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Showing 25 to 36 of 37 entries
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Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics.

Asia Pacific allergy

Kim SH, Lim KH, Park HK, Lee SY, Kim SH, Kang HR, Park HW, Chang YS, Cho SH.
PMID: 25938076
Asia Pac Allergy. 2015 Apr;5(2):114-22. doi: 10.5415/apallergy.2015.5.2.114. Epub 2015 Apr 29.

BACKGROUND: Human rhinoviruses are the major cause of asthma exacerbation in both children and adults. Recently, impaired antiviral interferon (IFN) response in asthmatics has been indicated as a primary reason of the susceptibility to respiratory virus, but the mechanism...

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Kim SH, Lim KH, Park HK, et al. Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics. Asia Pac Allergy. 2015;5(2):114-22doi: 10.5415/apallergy.2015.5.2.114.
Kim, S. H., Lim, K. H., Park, H. K., Lee, S. Y., Kim, S. H., Kang, H. R., Park, H. W., Chang, Y. S., & Cho, S. H. (2015). Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics. Asia Pacific allergy, 5(2), 114-22. https://doi.org/10.5415/apallergy.2015.5.2.114
Kim, Sae-Hoon, et al. "Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics." Asia Pacific allergy vol. 5,2 (2015): 114-22. doi: https://doi.org/10.5415/apallergy.2015.5.2.114
Kim SH, Lim KH, Park HK, Lee SY, Kim SH, Kang HR, Park HW, Chang YS, Cho SH. Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics. Asia Pac Allergy. 2015 Apr;5(2):114-22. doi: 10.5415/apallergy.2015.5.2.114. Epub 2015 Apr 29. PMID: 25938076; PMCID: PMC4415177.
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Safeguard against DNA sensing: the role of TREX1 in HIV-1 infection and autoimmune diseases.

Frontiers in microbiology

Hasan M, Yan N.
PMID: 24817865
Front Microbiol. 2014 Apr 30;5:193. doi: 10.3389/fmicb.2014.00193. eCollection 2014.

Innate immune recognition is crucial for host responses against viral infections, including infection by human immunodeficiency virus 1 (HIV-1). Human cells detect such invading pathogens with a collection of pattern recognition receptors that activate the production of antiviral proteins,...

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Hasan M, Yan N. Safeguard against DNA sensing: the role of TREX1 in HIV-1 infection and autoimmune diseases. Front Microbiol. 2014;5:193doi: 10.3389/fmicb.2014.00193.
Hasan, M., & Yan, N. (2014). Safeguard against DNA sensing: the role of TREX1 in HIV-1 infection and autoimmune diseases. Frontiers in microbiology, 5193. https://doi.org/10.3389/fmicb.2014.00193
Hasan, Maroof, and Yan, Nan. "Safeguard against DNA sensing: the role of TREX1 in HIV-1 infection and autoimmune diseases." Frontiers in microbiology vol. 5 (2014): 193. doi: https://doi.org/10.3389/fmicb.2014.00193
Hasan M, Yan N. Safeguard against DNA sensing: the role of TREX1 in HIV-1 infection and autoimmune diseases. Front Microbiol. 2014 Apr 30;5:193. doi: 10.3389/fmicb.2014.00193. eCollection 2014. PMID: 24817865; PMCID: PMC4012220.
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Integrated genomics point to immune vulnerabilities in pleural mesothelioma.

Scientific reports

Nastase A, Mandal A, Lu SK, Anbunathan H, Morris-Rosendahl D, Zhang YZ, Sun XM, Gennatas S, Rintoul RC, Edwards M, Bowman A, Chernova T, Benepal T, Lim E, Taylor AN, Nicholson AG, Popat S, Willis AE, MacFarlane M, Lathrop M, Bowcock AM, Moffatt MF, Cookson WOCM.
PMID: 34580349
Sci Rep. 2021 Sep 27;11(1):19138. doi: 10.1038/s41598-021-98414-w.

Pleural mesothelioma is an aggressive malignancy with limited effective therapies. In order to identify therapeutic targets, we integrated SNP genotyping, sequencing and transcriptomics from tumours and low-passage patient-derived cells. Previously unrecognised deletions of SUFU locus (10q24.32), observed in 21%...

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Nastase A, Mandal A, Lu SK, et al. Integrated genomics point to immune vulnerabilities in pleural mesothelioma. Sci Rep. 2021;11(1):19138doi: 10.1038/s41598-021-98414-w.
Nastase, A., Mandal, A., Lu, S. K., Anbunathan, H., Morris-Rosendahl, D., Zhang, Y. Z., Sun, X. M., Gennatas, S., Rintoul, R. C., Edwards, M., Bowman, A., Chernova, T., Benepal, T., Lim, E., Taylor, A. N., Nicholson, A. G., Popat, S., Willis, A. E., MacFarlane, M., Lathrop, M., Bowcock, A. M., Moffatt, M. F., & Cookson, W. O. C. M. (2021). Integrated genomics point to immune vulnerabilities in pleural mesothelioma. Scientific reports, 11(1), 19138. https://doi.org/10.1038/s41598-021-98414-w
Nastase, Anca, et al. "Integrated genomics point to immune vulnerabilities in pleural mesothelioma." Scientific reports vol. 11,1 (2021): 19138. doi: https://doi.org/10.1038/s41598-021-98414-w
Nastase A, Mandal A, Lu SK, Anbunathan H, Morris-Rosendahl D, Zhang YZ, Sun XM, Gennatas S, Rintoul RC, Edwards M, Bowman A, Chernova T, Benepal T, Lim E, Taylor AN, Nicholson AG, Popat S, Willis AE, MacFarlane M, Lathrop M, Bowcock AM, Moffatt MF, Cookson WOCM. Integrated genomics point to immune vulnerabilities in pleural mesothelioma. Sci Rep. 2021 Sep 27;11(1):19138. doi: 10.1038/s41598-021-98414-w. PMID: 34580349; PMCID: PMC8476593.
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Role of CD123 (+) Plasmacytoid Dendritic Cells in Etiologically Different Variants of Erythema Multiforme: A Monocentric Retrospective Study.

Dermatopathology (Basel, Switzerland)

Zengin HB, Pukhalskaya T, Smoller BR.
PMID: 33916862
Dermatopathology (Basel). 2021 Apr 03;8(2):89-96. doi: 10.3390/dermatopathology8020014.

Plasmacytoid dendritic cells (pDCs) constitute a subset of dendritic cells known to be the "professional" interferon type I (IFN-I) producers. pDCs play an important role in antiviral immunity, as well as linking innate and adaptive immunity. Under normal conditions...

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Zengin HB, Pukhalskaya T, Smoller BR. Role of CD123 (+) Plasmacytoid Dendritic Cells in Etiologically Different Variants of Erythema Multiforme: A Monocentric Retrospective Study. Dermatopathology (Basel). 2021;8(2):89-96doi: 10.3390/dermatopathology8020014.
Zengin, H. B., Pukhalskaya, T., & Smoller, B. R. (2021). Role of CD123 (+) Plasmacytoid Dendritic Cells in Etiologically Different Variants of Erythema Multiforme: A Monocentric Retrospective Study. Dermatopathology (Basel, Switzerland), 8(2), 89-96. https://doi.org/10.3390/dermatopathology8020014
Zengin, Hatice B, et al. "Role of CD123 (+) Plasmacytoid Dendritic Cells in Etiologically Different Variants of Erythema Multiforme: A Monocentric Retrospective Study." Dermatopathology (Basel, Switzerland) vol. 8,2 (2021): 89-96. doi: https://doi.org/10.3390/dermatopathology8020014
Zengin HB, Pukhalskaya T, Smoller BR. Role of CD123 (+) Plasmacytoid Dendritic Cells in Etiologically Different Variants of Erythema Multiforme: A Monocentric Retrospective Study. Dermatopathology (Basel). 2021 Apr 03;8(2):89-96. doi: 10.3390/dermatopathology8020014. PMID: 33916862; PMCID: PMC8167774.
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Dormant Tumor Cell Vaccination: A Mathematical Model of Immunological Dormancy in Triple-Negative Breast Cancer.

Cancers

Mehdizadeh R, Shariatpanahi SP, Goliaei B, Peyvandi S, Rüegg C.
PMID: 33440806
Cancers (Basel). 2021 Jan 11;13(2). doi: 10.3390/cancers13020245.

Triple-negative breast cancer (TNBC) is a molecular subtype of breast malignancy with a poor clinical prognosis. There is growing evidence that some chemotherapeutic agents induce an adaptive anti-tumor immune response. This reaction has been proposed to maintain the equilibrium...

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Mehdizadeh R, Shariatpanahi SP, Goliaei B, et al. Dormant Tumor Cell Vaccination: A Mathematical Model of Immunological Dormancy in Triple-Negative Breast Cancer. Cancers (Basel). 2021;13(2)doi: 10.3390/cancers13020245.
Mehdizadeh, R., Shariatpanahi, S. P., Goliaei, B., Peyvandi, S., & Rüegg, C. (2021). Dormant Tumor Cell Vaccination: A Mathematical Model of Immunological Dormancy in Triple-Negative Breast Cancer. Cancers, 13(2), . https://doi.org/10.3390/cancers13020245
Mehdizadeh, Reza, et al. "Dormant Tumor Cell Vaccination: A Mathematical Model of Immunological Dormancy in Triple-Negative Breast Cancer." Cancers vol. 13,2 (2021). doi: https://doi.org/10.3390/cancers13020245
Mehdizadeh R, Shariatpanahi SP, Goliaei B, Peyvandi S, Rüegg C. Dormant Tumor Cell Vaccination: A Mathematical Model of Immunological Dormancy in Triple-Negative Breast Cancer. Cancers (Basel). 2021 Jan 11;13(2). doi: 10.3390/cancers13020245. PMID: 33440806; PMCID: PMC7827392.
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Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus Reality.

Journal of inflammation research

Mdkhana B, Saheb Sharif-Askari N, Ramakrishnan RK, Goel S, Hamid Q, Halwani R.
PMID: 33531826
J Inflamm Res. 2021 Jan 26;14:199-216. doi: 10.2147/JIR.S277716. eCollection 2021.

The coronavirus disease 2019 (COVID-19) pandemic has affected millions of people and crippled economies worldwide. The severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) responsible for this pandemic has triggered avid research on its pathobiology to better understand the pathophysiology...

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Mdkhana B, Saheb Sharif-Askari N, Ramakrishnan RK, et al. Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus Reality. J Inflamm Res. 2021;14:199-216doi: 10.2147/JIR.S277716.
Mdkhana, B., Saheb Sharif-Askari, N., Ramakrishnan, R. K., Goel, S., Hamid, Q., & Halwani, R. (2021). Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus Reality. Journal of inflammation research, 14199-216. https://doi.org/10.2147/JIR.S277716
Mdkhana, Bushra, et al. "Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus Reality." Journal of inflammation research vol. 14 (2021): 199-216. doi: https://doi.org/10.2147/JIR.S277716
Mdkhana B, Saheb Sharif-Askari N, Ramakrishnan RK, Goel S, Hamid Q, Halwani R. Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus Reality. J Inflamm Res. 2021 Jan 26;14:199-216. doi: 10.2147/JIR.S277716. eCollection 2021. PMID: 33531826; PMCID: PMC7847386.
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Human TBK1 deficiency leads to autoinflammation driven by TNF-induced cell death.

Cell

Taft J, Markson M, Legarda D, Patel R, Chan M, Malle L, Richardson A, Gruber C, Martín-Fernández M, Mancini GMS, van Laar JAM, van Pelt P, Buta S, Wokke BHA, Sabli IKD, Sancho-Shimizu V, Chavan PP, Schnappauf O, Khubchandani R, Cüceoğlu MK, Özen S, Kastner DL, Ting AT, Aksentijevich I, Hollink IHIM, Bogunovic D.
PMID: 34363755
Cell. 2021 Aug 19;184(17):4447-4463.e20. doi: 10.1016/j.cell.2021.07.026. Epub 2021 Aug 06.

TANK binding kinase 1 (TBK1) regulates IFN-I, NF-κB, and TNF-induced RIPK1-dependent cell death (RCD). In mice, biallelic loss of TBK1 is embryonically lethal. We discovered four humans, ages 32, 26, 7, and 8 from three unrelated consanguineous families with...

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Taft J, Markson M, Legarda D, et al. Human TBK1 deficiency leads to autoinflammation driven by TNF-induced cell death. Cell. 2021;184(17):4447-4463.e20doi: 10.1016/j.cell.2021.07.026.
Taft, J., Markson, M., Legarda, D., Patel, R., Chan, M., Malle, L., Richardson, A., Gruber, C., Martín-Fernández, M., Mancini, G. M. S., van Laar, J. A. M., van Pelt, P., Buta, S., Wokke, B. H. A., Sabli, I. K. D., Sancho-Shimizu, V., Chavan, P. P., Schnappauf, O., Khubchandani, R., Cüceoğlu, M. K., Özen, S., Kastner, D. L., Ting, A. T., Aksentijevich, I., Hollink, I. H. I. M., & Bogunovic, D. (2021). Human TBK1 deficiency leads to autoinflammation driven by TNF-induced cell death. Cell, 184(17), 4447-4463.e20. https://doi.org/10.1016/j.cell.2021.07.026
Taft, Justin, et al. "Human TBK1 deficiency leads to autoinflammation driven by TNF-induced cell death." Cell vol. 184,17 (2021): 4447-4463.e20. doi: https://doi.org/10.1016/j.cell.2021.07.026
Taft J, Markson M, Legarda D, Patel R, Chan M, Malle L, Richardson A, Gruber C, Martín-Fernández M, Mancini GMS, van Laar JAM, van Pelt P, Buta S, Wokke BHA, Sabli IKD, Sancho-Shimizu V, Chavan PP, Schnappauf O, Khubchandani R, Cüceoğlu MK, Özen S, Kastner DL, Ting AT, Aksentijevich I, Hollink IHIM, Bogunovic D. Human TBK1 deficiency leads to autoinflammation driven by TNF-induced cell death. Cell. 2021 Aug 19;184(17):4447-4463.e20. doi: 10.1016/j.cell.2021.07.026. Epub 2021 Aug 06. PMID: 34363755; PMCID: PMC8380741.
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Immune Responses Against Classical Swine Fever Virus: Between Ignorance and Lunacy.

Frontiers in veterinary science

Summerfield A, Ruggli N.
PMID: 26664939
Front Vet Sci. 2015 May 07;2:10. doi: 10.3389/fvets.2015.00010. eCollection 2015.

Classical swine fever virus infection of pigs causes disease courses from life-threatening to asymptomatic, depending on the virulence of the virus strain and the immunocompetence of the host. The virus targets immune cells, which are central in orchestrating innate...

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Summerfield A, Ruggli N. Immune Responses Against Classical Swine Fever Virus: Between Ignorance and Lunacy. Front Vet Sci. 2015;2:10doi: 10.3389/fvets.2015.00010.
Summerfield, A., & Ruggli, N. (2015). Immune Responses Against Classical Swine Fever Virus: Between Ignorance and Lunacy. Frontiers in veterinary science, 210. https://doi.org/10.3389/fvets.2015.00010
Summerfield, Artur, and Ruggli, Nicolas. "Immune Responses Against Classical Swine Fever Virus: Between Ignorance and Lunacy." Frontiers in veterinary science vol. 2 (2015): 10. doi: https://doi.org/10.3389/fvets.2015.00010
Summerfield A, Ruggli N. Immune Responses Against Classical Swine Fever Virus: Between Ignorance and Lunacy. Front Vet Sci. 2015 May 07;2:10. doi: 10.3389/fvets.2015.00010. eCollection 2015. PMID: 26664939; PMCID: PMC4672165.
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Architectural B-cell organization in skeletal muscle identifies subtypes of dermatomyositis.

Neurology(R) neuroimmunology & neuroinflammation

Radke J, Koll R, Preuße C, Pehl D, Todorova K, Schönemann C, Allenbach Y, Aronica E, de Visser M, Heppner FL, Weis J, Doostkam S, Maisonobe T, Benveniste O, Goebel HH, Stenzel W.
PMID: 29520367
Neurol Neuroimmunol Neuroinflamm. 2018 Mar 06;5(3):e451. doi: 10.1212/NXI.0000000000000451. eCollection 2018 May.

OBJECTIVE: To study the B-cell content, organization, and existence of distinct B-cell subpopulations in relation to the expression of type 1 interferon signature related genes in dermatomyositis (DM).METHODS: Evaluation of skeletal muscle biopsies from patients with adult DM (aDM)...

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Radke J, Koll R, Preuße C, et al. Architectural B-cell organization in skeletal muscle identifies subtypes of dermatomyositis. Neurol Neuroimmunol Neuroinflamm. 2018;5(3):e451doi: 10.1212/NXI.0000000000000451.
Radke, J., Koll, R., Preuße, C., Pehl, D., Todorova, K., Schönemann, C., Allenbach, Y., Aronica, E., de Visser, M., Heppner, F. L., Weis, J., Doostkam, S., Maisonobe, T., Benveniste, O., Goebel, H. H., & Stenzel, W. (2018). Architectural B-cell organization in skeletal muscle identifies subtypes of dermatomyositis. Neurology(R) neuroimmunology & neuroinflammation, 5(3), e451. https://doi.org/10.1212/NXI.0000000000000451
Radke, Josefine, et al. "Architectural B-cell organization in skeletal muscle identifies subtypes of dermatomyositis." Neurology(R) neuroimmunology & neuroinflammation vol. 5,3 (2018): e451. doi: https://doi.org/10.1212/NXI.0000000000000451
Radke J, Koll R, Preuße C, Pehl D, Todorova K, Schönemann C, Allenbach Y, Aronica E, de Visser M, Heppner FL, Weis J, Doostkam S, Maisonobe T, Benveniste O, Goebel HH, Stenzel W. Architectural B-cell organization in skeletal muscle identifies subtypes of dermatomyositis. Neurol Neuroimmunol Neuroinflamm. 2018 Mar 06;5(3):e451. doi: 10.1212/NXI.0000000000000451. eCollection 2018 May. PMID: 29520367; PMCID: PMC5840889.
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Autoimmune Thyroiditis and Myasthenia Gravis.

Frontiers in endocrinology

Lopomo A, Berrih-Aknin S.
PMID: 28751878
Front Endocrinol (Lausanne). 2017 Jul 13;8:169. doi: 10.3389/fendo.2017.00169. eCollection 2017.

Autoimmune diseases (AIDs) are the result of specific immune responses directed against structures of the self. In normal conditions, the molecules recognized as "self" are tolerated by immune system, but when the self-tolerance is lost, the immune system could...

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Lopomo A, Berrih-Aknin S. Autoimmune Thyroiditis and Myasthenia Gravis. Front Endocrinol (Lausanne). 2017;8:169doi: 10.3389/fendo.2017.00169.
Lopomo, A., & Berrih-Aknin, S. (2017). Autoimmune Thyroiditis and Myasthenia Gravis. Frontiers in endocrinology, 8169. https://doi.org/10.3389/fendo.2017.00169
Lopomo, Angela, and Berrih-Aknin, Sonia. "Autoimmune Thyroiditis and Myasthenia Gravis." Frontiers in endocrinology vol. 8 (2017): 169. doi: https://doi.org/10.3389/fendo.2017.00169
Lopomo A, Berrih-Aknin S. Autoimmune Thyroiditis and Myasthenia Gravis. Front Endocrinol (Lausanne). 2017 Jul 13;8:169. doi: 10.3389/fendo.2017.00169. eCollection 2017. PMID: 28751878; PMCID: PMC5508005.
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TREX1 is a checkpoint for innate immune sensing of DNA damage that fosters cancer immune resistance.

Emerging topics in life sciences

Demaria S, Vanpouille-Box C.
PMID: 33525799
Emerg Top Life Sci. 2017 Dec 12;1(5):509-515. doi: 10.1042/ETLS20170063.

Genomic instability is a hallmark of neoplastic transformation that leads to the accumulation of mutations, and generates a state of replicative stress in neoplastic cells associated with dysregulated DNA damage repair (DDR) responses. The importance of increasing mutations in...

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Demaria S, Vanpouille-Box C. TREX1 is a checkpoint for innate immune sensing of DNA damage that fosters cancer immune resistance. Emerg Top Life Sci. 2017;1(5):509-515doi: 10.1042/ETLS20170063.
Demaria, S., & Vanpouille-Box, C. (2017). TREX1 is a checkpoint for innate immune sensing of DNA damage that fosters cancer immune resistance. Emerging topics in life sciences, 1(5), 509-515. https://doi.org/10.1042/ETLS20170063
Demaria, Sandra, and Vanpouille-Box, Claire. "TREX1 is a checkpoint for innate immune sensing of DNA damage that fosters cancer immune resistance." Emerging topics in life sciences vol. 1,5 (2017): 509-515. doi: https://doi.org/10.1042/ETLS20170063
Demaria S, Vanpouille-Box C. TREX1 is a checkpoint for innate immune sensing of DNA damage that fosters cancer immune resistance. Emerg Top Life Sci. 2017 Dec 12;1(5):509-515. doi: 10.1042/ETLS20170063. PMID: 33525799.
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Systems Immunology Analyses Following Porcine Respiratory and Reproductive Syndrome Virus Infection and Vaccination.

Frontiers in immunology

Bocard LV, Kick AR, Hug C, Lischer HEL, Käser T, Summerfield A.
PMID: 34975868
Front Immunol. 2021 Dec 16;12:779747. doi: 10.3389/fimmu.2021.779747. eCollection 2021.

This study was initiated to better understand the nature of innate immune responses and the relatively weak and delayed immune response against porcine reproductive and respiratory syndrome virus (PRRSV). Following modified live virus (MLV) vaccination or infection with two...

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Bocard LV, Kick AR, Hug C, et al. Systems Immunology Analyses Following Porcine Respiratory and Reproductive Syndrome Virus Infection and Vaccination. Front Immunol. 2021;12:779747doi: 10.3389/fimmu.2021.779747.
Bocard, L. V., Kick, A. R., Hug, C., Lischer, H. E. L., Käser, T., & Summerfield, A. (2021). Systems Immunology Analyses Following Porcine Respiratory and Reproductive Syndrome Virus Infection and Vaccination. Frontiers in immunology, 12779747. https://doi.org/10.3389/fimmu.2021.779747
Bocard, Loïc Vivien, et al. "Systems Immunology Analyses Following Porcine Respiratory and Reproductive Syndrome Virus Infection and Vaccination." Frontiers in immunology vol. 12 (2021): 779747. doi: https://doi.org/10.3389/fimmu.2021.779747
Bocard LV, Kick AR, Hug C, Lischer HEL, Käser T, Summerfield A. Systems Immunology Analyses Following Porcine Respiratory and Reproductive Syndrome Virus Infection and Vaccination. Front Immunol. 2021 Dec 16;12:779747. doi: 10.3389/fimmu.2021.779747. eCollection 2021. PMID: 34975868; PMCID: PMC8716554.
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