https://doi.org/10.21037/atm-21-1408
Armstrong-Jr R, Ricardo-da-Silva FY, Vidal-Dos-Santos M, Correia CJ, Anunciação LF, Coutinho E Silva RDS, Moreira LFP, Leuvenink HGD, Breithaupt-Faloppa AC. Protective role of 17β-estradiol treatment in renal injury on female rats submitted to brain death. Ann Transl Med. 2021 Jul;9(14):1125. doi: 10.21037/atm-21-1408. PMID: 34430566; PMCID: PMC8350685.
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Showing 5161 to 5168 of 5168 entries