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Showing 13 to 24 of 2513 entries
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Expression of 14-3-3sigma, p16 and p53 proteins in anal squamous intraepithelial neoplasm and squamous cell carcinoma.

International journal of clinical and experimental pathology

Roma AA, Goldblum JR, Fazio V, Yang B.
PMID: 18787619
Int J Clin Exp Pathol. 2008 Jan 10;1(5):419-25.

14-3-3sigma is a p53-regulated G2/M inhibitor involved in numerous cellular signaling pathways related to cell cycle, DNA repair and apoptosis. Recent studies have showed that 14-3-3sigma was silenced transcriptionally through promoter hypermethylation mainly in HPV-negative vulvar squamous cell carcinoma...

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Roma AA, Goldblum JR, Fazio V, et al. Expression of 14-3-3sigma, p16 and p53 proteins in anal squamous intraepithelial neoplasm and squamous cell carcinoma. Int J Clin Exp Pathol. 2008;1(5):419-25
Roma, A. A., Goldblum, J. R., Fazio, V., & Yang, B. (2008). Expression of 14-3-3sigma, p16 and p53 proteins in anal squamous intraepithelial neoplasm and squamous cell carcinoma. International journal of clinical and experimental pathology, 1(5), 419-25.
Roma, Andres A, et al. "Expression of 14-3-3sigma, p16 and p53 proteins in anal squamous intraepithelial neoplasm and squamous cell carcinoma." International journal of clinical and experimental pathology vol. 1,5 (2008): 419-25.
Roma AA, Goldblum JR, Fazio V, Yang B. Expression of 14-3-3sigma, p16 and p53 proteins in anal squamous intraepithelial neoplasm and squamous cell carcinoma. Int J Clin Exp Pathol. 2008 Jan 10;1(5):419-25. PMID: 18787619; PMCID: PMC2480570.
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Invasive micropapillary carcinoma of the sigmoid colon: distinct morphology and aggressive behavior.

International journal of clinical and experimental pathology

Wen P, Xu Y, Frankel WL, Shen R.
PMID: 18787620
Int J Clin Exp Pathol. 2008 Jan 01;1(5):457-60.

We report a case of invasive micropapillary carcinoma of the sigmoid colon in a 72-year-old female with anemia and abdominal pain. Grossly, the tumor demonstrated a deeply invasive, ulcerated fungating mass. Microscopically, the carcinoma was predominantly composed of micropapillae...

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Wen P, Xu Y, Frankel WL, et al. Invasive micropapillary carcinoma of the sigmoid colon: distinct morphology and aggressive behavior. Int J Clin Exp Pathol. 2008;1(5):457-60
Wen, P., Xu, Y., Frankel, W. L., & Shen, R. (2008). Invasive micropapillary carcinoma of the sigmoid colon: distinct morphology and aggressive behavior. International journal of clinical and experimental pathology, 1(5), 457-60.
Wen, Ping, et al. "Invasive micropapillary carcinoma of the sigmoid colon: distinct morphology and aggressive behavior." International journal of clinical and experimental pathology vol. 1,5 (2008): 457-60.
Wen P, Xu Y, Frankel WL, Shen R. Invasive micropapillary carcinoma of the sigmoid colon: distinct morphology and aggressive behavior. Int J Clin Exp Pathol. 2008 Jan 01;1(5):457-60. PMID: 18787620; PMCID: PMC2480571.
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In vitro healing of avascular meniscal injuries with fresh and frozen plugs treated with TGF-beta1 and IGF-1 in sheep.

International journal of clinical and experimental pathology

Izal I, Ripalda P, Acosta CA, Forriol F.
PMID: 18787623
Int J Clin Exp Pathol. 2008 Jan 01;1(5):426-34.

We studied the effect of freezing and inserting meniscal plugs in lesions generated in the avascular area of sheep menisci maintained in vitro, and whether the healing process can be improved by adding growth factors TGF-beta1 and IGF-1. Thirty...

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Izal I, Ripalda P, Acosta CA, et al. In vitro healing of avascular meniscal injuries with fresh and frozen plugs treated with TGF-beta1 and IGF-1 in sheep. Int J Clin Exp Pathol. 2008;1(5):426-34
Izal, I., Ripalda, P., Acosta, C. A., & Forriol, F. (2008). In vitro healing of avascular meniscal injuries with fresh and frozen plugs treated with TGF-beta1 and IGF-1 in sheep. International journal of clinical and experimental pathology, 1(5), 426-34.
Izal, Iñigo, et al. "In vitro healing of avascular meniscal injuries with fresh and frozen plugs treated with TGF-beta1 and IGF-1 in sheep." International journal of clinical and experimental pathology vol. 1,5 (2008): 426-34.
Izal I, Ripalda P, Acosta CA, Forriol F. In vitro healing of avascular meniscal injuries with fresh and frozen plugs treated with TGF-beta1 and IGF-1 in sheep. Int J Clin Exp Pathol. 2008 Jan 01;1(5):426-34. PMID: 18787623; PMCID: PMC2480574.
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The 8p11 myeloproliferative syndrome: review of literature and an illustrative case report.

International journal of clinical and experimental pathology

Goradia A, Bayerl M, Cornfield D.
PMID: 18787627
Int J Clin Exp Pathol. 2008 Jan 01;1(5):448-56.

The 8p11 myeloproliferative syndrome (EMS), also called stem cell leukemia/lymphoma (SCLL), is a relatively rare condition characterized in its typical form by the occurrence, either simultaneously or sequentially, of a bcr/abl-negative myeloproliferative disorder and a lymphoma, usually a precursor...

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Goradia A, Bayerl M, Cornfield D. The 8p11 myeloproliferative syndrome: review of literature and an illustrative case report. Int J Clin Exp Pathol. 2008;1(5):448-56
Goradia, A., Bayerl, M., & Cornfield, D. (2008). The 8p11 myeloproliferative syndrome: review of literature and an illustrative case report. International journal of clinical and experimental pathology, 1(5), 448-56.
Goradia, Ami, et al. "The 8p11 myeloproliferative syndrome: review of literature and an illustrative case report." International journal of clinical and experimental pathology vol. 1,5 (2008): 448-56.
Goradia A, Bayerl M, Cornfield D. The 8p11 myeloproliferative syndrome: review of literature and an illustrative case report. Int J Clin Exp Pathol. 2008 Jan 01;1(5):448-56. PMID: 18787627; PMCID: PMC2480578.
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The genetic character of O-D change in influenza A.

British journal of experimental pathology

BURNET FM, STONE JD.
PMID: 15403572
Br J Exp Pathol. 1949 Oct;30(5):419-25.

No abstract available.

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BURNET FM, STONE JD. The genetic character of O-D change in influenza A. Br J Exp Pathol. 1949;30(5):419-25
BURNET, F. M., & STONE, J. D. (1949). The genetic character of O-D change in influenza A. British journal of experimental pathology, 30(5), 419-25.
BURNET, F M, and STONE, J D. "The genetic character of O-D change in influenza A." British journal of experimental pathology vol. 30,5 (1949): 419-25.
BURNET FM, STONE JD. The genetic character of O-D change in influenza A. Br J Exp Pathol. 1949 Oct;30(5):419-25. PMID: 15403572; PMCID: PMC2073285.
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Anaplastic lymphoma kinase-positive large B-cell lymphoma: a distinct clinicopathological entity.

International journal of clinical and experimental pathology

Li S.
PMID: 19636398
Int J Clin Exp Pathol. 2009 Mar 13;2(6):508-18.

Anaplastic lymphoma kinase-positive large B-cell lymphoma (ALK(+) LBCL) represents a distinct subtype of mature B-cell neoplasms in the most recent WHO classification of hematolymphoid neoplasms. It has a characteristic immunoblastic/plasmablastic morphology, a distinct immunophenotypic profile and recurrent cytogenetic/molecular genetic...

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Li S. Anaplastic lymphoma kinase-positive large B-cell lymphoma: a distinct clinicopathological entity. Int J Clin Exp Pathol. 2009;2(6):508-18
Li, S. (2009). Anaplastic lymphoma kinase-positive large B-cell lymphoma: a distinct clinicopathological entity. International journal of clinical and experimental pathology, 2(6), 508-18.
Li, Shiyong. "Anaplastic lymphoma kinase-positive large B-cell lymphoma: a distinct clinicopathological entity." International journal of clinical and experimental pathology vol. 2,6 (2009): 508-18.
Li S. Anaplastic lymphoma kinase-positive large B-cell lymphoma: a distinct clinicopathological entity. Int J Clin Exp Pathol. 2009 Mar 13;2(6):508-18. PMID: 19636398; PMCID: PMC2713458.
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The role of T cells in osteoporosis, an update.

International journal of clinical and experimental pathology

Zhao W, Liu Y, Cahill CM, Yang W, Rogers JT, Huang X.
PMID: 19636401
Int J Clin Exp Pathol. 2009 May 20;2(6):544-52.

Emerging evidence highlights the importance of the interplay between the bone and immune systems. That evidence bolsters a longstanding recognition that estrogen deficiency, infection, inflammation, and autoimmune disorders are associated with systemic and local bone loss. Yet, only recently...

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Zhao W, Liu Y, Cahill CM, et al. The role of T cells in osteoporosis, an update. Int J Clin Exp Pathol. 2009;2(6):544-52
Zhao, W., Liu, Y., Cahill, C. M., Yang, W., Rogers, J. T., & Huang, X. (2009). The role of T cells in osteoporosis, an update. International journal of clinical and experimental pathology, 2(6), 544-52.
Zhao, Wen, et al. "The role of T cells in osteoporosis, an update." International journal of clinical and experimental pathology vol. 2,6 (2009): 544-52.
Zhao W, Liu Y, Cahill CM, Yang W, Rogers JT, Huang X. The role of T cells in osteoporosis, an update. Int J Clin Exp Pathol. 2009 May 20;2(6):544-52. PMID: 19636401; PMCID: PMC2713452.
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Dysplastic ("in-situ") Lesions in multofocal renal oncocytomas (oncocytosis).

International journal of clinical and experimental pathology

Huang J, Lee P, Mikami Y, Melamed J.
PMID: 19636405
Int J Clin Exp Pathol. 2009 Jun 18;2(6):583-7.

Preneoplastic lesions for renal oncocytosis have not been well defined. We have attempted to identify the putative in-situ or dysplastic change in nephrectomy specimens with oncocytosis. Cases of multiple oncocytoma previously identified in radical nephrectomy specimens (n = 5)...

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Huang J, Lee P, Mikami Y, et al. Dysplastic ("in-situ") Lesions in multofocal renal oncocytomas (oncocytosis). Int J Clin Exp Pathol. 2009;2(6):583-7
Huang, J., Lee, P., Mikami, Y., & Melamed, J. (2009). Dysplastic ("in-situ") Lesions in multofocal renal oncocytomas (oncocytosis). International journal of clinical and experimental pathology, 2(6), 583-7.
Huang, Jiaoti, et al. "Dysplastic ("in-situ") Lesions in multofocal renal oncocytomas (oncocytosis)." International journal of clinical and experimental pathology vol. 2,6 (2009): 583-7.
Huang J, Lee P, Mikami Y, Melamed J. Dysplastic ("in-situ") Lesions in multofocal renal oncocytomas (oncocytosis). Int J Clin Exp Pathol. 2009 Jun 18;2(6):583-7. PMID: 19636405; PMCID: PMC2713448.
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Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy.

International journal of clinical and experimental pathology

Oshima K, Dickson DW.
PMID: 19158937
Int J Clin Exp Pathol. 2009;2(4):399-406. Epub 2008 Dec 20.

Alzheimer disease (AD) is characterized by numerous senile plaques (SP) in addition to widespread neocortical neurofibrillary tangles (NFT). Some elderly have pathologic aging (PA), which is characterized by numerous SP composed of diffuse amyloid deposits with few or no...

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Oshima K, Dickson DW. Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy. Int J Clin Exp Pathol. 2008;2(4):399-406
Oshima, K., & Dickson, D. W. (2009). Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy. International journal of clinical and experimental pathology, 2(4), 399-406.
Oshima, Kenichi, and Dickson, Dennis W. "Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy." International journal of clinical and experimental pathology vol. 2,4 (2009): 399-406.
Oshima K, Dickson DW. Cortical Alzheimer type pathology does not influence tau pathology in progressive supranuclear palsy. Int J Clin Exp Pathol. 2009;2(4):399-406. Epub 2008 Dec 20. PMID: 19158937; PMCID: PMC2615597.
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Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties.

International journal of clinical and experimental pathology

Shiwany NA, Xie J, Guo Q.
PMID: 19158991
Int J Clin Exp Pathol. 2009;2(4):339-52. Epub 2008 Nov 26.

Alzheimer's disease (AD) is a leading cause of chronic dementia in the United States. Its incidence is increasing with an attendant increase in associated health care costs. Amyloid beta peptide (Abeta; a 39-42 amino acid molecule) is the major...

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Shiwany NA, Xie J, Guo Q. Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties. Int J Clin Exp Pathol. 2008;2(4):339-52
Shiwany, N. A., Xie, J., & Guo, Q. (2009). Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties. International journal of clinical and experimental pathology, 2(4), 339-52.
Shiwany, Najeeb A, et al. "Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties." International journal of clinical and experimental pathology vol. 2,4 (2009): 339-52.
Shiwany NA, Xie J, Guo Q. Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties. Int J Clin Exp Pathol. 2009;2(4):339-52. Epub 2008 Nov 26. PMID: 19158991; PMCID: PMC2615591.
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Disseminated coccidioidomycosis: a case report from the United kingdom.

International journal of clinical and experimental pathology

Al-Daraji WI, Al-Mahmoud RM, Ali MA.
PMID: 19294009
Int J Clin Exp Pathol. 2009;2(5):494-7. Epub 2008 Jun 04.

We reported the first case of disseminated coccidioidomycosis from the UK successfully treated with amphotericin B, where skin biopsy was the initial clue for the correct diagnosis.

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Al-Daraji WI, Al-Mahmoud RM, Ali MA. Disseminated coccidioidomycosis: a case report from the United kingdom. Int J Clin Exp Pathol. 2008;2(5):494-7
Al-Daraji, W. I., Al-Mahmoud, R. M., & Ali, M. A. (2009). Disseminated coccidioidomycosis: a case report from the United kingdom. International journal of clinical and experimental pathology, 2(5), 494-7.
Al-Daraji, Wael I, et al. "Disseminated coccidioidomycosis: a case report from the United kingdom." International journal of clinical and experimental pathology vol. 2,5 (2009): 494-7.
Al-Daraji WI, Al-Mahmoud RM, Ali MA. Disseminated coccidioidomycosis: a case report from the United kingdom. Int J Clin Exp Pathol. 2009;2(5):494-7. Epub 2008 Jun 04. PMID: 19294009; PMCID: PMC2655149.
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Detection of human herpesvirus DNA in Kikuchi-Fujimoto disease and reactive lymphoid hyperplasia.

International journal of clinical and experimental pathology

Hudnall SD, Chen T, Amr S, Young KH, Henry K.
PMID: 18787614
Int J Clin Exp Pathol. 2008 Jan 01;1(4):362-8.

Kikuchi-Fujimoto disease (KFD), or histiocytic necrotizing lymphadenitis, is a subacute inflammatory disorder most often seen in young women with clinicopathologic features suggestive of an infectious etiology. The most commonly suspected infectious agents in KFD are the human herpesviruses EBV,...

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Hudnall SD, Chen T, Amr S, et al. Detection of human herpesvirus DNA in Kikuchi-Fujimoto disease and reactive lymphoid hyperplasia. Int J Clin Exp Pathol. 2008;1(4):362-8
Hudnall, S. D., Chen, T., Amr, S., Young, K. H., & Henry, K. (2008). Detection of human herpesvirus DNA in Kikuchi-Fujimoto disease and reactive lymphoid hyperplasia. International journal of clinical and experimental pathology, 1(4), 362-8.
Hudnall, S David, et al. "Detection of human herpesvirus DNA in Kikuchi-Fujimoto disease and reactive lymphoid hyperplasia." International journal of clinical and experimental pathology vol. 1,4 (2008): 362-8.
Hudnall SD, Chen T, Amr S, Young KH, Henry K. Detection of human herpesvirus DNA in Kikuchi-Fujimoto disease and reactive lymphoid hyperplasia. Int J Clin Exp Pathol. 2008 Jan 01;1(4):362-8. PMID: 18787614; PMCID: PMC2480545.
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Showing 13 to 24 of 2513 entries