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Showing 97 to 107 of 107 entries
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HDL promotes adiponectin gene expression via the CAMKK/CAMKIV pathway.

Journal of molecular endocrinology

Kobayashi T, Imachi H, Fukunaga K, Lyu J, Sato S, Saheki T, Ibata T, Matsumoto M, Japar SB, Murao K.
PMID: 34845988
J Mol Endocrinol. 2022 Jan 10;68(2):89-98. doi: 10.1530/JME-20-0211.

Adiponectin (APN) is an adipokine that protects against diabetes and atherosclerosis. High-density lipoprotein (HDL) mediates reverse cholesterol transport, which also protects against atherosclerosis. In this process, the human homolog of the B class type I scavenger receptor (SR-BI/CLA-1) facilitates...

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Kobayashi T, Imachi H, Fukunaga K, et al. HDL promotes adiponectin gene expression via the CAMKK/CAMKIV pathway. J Mol Endocrinol. 2022;68(2):89-98doi: 10.1530/JME-20-0211.
Kobayashi, T., Imachi, H., Fukunaga, K., Lyu, J., Sato, S., Saheki, T., Ibata, T., Matsumoto, M., Japar, S. B., & Murao, K. (2022). HDL promotes adiponectin gene expression via the CAMKK/CAMKIV pathway. Journal of molecular endocrinology, 68(2), 89-98. https://doi.org/10.1530/JME-20-0211
Kobayashi, Toshihiro, et al. "HDL promotes adiponectin gene expression via the CAMKK/CAMKIV pathway." Journal of molecular endocrinology vol. 68,2 (2022): 89-98. doi: https://doi.org/10.1530/JME-20-0211
Kobayashi T, Imachi H, Fukunaga K, Lyu J, Sato S, Saheki T, Ibata T, Matsumoto M, Japar SB, Murao K. HDL promotes adiponectin gene expression via the CAMKK/CAMKIV pathway. J Mol Endocrinol. 2022 Jan 10;68(2):89-98. doi: 10.1530/JME-20-0211. PMID: 34845988.
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Pathological changes and oxidative stress of the HPG axis in hypothyroid rat.

Journal of molecular endocrinology

Wang L, He W, Xu X, Qi L, Lv B, Qin J, Xue Z, Xue J.
PMID: 34270447
J Mol Endocrinol. 2021 Aug 11;67(3):107-119. doi: 10.1530/JME-21-0095.

Hypothyroidism is a common endocrine disease caused by a deficiency of thyroid hormones, which could affect the hypothalamus-pituitary-gonadal (HPG) axis and cause additional severe fertility problems. However, the pathogenesis of abnormal reproductive capacity caused by hypothyroidism and whether there...

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Wang L, He W, Xu X, et al. Pathological changes and oxidative stress of the HPG axis in hypothyroid rat. J Mol Endocrinol. 2021;67(3):107-119doi: 10.1530/JME-21-0095.
Wang, L., He, W., Xu, X., Qi, L., Lv, B., Qin, J., Xue, Z., & Xue, J. (2021). Pathological changes and oxidative stress of the HPG axis in hypothyroid rat. Journal of molecular endocrinology, 67(3), 107-119. https://doi.org/10.1530/JME-21-0095
Wang, Lu, et al. "Pathological changes and oxidative stress of the HPG axis in hypothyroid rat." Journal of molecular endocrinology vol. 67,3 (2021): 107-119. doi: https://doi.org/10.1530/JME-21-0095
Wang L, He W, Xu X, Qi L, Lv B, Qin J, Xue Z, Xue J. Pathological changes and oxidative stress of the HPG axis in hypothyroid rat. J Mol Endocrinol. 2021 Aug 11;67(3):107-119. doi: 10.1530/JME-21-0095. PMID: 34270447.
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Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β.

Journal of molecular endocrinology

Jin Z, Xia F, Dong J, Lin T, Cai Y, Chen J, Chen X, Huang Z, Wang Q, Chen H, Zhang J.
PMID: 33739937
J Mol Endocrinol. 2021 Apr 22;66(4):273-283. doi: 10.1530/JME-20-0236.

Glucocorticoid excess often causes a variety of cardiovascular complications, including hypertension, atherosclerosis, and cardiac hypertrophy. To abrogate its cardiac side effects, it is necessary to fully disclose the pathophysiological role of glucocorticoid in cardiac remodelling. Previous clinical and experimental...

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Jin Z, Xia F, Dong J, et al. Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β. J Mol Endocrinol. 2021;66(4):273-283doi: 10.1530/JME-20-0236.
Jin, Z., Xia, F., Dong, J., Lin, T., Cai, Y., Chen, J., Chen, X., Huang, Z., Wang, Q., Chen, H., & Zhang, J. (2021). Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β. Journal of molecular endocrinology, 66(4), 273-283. https://doi.org/10.1530/JME-20-0236
Jin, Zhousheng, et al. "Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β." Journal of molecular endocrinology vol. 66,4 (2021): 273-283. doi: https://doi.org/10.1530/JME-20-0236
Jin Z, Xia F, Dong J, Lin T, Cai Y, Chen J, Chen X, Huang Z, Wang Q, Chen H, Zhang J. Omentin-1 attenuates glucocorticoid-induced cardiac injury by phosphorylating GSK3β. J Mol Endocrinol. 2021 Apr 22;66(4):273-283. doi: 10.1530/JME-20-0236. PMID: 33739937.
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FGF23-related hypophosphatemic rickets/osteomalacia: diagnosis and new treatment.

Journal of molecular endocrinology

Fukumoto S.
PMID: 33295878
J Mol Endocrinol. 2021 Feb;66(2):R57-R65. doi: 10.1530/JME-20-0089.

FGF23 is a phosphaturic hormone produced by bone. FGF23 reduces serum phosphate by suppressing proximal tubular phosphate reabsorption and intestinal phosphate absorption. After the identification of FGF23, several kinds of hypophosphatemic rickets/osteomalacia such as X-linked hypophosphatemia (XLH) and tumor-induced...

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Fukumoto S. FGF23-related hypophosphatemic rickets/osteomalacia: diagnosis and new treatment. J Mol Endocrinol. 2021;66(2):R57-R65doi: 10.1530/JME-20-0089.
Fukumoto, S. (2021). FGF23-related hypophosphatemic rickets/osteomalacia: diagnosis and new treatment. Journal of molecular endocrinology, 66(2), R57-R65. https://doi.org/10.1530/JME-20-0089
Fukumoto, Seiji. "FGF23-related hypophosphatemic rickets/osteomalacia: diagnosis and new treatment." Journal of molecular endocrinology vol. 66,2 (2021): R57-R65. doi: https://doi.org/10.1530/JME-20-0089
Fukumoto S. FGF23-related hypophosphatemic rickets/osteomalacia: diagnosis and new treatment. J Mol Endocrinol. 2021 Feb;66(2):R57-R65. doi: 10.1530/JME-20-0089. PMID: 33295878.
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Graves' disease: introducing new genetic and epigenetic contributors.

Journal of molecular endocrinology

Razmara E, Salehi M, Aslani S, Bitaraf A, Yousefi H, Colón JR, Mahmoudi M.
PMID: 33295879
J Mol Endocrinol. 2021 Feb;66(2):R33-R55. doi: 10.1530/JME-20-0078.

Autoimmune thyroid disease (AITD) accounts for 90% of all thyroid diseases and affects 2-5% of the population with remarkable familial clustering. Among AITDs, Graves' disease (GD) is a complex disease affecting thyroid function. Over the last two decades, case-control...

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Razmara E, Salehi M, Aslani S, et al. Graves' disease: introducing new genetic and epigenetic contributors. J Mol Endocrinol. 2021;66(2):R33-R55doi: 10.1530/JME-20-0078.
Razmara, E., Salehi, M., Aslani, S., Bitaraf, A., Yousefi, H., Colón, J. R., & Mahmoudi, M. (2021). Graves' disease: introducing new genetic and epigenetic contributors. Journal of molecular endocrinology, 66(2), R33-R55. https://doi.org/10.1530/JME-20-0078
Razmara, Ehsan, et al. "Graves' disease: introducing new genetic and epigenetic contributors." Journal of molecular endocrinology vol. 66,2 (2021): R33-R55. doi: https://doi.org/10.1530/JME-20-0078
Razmara E, Salehi M, Aslani S, Bitaraf A, Yousefi H, Colón JR, Mahmoudi M. Graves' disease: introducing new genetic and epigenetic contributors. J Mol Endocrinol. 2021 Feb;66(2):R33-R55. doi: 10.1530/JME-20-0078. PMID: 33295879.
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20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor.

Journal of molecular endocrinology

Lafont R, Serova M, Didry-Barca B, Raynal S, Guibout L, Dinan L, Veillet S, Latil M, Dioh W, Dilda PJ.
PMID: 34825653
J Mol Endocrinol. 2021 Dec 23;68(2):77-87. doi: 10.1530/JME-21-0033.

20-Hydroxyecdysone (20E) is a steroid hormone that plays a key role in insect development through nuclear ecdysteroid receptors (EcR/RXR complex) and at least one membrane GPCR receptor (DopEcR). It also displays numerous pharmacological effects in mammals, where its mechanism...

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Lafont R, Serova M, Didry-Barca B, et al. 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. J Mol Endocrinol. 2021;68(2):77-87doi: 10.1530/JME-21-0033.
Lafont, R., Serova, M., Didry-Barca, B., Raynal, S., Guibout, L., Dinan, L., Veillet, S., Latil, M., Dioh, W., & Dilda, P. J. (2021). 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. Journal of molecular endocrinology, 68(2), 77-87. https://doi.org/10.1530/JME-21-0033
Lafont, René, et al. "20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor." Journal of molecular endocrinology vol. 68,2 (2021): 77-87. doi: https://doi.org/10.1530/JME-21-0033
Lafont R, Serova M, Didry-Barca B, Raynal S, Guibout L, Dinan L, Veillet S, Latil M, Dioh W, Dilda PJ. 20-Hydroxyecdysone activates the protective arm of the RAAS via the MAS receptor. J Mol Endocrinol. 2021 Dec 23;68(2):77-87. doi: 10.1530/JME-21-0033. PMID: 34825653.
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GCgx: transcriptome-wide exploration of the response to glucocorticoids.

Journal of molecular endocrinology

Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM.
PMID: 34787097
J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107.

Glucocorticoids are the cornerstone of immunosuppressive and anti-inflammatory therapy in humans, yet the mechanisms of glucocorticoid immunoregulation and toxicity remain unclear. The response to glucocorticoids is highly cell type-dependent, so translating results from different experimental systems into a better...

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Cao Q, Boo Irizarry Y, Yazhuk S, et al. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021;68(2):B1-B4doi: 10.1530/JME-21-0107.
Cao, Q., Boo Irizarry, Y., Yazhuk, S., Tran, T., Gadkari, M., & Franco, L. M. (2021). GCgx: transcriptome-wide exploration of the response to glucocorticoids. Journal of molecular endocrinology, 68(2), B1-B4. https://doi.org/10.1530/JME-21-0107
Cao, Qilin, et al. "GCgx: transcriptome-wide exploration of the response to glucocorticoids." Journal of molecular endocrinology vol. 68,2 (2021): B1-B4. doi: https://doi.org/10.1530/JME-21-0107
Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107. PMID: 34787097; PMCID: PMC8691098.
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MicroRNA-21 enhances estradiol production by inhibiting WT1 expression in granulosa cells.

Journal of molecular endocrinology

Hilker RE, Pan B, Zhan X, Li J.
PMID: 34665763
J Mol Endocrinol. 2021 Nov 12;68(1):11-22. doi: 10.1530/JME-21-0162.

In antral follicles, the transition of proliferative granulosa cells to estradiol-producing is critical for proper oocyte maturation. MicroRNAs are noncoding RNAs that play important roles in ovarian follicular development; however, this has yet to be fully characterized. MicroRNA-21 is...

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Hilker RE, Pan B, Zhan X, et al. MicroRNA-21 enhances estradiol production by inhibiting WT1 expression in granulosa cells. J Mol Endocrinol. 2021;68(1):11-22doi: 10.1530/JME-21-0162.
Hilker, R. E., Pan, B., Zhan, X., & Li, J. (2021). MicroRNA-21 enhances estradiol production by inhibiting WT1 expression in granulosa cells. Journal of molecular endocrinology, 68(1), 11-22. https://doi.org/10.1530/JME-21-0162
Hilker, Renée Emily, et al. "MicroRNA-21 enhances estradiol production by inhibiting WT1 expression in granulosa cells." Journal of molecular endocrinology vol. 68,1 (2021): 11-22. doi: https://doi.org/10.1530/JME-21-0162
Hilker RE, Pan B, Zhan X, Li J. MicroRNA-21 enhances estradiol production by inhibiting WT1 expression in granulosa cells. J Mol Endocrinol. 2021 Nov 12;68(1):11-22. doi: 10.1530/JME-21-0162. PMID: 34665763.
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Age-dependent response to T4 overtreatment and recovery on systemic and organ level.

Journal of molecular endocrinology

Kerp H, Renko K, Hönes GS, Brix K, Köhrle J, Moeller LC, Führer D.
PMID: 34370696
J Mol Endocrinol. 2021 Sep 09;67(4):161-172. doi: 10.1530/JME-21-0109.

Thyroid hormone (TH) metabolism and cellular TH action are influenced by ageing. To investigate the response to thyroxine (T4) overtreatment, a kinetic study was conducted in young and aged mice with chronic hyperthyroidism and hormone withdrawal. Five and 22...

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Kerp H, Renko K, Hönes GS, et al. Age-dependent response to T4 overtreatment and recovery on systemic and organ level. J Mol Endocrinol. 2021;67(4):161-172doi: 10.1530/JME-21-0109.
Kerp, H., Renko, K., Hönes, G. S., Brix, K., Köhrle, J., Moeller, L. C., & Führer, D. (2021). Age-dependent response to T4 overtreatment and recovery on systemic and organ level. Journal of molecular endocrinology, 67(4), 161-172. https://doi.org/10.1530/JME-21-0109
Kerp, Helena, et al. "Age-dependent response to T4 overtreatment and recovery on systemic and organ level." Journal of molecular endocrinology vol. 67,4 (2021): 161-172. doi: https://doi.org/10.1530/JME-21-0109
Kerp H, Renko K, Hönes GS, Brix K, Köhrle J, Moeller LC, Führer D. Age-dependent response to T4 overtreatment and recovery on systemic and organ level. J Mol Endocrinol. 2021 Sep 09;67(4):161-172. doi: 10.1530/JME-21-0109. PMID: 34370696.
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GCgx: transcriptome-wide exploration of the response to glucocorticoids.

Journal of molecular endocrinology

Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM.
PMID: 34787097
J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107.

Glucocorticoids are the cornerstone of immunosuppressive and anti-inflammatory therapy in humans, yet the mechanisms of glucocorticoid immunoregulation and toxicity remain unclear. The response to glucocorticoids is highly cell type-dependent, so translating results from different experimental systems into a better...

Cite
Cao Q, Boo Irizarry Y, Yazhuk S, et al. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021;68(2):B1-B4doi: 10.1530/JME-21-0107.
Cao, Q., Boo Irizarry, Y., Yazhuk, S., Tran, T., Gadkari, M., & Franco, L. M. (2021). GCgx: transcriptome-wide exploration of the response to glucocorticoids. Journal of molecular endocrinology, 68(2), B1-B4. https://doi.org/10.1530/JME-21-0107
Cao, Qilin, et al. "GCgx: transcriptome-wide exploration of the response to glucocorticoids." Journal of molecular endocrinology vol. 68,2 (2021): B1-B4. doi: https://doi.org/10.1530/JME-21-0107
Cao Q, Boo Irizarry Y, Yazhuk S, Tran T, Gadkari M, Franco LM. GCgx: transcriptome-wide exploration of the response to glucocorticoids. J Mol Endocrinol. 2021 Dec 10;68(2):B1-B4. doi: 10.1530/JME-21-0107. PMID: 34787097.
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25 years of ERβ: a personal journey.

Journal of molecular endocrinology

Warner M, Fan X, Strom A, Wu W, Gustafsson JÅ.
PMID: 34546964
J Mol Endocrinol. 2021 Oct 15;68(1):R1-R9. doi: 10.1530/JME-21-0121.

SUMMARY: After the discovery of ERβ, a novel role for dihydrotestosterone (DHT) in estrogen signaling was revealed. Instead of just being a better androgen, DHT was found to be a precursor of the ERβ agonist 5α-androstane-3β, 17β-diol (3βAdiol), an...

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Warner M, Fan X, Strom A, et al. 25 years of ERβ: a personal journey. J Mol Endocrinol. 2021;68(1):R1-R9doi: 10.1530/JME-21-0121.
Warner, M., Fan, X., Strom, A., Wu, W., & Gustafsson, J. �. �. (2021). 25 years of ERβ: a personal journey. Journal of molecular endocrinology, 68(1), R1-R9. https://doi.org/10.1530/JME-21-0121
Warner, Margaret, et al. "25 years of ERβ: a personal journey." Journal of molecular endocrinology vol. 68,1 (2021): R1-R9. doi: https://doi.org/10.1530/JME-21-0121
Warner M, Fan X, Strom A, Wu W, Gustafsson JÅ. 25 years of ERβ: a personal journey. J Mol Endocrinol. 2021 Oct 15;68(1):R1-R9. doi: 10.1530/JME-21-0121. PMID: 34546964.
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Showing 97 to 107 of 107 entries