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Investigational drugs for eating disorders.

Expert opinion on investigational drugs

Crow S.
PMID: 15989610
Expert Opin Investig Drugs. 1997 Apr;6(4):427-36. doi: 10.1517/13543784.6.4.427.

Anorexia nervosa (AN), bulimia nervosa (BN) and binge eating disorder (BED) each occur in about 0.5-1.5% of the population in westernised countries, and primarily affect women. At present, a variety of pharmacological treatments are used in addition to psychotherapy,...

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Crow S. Investigational drugs for eating disorders. Expert Opin Investig Drugs. 1997;6(4):427-36doi: 10.1517/13543784.6.4.427.
Crow, S. (1997). Investigational drugs for eating disorders. Expert opinion on investigational drugs, 6(4), 427-36. https://doi.org/10.1517/13543784.6.4.427
Crow, S. "Investigational drugs for eating disorders." Expert opinion on investigational drugs vol. 6,4 (1997): 427-36. doi: https://doi.org/10.1517/13543784.6.4.427
Crow S. Investigational drugs for eating disorders. Expert Opin Investig Drugs. 1997 Apr;6(4):427-36. doi: 10.1517/13543784.6.4.427. PMID: 15989610.
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Role of Glucocorticoids in Fasting-induced Changes in Hypothalamic and Pituitary Components of the Growth Hormone (GH)-axis.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Kim E, Seo S, Chung H, Park S.
PMID: 19967059
Korean J Physiol Pharmacol. 2008 Oct;12(5):217-23. doi: 10.4196/kjpp.2008.12.5.217. Epub 2008 Oct 31.

To directly test if elevated glucocorticoids are required for fasting-induced regulation of growth hormone (GH)-releasing hormone (GHRH), GHRH receptors (GHRH-R) and ghrelin receptors (GHS-R) expression, male rats were bilaterally adrenalectomized or sham operated. After 7 days, animals were fed...

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Kim E, Seo S, Chung H, et al. Role of Glucocorticoids in Fasting-induced Changes in Hypothalamic and Pituitary Components of the Growth Hormone (GH)-axis. Korean J Physiol Pharmacol. 2008;12(5):217-23doi: 10.4196/kjpp.2008.12.5.217.
Kim, E., Seo, S., Chung, H., & Park, S. (2008). Role of Glucocorticoids in Fasting-induced Changes in Hypothalamic and Pituitary Components of the Growth Hormone (GH)-axis. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 12(5), 217-23. https://doi.org/10.4196/kjpp.2008.12.5.217
Kim, Eunhee, et al. "Role of Glucocorticoids in Fasting-induced Changes in Hypothalamic and Pituitary Components of the Growth Hormone (GH)-axis." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 12,5 (2008): 217-23. doi: https://doi.org/10.4196/kjpp.2008.12.5.217
Kim E, Seo S, Chung H, Park S. Role of Glucocorticoids in Fasting-induced Changes in Hypothalamic and Pituitary Components of the Growth Hormone (GH)-axis. Korean J Physiol Pharmacol. 2008 Oct;12(5):217-23. doi: 10.4196/kjpp.2008.12.5.217. Epub 2008 Oct 31. PMID: 19967059; PMCID: PMC2788639.
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Cell and gene therapies for refractory epilepsy.

Current neuropharmacology

Boison D.
PMID: 18615179
Curr Neuropharmacol. 2007;5(2):115-25. doi: 10.2174/157015907780866938.

Despite recent advances in the development of antiepileptic drugs, refractory epilepsy remains a major clinical problem affecting up to 35% of patients with partial epilepsy. Currently, there are few therapies that affect the underlying disease process. Therefore, novel therapeutic...

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Boison D. Cell and gene therapies for refractory epilepsy. Curr Neuropharmacol. 2007;5(2):115-25doi: 10.2174/157015907780866938.
Boison, D. (2007). Cell and gene therapies for refractory epilepsy. Current neuropharmacology, 5(2), 115-25. https://doi.org/10.2174/157015907780866938
Boison, Detlev. "Cell and gene therapies for refractory epilepsy." Current neuropharmacology vol. 5,2 (2007): 115-25. doi: https://doi.org/10.2174/157015907780866938
Boison D. Cell and gene therapies for refractory epilepsy. Curr Neuropharmacol. 2007;5(2):115-25. doi: 10.2174/157015907780866938. PMID: 18615179; PMCID: PMC2435346.
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Differential regulation of neuropeptide Y and catecholamine production in superior cervical ganglion cultures.

Molecular and cellular neurosciences

Marek KL, Mains RE.
PMID: 19912776
Mol Cell Neurosci. 1990 Dec;1(3):262-9. doi: 10.1016/1044-7431(90)90008-r.

Primary cultures of newborn rat superior cervical ganglion (SCG) neurons grown in complete serum-free medium have been used to study the regulation of catecholamine and neuropeptide Y (NPY) production. The levels of mRNA for preproNPY and for tyrosine hydroxylase...

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Marek KL, Mains RE. Differential regulation of neuropeptide Y and catecholamine production in superior cervical ganglion cultures. Mol Cell Neurosci. 1990;1(3):262-9doi: 10.1016/1044-7431(90)90008-r.
Marek, K. L., & Mains, R. E. (1990). Differential regulation of neuropeptide Y and catecholamine production in superior cervical ganglion cultures. Molecular and cellular neurosciences, 1(3), 262-9. https://doi.org/10.1016/1044-7431(90)90008-r
Marek, K L, and Mains, R E. "Differential regulation of neuropeptide Y and catecholamine production in superior cervical ganglion cultures." Molecular and cellular neurosciences vol. 1,3 (1990): 262-9. doi: https://doi.org/10.1016/1044-7431(90)90008-r
Marek KL, Mains RE. Differential regulation of neuropeptide Y and catecholamine production in superior cervical ganglion cultures. Mol Cell Neurosci. 1990 Dec;1(3):262-9. doi: 10.1016/1044-7431(90)90008-r. PMID: 19912776.
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Differential and independent manifestation within co-containing neurones of neuropeptide Y and tyrosine hydroxylase during ontogeny of the rat central nervous system.

Neurochemistry international

Foster GA, Schultzberg M, Goldstein M.
PMID: 20488105
Neurochem Int. 1984;6(6):761-71. doi: 10.1016/0197-0186(84)90008-1.

The development of tyrosine hydroxylase- and neuropeptide Y-immunoreactive cell bodies in the foetal rat brain was analyzed immunohistochemically using antibodies raised against tyrosine hydroxylase and neuropeptide Y. Possible co-existence of these two substance within the same neurones was investigated...

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Foster GA, Schultzberg M, Goldstein M. Differential and independent manifestation within co-containing neurones of neuropeptide Y and tyrosine hydroxylase during ontogeny of the rat central nervous system. Neurochem Int. 1984;6(6):761-71doi: 10.1016/0197-0186(84)90008-1.
Foster, G. A., Schultzberg, M., & Goldstein, M. (1984). Differential and independent manifestation within co-containing neurones of neuropeptide Y and tyrosine hydroxylase during ontogeny of the rat central nervous system. Neurochemistry international, 6(6), 761-71. https://doi.org/10.1016/0197-0186(84)90008-1
Foster, G A, et al. "Differential and independent manifestation within co-containing neurones of neuropeptide Y and tyrosine hydroxylase during ontogeny of the rat central nervous system." Neurochemistry international vol. 6,6 (1984): 761-71. doi: https://doi.org/10.1016/0197-0186(84)90008-1
Foster GA, Schultzberg M, Goldstein M. Differential and independent manifestation within co-containing neurones of neuropeptide Y and tyrosine hydroxylase during ontogeny of the rat central nervous system. Neurochem Int. 1984;6(6):761-71. doi: 10.1016/0197-0186(84)90008-1. PMID: 20488105.
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Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure.

Molecular and cellular neurosciences

White JD, Yount GL, Lauterborn JC, Krause JE, Gall CM.
PMID: 19912848
Mol Cell Neurosci. 1992 Feb;3(1):79-87. doi: 10.1016/1044-7431(92)90011-p.

The opioid peptide dynorphin (DYN) is expressed normally at high levels in dentate gyrus granule cells in hippocampus and in neurons in entorhinal and neocortex. In the present study, ribonuclease protection and in situ hybridization analyses were used to...

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White JD, Yount GL, Lauterborn JC, et al. Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure. Mol Cell Neurosci. 1992;3(1):79-87doi: 10.1016/1044-7431(92)90011-p.
White, J. D., Yount, G. L., Lauterborn, J. C., Krause, J. E., & Gall, C. M. (1992). Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure. Molecular and cellular neurosciences, 3(1), 79-87. https://doi.org/10.1016/1044-7431(92)90011-p
White, J D, et al. "Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure." Molecular and cellular neurosciences vol. 3,1 (1992): 79-87. doi: https://doi.org/10.1016/1044-7431(92)90011-p
White JD, Yount GL, Lauterborn JC, Krause JE, Gall CM. Biphasic response of hippocampal dynorphin expression following recurrent limbic seizure. Mol Cell Neurosci. 1992 Feb;3(1):79-87. doi: 10.1016/1044-7431(92)90011-p. PMID: 19912848.
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Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease.

Neurochemistry international

De Laet MH, Dassonville M, Steyaert H, Vanderveken E, Lotstra F, Vierendeels G, Przedborski S, Vaudry H, Vanderhaeghen JJ.
PMID: 20504409
Neurochem Int. 1989;14(2):135-41. doi: 10.1016/0197-0186(89)90113-7.

Ganglionic and aganglionic full-thickness samples, at 4 levels of the colon of 26 infants with Hirschsprung's disease, were studied by immunohistochemistry. In the distal part of the aganglionic bowel, we observe a decrease of substance P and vasoactive intestinal...

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De Laet MH, Dassonville M, Steyaert H, et al. Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochem Int. 1989;14(2):135-41doi: 10.1016/0197-0186(89)90113-7.
De Laet, M. H., Dassonville, M., Steyaert, H., Vanderveken, E., Lotstra, F., Vierendeels, G., Przedborski, S., Vaudry, H., & Vanderhaeghen, J. J. (1989). Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochemistry international, 14(2), 135-41. https://doi.org/10.1016/0197-0186(89)90113-7
De Laet, M H, et al. "Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease." Neurochemistry international vol. 14,2 (1989): 135-41. doi: https://doi.org/10.1016/0197-0186(89)90113-7
De Laet MH, Dassonville M, Steyaert H, Vanderveken E, Lotstra F, Vierendeels G, Przedborski S, Vaudry H, Vanderhaeghen JJ. Decrease of vasoactive intestinal peptide, methionine-enkephalin, substance P and increase of neuropeptide Y immunoreactive nerve fibres in aganglionic colon of Hirschsprung's disease. Neurochem Int. 1989;14(2):135-41. doi: 10.1016/0197-0186(89)90113-7. PMID: 20504409.
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5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression.

Experimental neurobiology

Moon YW, Choi SH, Yoo SB, Lee JH, Jahng JW.
PMID: 22110352
Exp Neurobiol. 2010 Dec;19(3):132-9. doi: 10.5607/en.2010.19.3.132. Epub 2010 Dec 31.

This study was conducted to define the underlying mechanism of hypophagia induced by increased central serotonergic action. Rats received 3 daily injections of 5-hydroxy-L-tryptophan (5-HTP), a serotonin precursor, at a dose of 100 mg/kg/10 ml saline at 1 h...

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Moon YW, Choi SH, Yoo SB, et al. 5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression. Exp Neurobiol. 2010;19(3):132-9doi: 10.5607/en.2010.19.3.132.
Moon, Y. W., Choi, S. H., Yoo, S. B., Lee, J. H., & Jahng, J. W. (2010). 5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression. Experimental neurobiology, 19(3), 132-9. https://doi.org/10.5607/en.2010.19.3.132
Moon, Young Wha, et al. "5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression." Experimental neurobiology vol. 19,3 (2010): 132-9. doi: https://doi.org/10.5607/en.2010.19.3.132
Moon YW, Choi SH, Yoo SB, Lee JH, Jahng JW. 5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression. Exp Neurobiol. 2010 Dec;19(3):132-9. doi: 10.5607/en.2010.19.3.132. Epub 2010 Dec 31. PMID: 22110352; PMCID: PMC3214782.
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Addiction is a Reward Deficit and Stress Surfeit Disorder.

Frontiers in psychiatry

Koob GF.
PMID: 23914176
Front Psychiatry. 2013 Aug 01;4:72. doi: 10.3389/fpsyt.2013.00072. eCollection 2013.

Drug addiction can be defined by a three-stage cycle - binge/intoxication, withdrawal/negative affect, and preoccupation/anticipation - that involves allostatic changes in the brain reward and stress systems. Two primary sources of reinforcement, positive and negative reinforcement, have been hypothesized...

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Koob GF. Addiction is a Reward Deficit and Stress Surfeit Disorder. Front Psychiatry. 2013;4:72doi: 10.3389/fpsyt.2013.00072.
Koob, G. F. (2013). Addiction is a Reward Deficit and Stress Surfeit Disorder. Frontiers in psychiatry, 472. https://doi.org/10.3389/fpsyt.2013.00072
Koob, George F. "Addiction is a Reward Deficit and Stress Surfeit Disorder." Frontiers in psychiatry vol. 4 (2013): 72. doi: https://doi.org/10.3389/fpsyt.2013.00072
Koob GF. Addiction is a Reward Deficit and Stress Surfeit Disorder. Front Psychiatry. 2013 Aug 01;4:72. doi: 10.3389/fpsyt.2013.00072. eCollection 2013. PMID: 23914176; PMCID: PMC3730086.
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Gut hormones in cyclostomes.

Fish physiology and biochemistry

Van Noorden S.
PMID: 24220989
Fish Physiol Biochem. 1990 Sep;8(5):399-408. doi: 10.1007/BF00003371.

The current state of knowledge about regulatory peptides in endocrine cells and nerves of the alimentary canal of lampreys and hagfishes is reviewed. Cyclostomes have a wide range of peptides similar immunochemically to those of higher vertebrates. They include,...

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Van Noorden S. Gut hormones in cyclostomes. Fish Physiol Biochem. 1990;8(5):399-408doi: 10.1007/BF00003371.
Van Noorden, S. (1990). Gut hormones in cyclostomes. Fish physiology and biochemistry, 8(5), 399-408. https://doi.org/10.1007/BF00003371
Van Noorden, S. "Gut hormones in cyclostomes." Fish physiology and biochemistry vol. 8,5 (1990): 399-408. doi: https://doi.org/10.1007/BF00003371
Van Noorden S. Gut hormones in cyclostomes. Fish Physiol Biochem. 1990 Sep;8(5):399-408. doi: 10.1007/BF00003371. PMID: 24220989.
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Metformin inhibits food intake and neuropeptide Y gene expression in the hypothalamus.

Neural regeneration research

Duan Y, Zhang R, Zhang M, Sun L, Dong S, Wang G, Zhang J, Zhao Z.
PMID: 25206548
Neural Regen Res. 2013 Sep 05;8(25):2379-88. doi: 10.3969/j.issn.1673-5374.2013.25.009.

Metformin may reduce food intake and body weight, but the anorexigenic effects of metformin are still poorly understood. In this study, Sprague-Dawley rats were administered a single intracere-broventricular dose of metformin and compound C, in a broader attempt to...

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Duan Y, Zhang R, Zhang M, et al. Metformin inhibits food intake and neuropeptide Y gene expression in the hypothalamus. Neural Regen Res. 2013;8(25):2379-88doi: 10.3969/j.issn.1673-5374.2013.25.009.
Duan, Y., Zhang, R., Zhang, M., Sun, L., Dong, S., Wang, G., Zhang, J., & Zhao, Z. (2013). Metformin inhibits food intake and neuropeptide Y gene expression in the hypothalamus. Neural regeneration research, 8(25), 2379-88. https://doi.org/10.3969/j.issn.1673-5374.2013.25.009
Duan, Yale, et al. "Metformin inhibits food intake and neuropeptide Y gene expression in the hypothalamus." Neural regeneration research vol. 8,25 (2013): 2379-88. doi: https://doi.org/10.3969/j.issn.1673-5374.2013.25.009
Duan Y, Zhang R, Zhang M, Sun L, Dong S, Wang G, Zhang J, Zhao Z. Metformin inhibits food intake and neuropeptide Y gene expression in the hypothalamus. Neural Regen Res. 2013 Sep 05;8(25):2379-88. doi: 10.3969/j.issn.1673-5374.2013.25.009. PMID: 25206548; PMCID: PMC4146045.
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Neuropeptide Y and leptin receptor expression in the hypothalamus of rats with chronic immobilization stress.

Neural regeneration research

Wang S, Chen J, Yue G, Bai M, Kou M, Jin Z.
PMID: 25206469
Neural Regen Res. 2013 Jun 25;8(18):1721-6. doi: 10.3969/j.issn.1673-5374.2013.18.010.

In this study, Sprague-Dawley rats were immobilized to a frame for 3 hours a day for 21 days to establish a model of chronic immobilization stress. The body weight and food intake of rats subjected to chronic immobilization stress...

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Wang S, Chen J, Yue G, et al. Neuropeptide Y and leptin receptor expression in the hypothalamus of rats with chronic immobilization stress. Neural Regen Res. 2013;8(18):1721-6doi: 10.3969/j.issn.1673-5374.2013.18.010.
Wang, S., Chen, J., Yue, G., Bai, M., Kou, M., & Jin, Z. (2013). Neuropeptide Y and leptin receptor expression in the hypothalamus of rats with chronic immobilization stress. Neural regeneration research, 8(18), 1721-6. https://doi.org/10.3969/j.issn.1673-5374.2013.18.010
Wang, Shaoxian, et al. "Neuropeptide Y and leptin receptor expression in the hypothalamus of rats with chronic immobilization stress." Neural regeneration research vol. 8,18 (2013): 1721-6. doi: https://doi.org/10.3969/j.issn.1673-5374.2013.18.010
Wang S, Chen J, Yue G, Bai M, Kou M, Jin Z. Neuropeptide Y and leptin receptor expression in the hypothalamus of rats with chronic immobilization stress. Neural Regen Res. 2013 Jun 25;8(18):1721-6. doi: 10.3969/j.issn.1673-5374.2013.18.010. PMID: 25206469; PMCID: PMC4145910.
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