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Effects of triazolodiazepine on the production of interleukin-6 from murine spleen cells and rabbit synovial cells in vitro.

Mediators of inflammation

Ju DW, Zheng QY, Cao XT, Zheng QH, Guan XJ, Wang HB.
PMID: 18475629
Mediators Inflamm. 1995;4(2):130-2. doi: 10.1155/S0962935195000226.

Interleukin-6 (IL-6) is a multifunctional cytokine that regulates the immune response, acute phase anaphylactic reaction, and haematopoiesis. Lipopolysaccharide (6-24 mug/ml) significantly induced IL-6 release from murine spleen cells. In cultured rabbit synovial cells interleukin-1 (IL-1, 1-10 U/ml) induced IL-6...

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Ju DW, Zheng QY, Cao XT, et al. Effects of triazolodiazepine on the production of interleukin-6 from murine spleen cells and rabbit synovial cells in vitro. Mediators Inflamm. 1995;4(2):130-2doi: 10.1155/S0962935195000226.
Ju, D. W., Zheng, Q. Y., Cao, X. T., Zheng, Q. H., Guan, X. J., & Wang, H. B. (1995). Effects of triazolodiazepine on the production of interleukin-6 from murine spleen cells and rabbit synovial cells in vitro. Mediators of inflammation, 4(2), 130-2. https://doi.org/10.1155/S0962935195000226
Ju, D W, et al. "Effects of triazolodiazepine on the production of interleukin-6 from murine spleen cells and rabbit synovial cells in vitro." Mediators of inflammation vol. 4,2 (1995): 130-2. doi: https://doi.org/10.1155/S0962935195000226
Ju DW, Zheng QY, Cao XT, Zheng QH, Guan XJ, Wang HB. Effects of triazolodiazepine on the production of interleukin-6 from murine spleen cells and rabbit synovial cells in vitro. Mediators Inflamm. 1995;4(2):130-2. doi: 10.1155/S0962935195000226. PMID: 18475629; PMCID: PMC2365624.
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Platelet-activating factor alters the dynamics of prostaglandin and protein synthesis by endometrial explants from pregnant and cyclic cows at day 17 following estrus.

Theriogenology

Gross TS, Thatcher WW, O'Neill C, Danet-Desnoyers G.
PMID: 16726831
Theriogenology. 1990 Aug;34(2):205-18. doi: 10.1016/0093-691x(90)90515-u.

Factors produced by bovine conceptuses alter prostaglandin (PG) and protein secretion by endometrial explants from cyclic cows and induce an intracellular inhibitor of PG synthesis. Endometrial explants from cyclic (n = 4) and pregnant (n = 3) cows at...

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Gross TS, Thatcher WW, O'Neill C, et al. Platelet-activating factor alters the dynamics of prostaglandin and protein synthesis by endometrial explants from pregnant and cyclic cows at day 17 following estrus. Theriogenology. 1990;34(2):205-18doi: 10.1016/0093-691x(90)90515-u.
Gross, T. S., Thatcher, W. W., O'Neill, C., & Danet-Desnoyers, G. (1990). Platelet-activating factor alters the dynamics of prostaglandin and protein synthesis by endometrial explants from pregnant and cyclic cows at day 17 following estrus. Theriogenology, 34(2), 205-18. https://doi.org/10.1016/0093-691x(90)90515-u
Gross, T S, et al. "Platelet-activating factor alters the dynamics of prostaglandin and protein synthesis by endometrial explants from pregnant and cyclic cows at day 17 following estrus." Theriogenology vol. 34,2 (1990): 205-18. doi: https://doi.org/10.1016/0093-691x(90)90515-u
Gross TS, Thatcher WW, O'Neill C, Danet-Desnoyers G. Platelet-activating factor alters the dynamics of prostaglandin and protein synthesis by endometrial explants from pregnant and cyclic cows at day 17 following estrus. Theriogenology. 1990 Aug;34(2):205-18. doi: 10.1016/0093-691x(90)90515-u. PMID: 16726831.
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The use of gene expression analysis and proteomic databases in the development of a screening system to determine the value of natural medicinal products.

Evidence-based complementary and alternative medicine : eCAM

Katz S, Harris R, Lau JT, Chau A.
PMID: 16550225
Evid Based Complement Alternat Med. 2006 Mar;3(1):65-70. doi: 10.1093/ecam/nek001. Epub 2006 Jan 16.

A rapid throughput screening system involving gene expression analysis was developed in order to investigate the potential of bioactive chemicals contained in natural health products as effective drug therapy, in particular the ability of these chemicals to alleviate the...

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Katz S, Harris R, Lau JT, et al. The use of gene expression analysis and proteomic databases in the development of a screening system to determine the value of natural medicinal products. Evid Based Complement Alternat Med. 2006;3(1):65-70doi: 10.1093/ecam/nek001.
Katz, S., Harris, R., Lau, J. T., & Chau, A. (2006). The use of gene expression analysis and proteomic databases in the development of a screening system to determine the value of natural medicinal products. Evidence-based complementary and alternative medicine : eCAM, 3(1), 65-70. https://doi.org/10.1093/ecam/nek001
Katz, Sidney, et al. "The use of gene expression analysis and proteomic databases in the development of a screening system to determine the value of natural medicinal products." Evidence-based complementary and alternative medicine : eCAM vol. 3,1 (2006): 65-70. doi: https://doi.org/10.1093/ecam/nek001
Katz S, Harris R, Lau JT, Chau A. The use of gene expression analysis and proteomic databases in the development of a screening system to determine the value of natural medicinal products. Evid Based Complement Alternat Med. 2006 Mar;3(1):65-70. doi: 10.1093/ecam/nek001. Epub 2006 Jan 16. PMID: 16550225; PMCID: PMC1375224.
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Apolipoprotein M.

Lipids in health and disease

Luo G, Zhang X, Nilsson-Ehle P, Xu N.
PMID: 15461812
Lipids Health Dis. 2004 Oct 04;3:21. doi: 10.1186/1476-511X-3-21.

Apolipoprotein M (apoM) is a 26-kDa protein that is mainly associated with high-density lipoprotein (HDL) in human plasma, with a small proportion present in triglyceride-rich lipoproteins (TGRLP) and low-density lipoproteins (LDL). Human apoM gene is located in p21.31 on...

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Luo G, Zhang X, Nilsson-Ehle P, et al. Apolipoprotein M. Lipids Health Dis. 2004;3:21doi: 10.1186/1476-511X-3-21.
Luo, G., Zhang, X., Nilsson-Ehle, P., & Xu, N. (2004). Apolipoprotein M. Lipids in health and disease, 321. https://doi.org/10.1186/1476-511X-3-21
Luo, Guanghua, et al. "Apolipoprotein M." Lipids in health and disease vol. 3 (2004): 21. doi: https://doi.org/10.1186/1476-511X-3-21
Luo G, Zhang X, Nilsson-Ehle P, Xu N. Apolipoprotein M. Lipids Health Dis. 2004 Oct 04;3:21. doi: 10.1186/1476-511X-3-21. PMID: 15461812; PMCID: PMC523857.
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Hypothesis: hemolytic transfusion reactions represent an alternative type of anaphylaxis.

International journal of clinical and experimental pathology

Hod EA, Sokol SA, Zimring JC, Spitalnik SL.
PMID: 18830382
Int J Clin Exp Pathol. 2009;2(1):71-82. Epub 2008 May 30.

Classical anaphylaxis is the most severe, and potentially fatal, type of allergic reaction, manifested by hypotension, bronchoconstriction, and vascular permeability. Similarly, a hemolytic transfusion reaction (HTR) is the most feared consequence of blood transfusion. Evidence for the existence of...

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Hod EA, Sokol SA, Zimring JC, et al. Hypothesis: hemolytic transfusion reactions represent an alternative type of anaphylaxis. Int J Clin Exp Pathol. 2008;2(1):71-82
Hod, E. A., Sokol, S. A., Zimring, J. C., & Spitalnik, S. L. (2009). Hypothesis: hemolytic transfusion reactions represent an alternative type of anaphylaxis. International journal of clinical and experimental pathology, 2(1), 71-82.
Hod, Eldad A, et al. "Hypothesis: hemolytic transfusion reactions represent an alternative type of anaphylaxis." International journal of clinical and experimental pathology vol. 2,1 (2009): 71-82.
Hod EA, Sokol SA, Zimring JC, Spitalnik SL. Hypothesis: hemolytic transfusion reactions represent an alternative type of anaphylaxis. Int J Clin Exp Pathol. 2009;2(1):71-82. Epub 2008 May 30. PMID: 18830382; PMCID: PMC2491388.
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Drug therapy for ocular allergy.

Expert opinion on investigational drugs

Hingorani M.
PMID: 15991917
Expert Opin Investig Drugs. 1998 Jan;7(1):27-55. doi: 10.1517/13543784.7.1.27.

The spectrum of ocular allergy ranges from mild, non-sight threatening disease, such as hay fever, to disorders such as atopic keratoconjunctivitis (AKC) which cause permanent ocular surface changes and reduced vision. The ideal treatment is with topical preparations. Launched...

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Hingorani M. Drug therapy for ocular allergy. Expert Opin Investig Drugs. 1998;7(1):27-55doi: 10.1517/13543784.7.1.27.
Hingorani, M. (1998). Drug therapy for ocular allergy. Expert opinion on investigational drugs, 7(1), 27-55. https://doi.org/10.1517/13543784.7.1.27
Hingorani, M. "Drug therapy for ocular allergy." Expert opinion on investigational drugs vol. 7,1 (1998): 27-55. doi: https://doi.org/10.1517/13543784.7.1.27
Hingorani M. Drug therapy for ocular allergy. Expert Opin Investig Drugs. 1998 Jan;7(1):27-55. doi: 10.1517/13543784.7.1.27. PMID: 15991917.
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Interferon-gamma increases neuronal death in response to amyloid-beta1-42.

Journal of neuroinflammation

Bate C, Kempster S, Last V, Williams A.
PMID: 16569229
J Neuroinflammation. 2006 Mar 28;3:7. doi: 10.1186/1742-2094-3-7.

BACKGROUND: Alzheimer's disease is a neurodegenerative disorder characterized by a progressive cognitive impairment, the consequence of neuronal dysfunction and ultimately the death of neurons. The amyloid hypothesis proposes that neuronal damage results from the accumulation of insoluble, hydrophobic, fibrillar...

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Bate C, Kempster S, Last V, et al. Interferon-gamma increases neuronal death in response to amyloid-beta1-42. J Neuroinflammation. 2006;3:7doi: 10.1186/1742-2094-3-7.
Bate, C., Kempster, S., Last, V., & Williams, A. (2006). Interferon-gamma increases neuronal death in response to amyloid-beta1-42. Journal of neuroinflammation, 37. https://doi.org/10.1186/1742-2094-3-7
Bate, Clive, et al. "Interferon-gamma increases neuronal death in response to amyloid-beta1-42." Journal of neuroinflammation vol. 3 (2006): 7. doi: https://doi.org/10.1186/1742-2094-3-7
Bate C, Kempster S, Last V, Williams A. Interferon-gamma increases neuronal death in response to amyloid-beta1-42. J Neuroinflammation. 2006 Mar 28;3:7. doi: 10.1186/1742-2094-3-7. PMID: 16569229; PMCID: PMC1435873.
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Cardiovascular effects of platelet-activating factor.

Trends in cardiovascular medicine

Holland MR, McIntyre TM, Zimmerman GA, Prescott SM.
PMID: 21239326
Trends Cardiovasc Med. 1991 Apr;1(3):117-21. doi: 10.1016/1050-1738(91)90005-Y.

Platelet-activating factor (PAF) is a unique biologically active phospholipid that is synthesized by many cells and tissues and has actions far more diverse than just platelet activation. PAF is probably a mediator of the normal inflammatory and thrombotic responses....

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Holland MR, McIntyre TM, Zimmerman GA, et al. Cardiovascular effects of platelet-activating factor. Trends Cardiovasc Med. 1991;1(3):117-21doi: 10.1016/1050-1738(91)90005-Y.
Holland, M. R., McIntyre, T. M., Zimmerman, G. A., & Prescott, S. M. (1991). Cardiovascular effects of platelet-activating factor. Trends in cardiovascular medicine, 1(3), 117-21. https://doi.org/10.1016/1050-1738(91)90005-Y
Holland, M R, et al. "Cardiovascular effects of platelet-activating factor." Trends in cardiovascular medicine vol. 1,3 (1991): 117-21. doi: https://doi.org/10.1016/1050-1738(91)90005-Y
Holland MR, McIntyre TM, Zimmerman GA, Prescott SM. Cardiovascular effects of platelet-activating factor. Trends Cardiovasc Med. 1991 Apr;1(3):117-21. doi: 10.1016/1050-1738(91)90005-Y. PMID: 21239326.
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Nongenomic actions of progesterone on human spermatozoa.

Trends in endocrinology and metabolism: TEM

Baldi E, Krausz C, Forti G.
PMID: 18406702
Trends Endocrinol Metab. 1995 Aug;6(6):198-205. doi: 10.1016/1043-2760(95)00083-t.

Progesterone's (P) stimulatory actions on human spermatozoa have been known for many years. P indeed appears to be the main sperm stimulator present in women's biological fluids, particularly the follicular fluid. The nongenomic nature of the biological effects of...

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Baldi E, Krausz C, Forti G. Nongenomic actions of progesterone on human spermatozoa. Trends Endocrinol Metab. 1995;6(6):198-205doi: 10.1016/1043-2760(95)00083-t.
Baldi, E., Krausz, C., & Forti, G. (1995). Nongenomic actions of progesterone on human spermatozoa. Trends in endocrinology and metabolism: TEM, 6(6), 198-205. https://doi.org/10.1016/1043-2760(95)00083-t
Baldi, E, et al. "Nongenomic actions of progesterone on human spermatozoa." Trends in endocrinology and metabolism: TEM vol. 6,6 (1995): 198-205. doi: https://doi.org/10.1016/1043-2760(95)00083-t
Baldi E, Krausz C, Forti G. Nongenomic actions of progesterone on human spermatozoa. Trends Endocrinol Metab. 1995 Aug;6(6):198-205. doi: 10.1016/1043-2760(95)00083-t. PMID: 18406702.
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Nitric oxide decreases intestinal haemorrhagic lesions in rat anaphylaxis independently of mast cell activation.

Mediators of inflammation

Tavares JC, Moreno A, Crespo MS.
PMID: 18472830
Mediators Inflamm. 1997;6(1):25-31. doi: 10.1080/09629359791893.

The purpose of this study is to assess the role of nitric oxide (NO) in the intestinal lesions of passive anaphylaxis, since this experimental model resembles necrotizing enterocolitis. Sprague-Dawley rats were sensitized with IgE anti-dinitrophenol monoclonal antibody. Extravasation of...

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Tavares JC, Moreno A, Crespo MS. Nitric oxide decreases intestinal haemorrhagic lesions in rat anaphylaxis independently of mast cell activation. Mediators Inflamm. 1997;6(1):25-31doi: 10.1080/09629359791893.
Tavares, J. C., Moreno, A., & Crespo, M. S. (1997). Nitric oxide decreases intestinal haemorrhagic lesions in rat anaphylaxis independently of mast cell activation. Mediators of inflammation, 6(1), 25-31. https://doi.org/10.1080/09629359791893
Tavares, J C, et al. "Nitric oxide decreases intestinal haemorrhagic lesions in rat anaphylaxis independently of mast cell activation." Mediators of inflammation vol. 6,1 (1997): 25-31. doi: https://doi.org/10.1080/09629359791893
Tavares JC, Moreno A, Crespo MS. Nitric oxide decreases intestinal haemorrhagic lesions in rat anaphylaxis independently of mast cell activation. Mediators Inflamm. 1997;6(1):25-31. doi: 10.1080/09629359791893. PMID: 18472830; PMCID: PMC2365848.
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Involvement of A pertussis Toxin Sensitive G-Protein in the Inhibition of Inwardly Rectifying K Currents by Platelet-Activating Factor in Guinea-Pig Atrial Cardiomyocytes.

Mediators of inflammation

Gollasch M, Kleppisch T, Krautwurst D, Lewinsohn D, Hescheler J.
PMID: 18472923
Mediators Inflamm. 1994;3(1):45-51. doi: 10.1155/S0962935194000086.

Platelet-activating factor (PAF) inhibits single inwardly rectifying K(+) channels in guinea-pig ventricular cells. There is currently little information as to the mechanism by which these channels are modulated. The effect of PAF on quasi steady-state inwardly rectifying K(+) currents...

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Gollasch M, Kleppisch T, Krautwurst D, et al. Involvement of A pertussis Toxin Sensitive G-Protein in the Inhibition of Inwardly Rectifying K Currents by Platelet-Activating Factor in Guinea-Pig Atrial Cardiomyocytes. Mediators Inflamm. 1994;3(1):45-51doi: 10.1155/S0962935194000086.
Gollasch, M., Kleppisch, T., Krautwurst, D., Lewinsohn, D., & Hescheler, J. (1994). Involvement of A pertussis Toxin Sensitive G-Protein in the Inhibition of Inwardly Rectifying K Currents by Platelet-Activating Factor in Guinea-Pig Atrial Cardiomyocytes. Mediators of inflammation, 3(1), 45-51. https://doi.org/10.1155/S0962935194000086
Gollasch, M, et al. "Involvement of A pertussis Toxin Sensitive G-Protein in the Inhibition of Inwardly Rectifying K Currents by Platelet-Activating Factor in Guinea-Pig Atrial Cardiomyocytes." Mediators of inflammation vol. 3,1 (1994): 45-51. doi: https://doi.org/10.1155/S0962935194000086
Gollasch M, Kleppisch T, Krautwurst D, Lewinsohn D, Hescheler J. Involvement of A pertussis Toxin Sensitive G-Protein in the Inhibition of Inwardly Rectifying K Currents by Platelet-Activating Factor in Guinea-Pig Atrial Cardiomyocytes. Mediators Inflamm. 1994;3(1):45-51. doi: 10.1155/S0962935194000086. PMID: 18472923; PMCID: PMC2367020.
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5-[6-1 -(Cyclohexyl-1 H-tetrazol-5-YL)hexyl]-1,8-naphthyridin-2-(1H)-one, SC-44368, a Potent Anti-aggregatory Agent which Selectively Inhibits Platelet Cyclic AMP Phosphodiesterase.

Platelets

Booth RF, Manley PW, Buckham SP, Hassall DG, Honey AC, Lad N, Lunt DO, Oswald S, Porter RA, Tuffin DP.
PMID: 21043905
Platelets. 1992;3(3):129-36. doi: 10.3109/09537109209013173.

SC-44368 (5-[6-(1-cyclohexyl-1H-tetrazol-5-y)hexyl]-1,8-naphthyridin-2(1H)-one) is a potent and selective competitive inhibitor of platelet cyclic AMP-dependent phosphodiesterase (cAMP-PDE) (Ki: 1.65 μM). For the phosphodiesterase isoenzyms from human platelets SC-44368 shows a 26-fold selectivity (IC50 ratio) for the inhibition of the cAMP-PDE over...

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Booth RF, Manley PW, Buckham SP, et al. 5-[6-1 -(Cyclohexyl-1 H-tetrazol-5-YL)hexyl]-1,8-naphthyridin-2-(1H)-one, SC-44368, a Potent Anti-aggregatory Agent which Selectively Inhibits Platelet Cyclic AMP Phosphodiesterase. Platelets. 1992;3(3):129-36doi: 10.3109/09537109209013173.
Booth, R. F., Manley, P. W., Buckham, S. P., Hassall, D. G., Honey, A. C., Lad, N., Lunt, D. O., Oswald, S., Porter, R. A., & Tuffin, D. P. (1992). 5-[6-1 -(Cyclohexyl-1 H-tetrazol-5-YL)hexyl]-1,8-naphthyridin-2-(1H)-one, SC-44368, a Potent Anti-aggregatory Agent which Selectively Inhibits Platelet Cyclic AMP Phosphodiesterase. Platelets, 3(3), 129-36. https://doi.org/10.3109/09537109209013173
Booth, R F, et al. "5-[6-1 -(Cyclohexyl-1 H-tetrazol-5-YL)hexyl]-1,8-naphthyridin-2-(1H)-one, SC-44368, a Potent Anti-aggregatory Agent which Selectively Inhibits Platelet Cyclic AMP Phosphodiesterase." Platelets vol. 3,3 (1992): 129-36. doi: https://doi.org/10.3109/09537109209013173
Booth RF, Manley PW, Buckham SP, Hassall DG, Honey AC, Lad N, Lunt DO, Oswald S, Porter RA, Tuffin DP. 5-[6-1 -(Cyclohexyl-1 H-tetrazol-5-YL)hexyl]-1,8-naphthyridin-2-(1H)-one, SC-44368, a Potent Anti-aggregatory Agent which Selectively Inhibits Platelet Cyclic AMP Phosphodiesterase. Platelets. 1992;3(3):129-36. doi: 10.3109/09537109209013173. PMID: 21043905.
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Showing 13 to 24 of 366 entries