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Mesoporous silica nanoparticles containing copper or silver synthesized with a new metal source: Determination of their structure parameters and cytotoxic and irritating effects.

Toxicology and applied pharmacology

Sierosławska A, Borówka A, Rymuszka A, Żukociński G, Sobczak K.
PMID: 34428444
Toxicol Appl Pharmacol. 2021 Oct 15;429:115685. doi: 10.1016/j.taap.2021.115685. Epub 2021 Aug 21.

One of the potential implementation of mesoporous silica nanomaterials (MSNs) is their use in biomedical applications as adsorbents or carriers of various bioactive substances. In this study, we attempted to fabricate silica nanomaterials containing copper and silver that were...

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Sierosławska A, Borówka A, Rymuszka A, et al. Mesoporous silica nanoparticles containing copper or silver synthesized with a new metal source: Determination of their structure parameters and cytotoxic and irritating effects. Toxicol Appl Pharmacol. 2021;429:115685doi: 10.1016/j.taap.2021.115685.
Sierosławska, A., Borówka, A., Rymuszka, A., Żukociński, G., & Sobczak, K. (2021). Mesoporous silica nanoparticles containing copper or silver synthesized with a new metal source: Determination of their structure parameters and cytotoxic and irritating effects. Toxicology and applied pharmacology, 429115685. https://doi.org/10.1016/j.taap.2021.115685
Sierosławska, Anna, et al. "Mesoporous silica nanoparticles containing copper or silver synthesized with a new metal source: Determination of their structure parameters and cytotoxic and irritating effects." Toxicology and applied pharmacology vol. 429 (2021): 115685. doi: https://doi.org/10.1016/j.taap.2021.115685
Sierosławska A, Borówka A, Rymuszka A, Żukociński G, Sobczak K. Mesoporous silica nanoparticles containing copper or silver synthesized with a new metal source: Determination of their structure parameters and cytotoxic and irritating effects. Toxicol Appl Pharmacol. 2021 Oct 15;429:115685. doi: 10.1016/j.taap.2021.115685. Epub 2021 Aug 21. PMID: 34428444.
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Tadalafil and bergapten mitigate streptozotocin-induced sporadic Alzheimer's disease in mice via modulating neuroinflammation, PI3K/Akt, Wnt/β-catenin, AMPK/mTOR signaling pathways.

Toxicology and applied pharmacology

Salem MA, Budzyńska B, Kowalczyk J, El Sayed NS, Mansour SM.
PMID: 34428446
Toxicol Appl Pharmacol. 2021 Oct 15;429:115697. doi: 10.1016/j.taap.2021.115697. Epub 2021 Aug 21.

Sporadic Alzheimer's disease (SAD) is a slowly progressive neurodegenerative disorder. This study aimed to investigate neuroprotective potential of tadalafil (TAD) and bergapten (BG) in SAD-induced cognitive impairment in mice. SAD was induced by single injection of streptozotocin (STZ; 3...

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Salem MA, Budzyńska B, Kowalczyk J, et al. Tadalafil and bergapten mitigate streptozotocin-induced sporadic Alzheimer's disease in mice via modulating neuroinflammation, PI3K/Akt, Wnt/β-catenin, AMPK/mTOR signaling pathways. Toxicol Appl Pharmacol. 2021;429:115697doi: 10.1016/j.taap.2021.115697.
Salem, M. A., Budzyńska, B., Kowalczyk, J., El Sayed, N. S., & Mansour, S. M. (2021). Tadalafil and bergapten mitigate streptozotocin-induced sporadic Alzheimer's disease in mice via modulating neuroinflammation, PI3K/Akt, Wnt/β-catenin, AMPK/mTOR signaling pathways. Toxicology and applied pharmacology, 429115697. https://doi.org/10.1016/j.taap.2021.115697
Salem, Mohamed A, et al. "Tadalafil and bergapten mitigate streptozotocin-induced sporadic Alzheimer's disease in mice via modulating neuroinflammation, PI3K/Akt, Wnt/β-catenin, AMPK/mTOR signaling pathways." Toxicology and applied pharmacology vol. 429 (2021): 115697. doi: https://doi.org/10.1016/j.taap.2021.115697
Salem MA, Budzyńska B, Kowalczyk J, El Sayed NS, Mansour SM. Tadalafil and bergapten mitigate streptozotocin-induced sporadic Alzheimer's disease in mice via modulating neuroinflammation, PI3K/Akt, Wnt/β-catenin, AMPK/mTOR signaling pathways. Toxicol Appl Pharmacol. 2021 Oct 15;429:115697. doi: 10.1016/j.taap.2021.115697. Epub 2021 Aug 21. PMID: 34428446.
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Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein.

Toxicology and applied pharmacology

Zhong J, Li M, Xu J, Dong W, Qin Y, Qiu S, Li X, Wang H.
PMID: 34990728
Toxicol Appl Pharmacol. 2022 Jan 04;436:115859. doi: 10.1016/j.taap.2021.115859. Epub 2022 Jan 04.

We have previously shown that inhibition of cAMP-specific 3',5'-cyclic phosphodiesterase 4 (PDE4) protects against cellular toxicity in neuronal cells. Since α-synuclein (α-syn) toxicity contributes to the neurodegeneration of Parkinson's disease (PD). The aim of this study was to explore...

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Zhong J, Li M, Xu J, et al. Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicol Appl Pharmacol. 2022;436:115859doi: 10.1016/j.taap.2021.115859.
Zhong, J., Li, M., Xu, J., Dong, W., Qin, Y., Qiu, S., Li, X., & Wang, H. (2022). Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicology and applied pharmacology, 436115859. https://doi.org/10.1016/j.taap.2021.115859
Zhong, Jiahong, et al. "Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein." Toxicology and applied pharmacology vol. 436 (2022): 115859. doi: https://doi.org/10.1016/j.taap.2021.115859
Zhong J, Li M, Xu J, Dong W, Qin Y, Qiu S, Li X, Wang H. Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicol Appl Pharmacol. 2022 Jan 04;436:115859. doi: 10.1016/j.taap.2021.115859. Epub 2022 Jan 04. PMID: 34990728.
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PARP inhibitor BMN-673 induced apoptosis by trapping PARP-1 and inhibiting base excision repair via modulation of pol-β in chromatin of breast cancer cells.

Toxicology and applied pharmacology

Sethy C, Kundu CN.
PMID: 34998856
Toxicol Appl Pharmacol. 2022 Jan 05;436:115860. doi: 10.1016/j.taap.2021.115860. Epub 2022 Jan 05.

PARP inhibitors emerged as clinically effective anti-tumor agents in combination with DNA damaging agents but the toxicity of DNA damaging agents and their off-target effects caused serious problems in cancer therapy. They confer cytotoxicity in cancer cells both by...

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Sethy C, Kundu CN. PARP inhibitor BMN-673 induced apoptosis by trapping PARP-1 and inhibiting base excision repair via modulation of pol-β in chromatin of breast cancer cells. Toxicol Appl Pharmacol. 2022;436:115860doi: 10.1016/j.taap.2021.115860.
Sethy, C., & Kundu, C. N. (2022). PARP inhibitor BMN-673 induced apoptosis by trapping PARP-1 and inhibiting base excision repair via modulation of pol-β in chromatin of breast cancer cells. Toxicology and applied pharmacology, 436115860. https://doi.org/10.1016/j.taap.2021.115860
Sethy, Chinmayee, and Kundu, Chanakya Nath. "PARP inhibitor BMN-673 induced apoptosis by trapping PARP-1 and inhibiting base excision repair via modulation of pol-β in chromatin of breast cancer cells." Toxicology and applied pharmacology vol. 436 (2022): 115860. doi: https://doi.org/10.1016/j.taap.2021.115860
Sethy C, Kundu CN. PARP inhibitor BMN-673 induced apoptosis by trapping PARP-1 and inhibiting base excision repair via modulation of pol-β in chromatin of breast cancer cells. Toxicol Appl Pharmacol. 2022 Jan 05;436:115860. doi: 10.1016/j.taap.2021.115860. Epub 2022 Jan 05. PMID: 34998856.
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Nonylphenol regulates TL1A through the AhR/HDAC2/HNF4α pathway in endothelial cells to promote the angiogenesis of colorectal cancer.

Toxicology and applied pharmacology

Zhang T, Ning WW, Zhang J, Xu FJ, Wang XQ, Li ZB, Xie M.
PMID: 34974051
Toxicol Appl Pharmacol. 2021 Dec 30;436:115854. doi: 10.1016/j.taap.2021.115854. Epub 2021 Dec 30.

BACKGROUND: Colorectal cancer (CRC) is one of the most malignant cancers worldwide. Nonylphenol (NP) is an endocrine-disruptor chemical and plays an important role in the development of cancers. However, the effects of NP on CRC remain unclear. In this...

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Zhang T, Ning WW, Zhang J, et al. Nonylphenol regulates TL1A through the AhR/HDAC2/HNF4α pathway in endothelial cells to promote the angiogenesis of colorectal cancer. Toxicol Appl Pharmacol. 2021;436:115854doi: 10.1016/j.taap.2021.115854.
Zhang, T., Ning, W. W., Zhang, J., Xu, F. J., Wang, X. Q., Li, Z. B., & Xie, M. (2021). Nonylphenol regulates TL1A through the AhR/HDAC2/HNF4α pathway in endothelial cells to promote the angiogenesis of colorectal cancer. Toxicology and applied pharmacology, 436115854. https://doi.org/10.1016/j.taap.2021.115854
Zhang, Tao, et al. "Nonylphenol regulates TL1A through the AhR/HDAC2/HNF4α pathway in endothelial cells to promote the angiogenesis of colorectal cancer." Toxicology and applied pharmacology vol. 436 (2021): 115854. doi: https://doi.org/10.1016/j.taap.2021.115854
Zhang T, Ning WW, Zhang J, Xu FJ, Wang XQ, Li ZB, Xie M. Nonylphenol regulates TL1A through the AhR/HDAC2/HNF4α pathway in endothelial cells to promote the angiogenesis of colorectal cancer. Toxicol Appl Pharmacol. 2021 Dec 30;436:115854. doi: 10.1016/j.taap.2021.115854. Epub 2021 Dec 30. PMID: 34974051.
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Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein.

Toxicology and applied pharmacology

Zhong J, Li M, Xu J, Dong W, Qin Y, Qiu S, Li X, Wang H.
PMID: 34990728
Toxicol Appl Pharmacol. 2022 Jan 04;436:115859. doi: 10.1016/j.taap.2021.115859. Epub 2022 Jan 04.

We have previously shown that inhibition of cAMP-specific 3',5'-cyclic phosphodiesterase 4 (PDE4) protects against cellular toxicity in neuronal cells. Since α-synuclein (α-syn) toxicity contributes to the neurodegeneration of Parkinson's disease (PD). The aim of this study was to explore...

Cite
Zhong J, Li M, Xu J, et al. Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicol Appl Pharmacol. 2022;436:115859doi: 10.1016/j.taap.2021.115859.
Zhong, J., Li, M., Xu, J., Dong, W., Qin, Y., Qiu, S., Li, X., & Wang, H. (2022). Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicology and applied pharmacology, 436115859. https://doi.org/10.1016/j.taap.2021.115859
Zhong, Jiahong, et al. "Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein." Toxicology and applied pharmacology vol. 436 (2022): 115859. doi: https://doi.org/10.1016/j.taap.2021.115859
Zhong J, Li M, Xu J, Dong W, Qin Y, Qiu S, Li X, Wang H. Roflupram attenuates α-synuclein-induced cytotoxicity and promotes the mitochondrial translocation of Parkin in SH-SY5Y cells overexpressing A53T mutant α-synuclein. Toxicol Appl Pharmacol. 2022 Jan 04;436:115859. doi: 10.1016/j.taap.2021.115859. Epub 2022 Jan 04. PMID: 34990728.
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Assessment of intestinal injury of hexavalent chromium using a modified in vitro gastrointestinal digestion model.

Toxicology and applied pharmacology

Wang Z, Peng H, Zhang R, Jiang X, Chen S, Guo P, Xiao Y, Zeng X, Wang Q, Aschner M, Chou WC, Li D, Chen W, Chen L.
PMID: 35016909
Toxicol Appl Pharmacol. 2022 Jan 08;115880. doi: 10.1016/j.taap.2022.115880. Epub 2022 Jan 08.

Intestinal injury assessment of hexavalent chromium (Cr-VI) in humans is crucial for quantifying assessment of adverse health risk posed by the intake of Cr (VI)-contaminated water. To overcome the deficiency in simulating human gastric reduction and intestinal absorption, we...

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Wang Z, Peng H, Zhang R, et al. Assessment of intestinal injury of hexavalent chromium using a modified in vitro gastrointestinal digestion model. Toxicol Appl Pharmacol. 2022;115880doi: 10.1016/j.taap.2022.115880.
Wang, Z., Peng, H., Zhang, R., Jiang, X., Chen, S., Guo, P., Xiao, Y., Zeng, X., Wang, Q., Aschner, M., Chou, W. C., Li, D., Chen, W., & Chen, L. (2022). Assessment of intestinal injury of hexavalent chromium using a modified in vitro gastrointestinal digestion model. Toxicology and applied pharmacology, 115880. https://doi.org/10.1016/j.taap.2022.115880
Wang, Ziwei, et al. "Assessment of intestinal injury of hexavalent chromium using a modified in vitro gastrointestinal digestion model." Toxicology and applied pharmacology vol. (2022): 115880. doi: https://doi.org/10.1016/j.taap.2022.115880
Wang Z, Peng H, Zhang R, Jiang X, Chen S, Guo P, Xiao Y, Zeng X, Wang Q, Aschner M, Chou WC, Li D, Chen W, Chen L. Assessment of intestinal injury of hexavalent chromium using a modified in vitro gastrointestinal digestion model. Toxicol Appl Pharmacol. 2022 Jan 08;115880. doi: 10.1016/j.taap.2022.115880. Epub 2022 Jan 08. PMID: 35016909.
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Scopoletin and umbelliferone protect hepatocytes against palmitate- and bile acid-induced cell death by reducing endoplasmic reticulum stress and oxidative stress.

Toxicology and applied pharmacology

Wu Z, Geng Y, Buist-Homan M, Moshage H.
PMID: 34979142
Toxicol Appl Pharmacol. 2021 Dec 31;436:115858. doi: 10.1016/j.taap.2021.115858. Epub 2021 Dec 31.

BACKGROUND: The number of patients with non-alcoholic fatty liver disease (NAFLD) is rapidly increasing due to the growing epidemic of obesity. Non-alcoholic steatohepatitis (NASH), the inflammatory stage of NAFLD, is characterized by lipid accumulation in hepatocytes, chronic inflammation and...

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Wu Z, Geng Y, Buist-Homan M, et al. Scopoletin and umbelliferone protect hepatocytes against palmitate- and bile acid-induced cell death by reducing endoplasmic reticulum stress and oxidative stress. Toxicol Appl Pharmacol. 2021;436:115858doi: 10.1016/j.taap.2021.115858.
Wu, Z., Geng, Y., Buist-Homan, M., & Moshage, H. (2021). Scopoletin and umbelliferone protect hepatocytes against palmitate- and bile acid-induced cell death by reducing endoplasmic reticulum stress and oxidative stress. Toxicology and applied pharmacology, 436115858. https://doi.org/10.1016/j.taap.2021.115858
Wu, Zongmei, et al. "Scopoletin and umbelliferone protect hepatocytes against palmitate- and bile acid-induced cell death by reducing endoplasmic reticulum stress and oxidative stress." Toxicology and applied pharmacology vol. 436 (2021): 115858. doi: https://doi.org/10.1016/j.taap.2021.115858
Wu Z, Geng Y, Buist-Homan M, Moshage H. Scopoletin and umbelliferone protect hepatocytes against palmitate- and bile acid-induced cell death by reducing endoplasmic reticulum stress and oxidative stress. Toxicol Appl Pharmacol. 2021 Dec 31;436:115858. doi: 10.1016/j.taap.2021.115858. Epub 2021 Dec 31. PMID: 34979142.
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Down-regulation of lncRNA MEG3 promotes chronic low dose cadmium exposure-induced cell transformation and cancer stem cell-like property.

Toxicology and applied pharmacology

Lin HP, Rea M, Wang Z, Yang C.
PMID: 34520792
Toxicol Appl Pharmacol. 2021 Nov 01;430:115724. doi: 10.1016/j.taap.2021.115724. Epub 2021 Sep 11.

Cadmium (Cd) is a toxic heavy metal and one of carcinogens that cause lung cancer. However, the exact mechanism of Cd carcinogenesis remains unclear. To investigate the mechanism of Cd carcinogenesis, we exposed human bronchial epithelial cells (BEAS-2B) to...

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Lin HP, Rea M, Wang Z, et al. Down-regulation of lncRNA MEG3 promotes chronic low dose cadmium exposure-induced cell transformation and cancer stem cell-like property. Toxicol Appl Pharmacol. 2021;430:115724doi: 10.1016/j.taap.2021.115724.
Lin, H. P., Rea, M., Wang, Z., & Yang, C. (2021). Down-regulation of lncRNA MEG3 promotes chronic low dose cadmium exposure-induced cell transformation and cancer stem cell-like property. Toxicology and applied pharmacology, 430115724. https://doi.org/10.1016/j.taap.2021.115724
Lin, Hsuan-Pei, et al. "Down-regulation of lncRNA MEG3 promotes chronic low dose cadmium exposure-induced cell transformation and cancer stem cell-like property." Toxicology and applied pharmacology vol. 430 (2021): 115724. doi: https://doi.org/10.1016/j.taap.2021.115724
Lin HP, Rea M, Wang Z, Yang C. Down-regulation of lncRNA MEG3 promotes chronic low dose cadmium exposure-induced cell transformation and cancer stem cell-like property. Toxicol Appl Pharmacol. 2021 Nov 01;430:115724. doi: 10.1016/j.taap.2021.115724. Epub 2021 Sep 11. PMID: 34520792; PMCID: PMC8511074.
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Nimbolide attenuated the inflammation in the liver of autoimmune hepatitis's mice through regulation of HDAC3.

Toxicology and applied pharmacology

Xia D, Chen D, Cai T, Zhu L, Lin Y, Yu S, Zhu K, Wang X, Xu L, Chen Y.
PMID: 34780724
Toxicol Appl Pharmacol. 2021 Nov 12;434:115795. doi: 10.1016/j.taap.2021.115795. Epub 2021 Nov 12.

A chronic liver disease named autoimmune hepatitis (AIH) will carry elevated levels of inflammatory cytokines, but there is currently no effective treatment to cure it. Histone deacetylase 3 (HDAC3) takes an important position in regulating the expression of inflammatory...

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Xia D, Chen D, Cai T, et al. Nimbolide attenuated the inflammation in the liver of autoimmune hepatitis's mice through regulation of HDAC3. Toxicol Appl Pharmacol. 2021;434:115795doi: 10.1016/j.taap.2021.115795.
Xia, D., Chen, D., Cai, T., Zhu, L., Lin, Y., Yu, S., Zhu, K., Wang, X., Xu, L., & Chen, Y. (2021). Nimbolide attenuated the inflammation in the liver of autoimmune hepatitis's mice through regulation of HDAC3. Toxicology and applied pharmacology, 434115795. https://doi.org/10.1016/j.taap.2021.115795
Xia, Dingchao, et al. "Nimbolide attenuated the inflammation in the liver of autoimmune hepatitis's mice through regulation of HDAC3." Toxicology and applied pharmacology vol. 434 (2021): 115795. doi: https://doi.org/10.1016/j.taap.2021.115795
Xia D, Chen D, Cai T, Zhu L, Lin Y, Yu S, Zhu K, Wang X, Xu L, Chen Y. Nimbolide attenuated the inflammation in the liver of autoimmune hepatitis's mice through regulation of HDAC3. Toxicol Appl Pharmacol. 2021 Nov 12;434:115795. doi: 10.1016/j.taap.2021.115795. Epub 2021 Nov 12. PMID: 34780724.
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Comparative study of estrogenic activities of phytoestrogens using OECD in vitro and in vivo testing methods.

Toxicology and applied pharmacology

Sim KS, Park S, Seo H, Lee SH, Lee HS, Park Y, Kim JH.
PMID: 34848279
Toxicol Appl Pharmacol. 2021 Nov 27;434:115815. doi: 10.1016/j.taap.2021.115815. Epub 2021 Nov 27.

With growing scientific interest in phytoestrogens, a number of studies have investigated the estrogenic potential of phytoestrogens in a wide variety of assay systems. However, evaluations of individual phytoestrogens with different assay systems make it difficult for predicting their...

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Sim KS, Park S, Seo H, et al. Comparative study of estrogenic activities of phytoestrogens using OECD in vitro and in vivo testing methods. Toxicol Appl Pharmacol. 2021;434:115815doi: 10.1016/j.taap.2021.115815.
Sim, K. S., Park, S., Seo, H., Lee, S. H., Lee, H. S., Park, Y., & Kim, J. H. (2021). Comparative study of estrogenic activities of phytoestrogens using OECD in vitro and in vivo testing methods. Toxicology and applied pharmacology, 434115815. https://doi.org/10.1016/j.taap.2021.115815
Sim, Kyu Sang, et al. "Comparative study of estrogenic activities of phytoestrogens using OECD in vitro and in vivo testing methods." Toxicology and applied pharmacology vol. 434 (2021): 115815. doi: https://doi.org/10.1016/j.taap.2021.115815
Sim KS, Park S, Seo H, Lee SH, Lee HS, Park Y, Kim JH. Comparative study of estrogenic activities of phytoestrogens using OECD in vitro and in vivo testing methods. Toxicol Appl Pharmacol. 2021 Nov 27;434:115815. doi: 10.1016/j.taap.2021.115815. Epub 2021 Nov 27. PMID: 34848279.
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1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway.

Toxicology and applied pharmacology

Liang B, Zhong Y, Wang B, Lin L, Liu J, Lin X, Huang Y, Hu M, Zhang B, Meng H, Jiang L, Jiang J, Wu J, Zhang Y, Rong W, Yang X, Huang Z.
PMID: 34560092
Toxicol Appl Pharmacol. 2021 Nov 01;430:115728. doi: 10.1016/j.taap.2021.115728. Epub 2021 Sep 22.

1,2-Dichloroethane (1,2-DCE) is a pervasive environmental pollutant found in ambient and residential air, as well as ground and drinking water. Overexposure to it results in cortex edema, in both animals and humans. 1,2-DCE induces apoptosis in the cerebellum, liver...

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Liang B, Zhong Y, Wang B, et al. 1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway. Toxicol Appl Pharmacol. 2021;430:115728doi: 10.1016/j.taap.2021.115728.
Liang, B., Zhong, Y., Wang, B., Lin, L., Liu, J., Lin, X., Huang, Y., Hu, M., Zhang, B., Meng, H., Jiang, L., Jiang, J., Wu, J., Zhang, Y., Rong, W., Yang, X., & Huang, Z. (2021). 1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway. Toxicology and applied pharmacology, 430115728. https://doi.org/10.1016/j.taap.2021.115728
Liang, Boxuan, et al. "1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway." Toxicology and applied pharmacology vol. 430 (2021): 115728. doi: https://doi.org/10.1016/j.taap.2021.115728
Liang B, Zhong Y, Wang B, Lin L, Liu J, Lin X, Huang Y, Hu M, Zhang B, Meng H, Jiang L, Jiang J, Wu J, Zhang Y, Rong W, Yang X, Huang Z. 1,2-Dichloroethane induces apoptosis in the cerebral cortexes of NIH Swiss mice through microRNA-182-5p targeting phospholipase D1 via a mitochondria-dependent pathway. Toxicol Appl Pharmacol. 2021 Nov 01;430:115728. doi: 10.1016/j.taap.2021.115728. Epub 2021 Sep 22. PMID: 34560092.
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