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Efficient Generation of P53 Biallelic Mutations in Diannan Miniature Pigs Using RNA-Guided Base Editing.

Life (Basel, Switzerland)

Li H, Cheng W, Chen B, Pu S, Fan N, Zhang X, Jiao D, Shi D, Guo J, Li Z, Qing Y, Jia B, Zhao HY, Wei HJ.
PMID: 34947951
Life (Basel). 2021 Dec 17;11(12). doi: 10.3390/life11121417.

The base editing 3 (BE3) system, a single-base gene editing technology developed using CRISPR/Cas9n, has a broad range of applications for human disease model construction and gene therapy, as it is highly efficient, accurate, and non-destructive. P53 mutations are...

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Li H, Cheng W, Chen B, et al. Efficient Generation of P53 Biallelic Mutations in Diannan Miniature Pigs Using RNA-Guided Base Editing. Life (Basel). 2021;11(12)doi: 10.3390/life11121417.
Li, H., Cheng, W., Chen, B., Pu, S., Fan, N., Zhang, X., Jiao, D., Shi, D., Guo, J., Li, Z., Qing, Y., Jia, B., Zhao, H. Y., & Wei, H. J. (2021). Efficient Generation of P53 Biallelic Mutations in Diannan Miniature Pigs Using RNA-Guided Base Editing. Life (Basel, Switzerland), 11(12), . https://doi.org/10.3390/life11121417
Li, Honghui, et al. "Efficient Generation of P53 Biallelic Mutations in Diannan Miniature Pigs Using RNA-Guided Base Editing." Life (Basel, Switzerland) vol. 11,12 (2021). doi: https://doi.org/10.3390/life11121417
Li H, Cheng W, Chen B, Pu S, Fan N, Zhang X, Jiao D, Shi D, Guo J, Li Z, Qing Y, Jia B, Zhao HY, Wei HJ. Efficient Generation of P53 Biallelic Mutations in Diannan Miniature Pigs Using RNA-Guided Base Editing. Life (Basel). 2021 Dec 17;11(12). doi: 10.3390/life11121417. PMID: 34947951; PMCID: PMC8706133.
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Establishment of the glioma polyploid giant cancer cell model by a modified PHA-DMSO-PEG fusion method following dual drug-fluorescence screening in vitro.

Journal of neuroscience methods

Mi R, Ji J, Zhang M, Zhang J, Li M, Hu Y, Liu F.
PMID: 34968625
J Neurosci Methods. 2021 Dec 28;368:109462. doi: 10.1016/j.jneumeth.2021.109462. Epub 2021 Dec 28.

BACKGROUND: In glioma, cell fusion and the number of the polyploid giant cancer cells (PGCC) were found to be augmented with tumor grades (WHO Ⅰ-Ⅳ) and closely related to poor prognosis. However, the pathological and molecular characteristics of glioma...

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Mi R, Ji J, Zhang M, et al. Establishment of the glioma polyploid giant cancer cell model by a modified PHA-DMSO-PEG fusion method following dual drug-fluorescence screening in vitro. J Neurosci Methods. 2021;368:109462doi: 10.1016/j.jneumeth.2021.109462.
Mi, R., Ji, J., Zhang, M., Zhang, J., Li, M., Hu, Y., & Liu, F. (2021). Establishment of the glioma polyploid giant cancer cell model by a modified PHA-DMSO-PEG fusion method following dual drug-fluorescence screening in vitro. Journal of neuroscience methods, 368109462. https://doi.org/10.1016/j.jneumeth.2021.109462
Mi, Ruifang, et al. "Establishment of the glioma polyploid giant cancer cell model by a modified PHA-DMSO-PEG fusion method following dual drug-fluorescence screening in vitro." Journal of neuroscience methods vol. 368 (2021): 109462. doi: https://doi.org/10.1016/j.jneumeth.2021.109462
Mi R, Ji J, Zhang M, Zhang J, Li M, Hu Y, Liu F. Establishment of the glioma polyploid giant cancer cell model by a modified PHA-DMSO-PEG fusion method following dual drug-fluorescence screening in vitro. J Neurosci Methods. 2021 Dec 28;368:109462. doi: 10.1016/j.jneumeth.2021.109462. Epub 2021 Dec 28. PMID: 34968625.
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MicroRNA-Mediated Drug Repurposing Unveiled Potential Candidate Drugs for Prolactinoma Treatment.

Neuroendocrinology

Aydin B, Arslan S, Bayraklı F, Karademir B, Arga KY.
PMID: 33706313
Neuroendocrinology. 2021 Mar 11; doi: 10.1159/000515801. Epub 2021 Mar 11.

INTRODUCTION: Prolactinomas, also called lactotroph adenomas, are the most encountered type of hormone-secreting pituitary neuroendocrine tumors in the clinic. The preferred first-line therapy is a medical treatment with dopamine agonists (DAs), mainly cabergoline, to reduce serum prolactin levels, tumor...

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Aydin B, Arslan S, Bayraklı F, et al. MicroRNA-Mediated Drug Repurposing Unveiled Potential Candidate Drugs for Prolactinoma Treatment. Neuroendocrinology. 2021;doi: 10.1159/000515801.
Aydin, B., Arslan, S., Bayraklı, F., Karademir, B., & Arga, K. Y. (2021). MicroRNA-Mediated Drug Repurposing Unveiled Potential Candidate Drugs for Prolactinoma Treatment. Neuroendocrinology, . https://doi.org/10.1159/000515801
Aydin, Busra, et al. "MicroRNA-Mediated Drug Repurposing Unveiled Potential Candidate Drugs for Prolactinoma Treatment." Neuroendocrinology vol. (2021). doi: https://doi.org/10.1159/000515801
Aydin B, Arslan S, Bayraklı F, Karademir B, Arga KY. MicroRNA-Mediated Drug Repurposing Unveiled Potential Candidate Drugs for Prolactinoma Treatment. Neuroendocrinology. 2021 Mar 11; doi: 10.1159/000515801. Epub 2021 Mar 11. PMID: 33706313.
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MCC Regulator of WNT Signaling Pathway (MCC) Is a Podocyte Essential Gene.

Frontiers in medicine

Song H, Zhuang L, Xu X, Shi J, Hu W, Liu Z, Shi S.
PMID: 34926519
Front Med (Lausanne). 2021 Dec 02;8:777563. doi: 10.3389/fmed.2021.777563. eCollection 2021.

Podocytes are an integral part of the glomerular filtration barrier. Many genes are already known to be essential for podocyte survival, structure and function, but there are more podocyte essential genes to be identified. By single-cell RNA-seq of mouse...

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Song H, Zhuang L, Xu X, et al. MCC Regulator of WNT Signaling Pathway (MCC) Is a Podocyte Essential Gene. Front Med (Lausanne). 2021;8:777563doi: 10.3389/fmed.2021.777563.
Song, H., Zhuang, L., Xu, X., Shi, J., Hu, W., Liu, Z., & Shi, S. (2021). MCC Regulator of WNT Signaling Pathway (MCC) Is a Podocyte Essential Gene. Frontiers in medicine, 8777563. https://doi.org/10.3389/fmed.2021.777563
Song, Hui, et al. "MCC Regulator of WNT Signaling Pathway (MCC) Is a Podocyte Essential Gene." Frontiers in medicine vol. 8 (2021): 777563. doi: https://doi.org/10.3389/fmed.2021.777563
Song H, Zhuang L, Xu X, Shi J, Hu W, Liu Z, Shi S. MCC Regulator of WNT Signaling Pathway (MCC) Is a Podocyte Essential Gene. Front Med (Lausanne). 2021 Dec 02;8:777563. doi: 10.3389/fmed.2021.777563. eCollection 2021. PMID: 34926519; PMCID: PMC8674659.
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