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Multi-platform characterization of the human cerebrospinal fluid metabolome: a comprehensive and quantitative update.

Genome medicine

Mandal R, Guo AC, Chaudhary KK, Liu P, Yallou FS, Dong E, Aziat F, Wishart DS.
PMID: 22546835
Genome Med. 2012 Apr 30;4(4):38. doi: 10.1186/gm337.

BACKGROUND: Human cerebral spinal fluid (CSF) is known to be a rich source of small molecule biomarkers for neurological and neurodegenerative diseases. In 2007, we conducted a comprehensive metabolomic study and performed a detailed literature review on metabolites that...

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Mandal R, Guo AC, Chaudhary KK, et al. Multi-platform characterization of the human cerebrospinal fluid metabolome: a comprehensive and quantitative update. Genome Med. 2012;4(4):38doi: 10.1186/gm337.
Mandal, R., Guo, A. C., Chaudhary, K. K., Liu, P., Yallou, F. S., Dong, E., Aziat, F., & Wishart, D. S. (2012). Multi-platform characterization of the human cerebrospinal fluid metabolome: a comprehensive and quantitative update. Genome medicine, 4(4), 38. https://doi.org/10.1186/gm337
Mandal, Rupasri, et al. "Multi-platform characterization of the human cerebrospinal fluid metabolome: a comprehensive and quantitative update." Genome medicine vol. 4,4 (2012): 38. doi: https://doi.org/10.1186/gm337
Mandal R, Guo AC, Chaudhary KK, Liu P, Yallou FS, Dong E, Aziat F, Wishart DS. Multi-platform characterization of the human cerebrospinal fluid metabolome: a comprehensive and quantitative update. Genome Med. 2012 Apr 30;4(4):38. doi: 10.1186/gm337. PMID: 22546835; PMCID: PMC3446266.
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The Lipid Asset Is Unbalanced in Peripheral Nerve Sheath Tumors.

International journal of molecular sciences

Vetrano IG, Dei Cas M, Nazzi V, Eoli M, Innocenti N, Saletti V, Potenza A, Carrozzini T, Pollaci G, Gorla G, Paroni R, Ghidoni R, Gatti L.
PMID: 35008487
Int J Mol Sci. 2021 Dec 22;23(1). doi: 10.3390/ijms23010061.

Peripheral nerve sheath tumors (PNSTs) include schwannomas, neurofibromas (NFs), and plexiform neurofibromas (PNFs), among others. While they are benign tumors, according to their biological behavior, some have the potential for malignant degeneration, mainly PNFs. The specific factors contributing to...

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Vetrano IG, Dei Cas M, Nazzi V, et al. The Lipid Asset Is Unbalanced in Peripheral Nerve Sheath Tumors. Int J Mol Sci. 2021;23(1)doi: 10.3390/ijms23010061.
Vetrano, I. G., Dei Cas, M., Nazzi, V., Eoli, M., Innocenti, N., Saletti, V., Potenza, A., Carrozzini, T., Pollaci, G., Gorla, G., Paroni, R., Ghidoni, R., & Gatti, L. (2021). The Lipid Asset Is Unbalanced in Peripheral Nerve Sheath Tumors. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010061
Vetrano, Ignazio G, et al. "The Lipid Asset Is Unbalanced in Peripheral Nerve Sheath Tumors." International journal of molecular sciences vol. 23,1 (2021). doi: https://doi.org/10.3390/ijms23010061
Vetrano IG, Dei Cas M, Nazzi V, Eoli M, Innocenti N, Saletti V, Potenza A, Carrozzini T, Pollaci G, Gorla G, Paroni R, Ghidoni R, Gatti L. The Lipid Asset Is Unbalanced in Peripheral Nerve Sheath Tumors. Int J Mol Sci. 2021 Dec 22;23(1). doi: 10.3390/ijms23010061. PMID: 35008487.
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Plasma Metabolomics and Lipidomics Differentiate Obese Individuals by Peripheral Neuropathy Status.

The Journal of clinical endocrinology and metabolism

Guo K, Savelieff MG, Rumora AE, Alakwaa FM, Callaghan BC, Hur J, Feldman EL.
PMID: 34878536
J Clin Endocrinol Metab. 2021 Nov 20; doi: 10.1210/clinem/dgab844. Epub 2021 Nov 20.

CONTEXT: Peripheral neuropathy (PN) is a frequent prediabetes and type 2 diabetes (T2D) complication. Multiple clinical studies reveal that obesity and dyslipidemia can also drive PN progression, independent of glycemia, suggesting a complex interplay of specific metabolite and/or lipid...

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Guo K, Savelieff MG, Rumora AE, et al. Plasma Metabolomics and Lipidomics Differentiate Obese Individuals by Peripheral Neuropathy Status. J Clin Endocrinol Metab. 2021;doi: 10.1210/clinem/dgab844.
Guo, K., Savelieff, M. G., Rumora, A. E., Alakwaa, F. M., Callaghan, B. C., Hur, J., & Feldman, E. L. (2021). Plasma Metabolomics and Lipidomics Differentiate Obese Individuals by Peripheral Neuropathy Status. The Journal of clinical endocrinology and metabolism, . https://doi.org/10.1210/clinem/dgab844
Guo, Kai, et al. "Plasma Metabolomics and Lipidomics Differentiate Obese Individuals by Peripheral Neuropathy Status." The Journal of clinical endocrinology and metabolism vol. (2021). doi: https://doi.org/10.1210/clinem/dgab844
Guo K, Savelieff MG, Rumora AE, Alakwaa FM, Callaghan BC, Hur J, Feldman EL. Plasma Metabolomics and Lipidomics Differentiate Obese Individuals by Peripheral Neuropathy Status. J Clin Endocrinol Metab. 2021 Nov 20; doi: 10.1210/clinem/dgab844. Epub 2021 Nov 20. PMID: 34878536.
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Hypertrophy of the ligamentum flavum in lumbar spinal canal stenosis is associated with abnormal accumulation of specific lipids.

Scientific reports

Yamada T, Horikawa M, Sato T, Kahyo T, Takanashi Y, Ushirozako H, Kurosu K, Al Mamun M, Mihara Y, Oe S, Arima H, Banno T, Yosida G, Hasegawa T, Yamato Y, Matsuyama Y, Setou M.
PMID: 34873238
Sci Rep. 2021 Dec 06;11(1):23515. doi: 10.1038/s41598-021-02818-7.

Ligamentum flavum hypertrophy (HLF) is the most important component of lumbar spinal canal stenosis (LSCS). Analysis of hypertrophied ligamentum flavum (HLF) samples from patients with LSCS can be an important que. The current study analyzed the surgical samples of...

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Yamada T, Horikawa M, Sato T, et al. Hypertrophy of the ligamentum flavum in lumbar spinal canal stenosis is associated with abnormal accumulation of specific lipids. Sci Rep. 2021;11(1):23515doi: 10.1038/s41598-021-02818-7.
Yamada, T., Horikawa, M., Sato, T., Kahyo, T., Takanashi, Y., Ushirozako, H., Kurosu, K., Al Mamun, M., Mihara, Y., Oe, S., Arima, H., Banno, T., Yosida, G., Hasegawa, T., Yamato, Y., Matsuyama, Y., & Setou, M. (2021). Hypertrophy of the ligamentum flavum in lumbar spinal canal stenosis is associated with abnormal accumulation of specific lipids. Scientific reports, 11(1), 23515. https://doi.org/10.1038/s41598-021-02818-7
Yamada, Tomohiro, et al. "Hypertrophy of the ligamentum flavum in lumbar spinal canal stenosis is associated with abnormal accumulation of specific lipids." Scientific reports vol. 11,1 (2021): 23515. doi: https://doi.org/10.1038/s41598-021-02818-7
Yamada T, Horikawa M, Sato T, Kahyo T, Takanashi Y, Ushirozako H, Kurosu K, Al Mamun M, Mihara Y, Oe S, Arima H, Banno T, Yosida G, Hasegawa T, Yamato Y, Matsuyama Y, Setou M. Hypertrophy of the ligamentum flavum in lumbar spinal canal stenosis is associated with abnormal accumulation of specific lipids. Sci Rep. 2021 Dec 06;11(1):23515. doi: 10.1038/s41598-021-02818-7. PMID: 34873238; PMCID: PMC8648848.
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A secretory phospholipase D hydrolyzes phosphatidylcholine to suppress rice heading time.

PLoS genetics

Qu L, Chu YJ, Lin WH, Xue HW.
PMID: 34879072
PLoS Genet. 2021 Dec 08;17(12):e1009905. doi: 10.1371/journal.pgen.1009905. eCollection 2021 Dec.

Phospholipase D (PLD) hydrolyzes membrane phospholipids and is crucial in various physiological processes and transduction of different signals. Secretory phospholipases play important roles in mammals, however, whose functions in plants remain largely unknown. We previously identified a rice secretory...

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Qu L, Chu YJ, Lin WH, et al. A secretory phospholipase D hydrolyzes phosphatidylcholine to suppress rice heading time. PLoS Genet. 2021;17(12):e1009905doi: 10.1371/journal.pgen.1009905.
Qu, L., Chu, Y. J., Lin, W. H., & Xue, H. W. (2021). A secretory phospholipase D hydrolyzes phosphatidylcholine to suppress rice heading time. PLoS genetics, 17(12), e1009905. https://doi.org/10.1371/journal.pgen.1009905
Qu, Li, et al. "A secretory phospholipase D hydrolyzes phosphatidylcholine to suppress rice heading time." PLoS genetics vol. 17,12 (2021): e1009905. doi: https://doi.org/10.1371/journal.pgen.1009905
Qu L, Chu YJ, Lin WH, Xue HW. A secretory phospholipase D hydrolyzes phosphatidylcholine to suppress rice heading time. PLoS Genet. 2021 Dec 08;17(12):e1009905. doi: 10.1371/journal.pgen.1009905. eCollection 2021 Dec. PMID: 34879072; PMCID: PMC8654219.
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Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride.

Cardiovascular diabetology

Jendle J, Hyötyläinen T, Orešič M, Nyström T.
PMID: 34920733
Cardiovasc Diabetol. 2021 Dec 17;20(1):237. doi: 10.1186/s12933-021-01431-2.

BACKGROUND: Treatment with glucagon-like peptide-1 receptor agonists (GLP-1 RAs) leads to multiple metabolic changes, reduction in glucose levels and body weight are well established. In people with type 2 diabetes, GLP-1 RAs reduce the risk of cardiovascular (CV) disease...

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Jendle J, Hyötyläinen T, Orešič M, et al. Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovasc Diabetol. 2021;20(1):237doi: 10.1186/s12933-021-01431-2.
Jendle, J., Hyötyläinen, T., Orešič, M., & Nyström, T. (2021). Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovascular diabetology, 20(1), 237. https://doi.org/10.1186/s12933-021-01431-2
Jendle, J, et al. "Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride." Cardiovascular diabetology vol. 20,1 (2021): 237. doi: https://doi.org/10.1186/s12933-021-01431-2
Jendle J, Hyötyläinen T, Orešič M, Nyström T. Pharmacometabolomic profiles in type 2 diabetic subjects treated with liraglutide or glimepiride. Cardiovasc Diabetol. 2021 Dec 17;20(1):237. doi: 10.1186/s12933-021-01431-2. PMID: 34920733; PMCID: PMC8684205.
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Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios.

The journals of gerontology. Series A, Biological sciences and medical sciences

Di Cesare F, Luchinat C, Tenori L, Saccenti E.
PMID: 34748631
J Gerontol A Biol Sci Med Sci. 2021 Nov 08; doi: 10.1093/gerona/glab335. Epub 2021 Nov 08.

In this study we investigated how the concentrations, pairwise correlations, and ratios of 202 free circulating blood metabolites and lipids vary with age in a panel of n=1882 subjects ranging from 48 to 94 years. We report a statistically...

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Di Cesare F, Luchinat C, Tenori L, et al. Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. J Gerontol A Biol Sci Med Sci. 2021;doi: 10.1093/gerona/glab335.
Di Cesare, F., Luchinat, C., Tenori, L., & Saccenti, E. (2021). Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. The journals of gerontology. Series A, Biological sciences and medical sciences, . https://doi.org/10.1093/gerona/glab335
Di Cesare, Francesca, et al. "Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios." The journals of gerontology. Series A, Biological sciences and medical sciences vol. (2021). doi: https://doi.org/10.1093/gerona/glab335
Di Cesare F, Luchinat C, Tenori L, Saccenti E. Age and sex dependent changes of free circulating blood metabolite and lipid abundances, correlations and ratios. J Gerontol A Biol Sci Med Sci. 2021 Nov 08; doi: 10.1093/gerona/glab335. Epub 2021 Nov 08. PMID: 34748631.
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Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages.

Biomedicines

Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, Baquero M, Cháfer-Pericás C.
PMID: 34944628
Biomedicines. 2021 Dec 02;9(12). doi: 10.3390/biomedicines9121812.

BACKGROUND: Alzheimer Disease (AD) is the most prevalent dementia. However, the physiopathological mechanisms involved in its development are unclear. In this sense, a multi-omics approach could provide some progress.METHODS: Epigenomic and lipidomic analysis were carried out in plasma samples...

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Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, et al. Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines. 2021;9(12)doi: 10.3390/biomedicines9121812.
Peña-Bautista, C., Álvarez-Sánchez, L., Cañada-Martínez, A. J., Baquero, M., & Cháfer-Pericás, C. (2021). Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines, 9(12), . https://doi.org/10.3390/biomedicines9121812
Peña-Bautista, Carmen, et al. "Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages." Biomedicines vol. 9,12 (2021). doi: https://doi.org/10.3390/biomedicines9121812
Peña-Bautista C, Álvarez-Sánchez L, Cañada-Martínez AJ, Baquero M, Cháfer-Pericás C. Epigenomics and Lipidomics Integration in Alzheimer Disease: Pathways Involved in Early Stages. Biomedicines. 2021 Dec 02;9(12). doi: 10.3390/biomedicines9121812. PMID: 34944628; PMCID: PMC8698767.
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Metabolomic Profiling of Blood-Derived Microvesicles in Breast Cancer Patients.

International journal of molecular sciences

Buentzel J, Klemp HG, Kraetzner R, Schulz M, Dihazi GH, Streit F, Bleckmann A, Menck K, Wlochowitz D, Binder C.
PMID: 34948336
Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413540.

Malignant cells differ from benign ones in their metabolome and it is largely unknown whether this difference is reflected in the metabolic profile of their microvesicles (MV), which are secreted into the blood of cancer patients. Here, they are...

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Buentzel J, Klemp HG, Kraetzner R, et al. Metabolomic Profiling of Blood-Derived Microvesicles in Breast Cancer Patients. Int J Mol Sci. 2021;22(24)doi: 10.3390/ijms222413540.
Buentzel, J., Klemp, H. G., Kraetzner, R., Schulz, M., Dihazi, G. H., Streit, F., Bleckmann, A., Menck, K., Wlochowitz, D., & Binder, C. (2021). Metabolomic Profiling of Blood-Derived Microvesicles in Breast Cancer Patients. International journal of molecular sciences, 22(24), . https://doi.org/10.3390/ijms222413540
Buentzel, Judith, et al. "Metabolomic Profiling of Blood-Derived Microvesicles in Breast Cancer Patients." International journal of molecular sciences vol. 22,24 (2021). doi: https://doi.org/10.3390/ijms222413540
Buentzel J, Klemp HG, Kraetzner R, Schulz M, Dihazi GH, Streit F, Bleckmann A, Menck K, Wlochowitz D, Binder C. Metabolomic Profiling of Blood-Derived Microvesicles in Breast Cancer Patients. Int J Mol Sci. 2021 Dec 17;22(24). doi: 10.3390/ijms222413540. PMID: 34948336; PMCID: PMC8707654.
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Extending the Lipidome Coverage by Combining Different Mass Spectrometric Platforms: An Innovative Strategy to Answer Chemical Food Safety Issues.

Foods (Basel, Switzerland)

Marchand J, Guitton Y, Martineau E, Royer AL, Balgoma D, Le Bizec B, Giraudeau P, Dervilly G.
PMID: 34071212
Foods. 2021 May 28;10(6). doi: 10.3390/foods10061218.

From a general public health perspective, a strategy combining non-targeted and targeted lipidomics MS-based approaches is proposed to identify disrupted patterns in serum lipidome upon growth promoter treatment in pigs. Evaluating the relative contributions of the platforms involved, the...

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Marchand J, Guitton Y, Martineau E, et al. Extending the Lipidome Coverage by Combining Different Mass Spectrometric Platforms: An Innovative Strategy to Answer Chemical Food Safety Issues. Foods. 2021;10(6)doi: 10.3390/foods10061218.
Marchand, J., Guitton, Y., Martineau, E., Royer, A. L., Balgoma, D., Le Bizec, B., Giraudeau, P., & Dervilly, G. (2021). Extending the Lipidome Coverage by Combining Different Mass Spectrometric Platforms: An Innovative Strategy to Answer Chemical Food Safety Issues. Foods (Basel, Switzerland), 10(6), . https://doi.org/10.3390/foods10061218
Marchand, Jérémy, et al. "Extending the Lipidome Coverage by Combining Different Mass Spectrometric Platforms: An Innovative Strategy to Answer Chemical Food Safety Issues." Foods (Basel, Switzerland) vol. 10,6 (2021). doi: https://doi.org/10.3390/foods10061218
Marchand J, Guitton Y, Martineau E, Royer AL, Balgoma D, Le Bizec B, Giraudeau P, Dervilly G. Extending the Lipidome Coverage by Combining Different Mass Spectrometric Platforms: An Innovative Strategy to Answer Chemical Food Safety Issues. Foods. 2021 May 28;10(6). doi: 10.3390/foods10061218. PMID: 34071212; PMCID: PMC8230090.
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Lipid profiling in malignant mesothelioma reveals promising signatures for diagnosis and prognosis: A plasma-based LC-MS lipidomics study.

Clinica chimica acta; international journal of clinical chemistry

Chen Z, Song S, Yang C, Dai Z, Gao Y, Li N, Zhu J, Mao W, Liu J.
PMID: 34843704
Clin Chim Acta. 2022 Jan 01;524:34-42. doi: 10.1016/j.cca.2021.11.024. Epub 2021 Nov 27.

BACKGROUND AND AIM: Malignant mesothelioma (MM), being a rare and aggressive carcinoma, can barely be cured. Incidence of this cancer will keep climbing up in the next few decades since its major carcinogen, asbestos, is still in use in...

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Chen Z, Song S, Yang C, et al. Lipid profiling in malignant mesothelioma reveals promising signatures for diagnosis and prognosis: A plasma-based LC-MS lipidomics study. Clin Chim Acta. 2021;524:34-42doi: 10.1016/j.cca.2021.11.024.
Chen, Z., Song, S., Yang, C., Dai, Z., Gao, Y., Li, N., Zhu, J., Mao, W., & Liu, J. (2022). Lipid profiling in malignant mesothelioma reveals promising signatures for diagnosis and prognosis: A plasma-based LC-MS lipidomics study. Clinica chimica acta; international journal of clinical chemistry, 52434-42. https://doi.org/10.1016/j.cca.2021.11.024
Chen, Zhongjian, et al. "Lipid profiling in malignant mesothelioma reveals promising signatures for diagnosis and prognosis: A plasma-based LC-MS lipidomics study." Clinica chimica acta; international journal of clinical chemistry vol. 524 (2022): 34-42. doi: https://doi.org/10.1016/j.cca.2021.11.024
Chen Z, Song S, Yang C, Dai Z, Gao Y, Li N, Zhu J, Mao W, Liu J. Lipid profiling in malignant mesothelioma reveals promising signatures for diagnosis and prognosis: A plasma-based LC-MS lipidomics study. Clin Chim Acta. 2022 Jan 01;524:34-42. doi: 10.1016/j.cca.2021.11.024. Epub 2021 Nov 27. PMID: 34843704.
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Comprehensive metabolomics profiling reveals common metabolic alterations underlying the four major non-communicable diseases in treated HIV infection.

EBioMedicine

Ding Y, Lin H, Chen X, Zhu B, Xu X, Xu X, Shen W, Gao M, He N.
PMID: 34419928
EBioMedicine. 2021 Sep;71:103548. doi: 10.1016/j.ebiom.2021.103548. Epub 2021 Aug 19.

BACKGROUND: HIV infection and normal aging share immune and inflammatory changes that result in premature aging and non-communicable diseases (NCDs), but the exact pathophysiology is not yet uncovered. We identified the common metabolic pathways underlying various NCDs in treated...

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Ding Y, Lin H, Chen X, et al. Comprehensive metabolomics profiling reveals common metabolic alterations underlying the four major non-communicable diseases in treated HIV infection. EBioMedicine. 2021;71:103548doi: 10.1016/j.ebiom.2021.103548.
Ding, Y., Lin, H., Chen, X., Zhu, B., Xu, X., Xu, X., Shen, W., Gao, M., & He, N. (2021). Comprehensive metabolomics profiling reveals common metabolic alterations underlying the four major non-communicable diseases in treated HIV infection. EBioMedicine, 71103548. https://doi.org/10.1016/j.ebiom.2021.103548
Ding, Yingying, et al. "Comprehensive metabolomics profiling reveals common metabolic alterations underlying the four major non-communicable diseases in treated HIV infection." EBioMedicine vol. 71 (2021): 103548. doi: https://doi.org/10.1016/j.ebiom.2021.103548
Ding Y, Lin H, Chen X, Zhu B, Xu X, Xu X, Shen W, Gao M, He N. Comprehensive metabolomics profiling reveals common metabolic alterations underlying the four major non-communicable diseases in treated HIV infection. EBioMedicine. 2021 Sep;71:103548. doi: 10.1016/j.ebiom.2021.103548. Epub 2021 Aug 19. PMID: 34419928; PMCID: PMC8385138.
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Showing 25 to 36 of 283 entries