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Showing 13 to 24 of 41 entries
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Diagnosis, Follow-Up and Treatment Results in Thyroid Ophthalmopathy.

Turkish journal of ophthalmology

Savku E, Gündüz K.
PMID: 27800224
Turk J Ophthalmol. 2015 Aug;45(4):156-163. doi: 10.4274/tjo.93609. Epub 2015 Aug 05.

OBJECTIVES: To discuss our follow-up and treatment results in thyroid-associated ophthalmopathy (TAO).MA­TE­RI­ALS AND MET­HODS: The records of 168 TAO cases who were followed at our clinic between October 1998 and October 2013 were reviewed retrospectively. The severity and activity...

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Savku E, Gündüz K. Diagnosis, Follow-Up and Treatment Results in Thyroid Ophthalmopathy. Turk J Ophthalmol. 2015;45(4):156-163doi: 10.4274/tjo.93609.
Savku, E., & Gündüz, K. (2015). Diagnosis, Follow-Up and Treatment Results in Thyroid Ophthalmopathy. Turkish journal of ophthalmology, 45(4), 156-163. https://doi.org/10.4274/tjo.93609
Savku, Esra, and Gündüz, Kaan. "Diagnosis, Follow-Up and Treatment Results in Thyroid Ophthalmopathy." Turkish journal of ophthalmology vol. 45,4 (2015): 156-163. doi: https://doi.org/10.4274/tjo.93609
Savku E, Gündüz K. Diagnosis, Follow-Up and Treatment Results in Thyroid Ophthalmopathy. Turk J Ophthalmol. 2015 Aug;45(4):156-163. doi: 10.4274/tjo.93609. Epub 2015 Aug 05. PMID: 27800224; PMCID: PMC5082274.
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On the mechanism of action of guanethidine and bretylium.

British journal of pharmacology and chemotherapy

Brodie BB, Chang CC, Costa E.
PMID: 19108194
Br J Pharmacol Chemother. 1965 Aug;25(1):171-8. doi: 10.1111/j.1476-5381.1965.tb01769.x.

No abstract available.

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Brodie BB, Chang CC, Costa E. On the mechanism of action of guanethidine and bretylium. Br J Pharmacol Chemother. 1965;25(1):171-8doi: 10.1111/j.1476-5381.1965.tb01769.x.
Brodie, B. B., Chang, C. C., & Costa, E. (1965). On the mechanism of action of guanethidine and bretylium. British journal of pharmacology and chemotherapy, 25(1), 171-8. https://doi.org/10.1111/j.1476-5381.1965.tb01769.x
Brodie, B B, et al. "On the mechanism of action of guanethidine and bretylium." British journal of pharmacology and chemotherapy vol. 25,1 (1965): 171-8. doi: https://doi.org/10.1111/j.1476-5381.1965.tb01769.x
Brodie BB, Chang CC, Costa E. On the mechanism of action of guanethidine and bretylium. Br J Pharmacol Chemother. 1965 Aug;25(1):171-8. doi: 10.1111/j.1476-5381.1965.tb01769.x. PMID: 19108194; PMCID: PMC1510669.
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Origin of the cholinergic response of the rabbit intestine to stimulation of its extrinsic sympathetic nerves after exposure to sympathetic blocking agents.

British journal of pharmacology and chemotherapy

Boyd G, Gillespie JS, Mackenna BR.
PMID: 19108166
Br J Pharmacol Chemother. 1962 Oct;19(2):258-70. doi: 10.1111/j.1476-5381.1962.tb01187.x.

The effect of guanethidine and of bretylium on the response to nerve stimulation has been studied on two types of isolated innervated preparations of rabbit intestine. One preparation was that of the rabbit ileum in which the periarterial (mainly...

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Boyd G, Gillespie JS, Mackenna BR. Origin of the cholinergic response of the rabbit intestine to stimulation of its extrinsic sympathetic nerves after exposure to sympathetic blocking agents. Br J Pharmacol Chemother. 1962;19(2):258-70doi: 10.1111/j.1476-5381.1962.tb01187.x.
Boyd, G., Gillespie, J. S., & Mackenna, B. R. (1962). Origin of the cholinergic response of the rabbit intestine to stimulation of its extrinsic sympathetic nerves after exposure to sympathetic blocking agents. British journal of pharmacology and chemotherapy, 19(2), 258-70. https://doi.org/10.1111/j.1476-5381.1962.tb01187.x
Boyd, G, et al. "Origin of the cholinergic response of the rabbit intestine to stimulation of its extrinsic sympathetic nerves after exposure to sympathetic blocking agents." British journal of pharmacology and chemotherapy vol. 19,2 (1962): 258-70. doi: https://doi.org/10.1111/j.1476-5381.1962.tb01187.x
Boyd G, Gillespie JS, Mackenna BR. Origin of the cholinergic response of the rabbit intestine to stimulation of its extrinsic sympathetic nerves after exposure to sympathetic blocking agents. Br J Pharmacol Chemother. 1962 Oct;19(2):258-70. doi: 10.1111/j.1476-5381.1962.tb01187.x. PMID: 19108166; PMCID: PMC1482143.
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Guanethidine in the Treatment of Hypertension.

Canadian Medical Association journal

[No authors listed]
PMID: 20326462
Can Med Assoc J. 1960 Sep 24;83(13):720-1.

No abstract available.

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Guanethidine in the Treatment of Hypertension. Can Med Assoc J. 1960;83(13):720-1
(1960). Guanethidine in the Treatment of Hypertension. Canadian Medical Association journal, 83(13), 720-1.
"Guanethidine in the Treatment of Hypertension." Canadian Medical Association journal vol. 83,13 (1960): 720-1.
Guanethidine in the Treatment of Hypertension. Can Med Assoc J. 1960 Sep 24;83(13):720-1. PMID: 20326462; PMCID: PMC1938694.
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Long-Term Follow-up of Patients with Hypertensive Disease Treated with Guanethidine.

Canadian Medical Association journal

Lewis JA, Kavelman DA.
PMID: 20327535
Can Med Assoc J. 1963 May 18;88(20):1010-3.

Sixty-three hypertensive patients treated with guanethidine, usually in conjunction with chlorthalidone, were followed up for periods as long as 2(1/2) years. The guanethidine dosage was not found to be related to the size of the patient, duration of hypertension...

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Lewis JA, Kavelman DA. Long-Term Follow-up of Patients with Hypertensive Disease Treated with Guanethidine. Can Med Assoc J. 1963;88(20):1010-3
Lewis, J. A., & Kavelman, D. A. (1963). Long-Term Follow-up of Patients with Hypertensive Disease Treated with Guanethidine. Canadian Medical Association journal, 88(20), 1010-3.
Lewis, J A, and Kavelman, D A. "Long-Term Follow-up of Patients with Hypertensive Disease Treated with Guanethidine." Canadian Medical Association journal vol. 88,20 (1963): 1010-3.
Lewis JA, Kavelman DA. Long-Term Follow-up of Patients with Hypertensive Disease Treated with Guanethidine. Can Med Assoc J. 1963 May 18;88(20):1010-3. PMID: 20327535; PMCID: PMC1921322.
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[Modification of the guanethidine block for diagnosing reflex sympathetic dystrophy.].

Schmerz (Berlin, Germany)

Hoffmann U, Blumberg H.
PMID: 18415442
Schmerz. 1994 Jun;8(2):95-9. doi: 10.1007/BF02530414.

UNLABELLED: For diagnosis of the pain origin in reflex sympathetic dystrophy (RSD), sympathetic blocks, including the intravenous regional guanethidine (IVRG) block after Hannington-Kiff, are recommended. Since the results obtained with this kind of block are sometimes unsatisfactory, modifications were...

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Hoffmann U, Blumberg H. Modifikationen der Guanethidin-Blockade zur Diagnostik der sympathischen Reflexdystrophie (Morbus Sudeck). [Modification of the guanethidine block for diagnosing reflex sympathetic dystrophy.]. Schmerz. 1994;8(2):95-9doi: 10.1007/BF02530414.
Hoffmann, U., & Blumberg, H. (1994). Modifikationen der Guanethidin-Blockade zur Diagnostik der sympathischen Reflexdystrophie (Morbus Sudeck). [Modification of the guanethidine block for diagnosing reflex sympathetic dystrophy.]. Schmerz (Berlin, Germany), 8(2), 95-9. https://doi.org/10.1007/BF02530414
Hoffmann, U, and Blumberg, H. "Modifikationen der Guanethidin-Blockade zur Diagnostik der sympathischen Reflexdystrophie (Morbus Sudeck)." [Modification of the guanethidine block for diagnosing reflex sympathetic dystrophy.]. Schmerz (Berlin, Germany) vol. 8,2 (1994): 95-9. doi: https://doi.org/10.1007/BF02530414
Hoffmann U, Blumberg H. Modifikationen der Guanethidin-Blockade zur Diagnostik der sympathischen Reflexdystrophie (Morbus Sudeck). [Modification of the guanethidine block for diagnosing reflex sympathetic dystrophy.]. Schmerz. 1994 Jun;8(2):95-9. doi: 10.1007/BF02530414. German. PMID: 18415442.
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Interactions of amitriptyline with guanethidine and thymoxamine in the human iris.

British journal of clinical pharmacology

Ghose K, Dobree C, Taylor P, Turner P.
PMID: 22454943
Br J Clin Pharmacol. 1974 Dec;1(6):516-7. doi: 10.1111/j.1365-2125.1974.tb01707.x.

No abstract available.

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Ghose K, Dobree C, Taylor P, et al. Interactions of amitriptyline with guanethidine and thymoxamine in the human iris. Br J Clin Pharmacol. 1974;1(6):516-7doi: 10.1111/j.1365-2125.1974.tb01707.x.
Ghose, K., Dobree, C., Taylor, P., & Turner, P. (1974). Interactions of amitriptyline with guanethidine and thymoxamine in the human iris. British journal of clinical pharmacology, 1(6), 516-7. https://doi.org/10.1111/j.1365-2125.1974.tb01707.x
Ghose, K, et al. "Interactions of amitriptyline with guanethidine and thymoxamine in the human iris." British journal of clinical pharmacology vol. 1,6 (1974): 516-7. doi: https://doi.org/10.1111/j.1365-2125.1974.tb01707.x
Ghose K, Dobree C, Taylor P, Turner P. Interactions of amitriptyline with guanethidine and thymoxamine in the human iris. Br J Clin Pharmacol. 1974 Dec;1(6):516-7. doi: 10.1111/j.1365-2125.1974.tb01707.x. PMID: 22454943; PMCID: PMC1402527.
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Electrical activation of common bile duct nerves modulates sphincter of Oddi motility in the Australian possum.

HPB : the official journal of the International Hepato Pancreato Biliary Association

Sonoda Y, Takahata S, Jabar F, Schloithe AC, Grivell MA, Woods CM, Simula ME, Toouli J, Saccone GT.
PMID: 18333212
HPB (Oxford). 2005;7(4):303-12. doi: 10.1080/13651820510037639.

BACKGROUND: Sphincter of Oddi (SO) motility is regulated by extrinsic and intrinsic nerves. The existence of neural circuits between the SO and the proximal extrahepatic biliary tree has been reported, but they are poorly understood. Using electrical field stimulation...

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Sonoda Y, Takahata S, Jabar F, et al. Electrical activation of common bile duct nerves modulates sphincter of Oddi motility in the Australian possum. HPB (Oxford). 2005;7(4):303-12doi: 10.1080/13651820510037639.
Sonoda, Y., Takahata, S., Jabar, F., Schloithe, A. C., Grivell, M. A., Woods, C. M., Simula, M. E., Toouli, J., & Saccone, G. T. (2005). Electrical activation of common bile duct nerves modulates sphincter of Oddi motility in the Australian possum. HPB : the official journal of the International Hepato Pancreato Biliary Association, 7(4), 303-12. https://doi.org/10.1080/13651820510037639
Sonoda, Y, et al. "Electrical activation of common bile duct nerves modulates sphincter of Oddi motility in the Australian possum." HPB : the official journal of the International Hepato Pancreato Biliary Association vol. 7,4 (2005): 303-12. doi: https://doi.org/10.1080/13651820510037639
Sonoda Y, Takahata S, Jabar F, Schloithe AC, Grivell MA, Woods CM, Simula ME, Toouli J, Saccone GT. Electrical activation of common bile duct nerves modulates sphincter of Oddi motility in the Australian possum. HPB (Oxford). 2005;7(4):303-12. doi: 10.1080/13651820510037639. PMID: 18333212; PMCID: PMC2043104.
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Iodine-131 meta-iodobezylguanidine single photon emission computed tomography/computerized tomography in diagnosis of neuro-endocrine tumors.

Indian journal of nuclear medicine : IJNM : the official journal of the Society of Nuclear Medicine, India

Harisankar CN, Mittal BR, Bhattacharya A, Kashyap R, Bhansali A.
PMID: 23599604
Indian J Nucl Med. 2012 Jan;27(1):55-8. doi: 10.4103/0972-3919.108877.

Metaiodobenzyl guanidine (MIBG) is a derivative of guanethidine and acts as an analogue of nor-epinephrine and is widely used in the imaging of tumors of neuro-endocrine origin. Iodine-123 MIBG has ideal imaging characteristics but is expensive with limited availability....

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Harisankar CN, Mittal BR, Bhattacharya A, et al. Iodine-131 meta-iodobezylguanidine single photon emission computed tomography/computerized tomography in diagnosis of neuro-endocrine tumors. Indian J Nucl Med. 2012;27(1):55-8doi: 10.4103/0972-3919.108877.
Harisankar, C. N., Mittal, B. R., Bhattacharya, A., Kashyap, R., & Bhansali, A. (2012). Iodine-131 meta-iodobezylguanidine single photon emission computed tomography/computerized tomography in diagnosis of neuro-endocrine tumors. Indian journal of nuclear medicine : IJNM : the official journal of the Society of Nuclear Medicine, India, 27(1), 55-8. https://doi.org/10.4103/0972-3919.108877
Harisankar, Chidambaram Natrajan Balasubramanian, et al. "Iodine-131 meta-iodobezylguanidine single photon emission computed tomography/computerized tomography in diagnosis of neuro-endocrine tumors." Indian journal of nuclear medicine : IJNM : the official journal of the Society of Nuclear Medicine, India vol. 27,1 (2012): 55-8. doi: https://doi.org/10.4103/0972-3919.108877
Harisankar CN, Mittal BR, Bhattacharya A, Kashyap R, Bhansali A. Iodine-131 meta-iodobezylguanidine single photon emission computed tomography/computerized tomography in diagnosis of neuro-endocrine tumors. Indian J Nucl Med. 2012 Jan;27(1):55-8. doi: 10.4103/0972-3919.108877. PMID: 23599604; PMCID: PMC3628268.
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The Inhibitory Mechanism of Gentamicin on Electrical Field Stimulation Response in Rat Bladder Smooth Muscle.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Min CH, Wang Y, Bae J, Han JH, Sohn UD.
PMID: 26330761
Korean J Physiol Pharmacol. 2015 Sep;19(5):473-8. doi: 10.4196/kjpp.2015.19.5.473. Epub 2015 Aug 20.

To see the inhibitory mechanism of gentamicin in response to electrical field stimulation (EFS) using the rat bladder smooth muscle, atropine or guanethidine was treated but had no effect. Methylsergide, a non-selective 5-HT1, 5-HT2 receptor antagonist was also treated...

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Min CH, Wang Y, Bae J, et al. The Inhibitory Mechanism of Gentamicin on Electrical Field Stimulation Response in Rat Bladder Smooth Muscle. Korean J Physiol Pharmacol. 2015;19(5):473-8doi: 10.4196/kjpp.2015.19.5.473.
Min, C. H., Wang, Y., Bae, J., Han, J. H., & Sohn, U. D. (2015). The Inhibitory Mechanism of Gentamicin on Electrical Field Stimulation Response in Rat Bladder Smooth Muscle. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 19(5), 473-8. https://doi.org/10.4196/kjpp.2015.19.5.473
Min, Chang Ho, et al. "The Inhibitory Mechanism of Gentamicin on Electrical Field Stimulation Response in Rat Bladder Smooth Muscle." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 19,5 (2015): 473-8. doi: https://doi.org/10.4196/kjpp.2015.19.5.473
Min CH, Wang Y, Bae J, Han JH, Sohn UD. The Inhibitory Mechanism of Gentamicin on Electrical Field Stimulation Response in Rat Bladder Smooth Muscle. Korean J Physiol Pharmacol. 2015 Sep;19(5):473-8. doi: 10.4196/kjpp.2015.19.5.473. Epub 2015 Aug 20. PMID: 26330761; PMCID: PMC4553408.
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Capsaicin-Sensitive Sensory Nerves Indirectly Modulate Motor Function of the Urinary Bladder.

International neurourology journal

Chang HH, Chang SJ, Hsieh CH, Hsu CK, Yang SS.
PMID: 29991229
Int Neurourol J. 2018 Jun;22(2):83-89. doi: 10.5213/inj.1836078.039. Epub 2018 Jun 30.

PURPOSE: The urinary bladder (UB) is innervated by both sensory and autonomic nerves. Recent studies have shown that sensory neuropeptides induced contractions in the detrusor muscle. Therefore, in a mouse model, we investigated the presence of interactions between the...

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Chang HH, Chang SJ, Hsieh CH, et al. Capsaicin-Sensitive Sensory Nerves Indirectly Modulate Motor Function of the Urinary Bladder. Int Neurourol J. 2018;22(2):83-89doi: 10.5213/inj.1836078.039.
Chang, H. H., Chang, S. J., Hsieh, C. H., Hsu, C. K., & Yang, S. S. (2018). Capsaicin-Sensitive Sensory Nerves Indirectly Modulate Motor Function of the Urinary Bladder. International neurourology journal, 22(2), 83-89. https://doi.org/10.5213/inj.1836078.039
Chang, Hsi-Hsien, et al. "Capsaicin-Sensitive Sensory Nerves Indirectly Modulate Motor Function of the Urinary Bladder." International neurourology journal vol. 22,2 (2018): 83-89. doi: https://doi.org/10.5213/inj.1836078.039
Chang HH, Chang SJ, Hsieh CH, Hsu CK, Yang SS. Capsaicin-Sensitive Sensory Nerves Indirectly Modulate Motor Function of the Urinary Bladder. Int Neurourol J. 2018 Jun;22(2):83-89. doi: 10.5213/inj.1836078.039. Epub 2018 Jun 30. PMID: 29991229; PMCID: PMC6059914.
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Displacement of one drug by another from carrier or receptor sites.

Proceedings of the Royal Society of Medicine

Brodie BB.
PMID: 5854428
Proc R Soc Med. 1965 Nov;58(11):946-55.
Free PMC Article

The medium of drug transfer is the water of plasma and extracellular fluid. Without complicating factors, the level of drug at a receptor site would be equal to that in the tissues and in plasma, and in dynamic equilibrium....

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Brodie BB. Displacement of one drug by another from carrier or receptor sites. Proc R Soc Med. 1965;58(11):946-55
Brodie, B. B. (1965). Displacement of one drug by another from carrier or receptor sites. Proceedings of the Royal Society of Medicine, 58(11), 946-55.
Brodie, B B. "Displacement of one drug by another from carrier or receptor sites." Proceedings of the Royal Society of Medicine vol. 58,11 (1965): 946-55.
Brodie BB. Displacement of one drug by another from carrier or receptor sites. Proc R Soc Med. 1965 Nov;58(11):946-55. PMID: 5854428; PMCID: PMC1898665.
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Showing 13 to 24 of 41 entries