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Mechanisms of lipase maturation.

Clinical lipidology

Doolittle MH, Péterfy M.
PMID: 20543905
Clin Lipidol. 2010 Feb 01;5(1):71-85. doi: 10.2217/clp.09.8.

Lipases are acyl hydrolases that represent a diverse group of enzymes present in organisms ranging from prokaryotes to humans. This article focuses on an evolutionarily related family of extracellular lipases that include lipoprotein lipase, hepatic lipase and endothelial lipase....

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Doolittle MH, Péterfy M. Mechanisms of lipase maturation. Clin Lipidol. 2010;5(1):71-85doi: 10.2217/clp.09.8.
Doolittle, M. H., & Péterfy, M. (2010). Mechanisms of lipase maturation. Clinical lipidology, 5(1), 71-85. https://doi.org/10.2217/clp.09.8
Doolittle, Mark H, and Péterfy, Miklós. "Mechanisms of lipase maturation." Clinical lipidology vol. 5,1 (2010): 71-85. doi: https://doi.org/10.2217/clp.09.8
Doolittle MH, Péterfy M. Mechanisms of lipase maturation. Clin Lipidol. 2010 Feb 01;5(1):71-85. doi: 10.2217/clp.09.8. PMID: 20543905; PMCID: PMC2883275.
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Tumor necrosis factor, cytokines, and the hyperlipidemia of infection.

Trends in endocrinology and metabolism: TEM

Grunfeld C, Feingold KR.
PMID: 18411185
Trends Endocrinol Metab. 1991 Nov-Dec;2(6):213-9. doi: 10.1016/1043-2760(91)90027-k.

The hypertriglyceridemia of infection is produced by many of the cytokines that regulate the immune response, including the tumor necrosis factors, interleukin-1, interleukin-6, and the interferons. In rodents, this hypertriglyceridemia is primarily due to the ability of these cytokines...

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Grunfeld C, Feingold KR. Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Trends Endocrinol Metab. 1991;2(6):213-9doi: 10.1016/1043-2760(91)90027-k.
Grunfeld, C., & Feingold, K. R. (1991). Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Trends in endocrinology and metabolism: TEM, 2(6), 213-9. https://doi.org/10.1016/1043-2760(91)90027-k
Grunfeld, C, and Feingold, K R. "Tumor necrosis factor, cytokines, and the hyperlipidemia of infection." Trends in endocrinology and metabolism: TEM vol. 2,6 (1991): 213-9. doi: https://doi.org/10.1016/1043-2760(91)90027-k
Grunfeld C, Feingold KR. Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Trends Endocrinol Metab. 1991 Nov-Dec;2(6):213-9. doi: 10.1016/1043-2760(91)90027-k. PMID: 18411185.
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Resistance to diet-induced adiposity in cannabinoid receptor-1 deficient mice is not due to impaired adipocyte function.

Nutrition & metabolism

Oosterveer MH, Koolman AH, de Boer PT, Bos T, Bleeker A, van Dijk TH, Bloks VW, Kuipers F, Sauer PJ, van Dijk G.
PMID: 22201701
Nutr Metab (Lond). 2011 Dec 27;8:93. doi: 10.1186/1743-7075-8-93.

BACKGROUND: Overactivity and/or dysregulation of the endocannabinoid system (ECS) contribute to development of obesity. In vitro studies indicate a regulatory role for the cannabinoid receptor 1 (CB1) in adipocyte function and CB1-receptor deficient (CB1-/-) mice are resistant to high...

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Oosterveer MH, Koolman AH, de Boer PT, et al. Resistance to diet-induced adiposity in cannabinoid receptor-1 deficient mice is not due to impaired adipocyte function. Nutr Metab (Lond). 2011;8:93doi: 10.1186/1743-7075-8-93.
Oosterveer, M. H., Koolman, A. H., de Boer, P. T., Bos, T., Bleeker, A., van Dijk, T. H., Bloks, V. W., Kuipers, F., Sauer, P. J., & van Dijk, G. (2011). Resistance to diet-induced adiposity in cannabinoid receptor-1 deficient mice is not due to impaired adipocyte function. Nutrition & metabolism, 893. https://doi.org/10.1186/1743-7075-8-93
Oosterveer, Maaike H, et al. "Resistance to diet-induced adiposity in cannabinoid receptor-1 deficient mice is not due to impaired adipocyte function." Nutrition & metabolism vol. 8 (2011): 93. doi: https://doi.org/10.1186/1743-7075-8-93
Oosterveer MH, Koolman AH, de Boer PT, Bos T, Bleeker A, van Dijk TH, Bloks VW, Kuipers F, Sauer PJ, van Dijk G. Resistance to diet-induced adiposity in cannabinoid receptor-1 deficient mice is not due to impaired adipocyte function. Nutr Metab (Lond). 2011 Dec 27;8:93. doi: 10.1186/1743-7075-8-93. PMID: 22201701; PMCID: PMC3307495.
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Antihyperlipidemic action of a Traditional Chinese Medicine (kampo medicine), Ogi-Keishi-Gomotsu-To-Ka-Kojin.

Phytomedicine : international journal of phytotherapy and phytopharmacology

Wu CZ, Inoue M, Ogihara Y.
PMID: 23195577
Phytomedicine. 1997 Dec;4(4):295-300. doi: 10.1016/S0944-7113(97)80036-0.

The antihyperlipidemic action of Ogi-Keishi-Gomotsu-To-Ka-Kojin (OKGK) was examined in rats with experimentally-induced hyperlipidemia. Oral administration of OKGK at 0.69 or 1.38 g/kg/day significantly reduced the increase of serum triglycerides (TG) and phospholipids (PL). OKGK given as a nutritional supplement...

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Wu CZ, Inoue M, Ogihara Y. Antihyperlipidemic action of a Traditional Chinese Medicine (kampo medicine), Ogi-Keishi-Gomotsu-To-Ka-Kojin. Phytomedicine. 1997;4(4):295-300doi: 10.1016/S0944-7113(97)80036-0.
Wu, C. Z., Inoue, M., & Ogihara, Y. (1997). Antihyperlipidemic action of a Traditional Chinese Medicine (kampo medicine), Ogi-Keishi-Gomotsu-To-Ka-Kojin. Phytomedicine : international journal of phytotherapy and phytopharmacology, 4(4), 295-300. https://doi.org/10.1016/S0944-7113(97)80036-0
Wu, C Z, et al. "Antihyperlipidemic action of a Traditional Chinese Medicine (kampo medicine), Ogi-Keishi-Gomotsu-To-Ka-Kojin." Phytomedicine : international journal of phytotherapy and phytopharmacology vol. 4,4 (1997): 295-300. doi: https://doi.org/10.1016/S0944-7113(97)80036-0
Wu CZ, Inoue M, Ogihara Y. Antihyperlipidemic action of a Traditional Chinese Medicine (kampo medicine), Ogi-Keishi-Gomotsu-To-Ka-Kojin. Phytomedicine. 1997 Dec;4(4):295-300. doi: 10.1016/S0944-7113(97)80036-0. PMID: 23195577.
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Do age and feeding levels have comparable effects on fat deposition in breast muscle of mule ducks?.

Animal : an international journal of animal bioscience

Chartrin P, Bernadet MD, Guy G, Mourot J, Hocquette JF, Rideau N, Duclos MJ, Baéza E.
PMID: 22444214
Animal. 2007 Feb;1(1):113-23. doi: 10.1017/S1751731107658029.

The effects of age (from 1 day post-hatch to 98 days of age) and feeding levels (feed restriction followed by overfeeding v. ad libitum feeding) on lipid deposition in breast muscle (quantity and quality, localisation) of mule ducks were...

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Chartrin P, Bernadet MD, Guy G, et al. Do age and feeding levels have comparable effects on fat deposition in breast muscle of mule ducks?. Animal. 2007;1(1):113-23doi: 10.1017/S1751731107658029.
Chartrin, P., Bernadet, M. D., Guy, G., Mourot, J., Hocquette, J. F., Rideau, N., Duclos, M. J., & Baéza, E. (2007). Do age and feeding levels have comparable effects on fat deposition in breast muscle of mule ducks?. Animal : an international journal of animal bioscience, 1(1), 113-23. https://doi.org/10.1017/S1751731107658029
Chartrin, P, et al. "Do age and feeding levels have comparable effects on fat deposition in breast muscle of mule ducks?." Animal : an international journal of animal bioscience vol. 1,1 (2007): 113-23. doi: https://doi.org/10.1017/S1751731107658029
Chartrin P, Bernadet MD, Guy G, Mourot J, Hocquette JF, Rideau N, Duclos MJ, Baéza E. Do age and feeding levels have comparable effects on fat deposition in breast muscle of mule ducks?. Animal. 2007 Feb;1(1):113-23. doi: 10.1017/S1751731107658029. PMID: 22444214.
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Once-a-day milking of Holstein and Montbéliarde cows for 7 weeks in mid-lactation.

Animal : an international journal of animal bioscience

Pomiès D, Martin B, Chilliard Y, Pradel P, Rémond B.
PMID: 22444923
Animal. 2007 Nov;1(10):1497-505. doi: 10.1017/S1751731107000778.

This study aimed at comparing the effects of once-a-day (OAD) milking during the descending phase of lactation between cows from the two most common breeds in France (Holstein and Montbéliarde). This study was carried on during two successive summers...

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Pomiès D, Martin B, Chilliard Y, et al. Once-a-day milking of Holstein and Montbéliarde cows for 7 weeks in mid-lactation. Animal. 2007;1(10):1497-505doi: 10.1017/S1751731107000778.
Pomiès, D., Martin, B., Chilliard, Y., Pradel, P., & Rémond, B. (2007). Once-a-day milking of Holstein and Montbéliarde cows for 7 weeks in mid-lactation. Animal : an international journal of animal bioscience, 1(10), 1497-505. https://doi.org/10.1017/S1751731107000778
Pomiès, D, et al. "Once-a-day milking of Holstein and Montbéliarde cows for 7 weeks in mid-lactation." Animal : an international journal of animal bioscience vol. 1,10 (2007): 1497-505. doi: https://doi.org/10.1017/S1751731107000778
Pomiès D, Martin B, Chilliard Y, Pradel P, Rémond B. Once-a-day milking of Holstein and Montbéliarde cows for 7 weeks in mid-lactation. Animal. 2007 Nov;1(10):1497-505. doi: 10.1017/S1751731107000778. PMID: 22444923.
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Proprotein convertases in high-density lipoprotein metabolism.

Biomarker research

Choi S, Korstanje R.
PMID: 24252756
Biomark Res. 2013 Sep 18;1(1):27. doi: 10.1186/2050-7771-1-27.

The proprotein convertase subtilisin/kexins (PCSKs) are a serine endopeptidase family. PCSK members cleave amino acid residues and modulate the activity of precursor proteins. Evidence from patients and animal models carrying genetic alterations in PCSK members show that PCSK members...

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Choi S, Korstanje R. Proprotein convertases in high-density lipoprotein metabolism. Biomark Res. 2013;1(1):27doi: 10.1186/2050-7771-1-27.
Choi, S., & Korstanje, R. (2013). Proprotein convertases in high-density lipoprotein metabolism. Biomarker research, 1(1), 27. https://doi.org/10.1186/2050-7771-1-27
Choi, Seungbum, and Korstanje, Ron. "Proprotein convertases in high-density lipoprotein metabolism." Biomarker research vol. 1,1 (2013): 27. doi: https://doi.org/10.1186/2050-7771-1-27
Choi S, Korstanje R. Proprotein convertases in high-density lipoprotein metabolism. Biomark Res. 2013 Sep 18;1(1):27. doi: 10.1186/2050-7771-1-27. PMID: 24252756; PMCID: PMC4177610.
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Thiazolidinedione induces a therapeutic effect on hepatosteatosis by regulating stearoyl-CoA desaturase-1, lipase activity, leptin and resistin.

Experimental and therapeutic medicine

Al-Muzafar HM, Amin KA.
PMID: 30214514
Exp Ther Med. 2018 Oct;16(4):2938-2948. doi: 10.3892/etm.2018.6563. Epub 2018 Aug 02.

Hepatosteatosis is a disease present worldwide, which presents a number of health problems. Recently, thiazolidinedione (TZD) has been used as a therapy for lipid disorders. The present study demonstrates the potential of TZD as a treatment for hepatosteatosis and...

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Al-Muzafar HM, Amin KA. Thiazolidinedione induces a therapeutic effect on hepatosteatosis by regulating stearoyl-CoA desaturase-1, lipase activity, leptin and resistin. Exp Ther Med. 2018;16(4):2938-2948doi: 10.3892/etm.2018.6563.
Al-Muzafar, H. M., & Amin, K. A. (2018). Thiazolidinedione induces a therapeutic effect on hepatosteatosis by regulating stearoyl-CoA desaturase-1, lipase activity, leptin and resistin. Experimental and therapeutic medicine, 16(4), 2938-2948. https://doi.org/10.3892/etm.2018.6563
Al-Muzafar, Hessah Mohammed, and Amin, Kamal Adel. "Thiazolidinedione induces a therapeutic effect on hepatosteatosis by regulating stearoyl-CoA desaturase-1, lipase activity, leptin and resistin." Experimental and therapeutic medicine vol. 16,4 (2018): 2938-2948. doi: https://doi.org/10.3892/etm.2018.6563
Al-Muzafar HM, Amin KA. Thiazolidinedione induces a therapeutic effect on hepatosteatosis by regulating stearoyl-CoA desaturase-1, lipase activity, leptin and resistin. Exp Ther Med. 2018 Oct;16(4):2938-2948. doi: 10.3892/etm.2018.6563. Epub 2018 Aug 02. PMID: 30214514; PMCID: PMC6125847.
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Stress during Lactation Induces Insulin Resistance Associated with an Increase in Type 1 Cannabinoid Receptors in Liver and Adipose Tissue.

Journal of nutrition and metabolism

Fonseca L, Castillo V, Aguirre C, Silva P, Ronco AM, Llanos M.
PMID: 30800481
J Nutr Metab. 2019 Jan 17;2019:2806519. doi: 10.1155/2019/2806519. eCollection 2019.

Several reports have shown that stress during lactation causes long-term metabolic and hormonal disruptions. In this study, we designed experiments to evaluate the effects of stress during lactation on the abundance of Type 1 cannabinoid/endocannabinoid receptors (CB1R) in epididymal...

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Fonseca L, Castillo V, Aguirre C, et al. Stress during Lactation Induces Insulin Resistance Associated with an Increase in Type 1 Cannabinoid Receptors in Liver and Adipose Tissue. J Nutr Metab. 2019;2019:2806519doi: 10.1155/2019/2806519.
Fonseca, L., Castillo, V., Aguirre, C., Silva, P., Ronco, A. M., & Llanos, M. (2019). Stress during Lactation Induces Insulin Resistance Associated with an Increase in Type 1 Cannabinoid Receptors in Liver and Adipose Tissue. Journal of nutrition and metabolism, 20192806519. https://doi.org/10.1155/2019/2806519
Fonseca, Liza, et al. "Stress during Lactation Induces Insulin Resistance Associated with an Increase in Type 1 Cannabinoid Receptors in Liver and Adipose Tissue." Journal of nutrition and metabolism vol. 2019 (2019): 2806519. doi: https://doi.org/10.1155/2019/2806519
Fonseca L, Castillo V, Aguirre C, Silva P, Ronco AM, Llanos M. Stress during Lactation Induces Insulin Resistance Associated with an Increase in Type 1 Cannabinoid Receptors in Liver and Adipose Tissue. J Nutr Metab. 2019 Jan 17;2019:2806519. doi: 10.1155/2019/2806519. eCollection 2019. PMID: 30800481; PMCID: PMC6360041.
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Relationship Between Angiopoietin-Like Protein 8 and Fasting Serum Triglyceride Level.

Journal of clinical medicine research

Yamada H, Kusaka I, Saikawa R, Hara K, Kakei M, Ishikawa SE.
PMID: 29317958
J Clin Med Res. 2018 Feb;10(2):134-136. doi: 10.14740/jocmr3286w. Epub 2017 Dec 30.

BACKGROUND: The aim of the study was to evaluate the correlation between angiopoietin-like protein 8 (ANGPTL8) and metabolic parameters in non-diabetic healthy humans.METHODS: We enrolled 30 healthy Japanese adults (25 men and five women). After 9 h of fasting,...

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Yamada H, Kusaka I, Saikawa R, et al. Relationship Between Angiopoietin-Like Protein 8 and Fasting Serum Triglyceride Level. J Clin Med Res. 2017;10(2):134-136doi: 10.14740/jocmr3286w.
Yamada, H., Kusaka, I., Saikawa, R., Hara, K., Kakei, M., & Ishikawa, S. E. (2018). Relationship Between Angiopoietin-Like Protein 8 and Fasting Serum Triglyceride Level. Journal of clinical medicine research, 10(2), 134-136. https://doi.org/10.14740/jocmr3286w
Yamada, Hodaka, et al. "Relationship Between Angiopoietin-Like Protein 8 and Fasting Serum Triglyceride Level." Journal of clinical medicine research vol. 10,2 (2018): 134-136. doi: https://doi.org/10.14740/jocmr3286w
Yamada H, Kusaka I, Saikawa R, Hara K, Kakei M, Ishikawa SE. Relationship Between Angiopoietin-Like Protein 8 and Fasting Serum Triglyceride Level. J Clin Med Res. 2018 Feb;10(2):134-136. doi: 10.14740/jocmr3286w. Epub 2017 Dec 30. PMID: 29317958; PMCID: PMC5755652.
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ANGPTL4 gene E40K variation protects against obesity-associated dyslipidemia in participants with obesity.

Obesity science & practice

Bailetti D, Bertoccini L, Mancina RM, Barchetta I, Capoccia D, Cossu E, Pujia A, Lenzi A, Leonetti F, Cavallo MG, Romeo S, Baroni MG.
PMID: 30820332
Obes Sci Pract. 2018 Dec 26;5(1):83-90. doi: 10.1002/osp4.311. eCollection 2019 Feb.

RESULTS: Participants with obesity carrying the E40K variant had significantly lower TG (CONCLUSIONS: In participants with obesity the

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Bailetti D, Bertoccini L, Mancina RM, et al. ANGPTL4 gene E40K variation protects against obesity-associated dyslipidemia in participants with obesity. Obes Sci Pract. 2018;5(1):83-90doi: 10.1002/osp4.311.
Bailetti, D., Bertoccini, L., Mancina, R. M., Barchetta, I., Capoccia, D., Cossu, E., Pujia, A., Lenzi, A., Leonetti, F., Cavallo, M. G., Romeo, S., & Baroni, M. G. (2019). ANGPTL4 gene E40K variation protects against obesity-associated dyslipidemia in participants with obesity. Obesity science & practice, 5(1), 83-90. https://doi.org/10.1002/osp4.311
Bailetti, D, et al. "ANGPTL4 gene E40K variation protects against obesity-associated dyslipidemia in participants with obesity." Obesity science & practice vol. 5,1 (2019): 83-90. doi: https://doi.org/10.1002/osp4.311
Bailetti D, Bertoccini L, Mancina RM, Barchetta I, Capoccia D, Cossu E, Pujia A, Lenzi A, Leonetti F, Cavallo MG, Romeo S, Baroni MG. ANGPTL4 gene E40K variation protects against obesity-associated dyslipidemia in participants with obesity. Obes Sci Pract. 2018 Dec 26;5(1):83-90. doi: 10.1002/osp4.311. eCollection 2019 Feb. PMID: 30820332; PMCID: PMC6381304.
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Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate.

Frontiers in cellular neuroscience

Zhang J, Li X, Ren Y, Zhao Y, Xing A, Jiang C, Chen Y, An L.
PMID: 29386999
Front Cell Neurosci. 2018 Jan 17;12:1. doi: 10.3389/fncel.2018.00001. eCollection 2018.

Intermittent fasting has been demonstrated to protect against Alzheimer's disease (AD), however, the mechanism is unclear. Histone acetylation and lipoprotein lipase (LPL) are involved in AD progression. Importantly, LPL has been documented to be regulated by histone deacetylases (HDACs)...

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Zhang J, Li X, Ren Y, et al. Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate. Front Cell Neurosci. 2018;12:1doi: 10.3389/fncel.2018.00001.
Zhang, J., Li, X., Ren, Y., Zhao, Y., Xing, A., Jiang, C., Chen, Y., & An, L. (2018). Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate. Frontiers in cellular neuroscience, 121. https://doi.org/10.3389/fncel.2018.00001
Zhang, Jingzhu, et al. "Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate." Frontiers in cellular neuroscience vol. 12 (2018): 1. doi: https://doi.org/10.3389/fncel.2018.00001
Zhang J, Li X, Ren Y, Zhao Y, Xing A, Jiang C, Chen Y, An L. Intermittent Fasting Alleviates the Increase of Lipoprotein Lipase Expression in Brain of a Mouse Model of Alzheimer's Disease: Possibly Mediated by β-hydroxybutyrate. Front Cell Neurosci. 2018 Jan 17;12:1. doi: 10.3389/fncel.2018.00001. eCollection 2018. PMID: 29386999; PMCID: PMC5776118.
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