Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 37 to 42 of 42 entries
Sorted by: Best Match Show Resources per page
Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice.

Frontiers in cellular neuroscience

Lee CH, Umemori H.
PMID: 23616746
Front Cell Neurosci. 2013 Apr 18;7:43. doi: 10.3389/fncel.2013.00043. eCollection 2013.

In the developing hippocampus, fibroblast growth factor (FGF) 22 promotes the formation of excitatory presynaptic terminals. Remarkably, FGF22 knockout (KO) mice show resistance to generalized seizures in adults as assessed by chemical kindling, a model that is widely used...

Cite
Lee CH, Umemori H. Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice. Front Cell Neurosci. 2013;7:43doi: 10.3389/fncel.2013.00043.
Lee, C. H., & Umemori, H. (2013). Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice. Frontiers in cellular neuroscience, 743. https://doi.org/10.3389/fncel.2013.00043
Lee, Clara H, and Umemori, Hisashi. "Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice." Frontiers in cellular neuroscience vol. 7 (2013): 43. doi: https://doi.org/10.3389/fncel.2013.00043
Lee CH, Umemori H. Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice. Front Cell Neurosci. 2013 Apr 18;7:43. doi: 10.3389/fncel.2013.00043. eCollection 2013. PMID: 23616746; PMCID: PMC3629311.
Copy Download .nbib Format: NLM
Transcriptional profiling of synovium in a porcine model of early post-traumatic osteoarthritis.

Journal of orthopaedic research : official publication of the Orthopaedic Research Society

Sieker JT, Proffen BL, Waller KA, Chin KE, Karamchedu NP, Akelman MR, Perrone GS, Kiapour AM, Konrad J, Fleming BC, Murray MM.
PMID: 29460983
J Orthop Res. 2018 Feb 20; doi: 10.1002/jor.23876. Epub 2018 Feb 20.

To determine the transcriptional profile of synovium during the molecular phase of post-traumatic osteoarthritis, anterior cruciate ligament transections (ACL) were performed in 36 Yucatan minipigs. Equal numbers were randomly assigned to no further treatment, ACL reconstruction or repair. Perimeniscal...

Cite
Sieker JT, Proffen BL, Waller KA, et al. Transcriptional profiling of synovium in a porcine model of early post-traumatic osteoarthritis. J Orthop Res. 2018;doi: 10.1002/jor.23876.
Sieker, J. T., Proffen, B. L., Waller, K. A., Chin, K. E., Karamchedu, N. P., Akelman, M. R., Perrone, G. S., Kiapour, A. M., Konrad, J., Fleming, B. C., & Murray, M. M. (2018). Transcriptional profiling of synovium in a porcine model of early post-traumatic osteoarthritis. Journal of orthopaedic research : official publication of the Orthopaedic Research Society, . https://doi.org/10.1002/jor.23876
Sieker, Jakob T, et al. "Transcriptional profiling of synovium in a porcine model of early post-traumatic osteoarthritis." Journal of orthopaedic research : official publication of the Orthopaedic Research Society vol. (2018). doi: https://doi.org/10.1002/jor.23876
Sieker JT, Proffen BL, Waller KA, Chin KE, Karamchedu NP, Akelman MR, Perrone GS, Kiapour AM, Konrad J, Fleming BC, Murray MM. Transcriptional profiling of synovium in a porcine model of early post-traumatic osteoarthritis. J Orthop Res. 2018 Feb 20; doi: 10.1002/jor.23876. Epub 2018 Feb 20. PMID: 29460983; PMCID: PMC6102098.
Copy Download .nbib Format: NLM
Sense and Immunity: Context-Dependent Neuro-Immune Interplay.

Frontiers in immunology

Foster SL, Seehus CR, Woolf CJ, Talbot S.
PMID: 29163530
Front Immunol. 2017 Nov 03;8:1463. doi: 10.3389/fimmu.2017.01463. eCollection 2017.

The sensory nervous and immune systems, historically considered autonomous, actually work in concert to promote host defense and tissue homeostasis. These systems interact with each other through a common language of cell surface G protein-coupled receptors and receptor tyrosine...

Cite
Foster SL, Seehus CR, Woolf CJ, et al. Sense and Immunity: Context-Dependent Neuro-Immune Interplay. Front Immunol. 2017;8:1463doi: 10.3389/fimmu.2017.01463.
Foster, S. L., Seehus, C. R., Woolf, C. J., & Talbot, S. (2017). Sense and Immunity: Context-Dependent Neuro-Immune Interplay. Frontiers in immunology, 81463. https://doi.org/10.3389/fimmu.2017.01463
Foster, Simmie L, et al. "Sense and Immunity: Context-Dependent Neuro-Immune Interplay." Frontiers in immunology vol. 8 (2017): 1463. doi: https://doi.org/10.3389/fimmu.2017.01463
Foster SL, Seehus CR, Woolf CJ, Talbot S. Sense and Immunity: Context-Dependent Neuro-Immune Interplay. Front Immunol. 2017 Nov 03;8:1463. doi: 10.3389/fimmu.2017.01463. eCollection 2017. PMID: 29163530; PMCID: PMC5675863.
Copy Download .nbib Format: NLM
Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients.

Pain reports

Sieberg CB, Taras C, Gomaa A, Nickerson C, Wong C, Ward C, Baskozos G, Bennett DLH, Ramirez JD, Themistocleous AC, Rice ASC, Shillo PR, Tesfaye S, Edwards RR, Andrews NA, Berde C, Costigan M.
PMID: 29922743
Pain Rep. 2018 May 24;3(3):e651. doi: 10.1097/PR9.0000000000000651. eCollection 2018 May.

BACKGROUND: Epidemiological studies in patients with neuropathic pain demonstrate a strong association with psychiatric conditions such as anxiety; however, the precipitating pathology between these symptoms remains unclear. To investigate this, we studied the effects of lifelong stress on levels...

Cite
Sieberg CB, Taras C, Gomaa A, et al. Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain Rep. 2018;3(3):e651doi: 10.1097/PR9.0000000000000651.
Sieberg, C. B., Taras, C., Gomaa, A., Nickerson, C., Wong, C., Ward, C., Baskozos, G., Bennett, D. L. H., Ramirez, J. D., Themistocleous, A. C., Rice, A. S. C., Shillo, P. R., Tesfaye, S., Edwards, R. R., Andrews, N. A., Berde, C., & Costigan, M. (2018). Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain reports, 3(3), e651. https://doi.org/10.1097/PR9.0000000000000651
Sieberg, Christine B, et al. "Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients." Pain reports vol. 3,3 (2018): e651. doi: https://doi.org/10.1097/PR9.0000000000000651
Sieberg CB, Taras C, Gomaa A, Nickerson C, Wong C, Ward C, Baskozos G, Bennett DLH, Ramirez JD, Themistocleous AC, Rice ASC, Shillo PR, Tesfaye S, Edwards RR, Andrews NA, Berde C, Costigan M. Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain Rep. 2018 May 24;3(3):e651. doi: 10.1097/PR9.0000000000000651. eCollection 2018 May. PMID: 29922743; PMCID: PMC5999418.
Copy Download .nbib Format: NLM
Cholinergic Activation of Primary Human Derived Intestinal Epithelium Does Not Ameliorate TNF-α Induced Injury.

Cellular and molecular bioengineering

Hosic S, Lake W, Stas E, Koppes R, Breault DT, Murthy SK, Koppes AN.
PMID: 33184579
Cell Mol Bioeng. 2020 Jun 24;13(5):487-505. doi: 10.1007/s12195-020-00633-0. eCollection 2020 Oct.

INTRODUCTION: The intestinal epithelium contains specialized cells including enterocytes, goblet, Paneth, enteroendocrine, and stem cells. Impaired barrier integrity in Inflammatory Bowel Disease is characterized by elevated levels of pro-inflammatory cytokines, including tumor necrosis factor-alpha (TNF-α). Prior studies in immortalized...

Cite
Hosic S, Lake W, Stas E, et al. Cholinergic Activation of Primary Human Derived Intestinal Epithelium Does Not Ameliorate TNF-α Induced Injury. Cell Mol Bioeng. 2020;13(5):487-505doi: 10.1007/s12195-020-00633-0.
Hosic, S., Lake, W., Stas, E., Koppes, R., Breault, D. T., Murthy, S. K., & Koppes, A. N. (2020). Cholinergic Activation of Primary Human Derived Intestinal Epithelium Does Not Ameliorate TNF-α Induced Injury. Cellular and molecular bioengineering, 13(5), 487-505. https://doi.org/10.1007/s12195-020-00633-0
Hosic, Sanjin, et al. "Cholinergic Activation of Primary Human Derived Intestinal Epithelium Does Not Ameliorate TNF-α Induced Injury." Cellular and molecular bioengineering vol. 13,5 (2020): 487-505. doi: https://doi.org/10.1007/s12195-020-00633-0
Hosic S, Lake W, Stas E, Koppes R, Breault DT, Murthy SK, Koppes AN. Cholinergic Activation of Primary Human Derived Intestinal Epithelium Does Not Ameliorate TNF-α Induced Injury. Cell Mol Bioeng. 2020 Jun 24;13(5):487-505. doi: 10.1007/s12195-020-00633-0. eCollection 2020 Oct. PMID: 33184579; PMCID: PMC7596162.
Copy Download .nbib Format: NLM
Notum Deletion From Late-Stage Skeletal Cells Increases Cortical Bone Formation and Potentiates Skeletal Effects of Sclerostin Inhibition.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

Choi RB, Bullock WA, Hoggatt AM, Horan DJ, Pemberton EZ, Hong JM, Zhang X, He X, Robling AG.
PMID: 34223673
J Bone Miner Res. 2021 Dec;36(12):2413-2425. doi: 10.1002/jbmr.4411. Epub 2021 Jul 23.

Wnt signaling plays a vital role in the cell biology of skeletal patterning, differentiation, and maintenance. Notum is a secreted member of the α/β-hydrolase superfamily that hydrolyzes the palmitoleoylate modification on Wnt proteins, thereby disrupting Wnt signaling. As a...

Cite
Choi RB, Bullock WA, Hoggatt AM, et al. Notum Deletion From Late-Stage Skeletal Cells Increases Cortical Bone Formation and Potentiates Skeletal Effects of Sclerostin Inhibition. J Bone Miner Res. 2021;36(12):2413-2425doi: 10.1002/jbmr.4411.
Choi, R. B., Bullock, W. A., Hoggatt, A. M., Horan, D. J., Pemberton, E. Z., Hong, J. M., Zhang, X., He, X., & Robling, A. G. (2021). Notum Deletion From Late-Stage Skeletal Cells Increases Cortical Bone Formation and Potentiates Skeletal Effects of Sclerostin Inhibition. Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 36(12), 2413-2425. https://doi.org/10.1002/jbmr.4411
Choi, Roy B, et al. "Notum Deletion From Late-Stage Skeletal Cells Increases Cortical Bone Formation and Potentiates Skeletal Effects of Sclerostin Inhibition." Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research vol. 36,12 (2021): 2413-2425. doi: https://doi.org/10.1002/jbmr.4411
Choi RB, Bullock WA, Hoggatt AM, Horan DJ, Pemberton EZ, Hong JM, Zhang X, He X, Robling AG. Notum Deletion From Late-Stage Skeletal Cells Increases Cortical Bone Formation and Potentiates Skeletal Effects of Sclerostin Inhibition. J Bone Miner Res. 2021 Dec;36(12):2413-2425. doi: 10.1002/jbmr.4411. Epub 2021 Jul 23. PMID: 34223673; PMCID: PMC8688238.
Copy Download .nbib Format: NLM
Showing 37 to 42 of 42 entries