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SIVsm quasispecies adaptation to a new simian host.

PLoS pathogens

Demma LJ, Logsdon JM, Vanderford TH, Feinberg MB, Staprans SI.
PMID: 16201015
PLoS Pathog. 2005 Sep;1(1):e3. doi: 10.1371/journal.ppat.0010003. Epub 2005 Sep 30.

Despite the potential for infectious agents harbored by other species to become emerging human pathogens, little is known about why some agents establish successful cross-species transmission, while others do not. The simian immunodeficiency viruses (SIVs), certain variants of which...

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Demma LJ, Logsdon JM, Vanderford TH, et al. SIVsm quasispecies adaptation to a new simian host. PLoS Pathog. 2005;1(1):e3doi: 10.1371/journal.ppat.0010003.
Demma, L. J., Logsdon, J. M., Vanderford, T. H., Feinberg, M. B., & Staprans, S. I. (2005). SIVsm quasispecies adaptation to a new simian host. PLoS pathogens, 1(1), e3. https://doi.org/10.1371/journal.ppat.0010003
Demma, Linda J, et al. "SIVsm quasispecies adaptation to a new simian host." PLoS pathogens vol. 1,1 (2005): e3. doi: https://doi.org/10.1371/journal.ppat.0010003
Demma LJ, Logsdon JM, Vanderford TH, Feinberg MB, Staprans SI. SIVsm quasispecies adaptation to a new simian host. PLoS Pathog. 2005 Sep;1(1):e3. doi: 10.1371/journal.ppat.0010003. Epub 2005 Sep 30. PMID: 16201015; PMCID: PMC1238738.
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Exit of Plasmodium sporozoites from oocysts is an active process that involves the circumsporozoite protein.

PLoS pathogens

Wang Q, Fujioka H, Nussenzweig V.
PMID: 16201021
PLoS Pathog. 2005 Sep;1(1):e9. doi: 10.1371/journal.ppat.0010009. Epub 2005 Sep 30.

Plasmodium sporozoites develop within oocysts residing in the mosquito midgut. Mature sporozoites exit the oocysts, enter the hemolymph, and invade the salivary glands. The circumsporozoite (CS) protein is the major surface protein of salivary gland and oocyst sporozoites. It...

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Wang Q, Fujioka H, Nussenzweig V. Exit of Plasmodium sporozoites from oocysts is an active process that involves the circumsporozoite protein. PLoS Pathog. 2005;1(1):e9doi: 10.1371/journal.ppat.0010009.
Wang, Q., Fujioka, H., & Nussenzweig, V. (2005). Exit of Plasmodium sporozoites from oocysts is an active process that involves the circumsporozoite protein. PLoS pathogens, 1(1), e9. https://doi.org/10.1371/journal.ppat.0010009
Wang, Qian, et al. "Exit of Plasmodium sporozoites from oocysts is an active process that involves the circumsporozoite protein." PLoS pathogens vol. 1,1 (2005): e9. doi: https://doi.org/10.1371/journal.ppat.0010009
Wang Q, Fujioka H, Nussenzweig V. Exit of Plasmodium sporozoites from oocysts is an active process that involves the circumsporozoite protein. PLoS Pathog. 2005 Sep;1(1):e9. doi: 10.1371/journal.ppat.0010009. Epub 2005 Sep 30. PMID: 16201021; PMCID: PMC1238744.
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Retraction. Identification of a novel gammaretrovirus in prostate tumors of patients homozygous for R462Q RNASEL variant.

PLoS pathogens

[No authors listed]
PMID: 23028303
PLoS Pathog. 2012 Sep;8(9). doi: 10.1371/annotation/7e2efc01-2e9b-4e9b-aef0-87ab0e4e4732. Epub 2012 Sep 18.

No abstract available.

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Retraction. Identification of a novel gammaretrovirus in prostate tumors of patients homozygous for R462Q RNASEL variant. PLoS Pathog. 2012;8(9)doi: 10.1371/annotation/7e2efc01-2e9b-4e9b-aef0-87ab0e4e4732.
(2012). Retraction. Identification of a novel gammaretrovirus in prostate tumors of patients homozygous for R462Q RNASEL variant. PLoS pathogens, 8(9), . https://doi.org/10.1371/annotation/7e2efc01-2e9b-4e9b-aef0-87ab0e4e4732
"Retraction. Identification of a novel gammaretrovirus in prostate tumors of patients homozygous for R462Q RNASEL variant." PLoS pathogens vol. 8,9 (2012). doi: https://doi.org/10.1371/annotation/7e2efc01-2e9b-4e9b-aef0-87ab0e4e4732
Retraction. Identification of a novel gammaretrovirus in prostate tumors of patients homozygous for R462Q RNASEL variant. PLoS Pathog. 2012 Sep;8(9). doi: 10.1371/annotation/7e2efc01-2e9b-4e9b-aef0-87ab0e4e4732. Epub 2012 Sep 18. PMID: 23028303; PMCID: PMC3445601.
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Correction: Treatment with native heterodimeric IL-15 increases cytotoxic lymphocytes and reduces SHIV RNA in lymph nodes.

PLoS pathogens

Watson DC, Moysi E, Valentin A, Bergamaschi C, Devasundaram S, Fortis SP, Bear J, Chertova E, Bess J, Sowder R, Venzon DJ, Deleage C, Estes JD, Lifson JD, Petrovas C, Felber BK, Pavlakis GN.
PMID: 30308061
PLoS Pathog. 2018 Oct 11;14(10):e1007345. doi: 10.1371/journal.ppat.1007345. eCollection 2018 Oct.

[This corrects the article DOI: 10.1371/journal.ppat.1006902.].

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Watson DC, Moysi E, Valentin A, et al. Correction: Treatment with native heterodimeric IL-15 increases cytotoxic lymphocytes and reduces SHIV RNA in lymph nodes. PLoS Pathog. 2018;14(10):e1007345doi: 10.1371/journal.ppat.1007345.
Watson, D. C., Moysi, E., Valentin, A., Bergamaschi, C., Devasundaram, S., Fortis, S. P., Bear, J., Chertova, E., Bess, J., Sowder, R., Venzon, D. J., Deleage, C., Estes, J. D., Lifson, J. D., Petrovas, C., Felber, B. K., & Pavlakis, G. N. (2018). Correction: Treatment with native heterodimeric IL-15 increases cytotoxic lymphocytes and reduces SHIV RNA in lymph nodes. PLoS pathogens, 14(10), e1007345. https://doi.org/10.1371/journal.ppat.1007345
Watson, Dionysios C, et al. "Correction: Treatment with native heterodimeric IL-15 increases cytotoxic lymphocytes and reduces SHIV RNA in lymph nodes." PLoS pathogens vol. 14,10 (2018): e1007345. doi: https://doi.org/10.1371/journal.ppat.1007345
Watson DC, Moysi E, Valentin A, Bergamaschi C, Devasundaram S, Fortis SP, Bear J, Chertova E, Bess J, Sowder R, Venzon DJ, Deleage C, Estes JD, Lifson JD, Petrovas C, Felber BK, Pavlakis GN. Correction: Treatment with native heterodimeric IL-15 increases cytotoxic lymphocytes and reduces SHIV RNA in lymph nodes. PLoS Pathog. 2018 Oct 11;14(10):e1007345. doi: 10.1371/journal.ppat.1007345. eCollection 2018 Oct. PMID: 30308061; PMCID: PMC6181417.
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Correction: Bacillus anthracis Spore Surface Protein BclA Mediates Complement Factor H Binding to Spores and Promotes Spore Persistence.

PLoS pathogens

Wang Y, Jenkins SA, Gu C, Shree A, Martinez-Moczygemba M, Herold J, Botto M, Wetsel RA, Xu Y.
PMID: 30735542
PLoS Pathog. 2019 Feb 08;15(2):e1007579. doi: 10.1371/journal.ppat.1007579. eCollection 2019 Feb.

[This corrects the article DOI: 10.1371/journal.ppat.1005678.].

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Wang Y, Jenkins SA, Gu C, et al. Correction: Bacillus anthracis Spore Surface Protein BclA Mediates Complement Factor H Binding to Spores and Promotes Spore Persistence. PLoS Pathog. 2019;15(2):e1007579doi: 10.1371/journal.ppat.1007579.
Wang, Y., Jenkins, S. A., Gu, C., Shree, A., Martinez-Moczygemba, M., Herold, J., Botto, M., Wetsel, R. A., & Xu, Y. (2019). Correction: Bacillus anthracis Spore Surface Protein BclA Mediates Complement Factor H Binding to Spores and Promotes Spore Persistence. PLoS pathogens, 15(2), e1007579. https://doi.org/10.1371/journal.ppat.1007579
Wang, Yanyu, et al. "Correction: Bacillus anthracis Spore Surface Protein BclA Mediates Complement Factor H Binding to Spores and Promotes Spore Persistence." PLoS pathogens vol. 15,2 (2019): e1007579. doi: https://doi.org/10.1371/journal.ppat.1007579
Wang Y, Jenkins SA, Gu C, Shree A, Martinez-Moczygemba M, Herold J, Botto M, Wetsel RA, Xu Y. Correction: Bacillus anthracis Spore Surface Protein BclA Mediates Complement Factor H Binding to Spores and Promotes Spore Persistence. PLoS Pathog. 2019 Feb 08;15(2):e1007579. doi: 10.1371/journal.ppat.1007579. eCollection 2019 Feb. PMID: 30735542; PMCID: PMC6368309.
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Correction: Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility.

PLoS pathogens

[No authors listed]
PMID: 28212438
PLoS Pathog. 2017 Feb 17;13(2):e1006222. doi: 10.1371/journal.ppat.1006222. eCollection 2017 Feb.

[This corrects the article DOI: 10.1371/journal.ppat.1006075.].

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Correction: Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility. PLoS Pathog. 2017;13(2):e1006222doi: 10.1371/journal.ppat.1006222.
(2017). Correction: Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility. PLoS pathogens, 13(2), e1006222. https://doi.org/10.1371/journal.ppat.1006222
"Correction: Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility." PLoS pathogens vol. 13,2 (2017): e1006222. doi: https://doi.org/10.1371/journal.ppat.1006222
Correction: Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease Susceptibility. PLoS Pathog. 2017 Feb 17;13(2):e1006222. doi: 10.1371/journal.ppat.1006222. eCollection 2017 Feb. PMID: 28212438; PMCID: PMC5315273.
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Correction: Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous Vacuole.

PLoS pathogens

Fougère A, Jackson AP, Bechtsi DP, Braks JA, Annoura T, Fonager J, Spaccapelo R, Ramesar J, Chevalley-Maurel S, Klop O, van der Laan AM, Tanke HJ, Kocken CH, Pasini EM, Khan SM, Böhme U, van Ooij C, Otto TD, Janse CJ, Franke-Fayard B.
PMID: 28095481
PLoS Pathog. 2017 Jan 17;13(1):e1006128. doi: 10.1371/journal.ppat.1006128. eCollection 2017 Jan.

[This corrects the article DOI: 10.1371/journal.ppat.1005917.].

Cite
Fougère A, Jackson AP, Bechtsi DP, et al. Correction: Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous Vacuole. PLoS Pathog. 2017;13(1):e1006128doi: 10.1371/journal.ppat.1006128.
Fougère, A., Jackson, A. P., Bechtsi, D. P., Braks, J. A., Annoura, T., Fonager, J., Spaccapelo, R., Ramesar, J., Chevalley-Maurel, S., Klop, O., van der Laan, A. M., Tanke, H. J., Kocken, C. H., Pasini, E. M., Khan, S. M., Böhme, U., van Ooij, C., Otto, T. D., Janse, C. J., & Franke-Fayard, B. (2017). Correction: Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous Vacuole. PLoS pathogens, 13(1), e1006128. https://doi.org/10.1371/journal.ppat.1006128
Fougère, Aurélie, et al. "Correction: Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous Vacuole." PLoS pathogens vol. 13,1 (2017): e1006128. doi: https://doi.org/10.1371/journal.ppat.1006128
Fougère A, Jackson AP, Bechtsi DP, Braks JA, Annoura T, Fonager J, Spaccapelo R, Ramesar J, Chevalley-Maurel S, Klop O, van der Laan AM, Tanke HJ, Kocken CH, Pasini EM, Khan SM, Böhme U, van Ooij C, Otto TD, Janse CJ, Franke-Fayard B. Correction: Variant Exported Blood-Stage Proteins Encoded by Plasmodium Multigene Families Are Expressed in Liver Stages Where They Are Exported into the Parasitophorous Vacuole. PLoS Pathog. 2017 Jan 17;13(1):e1006128. doi: 10.1371/journal.ppat.1006128. eCollection 2017 Jan. PMID: 28095481; PMCID: PMC5240906.
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Correction: Long-term persistence and function of hematopoietic stem cell-derived chimeric antigen receptor T cells in a nonhuman primate model of HIV/AIDS.

PLoS pathogens

Zhen A, Peterson CW, Carrillo MA, Reddy SS, Youn CS, Lam BB, Chang NY, Martin HA, Rick JW, Kim J, Neel NC, Rezek VK, Kamata M, Chen ISY, Zack JA, Kiem HP, Kitchen SG.
PMID: 29529058
PLoS Pathog. 2018 Mar 12;14(3):e1006891. doi: 10.1371/journal.ppat.1006891. eCollection 2018 Mar.

[This corrects the article DOI: 10.1371/journal.ppat.1006753.].

Cite
Zhen A, Peterson CW, Carrillo MA, et al. Correction: Long-term persistence and function of hematopoietic stem cell-derived chimeric antigen receptor T cells in a nonhuman primate model of HIV/AIDS. PLoS Pathog. 2018;14(3):e1006891doi: 10.1371/journal.ppat.1006891.
Zhen, A., Peterson, C. W., Carrillo, M. A., Reddy, S. S., Youn, C. S., Lam, B. B., Chang, N. Y., Martin, H. A., Rick, J. W., Kim, J., Neel, N. C., Rezek, V. K., Kamata, M., Chen, I. S. Y., Zack, J. A., Kiem, H. P., & Kitchen, S. G. (2018). Correction: Long-term persistence and function of hematopoietic stem cell-derived chimeric antigen receptor T cells in a nonhuman primate model of HIV/AIDS. PLoS pathogens, 14(3), e1006891. https://doi.org/10.1371/journal.ppat.1006891
Zhen, Anjie, et al. "Correction: Long-term persistence and function of hematopoietic stem cell-derived chimeric antigen receptor T cells in a nonhuman primate model of HIV/AIDS." PLoS pathogens vol. 14,3 (2018): e1006891. doi: https://doi.org/10.1371/journal.ppat.1006891
Zhen A, Peterson CW, Carrillo MA, Reddy SS, Youn CS, Lam BB, Chang NY, Martin HA, Rick JW, Kim J, Neel NC, Rezek VK, Kamata M, Chen ISY, Zack JA, Kiem HP, Kitchen SG. Correction: Long-term persistence and function of hematopoietic stem cell-derived chimeric antigen receptor T cells in a nonhuman primate model of HIV/AIDS. PLoS Pathog. 2018 Mar 12;14(3):e1006891. doi: 10.1371/journal.ppat.1006891. eCollection 2018 Mar. PMID: 29529058; PMCID: PMC5846784.
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Correction: GP73 represses host innate immune response to promote virus replication by facilitating MAVS and TRAF6 degradation.

PLoS pathogens

Zhang X, Zhu C, Wang T, Jiang H, Ren Y, Zhang Q, Wu K, Liu F, Liu Y, Wu J.
PMID: 29534116
PLoS Pathog. 2018 Mar 13;14(3):e1006938. doi: 10.1371/journal.ppat.1006938. eCollection 2018 Mar.

[This corrects the article DOI: 10.1371/journal.ppat.1006321.].

Cite
Zhang X, Zhu C, Wang T, et al. Correction: GP73 represses host innate immune response to promote virus replication by facilitating MAVS and TRAF6 degradation. PLoS Pathog. 2018;14(3):e1006938doi: 10.1371/journal.ppat.1006938.
Zhang, X., Zhu, C., Wang, T., Jiang, H., Ren, Y., Zhang, Q., Wu, K., Liu, F., Liu, Y., & Wu, J. (2018). Correction: GP73 represses host innate immune response to promote virus replication by facilitating MAVS and TRAF6 degradation. PLoS pathogens, 14(3), e1006938. https://doi.org/10.1371/journal.ppat.1006938
Zhang, Xuewu, et al. "Correction: GP73 represses host innate immune response to promote virus replication by facilitating MAVS and TRAF6 degradation." PLoS pathogens vol. 14,3 (2018): e1006938. doi: https://doi.org/10.1371/journal.ppat.1006938
Zhang X, Zhu C, Wang T, Jiang H, Ren Y, Zhang Q, Wu K, Liu F, Liu Y, Wu J. Correction: GP73 represses host innate immune response to promote virus replication by facilitating MAVS and TRAF6 degradation. PLoS Pathog. 2018 Mar 13;14(3):e1006938. doi: 10.1371/journal.ppat.1006938. eCollection 2018 Mar. PMID: 29534116; PMCID: PMC5849327.
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Correction: A Large Collection of Novel Nematode-Infecting Microsporidia and Their Diverse Interactions with Caenorhabditis elegans and Other Related Nematodes.

PLoS pathogens

[No authors listed]
PMID: 28199389
PLoS Pathog. 2017 Feb 15;13(2):e1006204. doi: 10.1371/journal.ppat.1006204. eCollection 2017 Feb.

[This corrects the article DOI: 10.1371/journal.ppat.1006093.].

Cite
Correction: A Large Collection of Novel Nematode-Infecting Microsporidia and Their Diverse Interactions with Caenorhabditis elegans and Other Related Nematodes. PLoS Pathog. 2017;13(2):e1006204doi: 10.1371/journal.ppat.1006204.
(2017). Correction: A Large Collection of Novel Nematode-Infecting Microsporidia and Their Diverse Interactions with Caenorhabditis elegans and Other Related Nematodes. PLoS pathogens, 13(2), e1006204. https://doi.org/10.1371/journal.ppat.1006204
"Correction: A Large Collection of Novel Nematode-Infecting Microsporidia and Their Diverse Interactions with Caenorhabditis elegans and Other Related Nematodes." PLoS pathogens vol. 13,2 (2017): e1006204. doi: https://doi.org/10.1371/journal.ppat.1006204
Correction: A Large Collection of Novel Nematode-Infecting Microsporidia and Their Diverse Interactions with Caenorhabditis elegans and Other Related Nematodes. PLoS Pathog. 2017 Feb 15;13(2):e1006204. doi: 10.1371/journal.ppat.1006204. eCollection 2017 Feb. PMID: 28199389; PMCID: PMC5310755.
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Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells.

PLoS pathogens

Rizkallah G, Alais S, Futsch N, Tanaka Y, Journo C, Mahieux R, Dutartre H.
PMID: 28700737
PLoS Pathog. 2017 Jul 11;13(7):e1006494. doi: 10.1371/journal.ppat.1006494. eCollection 2017 Jul.

[This corrects the article DOI: 10.1371/journal.ppat.1006353.].

Cite
Rizkallah G, Alais S, Futsch N, et al. Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells. PLoS Pathog. 2017;13(7):e1006494doi: 10.1371/journal.ppat.1006494.
Rizkallah, G., Alais, S., Futsch, N., Tanaka, Y., Journo, C., Mahieux, R., & Dutartre, H. (2017). Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells. PLoS pathogens, 13(7), e1006494. https://doi.org/10.1371/journal.ppat.1006494
Rizkallah, Gergès, et al. "Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells." PLoS pathogens vol. 13,7 (2017): e1006494. doi: https://doi.org/10.1371/journal.ppat.1006494
Rizkallah G, Alais S, Futsch N, Tanaka Y, Journo C, Mahieux R, Dutartre H. Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells. PLoS Pathog. 2017 Jul 11;13(7):e1006494. doi: 10.1371/journal.ppat.1006494. eCollection 2017 Jul. PMID: 28700737; PMCID: PMC5507459.
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Correction: The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis.

PLoS pathogens

[No authors listed]
PMID: 26928711
PLoS Pathog. 2016 Feb 29;12(2):e1005406. doi: 10.1371/journal.ppat.1005406. eCollection 2016 Feb.

No abstract available.

Cite
Correction: The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis. PLoS Pathog. 2016;12(2):e1005406doi: 10.1371/journal.ppat.1005406.
(2016). Correction: The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis. PLoS pathogens, 12(2), e1005406. https://doi.org/10.1371/journal.ppat.1005406
"Correction: The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis." PLoS pathogens vol. 12,2 (2016): e1005406. doi: https://doi.org/10.1371/journal.ppat.1005406
Correction: The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis. PLoS Pathog. 2016 Feb 29;12(2):e1005406. doi: 10.1371/journal.ppat.1005406. eCollection 2016 Feb. PMID: 26928711; PMCID: PMC4771199.
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Showing 25 to 36 of 634 entries