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Two new inflammatory markers related to the CURB-65 score for disease severity in patients with community-acquired pneumonia: The hypersensitive C-reactive protein to albumin ratio and fibrinogen to albumin ratio.

Open life sciences

Luo B, Sun M, Huo X, Wang Y.
PMID: 33817301
Open Life Sci. 2021 Jan 22;16(1):84-91. doi: 10.1515/biol-2021-0011. eCollection 2021.

BACKGROUND: The objective of this study was to investigate the relationship among hypersensitive C-reactive protein to albumin ratio (CAR), fibrinogen to albumin ratio (FAR), and the CURB-65 score for community-acquired pneumonia (CAP) severity.METHODS: Clinical data and laboratory indicators of...

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Luo B, Sun M, Huo X, et al. Two new inflammatory markers related to the CURB-65 score for disease severity in patients with community-acquired pneumonia: The hypersensitive C-reactive protein to albumin ratio and fibrinogen to albumin ratio. Open Life Sci. 2021;16(1):84-91doi: 10.1515/biol-2021-0011.
Luo, B., Sun, M., Huo, X., & Wang, Y. (2021). Two new inflammatory markers related to the CURB-65 score for disease severity in patients with community-acquired pneumonia: The hypersensitive C-reactive protein to albumin ratio and fibrinogen to albumin ratio. Open life sciences, 16(1), 84-91. https://doi.org/10.1515/biol-2021-0011
Luo, Bing, et al. "Two new inflammatory markers related to the CURB-65 score for disease severity in patients with community-acquired pneumonia: The hypersensitive C-reactive protein to albumin ratio and fibrinogen to albumin ratio." Open life sciences vol. 16,1 (2021): 84-91. doi: https://doi.org/10.1515/biol-2021-0011
Luo B, Sun M, Huo X, Wang Y. Two new inflammatory markers related to the CURB-65 score for disease severity in patients with community-acquired pneumonia: The hypersensitive C-reactive protein to albumin ratio and fibrinogen to albumin ratio. Open Life Sci. 2021 Jan 22;16(1):84-91. doi: 10.1515/biol-2021-0011. eCollection 2021. PMID: 33817301; PMCID: PMC7874604.
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Systemic low-grade inflammation and depressive symptomology at chronic phase of ischemic stroke: The chain mediating role of fibrinogen and neutrophil counts.

Brain, behavior, and immunity

Sun W, Miao J, Song Y, Wang Y, Pan C, Li G, Zhao X, Lan Y, Qiu X, Zhu S, Zhu Z.
PMID: 34728390
Brain Behav Immun. 2022 Feb;100:332-341. doi: 10.1016/j.bbi.2021.10.011. Epub 2021 Oct 30.

BACKGROUND: Post-stroke depression (PSD) is the most common psychological consequence of stroke. Increased inflammatory markers resulting from ischemic stroke may played an important role in the pathogenesis of depressive symptomology. The present study was conducted to further elucidate the...

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Sun W, Miao J, Song Y, et al. Systemic low-grade inflammation and depressive symptomology at chronic phase of ischemic stroke: The chain mediating role of fibrinogen and neutrophil counts. Brain Behav Immun. 2021;100:332-341doi: 10.1016/j.bbi.2021.10.011.
Sun, W., Miao, J., Song, Y., Wang, Y., Pan, C., Li, G., Zhao, X., Lan, Y., Qiu, X., Zhu, S., & Zhu, Z. (2022). Systemic low-grade inflammation and depressive symptomology at chronic phase of ischemic stroke: The chain mediating role of fibrinogen and neutrophil counts. Brain, behavior, and immunity, 100332-341. https://doi.org/10.1016/j.bbi.2021.10.011
Sun, Wenzhe, et al. "Systemic low-grade inflammation and depressive symptomology at chronic phase of ischemic stroke: The chain mediating role of fibrinogen and neutrophil counts." Brain, behavior, and immunity vol. 100 (2022): 332-341. doi: https://doi.org/10.1016/j.bbi.2021.10.011
Sun W, Miao J, Song Y, Wang Y, Pan C, Li G, Zhao X, Lan Y, Qiu X, Zhu S, Zhu Z. Systemic low-grade inflammation and depressive symptomology at chronic phase of ischemic stroke: The chain mediating role of fibrinogen and neutrophil counts. Brain Behav Immun. 2022 Feb;100:332-341. doi: 10.1016/j.bbi.2021.10.011. Epub 2021 Oct 30. PMID: 34728390.
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Evolution of a concept with enzymatic debridement and autologous in situ cell and platelet-rich fibrin therapy (BroKerF).

Scars, burns & healing

Waldner M, Ismail T, Lunger A, Klein HJ, Schweizer R, Alan O, Breckwoldt T, Giovanoli P, Plock JA.
PMID: 35024172
Scars Burn Heal. 2022 Jan 06;8:20595131211052394. doi: 10.1177/20595131211052394. eCollection 2022.

BACKGROUND: Deep partial-thickness burns are traditionally treated by tangential excision and split thickness skin graft (STSG) coverage. STSGs create donor site morbidity and increase the wound surface in burn patients. Herein, we present a novel concept consisting of enzymatic...

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Waldner M, Ismail T, Lunger A, et al. Evolution of a concept with enzymatic debridement and autologous in situ cell and platelet-rich fibrin therapy (BroKerF). Scars Burn Heal. 2022;8:20595131211052394doi: 10.1177/20595131211052394.
Waldner, M., Ismail, T., Lunger, A., Klein, H. J., Schweizer, R., Alan, O., Breckwoldt, T., Giovanoli, P., & Plock, J. A. (2022). Evolution of a concept with enzymatic debridement and autologous in situ cell and platelet-rich fibrin therapy (BroKerF). Scars, burns & healing, 820595131211052394. https://doi.org/10.1177/20595131211052394
Waldner, Matthias, et al. "Evolution of a concept with enzymatic debridement and autologous in situ cell and platelet-rich fibrin therapy (BroKerF)." Scars, burns & healing vol. 8 (2022): 20595131211052394. doi: https://doi.org/10.1177/20595131211052394
Waldner M, Ismail T, Lunger A, Klein HJ, Schweizer R, Alan O, Breckwoldt T, Giovanoli P, Plock JA. Evolution of a concept with enzymatic debridement and autologous in situ cell and platelet-rich fibrin therapy (BroKerF). Scars Burn Heal. 2022 Jan 06;8:20595131211052394. doi: 10.1177/20595131211052394. eCollection 2022. PMID: 35024172; PMCID: PMC8743980.
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Showing 8881 to 8883 of 8883 entries