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Showing 25 to 36 of 221 entries
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PGC-1α modulates denervation-induced mitophagy in skeletal muscle.

Skeletal muscle

Vainshtein A, Desjardins EM, Armani A, Sandri M, Hood DA.
PMID: 25834726
Skelet Muscle. 2015 Mar 18;5:9. doi: 10.1186/s13395-015-0033-y. eCollection 2015.

BACKGROUND: Alterations in skeletal muscle contractile activity necessitate an efficient remodeling mechanism. In particular, mitochondrial turnover is essential for tissue homeostasis during muscle adaptations to chronic use and disuse. While mitochondrial biogenesis appears to be largely governed by the...

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Vainshtein A, Desjardins EM, Armani A, et al. PGC-1α modulates denervation-induced mitophagy in skeletal muscle. Skelet Muscle. 2015;5:9doi: 10.1186/s13395-015-0033-y.
Vainshtein, A., Desjardins, E. M., Armani, A., Sandri, M., & Hood, D. A. (2015). PGC-1α modulates denervation-induced mitophagy in skeletal muscle. Skeletal muscle, 59. https://doi.org/10.1186/s13395-015-0033-y
Vainshtein, Anna, et al. "PGC-1α modulates denervation-induced mitophagy in skeletal muscle." Skeletal muscle vol. 5 (2015): 9. doi: https://doi.org/10.1186/s13395-015-0033-y
Vainshtein A, Desjardins EM, Armani A, Sandri M, Hood DA. PGC-1α modulates denervation-induced mitophagy in skeletal muscle. Skelet Muscle. 2015 Mar 18;5:9. doi: 10.1186/s13395-015-0033-y. eCollection 2015. PMID: 25834726; PMCID: PMC4381453.
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Control of mRNA stability contributes to low levels of nuclear poly(A) binding protein 1 (PABPN1) in skeletal muscle.

Skeletal muscle

Apponi LH, Corbett AH, Pavlath GK.
PMID: 24083404
Skelet Muscle. 2013 Oct 01;3(1):23. doi: 10.1186/2044-5040-3-23.

BACKGROUND: The nuclear poly(A) binding protein 1 (PABPN1) is a ubiquitously expressed protein that plays critical roles at multiple steps in post-transcriptional regulation of gene expression. Short expansions of the polyalanine tract in the N-terminus of PABPN1 lead to...

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Apponi LH, Corbett AH, Pavlath GK. Control of mRNA stability contributes to low levels of nuclear poly(A) binding protein 1 (PABPN1) in skeletal muscle. Skelet Muscle. 2013;3(1):23doi: 10.1186/2044-5040-3-23.
Apponi, L. H., Corbett, A. H., & Pavlath, G. K. (2013). Control of mRNA stability contributes to low levels of nuclear poly(A) binding protein 1 (PABPN1) in skeletal muscle. Skeletal muscle, 3(1), 23. https://doi.org/10.1186/2044-5040-3-23
Apponi, Luciano H, et al. "Control of mRNA stability contributes to low levels of nuclear poly(A) binding protein 1 (PABPN1) in skeletal muscle." Skeletal muscle vol. 3,1 (2013): 23. doi: https://doi.org/10.1186/2044-5040-3-23
Apponi LH, Corbett AH, Pavlath GK. Control of mRNA stability contributes to low levels of nuclear poly(A) binding protein 1 (PABPN1) in skeletal muscle. Skelet Muscle. 2013 Oct 01;3(1):23. doi: 10.1186/2044-5040-3-23. PMID: 24083404; PMCID: PMC3879409.
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Glypican-1 regulates myoblast response to HGF via Met in a lipid raft-dependent mechanism: effect on migration of skeletal muscle precursor cells.

Skeletal muscle

Gutiérrez J, Cabrera D, Brandan E.
PMID: 24517345
Skelet Muscle. 2014 Feb 12;4(1):5. doi: 10.1186/2044-5040-4-5.

BACKGROUND: Via the hepatocyte growth factor receptor (Met), hepatocyte growth factor (HGF) exerts key roles involving skeletal muscle development and regeneration. Heparan sulfate proteoglycans (HSPGs) are critical modulators of HGF activity, but the role of specific HSPGs in HGF...

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Gutiérrez J, Cabrera D, Brandan E. Glypican-1 regulates myoblast response to HGF via Met in a lipid raft-dependent mechanism: effect on migration of skeletal muscle precursor cells. Skelet Muscle. 2014;4(1):5doi: 10.1186/2044-5040-4-5.
Gutiérrez, J., Cabrera, D., & Brandan, E. (2014). Glypican-1 regulates myoblast response to HGF via Met in a lipid raft-dependent mechanism: effect on migration of skeletal muscle precursor cells. Skeletal muscle, 4(1), 5. https://doi.org/10.1186/2044-5040-4-5
Gutiérrez, Jaime, et al. "Glypican-1 regulates myoblast response to HGF via Met in a lipid raft-dependent mechanism: effect on migration of skeletal muscle precursor cells." Skeletal muscle vol. 4,1 (2014): 5. doi: https://doi.org/10.1186/2044-5040-4-5
Gutiérrez J, Cabrera D, Brandan E. Glypican-1 regulates myoblast response to HGF via Met in a lipid raft-dependent mechanism: effect on migration of skeletal muscle precursor cells. Skelet Muscle. 2014 Feb 12;4(1):5. doi: 10.1186/2044-5040-4-5. PMID: 24517345; PMCID: PMC3923899.
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Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration.

Skeletal muscle

Fuoco C, Salvatori ML, Biondo A, Shapira-Schweitzer K, Santoleri S, Antonini S, Bernardini S, Tedesco FS, Cannata S, Seliktar D, Cossu G, Gargioli C.
PMID: 23181356
Skelet Muscle. 2012 Nov 26;2(1):24. doi: 10.1186/2044-5040-2-24.

BACKGROUND: Cell-transplantation therapies have attracted attention as treatments for skeletal-muscle disorders; however, such research has been severely limited by poor cell survival. Tissue engineering offers a potential solution to this problem by providing biomaterial adjuvants that improve survival and...

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Fuoco C, Salvatori ML, Biondo A, et al. Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration. Skelet Muscle. 2012;2(1):24doi: 10.1186/2044-5040-2-24.
Fuoco, C., Salvatori, M. L., Biondo, A., Shapira-Schweitzer, K., Santoleri, S., Antonini, S., Bernardini, S., Tedesco, F. S., Cannata, S., Seliktar, D., Cossu, G., & Gargioli, C. (2012). Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration. Skeletal muscle, 2(1), 24. https://doi.org/10.1186/2044-5040-2-24
Fuoco, Claudia, et al. "Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration." Skeletal muscle vol. 2,1 (2012): 24. doi: https://doi.org/10.1186/2044-5040-2-24
Fuoco C, Salvatori ML, Biondo A, Shapira-Schweitzer K, Santoleri S, Antonini S, Bernardini S, Tedesco FS, Cannata S, Seliktar D, Cossu G, Gargioli C. Injectable polyethylene glycol-fibrinogen hydrogel adjuvant improves survival and differentiation of transplanted mesoangioblasts in acute and chronic skeletal-muscle degeneration. Skelet Muscle. 2012 Nov 26;2(1):24. doi: 10.1186/2044-5040-2-24. PMID: 23181356; PMCID: PMC3579757.
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Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy.

Skeletal muscle

Bentzinger CF, Lin S, Romanino K, Castets P, Guridi M, Summermatter S, Handschin C, Tintignac LA, Hall MN, Rüegg MA.
PMID: 23497627
Skelet Muscle. 2013 Mar 06;3(1):6. doi: 10.1186/2044-5040-3-6.

BACKGROUND: Skeletal muscle mass is determined by the balance between protein synthesis and degradation. Mammalian target of rapamycin complex 1 (mTORC1) is a master regulator of protein translation and has been implicated in the control of muscle mass. Inactivation...

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Bentzinger CF, Lin S, Romanino K, et al. Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy. Skelet Muscle. 2013;3(1):6doi: 10.1186/2044-5040-3-6.
Bentzinger, C. F., Lin, S., Romanino, K., Castets, P., Guridi, M., Summermatter, S., Handschin, C., Tintignac, L. A., Hall, M. N., & Rüegg, M. A. (2013). Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy. Skeletal muscle, 3(1), 6. https://doi.org/10.1186/2044-5040-3-6
Bentzinger, C Florian, et al. "Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy." Skeletal muscle vol. 3,1 (2013): 6. doi: https://doi.org/10.1186/2044-5040-3-6
Bentzinger CF, Lin S, Romanino K, Castets P, Guridi M, Summermatter S, Handschin C, Tintignac LA, Hall MN, Rüegg MA. Differential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy. Skelet Muscle. 2013 Mar 06;3(1):6. doi: 10.1186/2044-5040-3-6. PMID: 23497627; PMCID: PMC3622636.
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Long noncoding RNAs, emerging players in muscle differentiation and disease.

Skeletal muscle

Neguembor MV, Jothi M, Gabellini D.
PMID: 24685002
Skelet Muscle. 2014 Mar 31;4(1):8. doi: 10.1186/2044-5040-4-8.

The vast majority of the mammalian genome is transcribed giving rise to many different types of noncoding RNAs. Among them, long noncoding RNAs are the most numerous and functionally versatile class. Indeed, the lncRNA repertoire might be as rich...

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Neguembor MV, Jothi M, Gabellini D. Long noncoding RNAs, emerging players in muscle differentiation and disease. Skelet Muscle. 2014;4(1):8doi: 10.1186/2044-5040-4-8.
Neguembor, M. V., Jothi, M., & Gabellini, D. (2014). Long noncoding RNAs, emerging players in muscle differentiation and disease. Skeletal muscle, 4(1), 8. https://doi.org/10.1186/2044-5040-4-8
Neguembor, Maria Victoria, et al. "Long noncoding RNAs, emerging players in muscle differentiation and disease." Skeletal muscle vol. 4,1 (2014): 8. doi: https://doi.org/10.1186/2044-5040-4-8
Neguembor MV, Jothi M, Gabellini D. Long noncoding RNAs, emerging players in muscle differentiation and disease. Skelet Muscle. 2014 Mar 31;4(1):8. doi: 10.1186/2044-5040-4-8. PMID: 24685002; PMCID: PMC3973619.
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Live cell imaging reveals marked variability in myoblast proliferation and fate.

Skeletal muscle

Gross SM, Rotwein P.
PMID: 23638706
Skelet Muscle. 2013 May 02;3(1):10. doi: 10.1186/2044-5040-3-10.

BACKGROUND: During the process of muscle regeneration, activated stem cells termed satellite cells proliferate, and then differentiate to form new myofibers that restore the injured area. Yet not all satellite cells contribute to muscle repair. Some continue to proliferate,...

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Gross SM, Rotwein P. Live cell imaging reveals marked variability in myoblast proliferation and fate. Skelet Muscle. 2013;3(1):10doi: 10.1186/2044-5040-3-10.
Gross, S. M., & Rotwein, P. (2013). Live cell imaging reveals marked variability in myoblast proliferation and fate. Skeletal muscle, 3(1), 10. https://doi.org/10.1186/2044-5040-3-10
Gross, Sean M, and Rotwein, Peter. "Live cell imaging reveals marked variability in myoblast proliferation and fate." Skeletal muscle vol. 3,1 (2013): 10. doi: https://doi.org/10.1186/2044-5040-3-10
Gross SM, Rotwein P. Live cell imaging reveals marked variability in myoblast proliferation and fate. Skelet Muscle. 2013 May 02;3(1):10. doi: 10.1186/2044-5040-3-10. PMID: 23638706; PMCID: PMC3712004.
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Krüppel-like factor 6 (KLF6) promotes cell proliferation in skeletal myoblasts in response to TGFβ/Smad3 signaling.

Skeletal muscle

Dionyssiou MG, Salma J, Bevzyuk M, Wales S, Zakharyan L, McDermott JC.
PMID: 23547561
Skelet Muscle. 2013 Apr 02;3(1):7. doi: 10.1186/2044-5040-3-7.

BACKGROUND: Krüppel-like factor 6 (KLF6) has been recently identified as a MEF2D target gene involved in neuronal cell survival. In addition, KLF6 and TGFβ have been shown to regulate each other's expression in non-myogenic cell types. Since MEF2D and...

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Dionyssiou MG, Salma J, Bevzyuk M, et al. Krüppel-like factor 6 (KLF6) promotes cell proliferation in skeletal myoblasts in response to TGFβ/Smad3 signaling. Skelet Muscle. 2013;3(1):7doi: 10.1186/2044-5040-3-7.
Dionyssiou, M. G., Salma, J., Bevzyuk, M., Wales, S., Zakharyan, L., & McDermott, J. C. (2013). Krüppel-like factor 6 (KLF6) promotes cell proliferation in skeletal myoblasts in response to TGFβ/Smad3 signaling. Skeletal muscle, 3(1), 7. https://doi.org/10.1186/2044-5040-3-7
Dionyssiou, Mathew G, et al. "Krüppel-like factor 6 (KLF6) promotes cell proliferation in skeletal myoblasts in response to TGFβ/Smad3 signaling." Skeletal muscle vol. 3,1 (2013): 7. doi: https://doi.org/10.1186/2044-5040-3-7
Dionyssiou MG, Salma J, Bevzyuk M, Wales S, Zakharyan L, McDermott JC. Krüppel-like factor 6 (KLF6) promotes cell proliferation in skeletal myoblasts in response to TGFβ/Smad3 signaling. Skelet Muscle. 2013 Apr 02;3(1):7. doi: 10.1186/2044-5040-3-7. PMID: 23547561; PMCID: PMC3669038.
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ColVI myopathies: where do we stand, where do we go?.

Skeletal muscle

Allamand V, Briñas L, Richard P, Stojkovic T, Quijano-Roy S, Bonne G.
PMID: 21943391
Skelet Muscle. 2011 Sep 23;1:30. doi: 10.1186/2044-5040-1-30.

Collagen VI myopathies, caused by mutations in the genes encoding collagen type VI (ColVI), represent a clinical continuum with Ullrich congenital muscular dystrophy (UCMD) and Bethlem myopathy (BM) at each end of the spectrum, and less well-defined intermediate phenotypes...

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Allamand V, Briñas L, Richard P, et al. ColVI myopathies: where do we stand, where do we go?. Skelet Muscle. 2011;1:30doi: 10.1186/2044-5040-1-30.
Allamand, V., Briñas, L., Richard, P., Stojkovic, T., Quijano-Roy, S., & Bonne, G. (2011). ColVI myopathies: where do we stand, where do we go?. Skeletal muscle, 130. https://doi.org/10.1186/2044-5040-1-30
Allamand, Valérie, et al. "ColVI myopathies: where do we stand, where do we go?." Skeletal muscle vol. 1 (2011): 30. doi: https://doi.org/10.1186/2044-5040-1-30
Allamand V, Briñas L, Richard P, Stojkovic T, Quijano-Roy S, Bonne G. ColVI myopathies: where do we stand, where do we go?. Skelet Muscle. 2011 Sep 23;1:30. doi: 10.1186/2044-5040-1-30. PMID: 21943391; PMCID: PMC3189202.
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Analysis of human satellite cell dynamics on cultured adult skeletal muscle myofibers.

Skeletal muscle

Feige P, Tsai EC, Rudnicki MA.
PMID: 33397479
Skelet Muscle. 2021 Jan 04;11(1):1. doi: 10.1186/s13395-020-00256-z.

BACKGROUND: Maintaining stem cells in physiologically relevant states is necessary to understand cell and context-specific signalling paradigms and to understand complex interfaces between cells in situ. Understanding human stem cell function is largely based on tissue biopsies, cell culture,...

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Feige P, Tsai EC, Rudnicki MA. Analysis of human satellite cell dynamics on cultured adult skeletal muscle myofibers. Skelet Muscle. 2021;11(1):1doi: 10.1186/s13395-020-00256-z.
Feige, P., Tsai, E. C., & Rudnicki, M. A. (2021). Analysis of human satellite cell dynamics on cultured adult skeletal muscle myofibers. Skeletal muscle, 11(1), 1. https://doi.org/10.1186/s13395-020-00256-z
Feige, Peter, et al. "Analysis of human satellite cell dynamics on cultured adult skeletal muscle myofibers." Skeletal muscle vol. 11,1 (2021): 1. doi: https://doi.org/10.1186/s13395-020-00256-z
Feige P, Tsai EC, Rudnicki MA. Analysis of human satellite cell dynamics on cultured adult skeletal muscle myofibers. Skelet Muscle. 2021 Jan 04;11(1):1. doi: 10.1186/s13395-020-00256-z. PMID: 33397479; PMCID: PMC7780694.
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Retraction Note to: Exercise-induced mitochondrial p53 repairs mtDNA mutations in mutator mice.

Skeletal muscle

Safdar A, Khrapko K, Flynn JM, Saleem A, De Lisio M, Johnston APW, Kratysberg Y, Samjoo IA, Kitaoka Y, Ogborn DI, Little JP, Raha S, Parise G, Akhtar M, Hettinga BP, Rowe GC, Arany Z, Prolla TA, Tarnopolsky MA.
PMID: 33785048
Skelet Muscle. 2021 Mar 30;11(1):8. doi: 10.1186/s13395-021-00264-7.

No abstract available.

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Safdar A, Khrapko K, Flynn JM, et al. Retraction Note to: Exercise-induced mitochondrial p53 repairs mtDNA mutations in mutator mice. Skelet Muscle. 2021;11(1):8doi: 10.1186/s13395-021-00264-7.
Safdar, A., Khrapko, K., Flynn, J. M., Saleem, A., De Lisio, M., Johnston, A. P. W., Kratysberg, Y., Samjoo, I. A., Kitaoka, Y., Ogborn, D. I., Little, J. P., Raha, S., Parise, G., Akhtar, M., Hettinga, B. P., Rowe, G. C., Arany, Z., Prolla, T. A., & Tarnopolsky, M. A. (2021). Retraction Note to: Exercise-induced mitochondrial p53 repairs mtDNA mutations in mutator mice. Skeletal muscle, 11(1), 8. https://doi.org/10.1186/s13395-021-00264-7
Safdar, Adeel, et al. "Retraction Note to: Exercise-induced mitochondrial p53 repairs mtDNA mutations in mutator mice." Skeletal muscle vol. 11,1 (2021): 8. doi: https://doi.org/10.1186/s13395-021-00264-7
Safdar A, Khrapko K, Flynn JM, Saleem A, De Lisio M, Johnston APW, Kratysberg Y, Samjoo IA, Kitaoka Y, Ogborn DI, Little JP, Raha S, Parise G, Akhtar M, Hettinga BP, Rowe GC, Arany Z, Prolla TA, Tarnopolsky MA. Retraction Note to: Exercise-induced mitochondrial p53 repairs mtDNA mutations in mutator mice. Skelet Muscle. 2021 Mar 30;11(1):8. doi: 10.1186/s13395-021-00264-7. PMID: 33785048; PMCID: PMC8008634.
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Origins, potency, and heterogeneity of skeletal muscle fibro-adipogenic progenitors-time for new definitions.

Skeletal muscle

Contreras O, Rossi FMV, Theret M.
PMID: 34210364
Skelet Muscle. 2021 Jul 01;11(1):16. doi: 10.1186/s13395-021-00265-6.

Striated muscle is a highly plastic and regenerative organ that regulates body movement, temperature, and metabolism-all the functions needed for an individual's health and well-being. The muscle connective tissue's main components are the extracellular matrix and its resident stromal...

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Contreras O, Rossi FMV, Theret M. Origins, potency, and heterogeneity of skeletal muscle fibro-adipogenic progenitors-time for new definitions. Skelet Muscle. 2021;11(1):16doi: 10.1186/s13395-021-00265-6.
Contreras, O., Rossi, F. M. V., & Theret, M. (2021). Origins, potency, and heterogeneity of skeletal muscle fibro-adipogenic progenitors-time for new definitions. Skeletal muscle, 11(1), 16. https://doi.org/10.1186/s13395-021-00265-6
Contreras, Osvaldo, et al. "Origins, potency, and heterogeneity of skeletal muscle fibro-adipogenic progenitors-time for new definitions." Skeletal muscle vol. 11,1 (2021): 16. doi: https://doi.org/10.1186/s13395-021-00265-6
Contreras O, Rossi FMV, Theret M. Origins, potency, and heterogeneity of skeletal muscle fibro-adipogenic progenitors-time for new definitions. Skelet Muscle. 2021 Jul 01;11(1):16. doi: 10.1186/s13395-021-00265-6. PMID: 34210364; PMCID: PMC8247239.
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Showing 25 to 36 of 221 entries