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Showing 25 to 34 of 34 entries
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FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways.

Frontiers in cellular neuroscience

Devanna P, Middelbeek J, Vernes SC.
PMID: 25309332
Front Cell Neurosci. 2014 Sep 26;8:305. doi: 10.3389/fncel.2014.00305. eCollection 2014.

FOXP2 was the first gene shown to cause a Mendelian form of speech and language disorder. Although developmentally expressed in many organs, loss of a single copy of FOXP2 leads to a phenotype that is largely restricted to orofacial...

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Devanna P, Middelbeek J, Vernes SC. FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways. Front Cell Neurosci. 2014;8:305doi: 10.3389/fncel.2014.00305.
Devanna, P., Middelbeek, J., & Vernes, S. C. (2014). FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways. Frontiers in cellular neuroscience, 8305. https://doi.org/10.3389/fncel.2014.00305
Devanna, Paolo, et al. "FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways." Frontiers in cellular neuroscience vol. 8 (2014): 305. doi: https://doi.org/10.3389/fncel.2014.00305
Devanna P, Middelbeek J, Vernes SC. FOXP2 drives neuronal differentiation by interacting with retinoic acid signaling pathways. Front Cell Neurosci. 2014 Sep 26;8:305. doi: 10.3389/fncel.2014.00305. eCollection 2014. PMID: 25309332; PMCID: PMC4176457.
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Quantitative model of eukaryotic Cdk control through the Forkhead CONTROLLER.

NPJ systems biology and applications

Barberis M.
PMID: 34117265
NPJ Syst Biol Appl. 2021 Jun 11;7(1):28. doi: 10.1038/s41540-021-00187-5.

In budding yeast, synchronization of waves of mitotic cyclins that activate the Cdk1 kinase occur through Forkhead transcription factors. These molecules act as controllers of their sequential order and may account for the separation in time of incompatible processes....

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Barberis M. Quantitative model of eukaryotic Cdk control through the Forkhead CONTROLLER. NPJ Syst Biol Appl. 2021;7(1):28doi: 10.1038/s41540-021-00187-5.
Barberis, M. (2021). Quantitative model of eukaryotic Cdk control through the Forkhead CONTROLLER. NPJ systems biology and applications, 7(1), 28. https://doi.org/10.1038/s41540-021-00187-5
Barberis, Matteo. "Quantitative model of eukaryotic Cdk control through the Forkhead CONTROLLER." NPJ systems biology and applications vol. 7,1 (2021): 28. doi: https://doi.org/10.1038/s41540-021-00187-5
Barberis M. Quantitative model of eukaryotic Cdk control through the Forkhead CONTROLLER. NPJ Syst Biol Appl. 2021 Jun 11;7(1):28. doi: 10.1038/s41540-021-00187-5. PMID: 34117265; PMCID: PMC8196193.
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FOXO3a and Its Regulators in Prostate Cancer.

International journal of molecular sciences

Habrowska-Górczyńska DE, Kozieł MJ, Kowalska K, Piastowska-Ciesielska AW.
PMID: 34830408
Int J Mol Sci. 2021 Nov 20;22(22). doi: 10.3390/ijms222212530.

Forkhead box O3 (FOXO3a) is a member of a subfamily of forkhead transcription factors involved in the basic processes within a cell, including proliferation, apoptosis, cell cycle regulation, and DNA damage. As a transcription factor, FOXO3a is involved in...

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Habrowska-Górczyńska DE, Kozieł MJ, Kowalska K, et al. FOXO3a and Its Regulators in Prostate Cancer. Int J Mol Sci. 2021;22(22)doi: 10.3390/ijms222212530.
Habrowska-Górczyńska, D. E., Kozieł, M. J., Kowalska, K., & Piastowska-Ciesielska, A. W. (2021). FOXO3a and Its Regulators in Prostate Cancer. International journal of molecular sciences, 22(22), . https://doi.org/10.3390/ijms222212530
Habrowska-Górczyńska, Dominika Ewa, et al. "FOXO3a and Its Regulators in Prostate Cancer." International journal of molecular sciences vol. 22,22 (2021). doi: https://doi.org/10.3390/ijms222212530
Habrowska-Górczyńska DE, Kozieł MJ, Kowalska K, Piastowska-Ciesielska AW. FOXO3a and Its Regulators in Prostate Cancer. Int J Mol Sci. 2021 Nov 20;22(22). doi: 10.3390/ijms222212530. PMID: 34830408; PMCID: PMC8625444.
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Role of FoxO transcription factors in aging-associated cardiovascular diseases.

Vitamins and hormones

Mishra S, Ravi V, Sundaresan NR.
PMID: 33706958
Vitam Horm. 2021;115:449-475. doi: 10.1016/bs.vh.2020.12.018. Epub 2021 Feb 13.

Aging constitutes a major risk factor toward the development of cardiovascular diseases (CVDs). The aging heart undergoes several changes at the molecular, cellular and physiological levels, which diminishes its contractile function and weakens stress tolerance. Further, old age increases...

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Mishra S, Ravi V, Sundaresan NR. Role of FoxO transcription factors in aging-associated cardiovascular diseases. Vitam Horm. 2021;115:449-475doi: 10.1016/bs.vh.2020.12.018.
Mishra, S., Ravi, V., & Sundaresan, N. R. (2021). Role of FoxO transcription factors in aging-associated cardiovascular diseases. Vitamins and hormones, 115449-475. https://doi.org/10.1016/bs.vh.2020.12.018
Mishra, Sneha, et al. "Role of FoxO transcription factors in aging-associated cardiovascular diseases." Vitamins and hormones vol. 115 (2021): 449-475. doi: https://doi.org/10.1016/bs.vh.2020.12.018
Mishra S, Ravi V, Sundaresan NR. Role of FoxO transcription factors in aging-associated cardiovascular diseases. Vitam Horm. 2021;115:449-475. doi: 10.1016/bs.vh.2020.12.018. Epub 2021 Feb 13. PMID: 33706958.
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Cell cycle regulated transcription: from yeast to cancer.

F1000Research

McInerny CJ.
PMID: 27239285
F1000Res. 2016 May 12;5. doi: 10.12688/f1000research.8111.1. eCollection 2016.

Recent studies have revealed exciting new functions for forkhead transcription factors in cell proliferation and development. Cell proliferation is a fundamental process controlled by multiple overlapping mechanisms, and the control of gene expression plays a major role in the...

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McInerny CJ. Cell cycle regulated transcription: from yeast to cancer. F1000Res. 2016;5doi: 10.12688/f1000research.8111.1.
McInerny, C. J. (2016). Cell cycle regulated transcription: from yeast to cancer. F1000Research, 5. https://doi.org/10.12688/f1000research.8111.1
McInerny, Christopher J. "Cell cycle regulated transcription: from yeast to cancer." F1000Research vol. 5 (2016). doi: https://doi.org/10.12688/f1000research.8111.1
McInerny CJ. Cell cycle regulated transcription: from yeast to cancer. F1000Res. 2016 May 12;5. doi: 10.12688/f1000research.8111.1. eCollection 2016. PMID: 27239285; PMCID: PMC4870989.
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Modified Shenlingbaizhu decoction reduces intestinal adenoma formation in adenomatous polyposis coli multiple intestinal neoplasia mice by suppression of hypoxia-inducible factor 1α-induced CD4+CD25+forkhead box P3 regulatory T cells.

Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan

Xu W, Han Q, Liang S, Li L, Sun X, Shao M, Yao X, Xu W.
PMID: 32185948
J Tradit Chin Med. 2018 Feb;38(1):22-32.

OBJECTIVE: To test the hypothesis that modified Shenlingbaizhu decoction (MSD) attenuates the formation of intestinal adenomas by regulating activation of CD4+CD25+ forkhead box P3 (FoxP3) regulatory T cells (Tregs) by downregulation of hypoxia-inducible factor 1α (HIF-1α).METHODS: Chemical fingerprints of...

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Xu W, Han Q, Liang S, et al. Modified Shenlingbaizhu decoction reduces intestinal adenoma formation in adenomatous polyposis coli multiple intestinal neoplasia mice by suppression of hypoxia-inducible factor 1α-induced CD4+CD25+forkhead box P3 regulatory T cells. J Tradit Chin Med. 2018;38(1):22-32
Xu, W., Han, Q., Liang, S., Li, L., Sun, X., Shao, M., Yao, X., & Xu, W. (2018). Modified Shenlingbaizhu decoction reduces intestinal adenoma formation in adenomatous polyposis coli multiple intestinal neoplasia mice by suppression of hypoxia-inducible factor 1α-induced CD4+CD25+forkhead box P3 regulatory T cells. Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan, 38(1), 22-32.
Xu, Wenjuan, et al. "Modified Shenlingbaizhu decoction reduces intestinal adenoma formation in adenomatous polyposis coli multiple intestinal neoplasia mice by suppression of hypoxia-inducible factor 1α-induced CD4+CD25+forkhead box P3 regulatory T cells." Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan vol. 38,1 (2018): 22-32.
Xu W, Han Q, Liang S, Li L, Sun X, Shao M, Yao X, Xu W. Modified Shenlingbaizhu decoction reduces intestinal adenoma formation in adenomatous polyposis coli multiple intestinal neoplasia mice by suppression of hypoxia-inducible factor 1α-induced CD4+CD25+forkhead box P3 regulatory T cells. J Tradit Chin Med. 2018 Feb;38(1):22-32. PMID: 32185948.
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Erythropoietin and diabetes mellitus.

World journal of diabetes

Maiese K.
PMID: 26516410
World J Diabetes. 2015 Oct 25;6(14):1259-73. doi: 10.4239/wjd.v6.i14.1259.

Erythropoietin (EPO) is a 30.4 kDa growth factor and cytokine that governs cell proliferation, immune modulation, metabolic homeostasis, vascular function, and cytoprotection. EPO is under investigation for the treatment of variety of diseases, but appears especially suited for the...

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Maiese K. Erythropoietin and diabetes mellitus. World J Diabetes. 2015;6(14):1259-73doi: 10.4239/wjd.v6.i14.1259.
Maiese, K. (2015). Erythropoietin and diabetes mellitus. World journal of diabetes, 6(14), 1259-73. https://doi.org/10.4239/wjd.v6.i14.1259
Maiese, Kenneth. "Erythropoietin and diabetes mellitus." World journal of diabetes vol. 6,14 (2015): 1259-73. doi: https://doi.org/10.4239/wjd.v6.i14.1259
Maiese K. Erythropoietin and diabetes mellitus. World J Diabetes. 2015 Oct 25;6(14):1259-73. doi: 10.4239/wjd.v6.i14.1259. PMID: 26516410; PMCID: PMC4620106.
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Novel insights into FOXOlogy: FOXOs and their putative role in thyroid carcinogenesis.

Expert review of endocrinology & metabolism

Weidinger C, Krause K, Fuhrer D.
PMID: 30764036
Expert Rev Endocrinol Metab. 2011 Jan;6(1):63-69. doi: 10.1586/eem.10.70.

FOXO transcription factors regulate genes directly involved in the control of cell cycle arrest, apoptosis, DNA damage repair and antioxidative defense mechanisms. Genetic FOXO alterations and inactivation of FOXOs through oncogenic signaling cascades have been identified in several human...

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Weidinger C, Krause K, Fuhrer D. Novel insights into FOXOlogy: FOXOs and their putative role in thyroid carcinogenesis. Expert Rev Endocrinol Metab. 2011;6(1):63-69doi: 10.1586/eem.10.70.
Weidinger, C., Krause, K., & Fuhrer, D. (2011). Novel insights into FOXOlogy: FOXOs and their putative role in thyroid carcinogenesis. Expert review of endocrinology & metabolism, 6(1), 63-69. https://doi.org/10.1586/eem.10.70
Weidinger, Carl, et al. "Novel insights into FOXOlogy: FOXOs and their putative role in thyroid carcinogenesis." Expert review of endocrinology & metabolism vol. 6,1 (2011): 63-69. doi: https://doi.org/10.1586/eem.10.70
Weidinger C, Krause K, Fuhrer D. Novel insights into FOXOlogy: FOXOs and their putative role in thyroid carcinogenesis. Expert Rev Endocrinol Metab. 2011 Jan;6(1):63-69. doi: 10.1586/eem.10.70. PMID: 30764036.
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Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR.

Neural regeneration research

Maiese K.
PMID: 27127460
Neural Regen Res. 2016 Mar;11(3):372-85. doi: 10.4103/1673-5374.179032.

Throughout the globe, diabetes mellitus (DM) is increasing in incidence with limited therapies presently available to prevent or resolve the significant complications of this disorder. DM impacts multiple organs and affects all components of the central and peripheral nervous...

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Maiese K. Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR. Neural Regen Res. 2016;11(3):372-85doi: 10.4103/1673-5374.179032.
Maiese, K. (2016). Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR. Neural regeneration research, 11(3), 372-85. https://doi.org/10.4103/1673-5374.179032
Maiese, Kenneth. "Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR." Neural regeneration research vol. 11,3 (2016): 372-85. doi: https://doi.org/10.4103/1673-5374.179032
Maiese K. Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTOR. Neural Regen Res. 2016 Mar;11(3):372-85. doi: 10.4103/1673-5374.179032. PMID: 27127460; PMCID: PMC4828986.
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Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemia.

Military Medical Research

Han RH, Huang HM, Han H, Chen H, Zeng F, Xie X, Liu DY, Cai Y, Zhang LQ, Liu X, Xia ZY, Tang J.
PMID: 34753510
Mil Med Res. 2021 Nov 10;8(1):58. doi: 10.1186/s40779-021-00353-0.

BACKGROUND: Administration of propofol, an intravenous anesthetic with antioxidant property, immediately at the onset of post-ischemic reperfusion (propofol postconditioning, P-PostC) has been shown to confer cardioprotection against ischemia-reperfusion injury, while the underlying mechanism remains incompletely understood. The FoxO transcription...

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Han RH, Huang HM, Han H, et al. Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemia. Mil Med Res. 2021;8(1):58doi: 10.1186/s40779-021-00353-0.
Han, R. H., Huang, H. M., Han, H., Chen, H., Zeng, F., Xie, X., Liu, D. Y., Cai, Y., Zhang, L. Q., Liu, X., Xia, Z. Y., & Tang, J. (2021). Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemia. Military Medical Research, 8(1), 58. https://doi.org/10.1186/s40779-021-00353-0
Han, Rong-Hui, et al. "Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemia." Military Medical Research vol. 8,1 (2021): 58. doi: https://doi.org/10.1186/s40779-021-00353-0
Han RH, Huang HM, Han H, Chen H, Zeng F, Xie X, Liu DY, Cai Y, Zhang LQ, Liu X, Xia ZY, Tang J. Propofol postconditioning ameliorates hypoxia/reoxygenation induced H9c2 cell apoptosis and autophagy via upregulating forkhead transcription factors under hyperglycemia. Mil Med Res. 2021 Nov 10;8(1):58. doi: 10.1186/s40779-021-00353-0. PMID: 34753510; PMCID: PMC8579603.
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Showing 25 to 34 of 34 entries