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Ultrasound Assessment of Kidney Volume in Patients with Acute Decompensated Heart Failure: A Predictor of Diuretic Resistance.

Yonago acta medica

Sugihara S, Kinugasa Y, Takata T, Sugihara T, Hosho K, Imai C, Ito H, Yamada K, Kato M, Yamamoto K.
PMID: 28959123
Yonago Acta Med. 2017 Sep 15;60(3):135-144. eCollection 2017 Sep.

BACKGROUND: Diuretics are essential for treating acute decompensated heart failure (ADHF), but the response is inconsistent. This study aimed to clarify whether kidney volume as assessed by ultrasound (US) predicts diuretic resistance in patients with ADHF.METHODS: We enrolled 29...

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Sugihara S, Kinugasa Y, Takata T, et al. Ultrasound Assessment of Kidney Volume in Patients with Acute Decompensated Heart Failure: A Predictor of Diuretic Resistance. Yonago Acta Med. 2017;60(3):135-144
Sugihara, S., Kinugasa, Y., Takata, T., Sugihara, T., Hosho, K., Imai, C., Ito, H., Yamada, K., Kato, M., & Yamamoto, K. (2017). Ultrasound Assessment of Kidney Volume in Patients with Acute Decompensated Heart Failure: A Predictor of Diuretic Resistance. Yonago acta medica, 60(3), 135-144.
Sugihara, Shinobu, et al. "Ultrasound Assessment of Kidney Volume in Patients with Acute Decompensated Heart Failure: A Predictor of Diuretic Resistance." Yonago acta medica vol. 60,3 (2017): 135-144.
Sugihara S, Kinugasa Y, Takata T, Sugihara T, Hosho K, Imai C, Ito H, Yamada K, Kato M, Yamamoto K. Ultrasound Assessment of Kidney Volume in Patients with Acute Decompensated Heart Failure: A Predictor of Diuretic Resistance. Yonago Acta Med. 2017 Sep 15;60(3):135-144. eCollection 2017 Sep. PMID: 28959123; PMCID: PMC5611467.
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Association between plasma concentration of tolvaptan and urine volume in acute decompensated heart failure patients with fluid overload.

Cardiology journal

Kato M, Tohyama K, Ohya T, Hiro T, Hirayama A.
PMID: 27515480
Cardiol J. 2016;23(5):497-504. doi: 10.5603/CJ.a2016.0055. Epub 2016 Aug 12.

BACKGROUND: Tolvaptan (TLV) is a useful diuretic for acute decompensated heart failure (ADHF) with fluid overload, but its clinical response varies between patients. The aim of this study is to investigate whether plasma TLV concentrations correlate with the urine...

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Kato M, Tohyama K, Ohya T, et al. Association between plasma concentration of tolvaptan and urine volume in acute decompensated heart failure patients with fluid overload. Cardiol J. 2016;23(5):497-504doi: 10.5603/CJ.a2016.0055.
Kato, M., Tohyama, K., Ohya, T., Hiro, T., & Hirayama, A. (2016). Association between plasma concentration of tolvaptan and urine volume in acute decompensated heart failure patients with fluid overload. Cardiology journal, 23(5), 497-504. https://doi.org/10.5603/CJ.a2016.0055
Kato, Mahoto, et al. "Association between plasma concentration of tolvaptan and urine volume in acute decompensated heart failure patients with fluid overload." Cardiology journal vol. 23,5 (2016): 497-504. doi: https://doi.org/10.5603/CJ.a2016.0055
Kato M, Tohyama K, Ohya T, Hiro T, Hirayama A. Association between plasma concentration of tolvaptan and urine volume in acute decompensated heart failure patients with fluid overload. Cardiol J. 2016;23(5):497-504. doi: 10.5603/CJ.a2016.0055. Epub 2016 Aug 12. PMID: 27515480.
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Does Accidental Overcorrection of Symptomatic Hyponatremia in Chronic Heart Failure Require Specific Therapeutic Adjustments for Preventing Central Pontine Myelinolysis?.

Journal of clinical medicine research

De Vecchis R, Noutsias M, Ariano C, Cesaro A, Cioppa C, Giasi A, Maurea N.
PMID: 28270885
J Clin Med Res. 2017 Apr;9(4):266-272. doi: 10.14740/jocmr2933w. Epub 2017 Feb 21.

This review aims at summarizing essential aspects of epidemiology and pathophysiology of hyponatremia in chronic heart failure (CHF), to set the ground for a practical as well as evidence-based approach to treatment. As a guide through the discussion of...

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De Vecchis R, Noutsias M, Ariano C, et al. Does Accidental Overcorrection of Symptomatic Hyponatremia in Chronic Heart Failure Require Specific Therapeutic Adjustments for Preventing Central Pontine Myelinolysis?. J Clin Med Res. 2017;9(4):266-272doi: 10.14740/jocmr2933w.
De Vecchis, R., Noutsias, M., Ariano, C., Cesaro, A., Cioppa, C., Giasi, A., & Maurea, N. (2017). Does Accidental Overcorrection of Symptomatic Hyponatremia in Chronic Heart Failure Require Specific Therapeutic Adjustments for Preventing Central Pontine Myelinolysis?. Journal of clinical medicine research, 9(4), 266-272. https://doi.org/10.14740/jocmr2933w
De Vecchis, Renato, et al. "Does Accidental Overcorrection of Symptomatic Hyponatremia in Chronic Heart Failure Require Specific Therapeutic Adjustments for Preventing Central Pontine Myelinolysis?." Journal of clinical medicine research vol. 9,4 (2017): 266-272. doi: https://doi.org/10.14740/jocmr2933w
De Vecchis R, Noutsias M, Ariano C, Cesaro A, Cioppa C, Giasi A, Maurea N. Does Accidental Overcorrection of Symptomatic Hyponatremia in Chronic Heart Failure Require Specific Therapeutic Adjustments for Preventing Central Pontine Myelinolysis?. J Clin Med Res. 2017 Apr;9(4):266-272. doi: 10.14740/jocmr2933w. Epub 2017 Feb 21. PMID: 28270885; PMCID: PMC5330768.
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Organic anion transporter 2 transcript variant 1 shows broad ligand selectivity when expressed in multiple cell lines.

Frontiers in pharmacology

Hotchkiss AG, Berrigan L, Pelis RM.
PMID: 26500550
Front Pharmacol. 2015 Oct 06;6:216. doi: 10.3389/fphar.2015.00216. eCollection 2015.

Organic anion transporter 2 (OAT2) is likely important for renal and hepatic drug elimination. Three variants of the OAT2 peptide sequence have been described - OAT2 transcript variant 1 (OAT2-tv1), OAT2 transcript variant 2 (OAT2-tv2), and OAT2 transcript variant...

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Hotchkiss AG, Berrigan L, Pelis RM. Organic anion transporter 2 transcript variant 1 shows broad ligand selectivity when expressed in multiple cell lines. Front Pharmacol. 2015;6:216doi: 10.3389/fphar.2015.00216.
Hotchkiss, A. G., Berrigan, L., & Pelis, R. M. (2015). Organic anion transporter 2 transcript variant 1 shows broad ligand selectivity when expressed in multiple cell lines. Frontiers in pharmacology, 6216. https://doi.org/10.3389/fphar.2015.00216
Hotchkiss, Adam G, et al. "Organic anion transporter 2 transcript variant 1 shows broad ligand selectivity when expressed in multiple cell lines." Frontiers in pharmacology vol. 6 (2015): 216. doi: https://doi.org/10.3389/fphar.2015.00216
Hotchkiss AG, Berrigan L, Pelis RM. Organic anion transporter 2 transcript variant 1 shows broad ligand selectivity when expressed in multiple cell lines. Front Pharmacol. 2015 Oct 06;6:216. doi: 10.3389/fphar.2015.00216. eCollection 2015. PMID: 26500550; PMCID: PMC4594013.
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Evidence for an acute antimagnesiuretic effect of triamterene derivatives.

Pharmaceutical research

Priewer H, Kraft H, Mutschler E.
PMID: 24272618
Pharm Res. 1985 Mar;2(2):90-3. doi: 10.1023/A:1016398929981.

The acute natriuretic, antikaliuretic and antimagnesiuretic effects of two triamterene derivatives, carboxybutoxytriamterene ethyl amide and dimethylaminohydroxypropoxytriamterene (RPH 2823), are shown in male Wistar rats during urine collection periods of 1 to 2.5 h. In combination with furosemide both compounds...

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Priewer H, Kraft H, Mutschler E. Evidence for an acute antimagnesiuretic effect of triamterene derivatives. Pharm Res. 1985;2(2):90-3doi: 10.1023/A:1016398929981.
Priewer, H., Kraft, H., & Mutschler, E. (1985). Evidence for an acute antimagnesiuretic effect of triamterene derivatives. Pharmaceutical research, 2(2), 90-3. https://doi.org/10.1023/A:1016398929981
Priewer, H, et al. "Evidence for an acute antimagnesiuretic effect of triamterene derivatives." Pharmaceutical research vol. 2,2 (1985): 90-3. doi: https://doi.org/10.1023/A:1016398929981
Priewer H, Kraft H, Mutschler E. Evidence for an acute antimagnesiuretic effect of triamterene derivatives. Pharm Res. 1985 Mar;2(2):90-3. doi: 10.1023/A:1016398929981. PMID: 24272618.
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An efficient strategy for establishing a model of sensorineural deafness in rats.

Neural regeneration research

Ma L, Yi HJ, Yuan FQ, Guo WW, Yang SM.
PMID: 26692870
Neural Regen Res. 2015 Oct;10(10):1683-9. doi: 10.4103/1673-5374.153704.

Ototoxic drugs can be used to produce a loss of cochlear hair cells to create animal models of deafness. However, to the best of our knowledge, there is no report on the establishment of a rat deafness model through...

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Ma L, Yi HJ, Yuan FQ, et al. An efficient strategy for establishing a model of sensorineural deafness in rats. Neural Regen Res. 2015;10(10):1683-9doi: 10.4103/1673-5374.153704.
Ma, L., Yi, H. J., Yuan, F. Q., Guo, W. W., & Yang, S. M. (2015). An efficient strategy for establishing a model of sensorineural deafness in rats. Neural regeneration research, 10(10), 1683-9. https://doi.org/10.4103/1673-5374.153704
Ma, Long, et al. "An efficient strategy for establishing a model of sensorineural deafness in rats." Neural regeneration research vol. 10,10 (2015): 1683-9. doi: https://doi.org/10.4103/1673-5374.153704
Ma L, Yi HJ, Yuan FQ, Guo WW, Yang SM. An efficient strategy for establishing a model of sensorineural deafness in rats. Neural Regen Res. 2015 Oct;10(10):1683-9. doi: 10.4103/1673-5374.153704. PMID: 26692870; PMCID: PMC4660766.
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Predicting prognosis based on the shape of the left ventricular cavity in dilated cardiomyopathy: analysis using rate of improvement in the circle index.

Journal of medical ultrasonics (2001)

Hirano Y, Kayano H, Kawamata T, Toshida T, Ueda H, Ando H, Ozawa M, Katagiri T, Abe K.
PMID: 27277978
J Med Ultrason (2001). 2006 Dec;33(4):217-24. doi: 10.1007/s10396-006-0113-z. Epub 2006 Dec 22.

PURPOSE: To elucidate the relation between a quantitative measure of the shape of the left ventricular cavity, cardiac function, and prognosis in patients with dilated cardiomyopathy (DCM).METHODS: The hearts of 20 healthy individuals and 18 patients with DCM were...

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Hirano Y, Kayano H, Kawamata T, et al. Predicting prognosis based on the shape of the left ventricular cavity in dilated cardiomyopathy: analysis using rate of improvement in the circle index. J Med Ultrason (2001). 2006;33(4):217-24doi: 10.1007/s10396-006-0113-z.
Hirano, Y., Kayano, H., Kawamata, T., Toshida, T., Ueda, H., Ando, H., Ozawa, M., Katagiri, T., & Abe, K. (2006). Predicting prognosis based on the shape of the left ventricular cavity in dilated cardiomyopathy: analysis using rate of improvement in the circle index. Journal of medical ultrasonics (2001), 33(4), 217-24. https://doi.org/10.1007/s10396-006-0113-z
Hirano, Yuichi, et al. "Predicting prognosis based on the shape of the left ventricular cavity in dilated cardiomyopathy: analysis using rate of improvement in the circle index." Journal of medical ultrasonics (2001) vol. 33,4 (2006): 217-24. doi: https://doi.org/10.1007/s10396-006-0113-z
Hirano Y, Kayano H, Kawamata T, Toshida T, Ueda H, Ando H, Ozawa M, Katagiri T, Abe K. Predicting prognosis based on the shape of the left ventricular cavity in dilated cardiomyopathy: analysis using rate of improvement in the circle index. J Med Ultrason (2001). 2006 Dec;33(4):217-24. doi: 10.1007/s10396-006-0113-z. Epub 2006 Dec 22. PMID: 27277978.
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Acute kidney injury in neonatal intensive care: Medicines involved.

The International journal of risk & safety in medicine

Safina AI, Daminova MA, Abdullina GA.
PMID: 26639729
Int J Risk Saf Med. 2015;27:S9-S10. doi: 10.3233/JRS-150669.

BACKGROUND: The incidence of acute kidney injury (AKI) in neonates in the intensive care units and neonatal intensive care (NICU) according Plotz et al. ranges from 8% to 22% [3]. According to Andreoli, neonatal death due to AKI in...

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Safina AI, Daminova MA, Abdullina GA. Acute kidney injury in neonatal intensive care: Medicines involved. Int J Risk Saf Med. 2015;27:S9-S10doi: 10.3233/JRS-150669.
Safina, A. I., Daminova, M. A., & Abdullina, G. A. (2015). Acute kidney injury in neonatal intensive care: Medicines involved. The International journal of risk & safety in medicine, 27S9-S10. https://doi.org/10.3233/JRS-150669
Safina, A I, et al. "Acute kidney injury in neonatal intensive care: Medicines involved." The International journal of risk & safety in medicine vol. 27 (2015): S9-S10. doi: https://doi.org/10.3233/JRS-150669
Safina AI, Daminova MA, Abdullina GA. Acute kidney injury in neonatal intensive care: Medicines involved. Int J Risk Saf Med. 2015;27:S9-S10. doi: 10.3233/JRS-150669. PMID: 26639729.
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Synaptic Activation of GABAA Receptors Causes a Depolarizing Potential Under Physiological Conditions in Rat Hippocampal Pyramidal Cells.

The European journal of neuroscience

Avoli M.
PMID: 12106438
Eur J Neurosci. 1992 Oct;4(1):16-26. doi: 10.1111/j.1460-9568.1992.tb00105.x.

Intracellular recordings with K-acetate-filled microelectrodes were performed in slices of the adult rat hippocampus maintained in vitro at 35 - 36 degrees C to analyse the potentials associated with the orthodromic inhibitory sequence generated by CA1 pyramidal cells. In...

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Avoli M. Synaptic Activation of GABAA Receptors Causes a Depolarizing Potential Under Physiological Conditions in Rat Hippocampal Pyramidal Cells. Eur J Neurosci. 1992;4(1):16-26doi: 10.1111/j.1460-9568.1992.tb00105.x.
Avoli, M. (1992). Synaptic Activation of GABAA Receptors Causes a Depolarizing Potential Under Physiological Conditions in Rat Hippocampal Pyramidal Cells. The European journal of neuroscience, 4(1), 16-26. https://doi.org/10.1111/j.1460-9568.1992.tb00105.x
Avoli, Massimo. "Synaptic Activation of GABAA Receptors Causes a Depolarizing Potential Under Physiological Conditions in Rat Hippocampal Pyramidal Cells." The European journal of neuroscience vol. 4,1 (1992): 16-26. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00105.x
Avoli M. Synaptic Activation of GABAA Receptors Causes a Depolarizing Potential Under Physiological Conditions in Rat Hippocampal Pyramidal Cells. Eur J Neurosci. 1992 Oct;4(1):16-26. doi: 10.1111/j.1460-9568.1992.tb00105.x. PMID: 12106438.
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Atherosclerotic Renovascular Disease.

American journal of therapeutics

Spitalewitz S, Reiser IW.
PMID: 11862267
Am J Ther. 1996 Apr;3(4):321-328. doi: 10.1097/00045391-199604000-00010.

The patient, a 78-year-old Asian male, was brought to the hospital because of acute shortness of breath that had progressively worsened over the course of the day. He complained of a nonproductive cough and claudication after walking 1 block....

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Spitalewitz S, Reiser IW. Atherosclerotic Renovascular Disease. Am J Ther. 1996;3(4):321-328doi: 10.1097/00045391-199604000-00010.
Spitalewitz, S., & Reiser, I. W. (1996). Atherosclerotic Renovascular Disease. American journal of therapeutics, 3(4), 321-328. https://doi.org/10.1097/00045391-199604000-00010
Spitalewitz, Samuel, and Reiser, Ira W.. "Atherosclerotic Renovascular Disease." American journal of therapeutics vol. 3,4 (1996): 321-328. doi: https://doi.org/10.1097/00045391-199604000-00010
Spitalewitz S, Reiser IW. Atherosclerotic Renovascular Disease. Am J Ther. 1996 Apr;3(4):321-328. doi: 10.1097/00045391-199604000-00010. PMID: 11862267.
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Cytomegalovirus myocarditis in a healthy infant: Complete recovery after ganciclovir treatment.

Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies

Dehtiar N, Eherlichman M, Picard E, Kleid D, Glaser J, Raveh D, Schlesinger Y.
PMID: 12793954
Pediatr Crit Care Med. 2001 Jul;2(3):271-273. doi: 10.1097/00130478-200107000-00015.

OBJECTIVES: To report a case of acute myocarditis caused by cytomegalovirus infection in a 15-month-old immunocompetent infant completely recovered with ganciclovir treatment. DESIGN: Descriptive case report. SETTING: Pediatric intensive care unit in a general hospital. Patient: A 15-month-old healthy...

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Dehtiar N, Eherlichman M, Picard E, et al. Cytomegalovirus myocarditis in a healthy infant: Complete recovery after ganciclovir treatment. Pediatr Crit Care Med. 2001;2(3):271-273doi: 10.1097/00130478-200107000-00015.
Dehtiar, N., Eherlichman, M., Picard, E., Kleid, D., Glaser, J., Raveh, D., & Schlesinger, Y. (2001). Cytomegalovirus myocarditis in a healthy infant: Complete recovery after ganciclovir treatment. Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies, 2(3), 271-273. https://doi.org/10.1097/00130478-200107000-00015
Dehtiar, Natalie, et al. "Cytomegalovirus myocarditis in a healthy infant: Complete recovery after ganciclovir treatment." Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies vol. 2,3 (2001): 271-273. doi: https://doi.org/10.1097/00130478-200107000-00015
Dehtiar N, Eherlichman M, Picard E, Kleid D, Glaser J, Raveh D, Schlesinger Y. Cytomegalovirus myocarditis in a healthy infant: Complete recovery after ganciclovir treatment. Pediatr Crit Care Med. 2001 Jul;2(3):271-273. doi: 10.1097/00130478-200107000-00015. PMID: 12793954.
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Biopsychosocial Aspects of Chronic Illness.

Adolescent medicine (Philadelphia, Pa.)

Neal W, Coupey SM.
PMID: 10359996
Adolesc Med. 1996 Oct;7(3):427-432.

A 17-year-old female was brought in complaining of severe chest pain and difficulty breathing after being raped in the subway. During the assault, she developed palpitations, followed by dizziness, and ultimately loss of consciousness. Her medical history is significant...

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Neal W, Coupey SM. Biopsychosocial Aspects of Chronic Illness. Adolesc Med. 1996;7(3):427-432
Neal, W., & Coupey, S. M. (1996). Biopsychosocial Aspects of Chronic Illness. Adolescent medicine (Philadelphia, Pa.), 7(3), 427-432.
Neal, and Coupey. "Biopsychosocial Aspects of Chronic Illness." Adolescent medicine (Philadelphia, Pa.) vol. 7,3 (1996): 427-432.
Neal W, Coupey SM. Biopsychosocial Aspects of Chronic Illness. Adolesc Med. 1996 Oct;7(3):427-432. PMID: 10359996.
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Showing 25 to 36 of 786 entries