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Showing 13 to 24 of 107 entries
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RETRACTION: Hypochlorous acid via peroxynitrite activates protein kinase Cθ and insulin resistance in adipocytes.

Journal of molecular endocrinology

Zhou J, Wang Q, Ding Y, Zou MH.
PMID: 33492241
J Mol Endocrinol. 2021 Feb;66(2):Z1. doi: 10.1530/JME-14-0213r.

No abstract available.

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Zhou J, Wang Q, Ding Y, et al. RETRACTION: Hypochlorous acid via peroxynitrite activates protein kinase Cθ and insulin resistance in adipocytes. J Mol Endocrinol. 2021;66(2):Z1doi: 10.1530/JME-14-0213r.
Zhou, J., Wang, Q., Ding, Y., & Zou, M. H. (2021). RETRACTION: Hypochlorous acid via peroxynitrite activates protein kinase Cθ and insulin resistance in adipocytes. Journal of molecular endocrinology, 66(2), Z1. https://doi.org/10.1530/JME-14-0213r
Zhou, Jun, et al. "RETRACTION: Hypochlorous acid via peroxynitrite activates protein kinase Cθ and insulin resistance in adipocytes." Journal of molecular endocrinology vol. 66,2 (2021): Z1. doi: https://doi.org/10.1530/JME-14-0213r
Zhou J, Wang Q, Ding Y, Zou MH. RETRACTION: Hypochlorous acid via peroxynitrite activates protein kinase Cθ and insulin resistance in adipocytes. J Mol Endocrinol. 2021 Feb;66(2):Z1. doi: 10.1530/JME-14-0213r. PMID: 33492241; PMCID: PMC8475555.
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ACOX1, regulated by C/EBPα and miR-25-3p, promotes bovine preadipocyte adipogenesis.

Journal of molecular endocrinology

Zhang F, Xiong Q, Tao H, Liu Y, Zhang N, Li XF, Suo XJ, Yang QP, Chen MX.
PMID: 33502338
J Mol Endocrinol. 2021 Mar;66(3):195-205. doi: 10.1530/JME-20-0250.

Acyl-coenzyme A oxidase 1 (ACOX1) is the first and rate-limiting enzyme in peroxisomal fatty acid β-oxidation of fatty acids. Previous studies have reported that ACOX1 was correlated with the meat quality of livestock, while the role of ACOX1 in...

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Zhang F, Xiong Q, Tao H, et al. ACOX1, regulated by C/EBPα and miR-25-3p, promotes bovine preadipocyte adipogenesis. J Mol Endocrinol. 2021;66(3):195-205doi: 10.1530/JME-20-0250.
Zhang, F., Xiong, Q., Tao, H., Liu, Y., Zhang, N., Li, X. F., Suo, X. J., Yang, Q. P., & Chen, M. X. (2021). ACOX1, regulated by C/EBPα and miR-25-3p, promotes bovine preadipocyte adipogenesis. Journal of molecular endocrinology, 66(3), 195-205. https://doi.org/10.1530/JME-20-0250
Zhang, Feng, et al. "ACOX1, regulated by C/EBPα and miR-25-3p, promotes bovine preadipocyte adipogenesis." Journal of molecular endocrinology vol. 66,3 (2021): 195-205. doi: https://doi.org/10.1530/JME-20-0250
Zhang F, Xiong Q, Tao H, Liu Y, Zhang N, Li XF, Suo XJ, Yang QP, Chen MX. ACOX1, regulated by C/EBPα and miR-25-3p, promotes bovine preadipocyte adipogenesis. J Mol Endocrinol. 2021 Mar;66(3):195-205. doi: 10.1530/JME-20-0250. PMID: 33502338; PMCID: PMC8052523.
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A20 controls expression of beta-cell regulatory genes and transcription factors.

Journal of molecular endocrinology

Ratajczak W, Atkinson SD, Kelly C.
PMID: 34463643
J Mol Endocrinol. 2021 Sep 28;67(4):189-201. doi: 10.1530/JME-21-0076.

TNFAIP3 encodes a zinc finger protein called A20, which has potent anti-inflammatory and anti-apoptotic properties. A20 promotes beta-cell survival and protects against islet graft rejection in experimental models. The current study sought to investigate the mechanisms underlying the protective...

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Ratajczak W, Atkinson SD, Kelly C. A20 controls expression of beta-cell regulatory genes and transcription factors. J Mol Endocrinol. 2021;67(4):189-201doi: 10.1530/JME-21-0076.
Ratajczak, W., Atkinson, S. D., & Kelly, C. (2021). A20 controls expression of beta-cell regulatory genes and transcription factors. Journal of molecular endocrinology, 67(4), 189-201. https://doi.org/10.1530/JME-21-0076
Ratajczak, Wiktoria, et al. "A20 controls expression of beta-cell regulatory genes and transcription factors." Journal of molecular endocrinology vol. 67,4 (2021): 189-201. doi: https://doi.org/10.1530/JME-21-0076
Ratajczak W, Atkinson SD, Kelly C. A20 controls expression of beta-cell regulatory genes and transcription factors. J Mol Endocrinol. 2021 Sep 28;67(4):189-201. doi: 10.1530/JME-21-0076. PMID: 34463643.
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Obesity enhances the recruitment of mesenchymal stem cells to visceral adipose tissue.

Journal of molecular endocrinology

de Assis-Ferreira A, Saldanha-Gama R, de Brito NM, Renovato-Martins M, Simões RL, Barja-Fidalgo C, Vargas da Silva S.
PMID: 34045365
J Mol Endocrinol. 2021 Jun 17;67(1):15-26. doi: 10.1530/JME-20-0229.

In obesity, high levels of TNF-α in the bone marrow microenvironment induce the bone marrow-mesenchymal stem cells (BM-MSCs) towards a pro-adipogenic phenotype. Here, we investigated the effect of obesity on the migratory potential of BM-MSCs and their fate towards...

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de Assis-Ferreira A, Saldanha-Gama R, de Brito NM, et al. Obesity enhances the recruitment of mesenchymal stem cells to visceral adipose tissue. J Mol Endocrinol. 2021;67(1):15-26doi: 10.1530/JME-20-0229.
de Assis-Ferreira, A., Saldanha-Gama, R., de Brito, N. M., Renovato-Martins, M., Simões, R. L., Barja-Fidalgo, C., & Vargas da Silva, S. (2021). Obesity enhances the recruitment of mesenchymal stem cells to visceral adipose tissue. Journal of molecular endocrinology, 67(1), 15-26. https://doi.org/10.1530/JME-20-0229
de Assis-Ferreira, Agatha, et al. "Obesity enhances the recruitment of mesenchymal stem cells to visceral adipose tissue." Journal of molecular endocrinology vol. 67,1 (2021): 15-26. doi: https://doi.org/10.1530/JME-20-0229
de Assis-Ferreira A, Saldanha-Gama R, de Brito NM, Renovato-Martins M, Simões RL, Barja-Fidalgo C, Vargas da Silva S. Obesity enhances the recruitment of mesenchymal stem cells to visceral adipose tissue. J Mol Endocrinol. 2021 Jun 17;67(1):15-26. doi: 10.1530/JME-20-0229. PMID: 34045365.
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Phytohemagglutinin ameliorates HFD-induced obesity by increasing energy expenditure.

Journal of molecular endocrinology

Zhang Y, Li J, Wang HH, Li J, Yu Y, Li B, Huang L, Wu C, Liu X.
PMID: 33983894
J Mol Endocrinol. 2021 Jun 17;67(1):1-14. doi: 10.1530/JME-20-0349.

Despite all modern advances in medicine, there are few reports of effective and safe drugs to treat obesity. Our objective was to screen anti-obesity natural compounds, and to verify whether they can reduce the body weight gain and investigate...

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Zhang Y, Li J, Wang HH, et al. Phytohemagglutinin ameliorates HFD-induced obesity by increasing energy expenditure. J Mol Endocrinol. 2021;67(1):1-14doi: 10.1530/JME-20-0349.
Zhang, Y., Li, J., Wang, H. H., Li, J., Yu, Y., Li, B., Huang, L., Wu, C., & Liu, X. (2021). Phytohemagglutinin ameliorates HFD-induced obesity by increasing energy expenditure. Journal of molecular endocrinology, 67(1), 1-14. https://doi.org/10.1530/JME-20-0349
Zhang, Yunxia, et al. "Phytohemagglutinin ameliorates HFD-induced obesity by increasing energy expenditure." Journal of molecular endocrinology vol. 67,1 (2021): 1-14. doi: https://doi.org/10.1530/JME-20-0349
Zhang Y, Li J, Wang HH, Li J, Yu Y, Li B, Huang L, Wu C, Liu X. Phytohemagglutinin ameliorates HFD-induced obesity by increasing energy expenditure. J Mol Endocrinol. 2021 Jun 17;67(1):1-14. doi: 10.1530/JME-20-0349. PMID: 33983894; PMCID: PMC8240727.
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Endocrine-Related Resources from the National Institutes of Health.

Molecular endocrinology (Baltimore, Md.)

[No authors listed]
PMID: 11145750
Mol Endocrinol. 2001 Jan 01;15(1):201-203. doi: 10.1210/mend.15.1.5856.

No abstract available.

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Endocrine-Related Resources from the National Institutes of Health. Mol Endocrinol. 2001;15(1):201-203doi: 10.1210/mend.15.1.5856.
(2001). Endocrine-Related Resources from the National Institutes of Health. Molecular endocrinology (Baltimore, Md.), 15(1), 201-203. https://doi.org/10.1210/mend.15.1.5856
"Endocrine-Related Resources from the National Institutes of Health." Molecular endocrinology (Baltimore, Md.) vol. 15,1 (2001): 201-203. doi: https://doi.org/10.1210/mend.15.1.5856
Endocrine-Related Resources from the National Institutes of Health. Mol Endocrinol. 2001 Jan 01;15(1):201-203. doi: 10.1210/mend.15.1.5856. PMID: 11145750.
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AGEs inhibit scavenger receptor class B type I gene expression via Smad1 in HUVECs.

Journal of molecular endocrinology

Nagata H, Lyu J, Imachi H, Fukunaga K, Sato S, Kobayashi T, Saheki T, Seo K, Salimah JB, Iwama H, Sakamoto R, Ogawa Y, Murao K.
PMID: 33638940
J Mol Endocrinol. 2021 Mar;66(3):223-231. doi: 10.1530/JME-20-0177.

Vascular complications are the main cause of morbidity and mortality in diabetic patients, and advanced glycation end products (AGEs) play a critical role in promoting diabetic vascular dysfunction. The human homolog of scavenger receptor class B type I (SR-BI),...

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Nagata H, Lyu J, Imachi H, et al. AGEs inhibit scavenger receptor class B type I gene expression via Smad1 in HUVECs. J Mol Endocrinol. 2021;66(3):223-231doi: 10.1530/JME-20-0177.
Nagata, H., Lyu, J., Imachi, H., Fukunaga, K., Sato, S., Kobayashi, T., Saheki, T., Seo, K., Salimah, J. B., Iwama, H., Sakamoto, R., Ogawa, Y., & Murao, K. (2021). AGEs inhibit scavenger receptor class B type I gene expression via Smad1 in HUVECs. Journal of molecular endocrinology, 66(3), 223-231. https://doi.org/10.1530/JME-20-0177
Nagata, Hiromi, et al. "AGEs inhibit scavenger receptor class B type I gene expression via Smad1 in HUVECs." Journal of molecular endocrinology vol. 66,3 (2021): 223-231. doi: https://doi.org/10.1530/JME-20-0177
Nagata H, Lyu J, Imachi H, Fukunaga K, Sato S, Kobayashi T, Saheki T, Seo K, Salimah JB, Iwama H, Sakamoto R, Ogawa Y, Murao K. AGEs inhibit scavenger receptor class B type I gene expression via Smad1 in HUVECs. J Mol Endocrinol. 2021 Mar;66(3):223-231. doi: 10.1530/JME-20-0177. PMID: 33638940.
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Dexamethasone induces pancreatic β-cell apoptosis through upregulation of TRAIL death receptor.

Journal of molecular endocrinology

Suksri K, Semprasert N, Junking M, Kutpruek S, Limjindaporn T, Yenchitsomanus PT, Kooptiwut S.
PMID: 34223821
J Mol Endocrinol. 2021 Aug 05;67(3):95-106. doi: 10.1530/JME-20-0238.

Long-term medication with dexamethasone - a synthetic glucocorticoid (GC) drug - results in hyperglycemia, or steroid-induced diabetes. Although recent studies revealed that dexamethasone directly induces pancreatic β-cell apoptosis, its molecular mechanisms remain unclear. In our initial analysis of mRNA...

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Suksri K, Semprasert N, Junking M, et al. Dexamethasone induces pancreatic β-cell apoptosis through upregulation of TRAIL death receptor. J Mol Endocrinol. 2021;67(3):95-106doi: 10.1530/JME-20-0238.
Suksri, K., Semprasert, N., Junking, M., Kutpruek, S., Limjindaporn, T., Yenchitsomanus, P. T., & Kooptiwut, S. (2021). Dexamethasone induces pancreatic β-cell apoptosis through upregulation of TRAIL death receptor. Journal of molecular endocrinology, 67(3), 95-106. https://doi.org/10.1530/JME-20-0238
Suksri, Kanchana, et al. "Dexamethasone induces pancreatic β-cell apoptosis through upregulation of TRAIL death receptor." Journal of molecular endocrinology vol. 67,3 (2021): 95-106. doi: https://doi.org/10.1530/JME-20-0238
Suksri K, Semprasert N, Junking M, Kutpruek S, Limjindaporn T, Yenchitsomanus PT, Kooptiwut S. Dexamethasone induces pancreatic β-cell apoptosis through upregulation of TRAIL death receptor. J Mol Endocrinol. 2021 Aug 05;67(3):95-106. doi: 10.1530/JME-20-0238. PMID: 34223821.
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20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor.

Journal of molecular endocrinology

Lafont R, Serova M, Didry-Barca B, Raynal S, Guibout L, Dinan L, Veillet S, Latil M, Dioh W, Dilda PJ.
PMID: 34825653
J Mol Endocrinol. 2021 Dec 23;68(2):77-87. doi: 10.1530/JME-21-0033.

20-Hydroxyecdysone (20E) is a steroid hormone that plays a key role in insect development through nuclear ecdysteroid receptors (EcR/RXR complex) and at least one membrane GPCR receptor (DopEcR). It also displays numerous pharmacological effects in mammals, where its mechanism...

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Lafont R, Serova M, Didry-Barca B, et al. 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. J Mol Endocrinol. 2021;68(2):77-87doi: 10.1530/JME-21-0033.
Lafont, R., Serova, M., Didry-Barca, B., Raynal, S., Guibout, L., Dinan, L., Veillet, S., Latil, M., Dioh, W., & Dilda, P. J. (2021). 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. Journal of molecular endocrinology, 68(2), 77-87. https://doi.org/10.1530/JME-21-0033
Lafont, René, et al. "20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor." Journal of molecular endocrinology vol. 68,2 (2021): 77-87. doi: https://doi.org/10.1530/JME-21-0033
Lafont R, Serova M, Didry-Barca B, Raynal S, Guibout L, Dinan L, Veillet S, Latil M, Dioh W, Dilda PJ. 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. J Mol Endocrinol. 2021 Dec 23;68(2):77-87. doi: 10.1530/JME-21-0033. PMID: 34825653.
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Bone morphogenetic protein 2 controls steroid-induced osteonecrosis of the femoral head via directly inhibiting interleukin-34 expression.

Journal of molecular endocrinology

Wang M, Min HS, Shan H, Lin Y, Xia W, Yin F, Jiang C, Yu X.
PMID: 34582356
J Mol Endocrinol. 2021 Oct 21;68(1):1-9. doi: 10.1530/JME-21-0163.

Increased inflammatory response is one of the major characteristics of osteonecrosis of the femoral head (ONFH). We aimed to investigate the function of bone morphogenetic protein 2 (BMP-2)/interleukin (IL)-34 axis in the inflammatory responses of ONFH. The systemic and...

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Wang M, Min HS, Shan H, et al. Bone morphogenetic protein 2 controls steroid-induced osteonecrosis of the femoral head via directly inhibiting interleukin-34 expression. J Mol Endocrinol. 2021;68(1):1-9doi: 10.1530/JME-21-0163.
Wang, M., Min, H. S., Shan, H., Lin, Y., Xia, W., Yin, F., Jiang, C., & Yu, X. (2021). Bone morphogenetic protein 2 controls steroid-induced osteonecrosis of the femoral head via directly inhibiting interleukin-34 expression. Journal of molecular endocrinology, 68(1), 1-9. https://doi.org/10.1530/JME-21-0163
Wang, Meng, et al. "Bone morphogenetic protein 2 controls steroid-induced osteonecrosis of the femoral head via directly inhibiting interleukin-34 expression." Journal of molecular endocrinology vol. 68,1 (2021): 1-9. doi: https://doi.org/10.1530/JME-21-0163
Wang M, Min HS, Shan H, Lin Y, Xia W, Yin F, Jiang C, Yu X. Bone morphogenetic protein 2 controls steroid-induced osteonecrosis of the femoral head via directly inhibiting interleukin-34 expression. J Mol Endocrinol. 2021 Oct 21;68(1):1-9. doi: 10.1530/JME-21-0163. PMID: 34582356.
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Defining lipid mediators of insulin resistance - controversies and challenges.

Journal of molecular endocrinology

Metcalfe LK, Smith GC, Turner N.
PMID: 30068522
J Mol Endocrinol. 2018 Aug 01; doi: 10.1530/JME-18-0023. Epub 2018 Aug 01.

Essential elements of all cells, lipids play important roles in energy production, signalling and as structural components. Despite these critical functions, excessive availability and intracellular accumulation of lipid is now recognised as a major factor contributing to many human...

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Metcalfe LK, Smith GC, Turner N. Defining lipid mediators of insulin resistance - controversies and challenges. J Mol Endocrinol. 2018;doi: 10.1530/JME-18-0023.
Metcalfe, L. K., Smith, G. C., & Turner, N. (2018). Defining lipid mediators of insulin resistance - controversies and challenges. Journal of molecular endocrinology, . https://doi.org/10.1530/JME-18-0023
Metcalfe, Louise K, et al. "Defining lipid mediators of insulin resistance - controversies and challenges." Journal of molecular endocrinology vol. (2018). doi: https://doi.org/10.1530/JME-18-0023
Metcalfe LK, Smith GC, Turner N. Defining lipid mediators of insulin resistance - controversies and challenges. J Mol Endocrinol. 2018 Aug 01; doi: 10.1530/JME-18-0023. Epub 2018 Aug 01. PMID: 30068522.
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Resting-Exercise Salivary Cortisol Responses: Detecting the Magnitude of Hormonal Change Over Time.

Journal of clinical and molecular endocrinology

Hackney AC, Anderson T.
PMID: 29652407
J Clin Mol Endocrinol. 2016;1(1). Epub 2016 Jan 08.

This study investigated the validity of salivary cortisol responses to reflect blood cortisol responses relative to the magnitude of change observed over time in the hormone. Male subjects (n=25) conducted four experimental sessions (ES) where blood (B) and saliva...

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Hackney AC, Anderson T. Resting-Exercise Salivary Cortisol Responses: Detecting the Magnitude of Hormonal Change Over Time. J Clin Mol Endocrinol. 2016;1(1)
Hackney, A. C., & Anderson, T. (2016). Resting-Exercise Salivary Cortisol Responses: Detecting the Magnitude of Hormonal Change Over Time. Journal of clinical and molecular endocrinology, 1(1), .
Hackney, Anthony C, and Anderson, Travis. "Resting-Exercise Salivary Cortisol Responses: Detecting the Magnitude of Hormonal Change Over Time." Journal of clinical and molecular endocrinology vol. 1,1 (2016).
Hackney AC, Anderson T. Resting-Exercise Salivary Cortisol Responses: Detecting the Magnitude of Hormonal Change Over Time. J Clin Mol Endocrinol. 2016;1(1). Epub 2016 Jan 08. PMID: 29652407; PMCID: PMC5892455.
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Showing 13 to 24 of 107 entries