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Protective Effect of Oligonol on Dimethylnitrosamine-Induced Liver Fibrosis in Rats via the JNK/NF-κB and PI3K/Akt/Nrf2 Signaling Pathways.

Antioxidants (Basel, Switzerland)

Lee C, Bak J, Yoon S, Moon JO.
PMID: 33671028
Antioxidants (Basel). 2021 Feb 28;10(3). doi: 10.3390/antiox10030366.

Oligonol is a low molecular weight polyphenol product derived from lychee fruit by a manufacturing process. We investigated oligonol's anti-fibrotic effect and the underlying mechanism in dimethylnitrosamine (DMN)-induced chronic liver damage in male Sprague-Dawley rats. Oral administration of oligonol...

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Lee C, Bak J, Yoon S, et al. Protective Effect of Oligonol on Dimethylnitrosamine-Induced Liver Fibrosis in Rats via the JNK/NF-κB and PI3K/Akt/Nrf2 Signaling Pathways. Antioxidants (Basel). 2021;10(3)doi: 10.3390/antiox10030366.
Lee, C., Bak, J., Yoon, S., & Moon, J. O. (2021). Protective Effect of Oligonol on Dimethylnitrosamine-Induced Liver Fibrosis in Rats via the JNK/NF-κB and PI3K/Akt/Nrf2 Signaling Pathways. Antioxidants (Basel, Switzerland), 10(3), . https://doi.org/10.3390/antiox10030366
Lee, Changyong, et al. "Protective Effect of Oligonol on Dimethylnitrosamine-Induced Liver Fibrosis in Rats via the JNK/NF-κB and PI3K/Akt/Nrf2 Signaling Pathways." Antioxidants (Basel, Switzerland) vol. 10,3 (2021). doi: https://doi.org/10.3390/antiox10030366
Lee C, Bak J, Yoon S, Moon JO. Protective Effect of Oligonol on Dimethylnitrosamine-Induced Liver Fibrosis in Rats via the JNK/NF-κB and PI3K/Akt/Nrf2 Signaling Pathways. Antioxidants (Basel). 2021 Feb 28;10(3). doi: 10.3390/antiox10030366. PMID: 33671028; PMCID: PMC7997446.
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The relationship of retinopathy of prematurity with brain-derivated neurotrophic factor, vascular endotelial growth factor-A, endothelial PAD domain protein 1 and nitric oxide synthase 3 gene polymorphisms.

Ophthalmic genetics

Ilguy S, Cilingir O, Bilgec MD, Ozalp O, Erzurumluoglu Gokalp E, Arslan S, Tekin N, Aydemir O, Erol N, Colak E, Gursoy H.
PMID: 34346275
Ophthalmic Genet. 2021 Dec;42(6):725-731. doi: 10.1080/13816810.2021.1961279. Epub 2021 Aug 04.

BACKGROUND: In addition to risk factors such as low birth weight and uncontrolled oxygen therapy, genetic predisposition is also thought to play a role in the development of retinopathy of prematurity (ROP). In our study, we aimed to analyze...

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Ilguy S, Cilingir O, Bilgec MD, et al. The relationship of retinopathy of prematurity with brain-derivated neurotrophic factor, vascular endotelial growth factor-A, endothelial PAD domain protein 1 and nitric oxide synthase 3 gene polymorphisms. Ophthalmic Genet. 2021;42(6):725-731doi: 10.1080/13816810.2021.1961279.
Ilguy, S., Cilingir, O., Bilgec, M. D., Ozalp, O., Erzurumluoglu Gokalp, E., Arslan, S., Tekin, N., Aydemir, O., Erol, N., Colak, E., & Gursoy, H. (2021). The relationship of retinopathy of prematurity with brain-derivated neurotrophic factor, vascular endotelial growth factor-A, endothelial PAD domain protein 1 and nitric oxide synthase 3 gene polymorphisms. Ophthalmic genetics, 42(6), 725-731. https://doi.org/10.1080/13816810.2021.1961279
Ilguy, Serdar, et al. "The relationship of retinopathy of prematurity with brain-derivated neurotrophic factor, vascular endotelial growth factor-A, endothelial PAD domain protein 1 and nitric oxide synthase 3 gene polymorphisms." Ophthalmic genetics vol. 42,6 (2021): 725-731. doi: https://doi.org/10.1080/13816810.2021.1961279
Ilguy S, Cilingir O, Bilgec MD, Ozalp O, Erzurumluoglu Gokalp E, Arslan S, Tekin N, Aydemir O, Erol N, Colak E, Gursoy H. The relationship of retinopathy of prematurity with brain-derivated neurotrophic factor, vascular endotelial growth factor-A, endothelial PAD domain protein 1 and nitric oxide synthase 3 gene polymorphisms. Ophthalmic Genet. 2021 Dec;42(6):725-731. doi: 10.1080/13816810.2021.1961279. Epub 2021 Aug 04. PMID: 34346275.
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Myeloid-associated differentiation marker is a novel SP-A-associated transmembrane protein whose expression on airway epithelial cells correlates with asthma severity.

Scientific reports

Dy ABC, Langlais PR, Barker NK, Addison KJ, Tanyaratsrisakul S, Boitano S, Christenson SA, Kraft M, Meyers D, Bleecker ER, Li X, Ledford JG.
PMID: 34862427
Sci Rep. 2021 Dec 03;11(1):23392. doi: 10.1038/s41598-021-02869-w.

Surfactant protein A (SP-A) is well-known for its protective role in pulmonary immunity. Previous studies from our group have shown that SP-A mediates eosinophil activities, including degranulation and apoptosis. In order to identify potential binding partners on eosinophils for...

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Dy ABC, Langlais PR, Barker NK, et al. Myeloid-associated differentiation marker is a novel SP-A-associated transmembrane protein whose expression on airway epithelial cells correlates with asthma severity. Sci Rep. 2021;11(1):23392doi: 10.1038/s41598-021-02869-w.
Dy, A. B. C., Langlais, P. R., Barker, N. K., Addison, K. J., Tanyaratsrisakul, S., Boitano, S., Christenson, S. A., Kraft, M., Meyers, D., Bleecker, E. R., Li, X., & Ledford, J. G. (2021). Myeloid-associated differentiation marker is a novel SP-A-associated transmembrane protein whose expression on airway epithelial cells correlates with asthma severity. Scientific reports, 11(1), 23392. https://doi.org/10.1038/s41598-021-02869-w
Dy, Alane Blythe C, et al. "Myeloid-associated differentiation marker is a novel SP-A-associated transmembrane protein whose expression on airway epithelial cells correlates with asthma severity." Scientific reports vol. 11,1 (2021): 23392. doi: https://doi.org/10.1038/s41598-021-02869-w
Dy ABC, Langlais PR, Barker NK, Addison KJ, Tanyaratsrisakul S, Boitano S, Christenson SA, Kraft M, Meyers D, Bleecker ER, Li X, Ledford JG. Myeloid-associated differentiation marker is a novel SP-A-associated transmembrane protein whose expression on airway epithelial cells correlates with asthma severity. Sci Rep. 2021 Dec 03;11(1):23392. doi: 10.1038/s41598-021-02869-w. PMID: 34862427; PMCID: PMC8642528.
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The place of vericiguat in the landscape of treatment for heart failure with reduced ejection fraction.

Heart failure reviews

Aimo A, Castiglione V, Vergaro G, Panichella G, Senni M, Lombardi CM, Emdin M.
PMID: 34291399
Heart Fail Rev. 2021 Jul 21; doi: 10.1007/s10741-021-10146-1. Epub 2021 Jul 21.

The significant morbidity and mortality associated with heart failure with reduced (HFrEF) or preserved ejection fraction (HFpEF) justify the search for novel therapeutic agents. The nitric oxide (NO)-soluble guanylate cyclase (sGC)-cyclic guanosine monophosphate (cGMP) pathway plays an important role...

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Aimo A, Castiglione V, Vergaro G, et al. The place of vericiguat in the landscape of treatment for heart failure with reduced ejection fraction. Heart Fail Rev. 2021;doi: 10.1007/s10741-021-10146-1.
Aimo, A., Castiglione, V., Vergaro, G., Panichella, G., Senni, M., Lombardi, C. M., & Emdin, M. (2021). The place of vericiguat in the landscape of treatment for heart failure with reduced ejection fraction. Heart failure reviews, . https://doi.org/10.1007/s10741-021-10146-1
Aimo, Alberto, et al. "The place of vericiguat in the landscape of treatment for heart failure with reduced ejection fraction." Heart failure reviews vol. (2021). doi: https://doi.org/10.1007/s10741-021-10146-1
Aimo A, Castiglione V, Vergaro G, Panichella G, Senni M, Lombardi CM, Emdin M. The place of vericiguat in the landscape of treatment for heart failure with reduced ejection fraction. Heart Fail Rev. 2021 Jul 21; doi: 10.1007/s10741-021-10146-1. Epub 2021 Jul 21. PMID: 34291399.
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Violet LED induces vasodilation in rat aortic rings by soluble guanylate cyclase-dependent mechanism and increases SOD activity.

Lasers in medical science

de Moraes LHO, Mancini MW, Almeida-Lopes L, Rodrigues GJ.
PMID: 33735421
Lasers Med Sci. 2021 Mar 18; doi: 10.1007/s10103-021-03293-2. Epub 2021 Mar 18.

We found several studies that have used the aortic rings as an experimental model, mainly for the testing of new drugs or new therapies that try to reverse or prevent endothelial dysfunction or characterize its mechanism of action in...

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de Moraes LHO, Mancini MW, Almeida-Lopes L, et al. Violet LED induces vasodilation in rat aortic rings by soluble guanylate cyclase-dependent mechanism and increases SOD activity. Lasers Med Sci. 2021;doi: 10.1007/s10103-021-03293-2.
de Moraes, L. H. O., Mancini, M. W., Almeida-Lopes, L., & Rodrigues, G. J. (2021). Violet LED induces vasodilation in rat aortic rings by soluble guanylate cyclase-dependent mechanism and increases SOD activity. Lasers in medical science, . https://doi.org/10.1007/s10103-021-03293-2
de Moraes, Luis Henrique Oliveira, et al. "Violet LED induces vasodilation in rat aortic rings by soluble guanylate cyclase-dependent mechanism and increases SOD activity." Lasers in medical science vol. (2021). doi: https://doi.org/10.1007/s10103-021-03293-2
de Moraes LHO, Mancini MW, Almeida-Lopes L, Rodrigues GJ. Violet LED induces vasodilation in rat aortic rings by soluble guanylate cyclase-dependent mechanism and increases SOD activity. Lasers Med Sci. 2021 Mar 18; doi: 10.1007/s10103-021-03293-2. Epub 2021 Mar 18. PMID: 33735421.
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Pathogenesis of aortic stenosis: not just a matter of wear and tear.

American journal of cardiovascular disease

Sverdlov AL, Ngo DT, Chapman MJ, Ali OA, Chirkov YY, Horowitz JD.
PMID: 22254198
Am J Cardiovasc Dis. 2011;1(2):185-99. Epub 2011 Jul 28.

Aortic valve stenosis (AS) is the commonest form of valvular heart disease in the Western world. Its prevalence increases exponentially with age and it is present in 2-7% of all patients over 65 years of age. In view of...

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Sverdlov AL, Ngo DT, Chapman MJ, et al. Pathogenesis of aortic stenosis: not just a matter of wear and tear. Am J Cardiovasc Dis. 2011;1(2):185-99
Sverdlov, A. L., Ngo, D. T., Chapman, M. J., Ali, O. A., Chirkov, Y. Y., & Horowitz, J. D. (2011). Pathogenesis of aortic stenosis: not just a matter of wear and tear. American journal of cardiovascular disease, 1(2), 185-99.
Sverdlov, Aaron L, et al. "Pathogenesis of aortic stenosis: not just a matter of wear and tear." American journal of cardiovascular disease vol. 1,2 (2011): 185-99.
Sverdlov AL, Ngo DT, Chapman MJ, Ali OA, Chirkov YY, Horowitz JD. Pathogenesis of aortic stenosis: not just a matter of wear and tear. Am J Cardiovasc Dis. 2011;1(2):185-99. Epub 2011 Jul 28. PMID: 22254198; PMCID: PMC3253493.
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Role of human glutathione transferases in biotransformation of the nitric oxide prodrug JS-K.

Scientific reports

Sjödin B, Mannervik B.
PMID: 34675290
Sci Rep. 2021 Oct 21;11(1):20765. doi: 10.1038/s41598-021-00327-1.

Nitric oxide (NO) plays a prominent physiological role as a low-molecular-mass signal molecule involved in diverse biological functions. Great attention has been directed to pharmacologically modulating the release of NO for various therapeutic applications. We have focused on O

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Sjödin B, Mannervik B. Role of human glutathione transferases in biotransformation of the nitric oxide prodrug JS-K. Sci Rep. 2021;11(1):20765doi: 10.1038/s41598-021-00327-1.
Sjödin, B., & Mannervik, B. (2021). Role of human glutathione transferases in biotransformation of the nitric oxide prodrug JS-K. Scientific reports, 11(1), 20765. https://doi.org/10.1038/s41598-021-00327-1
Sjödin, Birgitta, and Mannervik, Bengt. "Role of human glutathione transferases in biotransformation of the nitric oxide prodrug JS-K." Scientific reports vol. 11,1 (2021): 20765. doi: https://doi.org/10.1038/s41598-021-00327-1
Sjödin B, Mannervik B. Role of human glutathione transferases in biotransformation of the nitric oxide prodrug JS-K. Sci Rep. 2021 Oct 21;11(1):20765. doi: 10.1038/s41598-021-00327-1. PMID: 34675290; PMCID: PMC8531399.
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Exploring the paradox of defense between host and Leishmania parasite.

International immunopharmacology

Kumari D, Singh K.
PMID: 34890999
Int Immunopharmacol. 2021 Dec 07;102:108400. doi: 10.1016/j.intimp.2021.108400. Epub 2021 Dec 07.

Leishmaniasis, a neglected tropical disease, still remains a global concern for the healthcare sector. The primary causative agents of the disease comprise diverse leishmanial species, leading to recurring failures in disease diagnosis and delaying the initiation of appropriate chemotherapy....

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Kumari D, Singh K. Exploring the paradox of defense between host and Leishmania parasite. Int Immunopharmacol. 2021;102:108400doi: 10.1016/j.intimp.2021.108400.
Kumari, D., & Singh, K. (2021). Exploring the paradox of defense between host and Leishmania parasite. International immunopharmacology, 102108400. https://doi.org/10.1016/j.intimp.2021.108400
Kumari, Diksha, and Singh, Kuljit. "Exploring the paradox of defense between host and Leishmania parasite." International immunopharmacology vol. 102 (2021): 108400. doi: https://doi.org/10.1016/j.intimp.2021.108400
Kumari D, Singh K. Exploring the paradox of defense between host and Leishmania parasite. Int Immunopharmacol. 2021 Dec 07;102:108400. doi: 10.1016/j.intimp.2021.108400. Epub 2021 Dec 07. PMID: 34890999.
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Chimyl Alcohol Exhibits Proinflammatory Activity in vivo and in vitro.

Cells, tissues, organs

Mischenko P, Egoraeva A, Tyrtyshnaia A, Kasyanov S, Ponomarenko A, Manzhulo I.
PMID: 34571511
Cells Tissues Organs. 2021 Sep 27; doi: 10.1159/000519832. Epub 2021 Sep 27.

Marine organisms are among the prominent and abundant sources of 1-O-alkyl-sn-glycerols, including chimyl alcohol (CA), batyl alcohol and selachyl alcohol. These biologically active compounds are precursors in plasmalogen and phospholipid biosynthesis, which are the main irreplaceable components of cell...

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Mischenko P, Egoraeva A, Tyrtyshnaia A, et al. Chimyl Alcohol Exhibits Proinflammatory Activity in vivo and in vitro. Cells Tissues Organs. 2021;doi: 10.1159/000519832.
Mischenko, P., Egoraeva, A., Tyrtyshnaia, A., Kasyanov, S., Ponomarenko, A., & Manzhulo, I. (2021). Chimyl Alcohol Exhibits Proinflammatory Activity in vivo and in vitro. Cells, tissues, organs, . https://doi.org/10.1159/000519832
Mischenko, Polina, et al. "Chimyl Alcohol Exhibits Proinflammatory Activity in vivo and in vitro." Cells, tissues, organs vol. (2021). doi: https://doi.org/10.1159/000519832
Mischenko P, Egoraeva A, Tyrtyshnaia A, Kasyanov S, Ponomarenko A, Manzhulo I. Chimyl Alcohol Exhibits Proinflammatory Activity in vivo and in vitro. Cells Tissues Organs. 2021 Sep 27; doi: 10.1159/000519832. Epub 2021 Sep 27. PMID: 34571511.
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Vitamin D deficiency attenuates endothelial function by reducing antioxidant activity and vascular eNOS expression in the rat microcirculation.

Microvascular research

Wee CL, Mokhtar SS, Banga Singh KK, Rasool AHG.
PMID: 34324883
Microvasc Res. 2021 Nov;138:104227. doi: 10.1016/j.mvr.2021.104227. Epub 2021 Jul 27.

This study examined the effects of vitamin D deficiency on vascular function and tissue oxidative status in the microcirculation; and whether or not these effects can be ameliorated with calcitriol, the active vitamin D metabolite. Three groups (n =...

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Wee CL, Mokhtar SS, Banga Singh KK, et al. Vitamin D deficiency attenuates endothelial function by reducing antioxidant activity and vascular eNOS expression in the rat microcirculation. Microvasc Res. 2021;138:104227doi: 10.1016/j.mvr.2021.104227.
Wee, C. L., Mokhtar, S. S., Banga Singh, K. K., & Rasool, A. H. G. (2021). Vitamin D deficiency attenuates endothelial function by reducing antioxidant activity and vascular eNOS expression in the rat microcirculation. Microvascular research, 138104227. https://doi.org/10.1016/j.mvr.2021.104227
Wee, Chee Lee, et al. "Vitamin D deficiency attenuates endothelial function by reducing antioxidant activity and vascular eNOS expression in the rat microcirculation." Microvascular research vol. 138 (2021): 104227. doi: https://doi.org/10.1016/j.mvr.2021.104227
Wee CL, Mokhtar SS, Banga Singh KK, Rasool AHG. Vitamin D deficiency attenuates endothelial function by reducing antioxidant activity and vascular eNOS expression in the rat microcirculation. Microvasc Res. 2021 Nov;138:104227. doi: 10.1016/j.mvr.2021.104227. Epub 2021 Jul 27. PMID: 34324883.
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Current Understanding of Pressure Natriuresis.

Electrolyte & blood pressure : E & BP

Baek EJ, Kim S.
PMID: 35003284
Electrolyte Blood Press. 2021 Dec;19(2):38-45. doi: 10.5049/EBP.2021.19.2.38. Epub 2021 Dec 23.

Pressure natriuresis refers to the concept that increased renal perfusion pressure leads to a decrease in tubular reabsorption of sodium and an increased sodium excretion. The set point of blood pressure is the point at which pressure natriuresis and...

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Baek EJ, Kim S. Current Understanding of Pressure Natriuresis. Electrolyte Blood Press. 2021;19(2):38-45doi: 10.5049/EBP.2021.19.2.38.
Baek, E. J., & Kim, S. (2021). Current Understanding of Pressure Natriuresis. Electrolyte & blood pressure : E & BP, 19(2), 38-45. https://doi.org/10.5049/EBP.2021.19.2.38
Baek, Eun Ji, and Kim, Sejoong. "Current Understanding of Pressure Natriuresis." Electrolyte & blood pressure : E & BP vol. 19,2 (2021): 38-45. doi: https://doi.org/10.5049/EBP.2021.19.2.38
Baek EJ, Kim S. Current Understanding of Pressure Natriuresis. Electrolyte Blood Press. 2021 Dec;19(2):38-45. doi: 10.5049/EBP.2021.19.2.38. Epub 2021 Dec 23. PMID: 35003284; PMCID: PMC8715224.
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Betaine attenuates sodium arsenite-induced renal dysfunction in rats.

Drug and chemical toxicology

Sharma S, Kaur T, Sharma AK, Singh B, Pathak D, Yadav HN, Singh AP.
PMID: 34380335
Drug Chem Toxicol. 2021 Aug 11;1-8. doi: 10.1080/01480545.2021.1959699. Epub 2021 Aug 11.

Exposure to higher levels of arsenic is a serious threat affecting human health worldwide. We investigated the protective role of betaine (N,N,N-trimethylglycine) against sodium arsenite-induced renal dysfunction in rats. Sodium arsenite (5 mg/kg, oral) was given to rats for...

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Sharma S, Kaur T, Sharma AK, et al. Betaine attenuates sodium arsenite-induced renal dysfunction in rats. Drug Chem Toxicol. 2021;1-8doi: 10.1080/01480545.2021.1959699.
Sharma, S., Kaur, T., Sharma, A. K., Singh, B., Pathak, D., Yadav, H. N., & Singh, A. P. (2021). Betaine attenuates sodium arsenite-induced renal dysfunction in rats. Drug and chemical toxicology, 1-8. https://doi.org/10.1080/01480545.2021.1959699
Sharma, Sumedha, et al. "Betaine attenuates sodium arsenite-induced renal dysfunction in rats." Drug and chemical toxicology vol. (2021): 1-8. doi: https://doi.org/10.1080/01480545.2021.1959699
Sharma S, Kaur T, Sharma AK, Singh B, Pathak D, Yadav HN, Singh AP. Betaine attenuates sodium arsenite-induced renal dysfunction in rats. Drug Chem Toxicol. 2021 Aug 11;1-8. doi: 10.1080/01480545.2021.1959699. Epub 2021 Aug 11. PMID: 34380335.
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Showing 9985 to 9996 of 12726 entries