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A plausible explanation for male dominance in typhoid ileal perforation.

Clinical and experimental gastroenterology

Khan M.
PMID: 23180972
Clin Exp Gastroenterol. 2012;5:213-7. doi: 10.2147/CEG.S36569. Epub 2012 Nov 12.

The phenomenon of consistent male dominance in typhoid ileal perforation (TIP) is not well understood. It cannot be explained on the basis of microbial virulence, Peyer's patch anatomy, ileal wall thickness, gastric acidity, host genetic factors, or sex-linked bias...

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Khan M. A plausible explanation for male dominance in typhoid ileal perforation. Clin Exp Gastroenterol. 2012;5:213-7doi: 10.2147/CEG.S36569.
Khan, M. (2012). A plausible explanation for male dominance in typhoid ileal perforation. Clinical and experimental gastroenterology, 5213-7. https://doi.org/10.2147/CEG.S36569
Khan, Mohammad. "A plausible explanation for male dominance in typhoid ileal perforation." Clinical and experimental gastroenterology vol. 5 (2012): 213-7. doi: https://doi.org/10.2147/CEG.S36569
Khan M. A plausible explanation for male dominance in typhoid ileal perforation. Clin Exp Gastroenterol. 2012;5:213-7. doi: 10.2147/CEG.S36569. Epub 2012 Nov 12. PMID: 23180972; PMCID: PMC3501370.
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Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report.

Journal of medical case reports

Cheungpasitporn W, Jirajariyavej T, Howarth CB, Rosen RM.
PMID: 21831272
J Med Case Rep. 2011 Aug 10;5:364. doi: 10.1186/1752-1947-5-364.

INTRODUCTION: Henoch-Schönlein purpura is the most common systemic vasculitis in children. Typical presentations are palpable purpura, abdominal pain, arthritis, and hematuria. This vasculitic syndrome can present as an uncommon cause of rectal bleeding in older patients. We report a...

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Cheungpasitporn W, Jirajariyavej T, Howarth CB, et al. Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report. J Med Case Rep. 2011;5:364doi: 10.1186/1752-1947-5-364.
Cheungpasitporn, W., Jirajariyavej, T., Howarth, C. B., & Rosen, R. M. (2011). Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report. Journal of medical case reports, 5364. https://doi.org/10.1186/1752-1947-5-364
Cheungpasitporn, Wisit, et al. "Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report." Journal of medical case reports vol. 5 (2011): 364. doi: https://doi.org/10.1186/1752-1947-5-364
Cheungpasitporn W, Jirajariyavej T, Howarth CB, Rosen RM. Henoch-Schönlein purpura in an older man presenting as rectal bleeding and IgA mesangioproliferative glomerulonephritis: a case report. J Med Case Rep. 2011 Aug 10;5:364. doi: 10.1186/1752-1947-5-364. PMID: 21831272; PMCID: PMC3163221.
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Inflammation-induced atherosclerosis as a target for prevention of cardiovascular diseases from early life.

The open cardiovascular medicine journal

Kelishadi R.
PMID: 21804638
Open Cardiovasc Med J. 2010;4:24-9. doi: 10.2174/1874192401004020024. Epub 2010 Feb 23.

Atherogenesis starts from the fetal life, and its natural course consists of interrelations between traditional risk factors and inflammatory, immune, and endothelial biomarkers. Even the early-stages of atherosclerotic lesions, i.e. fatty streaks present the features of chronic inflammation. Markers...

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Kelishadi R. Inflammation-induced atherosclerosis as a target for prevention of cardiovascular diseases from early life. Open Cardiovasc Med J. 2010;4:24-9doi: 10.2174/1874192401004020024.
Kelishadi, R. (2010). Inflammation-induced atherosclerosis as a target for prevention of cardiovascular diseases from early life. The open cardiovascular medicine journal, 424-9. https://doi.org/10.2174/1874192401004020024
Kelishadi, Roya. "Inflammation-induced atherosclerosis as a target for prevention of cardiovascular diseases from early life." The open cardiovascular medicine journal vol. 4 (2010): 24-9. doi: https://doi.org/10.2174/1874192401004020024
Kelishadi R. Inflammation-induced atherosclerosis as a target for prevention of cardiovascular diseases from early life. Open Cardiovasc Med J. 2010;4:24-9. doi: 10.2174/1874192401004020024. Epub 2010 Feb 23. PMID: 21804638; PMCID: PMC2840608.
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Persistence of systemic inflammation in COPD in spite of smoking cessation.

Multidisciplinary respiratory medicine

Invernizzi G.
PMID: 22958349
Multidiscip Respir Med. 2011 Aug 31;6(4):210-1. doi: 10.1186/2049-6958-6-4-210.

No abstract available.

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Invernizzi G. Persistence of systemic inflammation in COPD in spite of smoking cessation. Multidiscip Respir Med. 2011;6(4):210-1doi: 10.1186/2049-6958-6-4-210.
Invernizzi, G. (2011). Persistence of systemic inflammation in COPD in spite of smoking cessation. Multidisciplinary respiratory medicine, 6(4), 210-1. https://doi.org/10.1186/2049-6958-6-4-210
Invernizzi, Giovanni. "Persistence of systemic inflammation in COPD in spite of smoking cessation." Multidisciplinary respiratory medicine vol. 6,4 (2011): 210-1. doi: https://doi.org/10.1186/2049-6958-6-4-210
Invernizzi G. Persistence of systemic inflammation in COPD in spite of smoking cessation. Multidiscip Respir Med. 2011 Aug 31;6(4):210-1. doi: 10.1186/2049-6958-6-4-210. PMID: 22958349; PMCID: PMC3463078.
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Systemic inflammation in COPD in relation to smoking status.

Multidisciplinary respiratory medicine

Serapinas D, Narbekovas A, Juskevicius J, Sakalauskas R.
PMID: 22958407
Multidiscip Respir Med. 2011 Aug 31;6(4):214-9. doi: 10.1186/2049-6958-6-4-214.

BACKGROUND AND AIMS: Smoking is the main risk factor for the development of chronic obstructive pulmonary disease (COPD) that has been recently defined as a systemic pulmonary inflammatory disease. However, the impact of smoking itself on systemic inflammation in...

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Serapinas D, Narbekovas A, Juskevicius J, et al. Systemic inflammation in COPD in relation to smoking status. Multidiscip Respir Med. 2011;6(4):214-9doi: 10.1186/2049-6958-6-4-214.
Serapinas, D., Narbekovas, A., Juskevicius, J., & Sakalauskas, R. (2011). Systemic inflammation in COPD in relation to smoking status. Multidisciplinary respiratory medicine, 6(4), 214-9. https://doi.org/10.1186/2049-6958-6-4-214
Serapinas, Danielius, et al. "Systemic inflammation in COPD in relation to smoking status." Multidisciplinary respiratory medicine vol. 6,4 (2011): 214-9. doi: https://doi.org/10.1186/2049-6958-6-4-214
Serapinas D, Narbekovas A, Juskevicius J, Sakalauskas R. Systemic inflammation in COPD in relation to smoking status. Multidiscip Respir Med. 2011 Aug 31;6(4):214-9. doi: 10.1186/2049-6958-6-4-214. PMID: 22958407; PMCID: PMC3463080.
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Fractalkine Mediates Communication between Pathogenic Proteins and Microglia: Implications of Anti-Inflammatory Treatments in Different Stages of Neurodegenerative Diseases.

International journal of Alzheimer's disease

Desforges NM, Hebron ML, Algarzae NK, Lonskaya I, Moussa CE.
PMID: 22919540
Int J Alzheimers Dis. 2012;2012:345472. doi: 10.1155/2012/345472. Epub 2012 Aug 05.

The role of inflammation in neurodegenerative diseases has been widely demonstrated. Intraneuronal protein accumulation may regulate microglial activity via the fractalkine (CX3CL1) signaling pathway that provides a mechanism through which neurons communicate with microglia. CX3CL1 levels fluctuate in different...

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Desforges NM, Hebron ML, Algarzae NK, et al. Fractalkine Mediates Communication between Pathogenic Proteins and Microglia: Implications of Anti-Inflammatory Treatments in Different Stages of Neurodegenerative Diseases. Int J Alzheimers Dis. 2012;2012:345472doi: 10.1155/2012/345472.
Desforges, N. M., Hebron, M. L., Algarzae, N. K., Lonskaya, I., & Moussa, C. E. (2012). Fractalkine Mediates Communication between Pathogenic Proteins and Microglia: Implications of Anti-Inflammatory Treatments in Different Stages of Neurodegenerative Diseases. International journal of Alzheimer's disease, 2012345472. https://doi.org/10.1155/2012/345472
Desforges, Nicole M, et al. "Fractalkine Mediates Communication between Pathogenic Proteins and Microglia: Implications of Anti-Inflammatory Treatments in Different Stages of Neurodegenerative Diseases." International journal of Alzheimer's disease vol. 2012 (2012): 345472. doi: https://doi.org/10.1155/2012/345472
Desforges NM, Hebron ML, Algarzae NK, Lonskaya I, Moussa CE. Fractalkine Mediates Communication between Pathogenic Proteins and Microglia: Implications of Anti-Inflammatory Treatments in Different Stages of Neurodegenerative Diseases. Int J Alzheimers Dis. 2012;2012:345472. doi: 10.1155/2012/345472. Epub 2012 Aug 05. PMID: 22919540; PMCID: PMC3420133.
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The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis.

Frontiers in aging neuroscience

Lovejoy DB, Guillemin GJ.
PMID: 25100994
Front Aging Neurosci. 2014 Jul 23;6:173. doi: 10.3389/fnagi.2014.00173. eCollection 2014.

Modulations of the potentially toxic transition metals iron (Fe) and copper (Cu) are implicated in the neurodegenerative process in a variety of human disease states including amyotrophic lateral sclerosis (ALS). However, the precise role played by these metals is...

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Lovejoy DB, Guillemin GJ. The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis. Front Aging Neurosci. 2014;6:173doi: 10.3389/fnagi.2014.00173.
Lovejoy, D. B., & Guillemin, G. J. (2014). The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis. Frontiers in aging neuroscience, 6173. https://doi.org/10.3389/fnagi.2014.00173
Lovejoy, David B, and Guillemin, Gilles J. "The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis." Frontiers in aging neuroscience vol. 6 (2014): 173. doi: https://doi.org/10.3389/fnagi.2014.00173
Lovejoy DB, Guillemin GJ. The potential for transition metal-mediated neurodegeneration in amyotrophic lateral sclerosis. Front Aging Neurosci. 2014 Jul 23;6:173. doi: 10.3389/fnagi.2014.00173. eCollection 2014. PMID: 25100994; PMCID: PMC4107949.
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Toll-like receptor 9 in breast cancer.

Frontiers in immunology

Sandholm J, Selander KS.
PMID: 25101078
Front Immunol. 2014 Jul 22;5:330. doi: 10.3389/fimmu.2014.00330. eCollection 2014.

Toll-like receptor 9 (TLR9) is a cellular DNA receptor of the innate immune system. DNA recognition via TLR9 results in an inflammatory reaction, which eventually also activates a Th1-biased adaptive immune attack. In addition to cells of the immune...

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Sandholm J, Selander KS. Toll-like receptor 9 in breast cancer. Front Immunol. 2014;5:330doi: 10.3389/fimmu.2014.00330.
Sandholm, J., & Selander, K. S. (2014). Toll-like receptor 9 in breast cancer. Frontiers in immunology, 5330. https://doi.org/10.3389/fimmu.2014.00330
Sandholm, Jouko, and Selander, Katri S. "Toll-like receptor 9 in breast cancer." Frontiers in immunology vol. 5 (2014): 330. doi: https://doi.org/10.3389/fimmu.2014.00330
Sandholm J, Selander KS. Toll-like receptor 9 in breast cancer. Front Immunol. 2014 Jul 22;5:330. doi: 10.3389/fimmu.2014.00330. eCollection 2014. PMID: 25101078; PMCID: PMC4105583.
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Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men.

Genes & nutrition

Pasman WJ, van Erk MJ, Klöpping WA, Pellis L, Wopereis S, Bijlsma S, Hendriks HF, Kardinaal AF.
PMID: 23595524
Genes Nutr. 2013 Sep;8(5):507-21. doi: 10.1007/s12263-013-0343-9. Epub 2013 Apr 18.

We aimed to explore whether vegetable consumption according to guidelines has beneficial health effects determined with classical biomarkers and nutrigenomics technologies. Fifteen lean (age 36 ± 7 years; BMI 23.4 ± 1.7 kg m(-2)) and 17 obese (age 40...

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Pasman WJ, van Erk MJ, Klöpping WA, et al. Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men. Genes Nutr. 2013;8(5):507-21doi: 10.1007/s12263-013-0343-9.
Pasman, W. J., van Erk, M. J., Klöpping, W. A., Pellis, L., Wopereis, S., Bijlsma, S., Hendriks, H. F., & Kardinaal, A. F. (2013). Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men. Genes & nutrition, 8(5), 507-21. https://doi.org/10.1007/s12263-013-0343-9
Pasman, W J, et al. "Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men." Genes & nutrition vol. 8,5 (2013): 507-21. doi: https://doi.org/10.1007/s12263-013-0343-9
Pasman WJ, van Erk MJ, Klöpping WA, Pellis L, Wopereis S, Bijlsma S, Hendriks HF, Kardinaal AF. Nutrigenomics approach elucidates health-promoting effects of high vegetable intake in lean and obese men. Genes Nutr. 2013 Sep;8(5):507-21. doi: 10.1007/s12263-013-0343-9. Epub 2013 Apr 18. PMID: 23595524; PMCID: PMC3755133.
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Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer.

Frontiers in pharmacology

Rak J.
PMID: 23508692
Front Pharmacol. 2013 Mar 06;4:21. doi: 10.3389/fphar.2013.00021. eCollection 2013.

In multicellular organisms both health and disease are defined by patterns of communication between the constituent cells. In addition to networks of soluble mediators, cells are also programed to exchange complex messages pre-assembled as multimolecular cargo of membraneous structures...

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Rak J. Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Front Pharmacol. 2013;4:21doi: 10.3389/fphar.2013.00021.
Rak, J. (2013). Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Frontiers in pharmacology, 421. https://doi.org/10.3389/fphar.2013.00021
Rak, Janusz. "Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer." Frontiers in pharmacology vol. 4 (2013): 21. doi: https://doi.org/10.3389/fphar.2013.00021
Rak J. Extracellular vesicles - biomarkers and effectors of the cellular interactome in cancer. Front Pharmacol. 2013 Mar 06;4:21. doi: 10.3389/fphar.2013.00021. eCollection 2013. PMID: 23508692; PMCID: PMC3589665.
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A Community-based Oral Health Promotion Model for HIV Patients in Nairobi, East District in Kenya: a Study Protocol.

Journal of public health research

Koyio LN, van der Sanden WJ, van der Ven A, Mulder J, Creugers NH, Merkx MA, Frencken JE.
PMID: 25170476
J Public Health Res. 2013 Jun 07;2(1):22-8. doi: 10.4081/jphr.2013.e5. eCollection 2013 Apr 28.

BACKGROUND: General HIV-related orofacial lesions, most commonly oropharyngeal candidiasis, have a typical clinical appearance and can be recognised by members of the community. Although affected patients often experience pain leading to compromised eating and swallowing, barriers such as social...

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Koyio LN, van der Sanden WJ, van der Ven A, et al. A Community-based Oral Health Promotion Model for HIV Patients in Nairobi, East District in Kenya: a Study Protocol. J Public Health Res. 2013;2(1):22-8doi: 10.4081/jphr.2013.e5.
Koyio, L. N., van der Sanden, W. J., van der Ven, A., Mulder, J., Creugers, N. H., Merkx, M. A., & Frencken, J. E. (2013). A Community-based Oral Health Promotion Model for HIV Patients in Nairobi, East District in Kenya: a Study Protocol. Journal of public health research, 2(1), 22-8. https://doi.org/10.4081/jphr.2013.e5
Koyio, Lucina N, et al. "A Community-based Oral Health Promotion Model for HIV Patients in Nairobi, East District in Kenya: a Study Protocol." Journal of public health research vol. 2,1 (2013): 22-8. doi: https://doi.org/10.4081/jphr.2013.e5
Koyio LN, van der Sanden WJ, van der Ven A, Mulder J, Creugers NH, Merkx MA, Frencken JE. A Community-based Oral Health Promotion Model for HIV Patients in Nairobi, East District in Kenya: a Study Protocol. J Public Health Res. 2013 Jun 07;2(1):22-8. doi: 10.4081/jphr.2013.e5. eCollection 2013 Apr 28. PMID: 25170476; PMCID: PMC4140327.
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Disseminated tuberculosis presenting as irido-ciliary granuloma in an immunocompetent patient.

Journal of ophthalmic inflammation and infection

Basu S, Mittal R, Rath S, Balne PK, Sharma S.
PMID: 22454250
J Ophthalmic Inflamm Infect. 2012 Sep;2(3):173-5. doi: 10.1007/s12348-012-0068-8. Epub 2012 Mar 28.

No abstract available.

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Basu S, Mittal R, Rath S, et al. Disseminated tuberculosis presenting as irido-ciliary granuloma in an immunocompetent patient. J Ophthalmic Inflamm Infect. 2012;2(3):173-5doi: 10.1007/s12348-012-0068-8.
Basu, S., Mittal, R., Rath, S., Balne, P. K., & Sharma, S. (2012). Disseminated tuberculosis presenting as irido-ciliary granuloma in an immunocompetent patient. Journal of ophthalmic inflammation and infection, 2(3), 173-5. https://doi.org/10.1007/s12348-012-0068-8
Basu, Soumyava, et al. "Disseminated tuberculosis presenting as irido-ciliary granuloma in an immunocompetent patient." Journal of ophthalmic inflammation and infection vol. 2,3 (2012): 173-5. doi: https://doi.org/10.1007/s12348-012-0068-8
Basu S, Mittal R, Rath S, Balne PK, Sharma S. Disseminated tuberculosis presenting as irido-ciliary granuloma in an immunocompetent patient. J Ophthalmic Inflamm Infect. 2012 Sep;2(3):173-5. doi: 10.1007/s12348-012-0068-8. Epub 2012 Mar 28. PMID: 22454250; PMCID: PMC3438301.
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Showing 25 to 36 of 94475 entries