Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 25 to 36 of 2513 entries
Sorted by: Best Match Show Resources per page
Loss of DNA mismatch repair protein hMSH6 in ovarian cancer is histotype-specific.

International journal of clinical and experimental pathology

Zhai QJ, Rosen DG, Lu K, Liu J.
PMID: 18787632
Int J Clin Exp Pathol. 2008 Jan 31;1(6):502-9.

Microsatellite instability (MSI) due to defects in DNA mismatch repair genes may be involved in the development of a subset of human ovarian carcinomas. The role of one such gene, hMSH6, in ovarian cancer is not well documented. We...

Cite
Zhai QJ, Rosen DG, Lu K, et al. Loss of DNA mismatch repair protein hMSH6 in ovarian cancer is histotype-specific. Int J Clin Exp Pathol. 2008;1(6):502-9
Zhai, Q. J., Rosen, D. G., Lu, K., & Liu, J. (2008). Loss of DNA mismatch repair protein hMSH6 in ovarian cancer is histotype-specific. International journal of clinical and experimental pathology, 1(6), 502-9.
Zhai, Qihui Jim, et al. "Loss of DNA mismatch repair protein hMSH6 in ovarian cancer is histotype-specific." International journal of clinical and experimental pathology vol. 1,6 (2008): 502-9.
Zhai QJ, Rosen DG, Lu K, Liu J. Loss of DNA mismatch repair protein hMSH6 in ovarian cancer is histotype-specific. Int J Clin Exp Pathol. 2008 Jan 31;1(6):502-9. PMID: 18787632; PMCID: PMC2480583.
Copy Download .nbib Format: NLM
Alpha-methylacyl-CoA racemase expression is upregulated in gastric adenocarcinoma: a study of 249 cases.

International journal of clinical and experimental pathology

Truong CD, Li W, Feng W, Cagle P, Khoury T, Alrawi S, Xie K, Yao J, Tan D.
PMID: 18787636
Int J Clin Exp Pathol. 2008 Apr 10;1(6):518-23.

Alpha-methylacyl-CoA racemase (AMACR [P504S]) is a mitochondrial and peroxisomal enzyme involved in beta-oxidation of dietary branched-chain fatty acids and their derivatives. Recent studies showed that AMACR is expressed in several neoplasms, including prostate and colon cancer. However, AMACR expression...

Cite
Truong CD, Li W, Feng W, et al. Alpha-methylacyl-CoA racemase expression is upregulated in gastric adenocarcinoma: a study of 249 cases. Int J Clin Exp Pathol. 2008;1(6):518-23
Truong, C. D., Li, W., Feng, W., Cagle, P., Khoury, T., Alrawi, S., Xie, K., Yao, J., & Tan, D. (2008). Alpha-methylacyl-CoA racemase expression is upregulated in gastric adenocarcinoma: a study of 249 cases. International journal of clinical and experimental pathology, 1(6), 518-23.
Truong, Camtu D, et al. "Alpha-methylacyl-CoA racemase expression is upregulated in gastric adenocarcinoma: a study of 249 cases." International journal of clinical and experimental pathology vol. 1,6 (2008): 518-23.
Truong CD, Li W, Feng W, Cagle P, Khoury T, Alrawi S, Xie K, Yao J, Tan D. Alpha-methylacyl-CoA racemase expression is upregulated in gastric adenocarcinoma: a study of 249 cases. Int J Clin Exp Pathol. 2008 Apr 10;1(6):518-23. PMID: 18787636; PMCID: PMC2480587.
Copy Download .nbib Format: NLM
HOX proteins and leukemia.

International journal of clinical and experimental pathology

Sitwala KV, Dandekar MN, Hess JL.
PMID: 18787682
Int J Clin Exp Pathol. 2008 Mar 30;1(6):461-74.

HOX and three amino acid loop extension (TALE) proteins cooperate to induce transformation in mouse leukemia models, and are dysregulated in a variety of human leukemias. Despite decades of research, the mechanism of action for Hox proteins in embryogenesis...

Cite
Sitwala KV, Dandekar MN, Hess JL. HOX proteins and leukemia. Int J Clin Exp Pathol. 2008;1(6):461-74
Sitwala, K. V., Dandekar, M. N., & Hess, J. L. (2008). HOX proteins and leukemia. International journal of clinical and experimental pathology, 1(6), 461-74.
Sitwala, Kajal V, et al. "HOX proteins and leukemia." International journal of clinical and experimental pathology vol. 1,6 (2008): 461-74.
Sitwala KV, Dandekar MN, Hess JL. HOX proteins and leukemia. Int J Clin Exp Pathol. 2008 Mar 30;1(6):461-74. PMID: 18787682; PMCID: PMC2480589.
Copy Download .nbib Format: NLM
Authors' Reply.

International journal of experimental pathology

Yu M, Zou Q.
PMID: 23772981
Int J Exp Pathol. 2013 Jun 17; doi: 10.1111/iep.12032b. Epub 2013 Jun 17.

No abstract available.

Cite
Yu M, Zou Q. Authors' Reply. Int J Exp Pathol. 2013;doi: 10.1111/iep.12032b.
Yu, M., & Zou, Q. (2013). Authors' Reply. International journal of experimental pathology, . https://doi.org/10.1111/iep.12032b
Yu, Man, and Zou, Qinghua. "Authors' Reply." International journal of experimental pathology vol. (2013). doi: https://doi.org/10.1111/iep.12032b
Yu M, Zou Q. Authors' Reply. Int J Exp Pathol. 2013 Jun 17; doi: 10.1111/iep.12032b. Epub 2013 Jun 17. PMID: 23772981.
Copy Download .nbib Format: NLM
Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling.

Journal of clinical & experimental pathology

de la Monte SM, Tong M, Agarwal AR, Cadenas E.
PMID: 27525191
J Clin Exp Pathol. 2016 Apr;6(2). doi: 10.4172/2161-0681.1000269. Epub 2016 Apr 17.

BACKGROUND: Alcoholic liver disease (ALD) is associated with impairments in hepatic insulin and insulin-like growth factor (IGF) signaling through cell growth, survival, and metabolic pathways. Since not all heavy drinkers develop ALD, co-factors may be important. Epidemiologic data indicate...

Cite
de la Monte SM, Tong M, Agarwal AR, et al. Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. J Clin Exp Pathol. 2016;6(2)doi: 10.4172/2161-0681.1000269.
de la Monte, S. M., Tong, M., Agarwal, A. R., & Cadenas, E. (2016). Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. Journal of clinical & experimental pathology, 6(2), . https://doi.org/10.4172/2161-0681.1000269
de la Monte, Suzanne M, et al. "Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling." Journal of clinical & experimental pathology vol. 6,2 (2016). doi: https://doi.org/10.4172/2161-0681.1000269
de la Monte SM, Tong M, Agarwal AR, Cadenas E. Tobacco Smoke-Induced Hepatic Injury with Steatosis, Inflammation, and Impairments in Insulin and Insulin-Like Growth Factor Signaling. J Clin Exp Pathol. 2016 Apr;6(2). doi: 10.4172/2161-0681.1000269. Epub 2016 Apr 17. PMID: 27525191; PMCID: PMC4979551.
Copy Download .nbib Format: NLM
TGF-β1 induces airway smooth muscle cell proliferation and remodeling in asthmatic mice by up-regulating miR-181a and suppressing PTEN.

International journal of clinical and experimental pathology

Lv X, Li Y, Gong Q, Jiang Z.
PMID: 31933731
Int J Clin Exp Pathol. 2019 Jan 01;12(1):173-181. eCollection 2019.

Several studies have shown that transforming growth factor β1 (TGF-β1) plays a crucial role in remodeling and proliferation of airway smooth muscle cells (ASMCs). However, its molecular mechanism needs to be further studied. TGF-β1 can up-regulate the level of...

Cite
Lv X, Li Y, Gong Q, et al. TGF-β1 induces airway smooth muscle cell proliferation and remodeling in asthmatic mice by up-regulating miR-181a and suppressing PTEN. Int J Clin Exp Pathol. 2019;12(1):173-181
Lv, X., Li, Y., Gong, Q., & Jiang, Z. (2019). TGF-β1 induces airway smooth muscle cell proliferation and remodeling in asthmatic mice by up-regulating miR-181a and suppressing PTEN. International journal of clinical and experimental pathology, 12(1), 173-181.
Lv, Xianghong, et al. "TGF-β1 induces airway smooth muscle cell proliferation and remodeling in asthmatic mice by up-regulating miR-181a and suppressing PTEN." International journal of clinical and experimental pathology vol. 12,1 (2019): 173-181.
Lv X, Li Y, Gong Q, Jiang Z. TGF-β1 induces airway smooth muscle cell proliferation and remodeling in asthmatic mice by up-regulating miR-181a and suppressing PTEN. Int J Clin Exp Pathol. 2019 Jan 01;12(1):173-181. eCollection 2019. PMID: 31933731; PMCID: PMC6944012.
Copy Download .nbib Format: NLM
Synergistic anticancer effect of Grb2 and ITGA1 on cancer cells highly expressing Grb2 through suppressing ERK phosphorylation.

International journal of clinical and experimental pathology

Ma Q, Song J, Ma H, Gao K, Yang Y, He N.
PMID: 31933732
Int J Clin Exp Pathol. 2019 Jan 01;12(1):182-189. eCollection 2019.

BACKGROUND: Growth factor receptor bound protein 2 (Grb2) is known to be an adaptor protein that provides a critical link between cell surface growth factor receptors and the MAPK signaling. It was shown that high expression of Grb2 enhances...

Cite
Ma Q, Song J, Ma H, et al. Synergistic anticancer effect of Grb2 and ITGA1 on cancer cells highly expressing Grb2 through suppressing ERK phosphorylation. Int J Clin Exp Pathol. 2019;12(1):182-189
Ma, Q., Song, J., Ma, H., Gao, K., Yang, Y., & He, N. (2019). Synergistic anticancer effect of Grb2 and ITGA1 on cancer cells highly expressing Grb2 through suppressing ERK phosphorylation. International journal of clinical and experimental pathology, 12(1), 182-189.
Ma, Qingxia, et al. "Synergistic anticancer effect of Grb2 and ITGA1 on cancer cells highly expressing Grb2 through suppressing ERK phosphorylation." International journal of clinical and experimental pathology vol. 12,1 (2019): 182-189.
Ma Q, Song J, Ma H, Gao K, Yang Y, He N. Synergistic anticancer effect of Grb2 and ITGA1 on cancer cells highly expressing Grb2 through suppressing ERK phosphorylation. Int J Clin Exp Pathol. 2019 Jan 01;12(1):182-189. eCollection 2019. PMID: 31933732; PMCID: PMC6944030.
Copy Download .nbib Format: NLM
Matrilin-2 is a novel prognostic marker in osteosarcoma.

International journal of clinical and experimental pathology

Jiang H, Guo W, Yuan S, Song L.
PMID: 31933763
Int J Clin Exp Pathol. 2019 Oct 01;12(10):3752-3760. eCollection 2019.

OBJECTIVE: Osteosarcoma is the most malignant bone tumor in adolescents. Molecules that are significantly associated with prognosis of osteosarcoma remain to be elucidated. In this study, we aimed to explore the prognostic role of Matrilin-2 (MATN2) in osteosarcoma.METHODS: By...

Cite
Jiang H, Guo W, Yuan S, et al. Matrilin-2 is a novel prognostic marker in osteosarcoma. Int J Clin Exp Pathol. 2019;12(10):3752-3760
Jiang, H., Guo, W., Yuan, S., & Song, L. (2019). Matrilin-2 is a novel prognostic marker in osteosarcoma. International journal of clinical and experimental pathology, 12(10), 3752-3760.
Jiang, Haoli, et al. "Matrilin-2 is a novel prognostic marker in osteosarcoma." International journal of clinical and experimental pathology vol. 12,10 (2019): 3752-3760.
Jiang H, Guo W, Yuan S, Song L. Matrilin-2 is a novel prognostic marker in osteosarcoma. Int J Clin Exp Pathol. 2019 Oct 01;12(10):3752-3760. eCollection 2019. PMID: 31933763; PMCID: PMC6949765.
Copy Download .nbib Format: NLM
Aquaporin 5 expression correlates with tumor multiplicity and vascular invasion in hepatocellular carcinoma.

International journal of clinical and experimental pathology

Vireak C, Seo AN, Han MH, Park TI, Kim YJ, Jeong JY.
PMID: 31933856
Int J Clin Exp Pathol. 2019 Feb 01;12(2):516-527. eCollection 2019.

Aquaporins (AQPs) are a family of water channel transmembrane proteins that play a key role in transcellular water movement and transport. Recent studies have reported that AQPs are involved in cancer biology and can be a novel biomarker for...

Cite
Vireak C, Seo AN, Han MH, et al. Aquaporin 5 expression correlates with tumor multiplicity and vascular invasion in hepatocellular carcinoma. Int J Clin Exp Pathol. 2019;12(2):516-527
Vireak, C., Seo, A. N., Han, M. H., Park, T. I., Kim, Y. J., & Jeong, J. Y. (2019). Aquaporin 5 expression correlates with tumor multiplicity and vascular invasion in hepatocellular carcinoma. International journal of clinical and experimental pathology, 12(2), 516-527.
Vireak, Chhun, et al. "Aquaporin 5 expression correlates with tumor multiplicity and vascular invasion in hepatocellular carcinoma." International journal of clinical and experimental pathology vol. 12,2 (2019): 516-527.
Vireak C, Seo AN, Han MH, Park TI, Kim YJ, Jeong JY. Aquaporin 5 expression correlates with tumor multiplicity and vascular invasion in hepatocellular carcinoma. Int J Clin Exp Pathol. 2019 Feb 01;12(2):516-527. eCollection 2019. PMID: 31933856; PMCID: PMC6945080.
Copy Download .nbib Format: NLM
Protective role of JNK inhibitor SP600125 in sepsis-induced acute lung injury.

International journal of clinical and experimental pathology

Lou L, Hu D, Chen S, Wang S, Xu Y, Huang Y, Shi Y, Zhang H.
PMID: 31933857
Int J Clin Exp Pathol. 2019 Feb 01;12(2):528-538. eCollection 2019.

BACKGROUND: Whether the c-Jun N-terminal kinase (JNK) pathway mediates apoptosis in sepsis-induced acute lung injury is not known. Here we investigated the effect of JNK inhibition in a rat model of sepsis-induced lung injury, and assessed expression of JNK...

Cite
Lou L, Hu D, Chen S, et al. Protective role of JNK inhibitor SP600125 in sepsis-induced acute lung injury. Int J Clin Exp Pathol. 2019;12(2):528-538
Lou, L., Hu, D., Chen, S., Wang, S., Xu, Y., Huang, Y., Shi, Y., & Zhang, H. (2019). Protective role of JNK inhibitor SP600125 in sepsis-induced acute lung injury. International journal of clinical and experimental pathology, 12(2), 528-538.
Lou, Liming, et al. "Protective role of JNK inhibitor SP600125 in sepsis-induced acute lung injury." International journal of clinical and experimental pathology vol. 12,2 (2019): 528-538.
Lou L, Hu D, Chen S, Wang S, Xu Y, Huang Y, Shi Y, Zhang H. Protective role of JNK inhibitor SP600125 in sepsis-induced acute lung injury. Int J Clin Exp Pathol. 2019 Feb 01;12(2):528-538. eCollection 2019. PMID: 31933857; PMCID: PMC6945091.
Copy Download .nbib Format: NLM
Abnormality of intestinal cholesterol absorption in .

International journal of clinical and experimental pathology

Yu B, Peng XH, Wang LY, Wang AB, Su YY, Chen JH, Zhang XW, Zhao DZ, Wang H, Pang DX, Ouyang HS, Tang XC, Zhang MJ.
PMID: 31933883
Int J Clin Exp Pathol. 2019 Mar 01;12(3):759-767. eCollection 2019.

Colorectal cancer syndrome has been one of the greatest concerns in the world, particularly in developed countries. Several epidemiological studies have shown that dyslipidemia may be associated with the progression of intestinal cachexia, but there is little research on...

Cite
Yu B, Peng XH, Wang LY, et al. Abnormality of intestinal cholesterol absorption in . Int J Clin Exp Pathol. 2019;12(3):759-767
Yu, B., Peng, X. H., Wang, L. Y., Wang, A. B., Su, Y. Y., Chen, J. H., Zhang, X. W., Zhao, D. Z., Wang, H., Pang, D. X., Ouyang, H. S., Tang, X. C., & Zhang, M. J. (2019). Abnormality of intestinal cholesterol absorption in . International journal of clinical and experimental pathology, 12(3), 759-767.
Yu, Biao, et al. "Abnormality of intestinal cholesterol absorption in ." International journal of clinical and experimental pathology vol. 12,3 (2019): 759-767.
Yu B, Peng XH, Wang LY, Wang AB, Su YY, Chen JH, Zhang XW, Zhao DZ, Wang H, Pang DX, Ouyang HS, Tang XC, Zhang MJ. Abnormality of intestinal cholesterol absorption in . Int J Clin Exp Pathol. 2019 Mar 01;12(3):759-767. eCollection 2019. PMID: 31933883; PMCID: PMC6945172.
Copy Download .nbib Format: NLM
CD163.

International journal of clinical and experimental pathology

Du X, Gao Y, Sun P, Chen Y, Chang H, Wei B.
PMID: 31938319
Int J Clin Exp Pathol. 2018 Apr 01;11(4):2106-2111. eCollection 2018.

Angiosarcoma of soft tissue is a group of aggressive malignancies with high mortality. However, molecular pathogenesis and therapeutic targets of angiosarcoma remain to be established. We explored the influence of M2-polarized tumor-associated macrophages (TAMs) on the formation of angiosarcoma....

Cite
Du X, Gao Y, Sun P, et al. CD163. Int J Clin Exp Pathol. 2018;11(4):2106-2111
Du, X., Gao, Y., Sun, P., Chen, Y., Chang, H., & Wei, B. (2018). CD163. International journal of clinical and experimental pathology, 11(4), 2106-2111.
Du, Xuemei, et al. "CD163." International journal of clinical and experimental pathology vol. 11,4 (2018): 2106-2111.
Du X, Gao Y, Sun P, Chen Y, Chang H, Wei B. CD163. Int J Clin Exp Pathol. 2018 Apr 01;11(4):2106-2111. eCollection 2018. PMID: 31938319; PMCID: PMC6958200.
Copy Download .nbib Format: NLM
Showing 25 to 36 of 2513 entries