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Lyl-1 regulates primitive macrophages and microglia development.

Communications biology

Wang S, Ren D, Arkoun B, Kaushik AL, Matherat G, Lécluse Y, Filipp D, Vainchenker W, Raslova H, Plo I, Godin I.
PMID: 34887504
Commun Biol. 2021 Dec 09;4(1):1382. doi: 10.1038/s42003-021-02886-5.

During ontogeny, macrophage populations emerge in the Yolk Sac (YS) via two distinct progenitor waves, prior to hematopoietic stem cell development. Macrophage progenitors from the primitive/"early EMP" and transient-definitive/"late EMP" waves both contribute to various resident primitive macrophage populations...

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Wang S, Ren D, Arkoun B, et al. Lyl-1 regulates primitive macrophages and microglia development. Commun Biol. 2021;4(1):1382doi: 10.1038/s42003-021-02886-5.
Wang, S., Ren, D., Arkoun, B., Kaushik, A. L., Matherat, G., Lécluse, Y., Filipp, D., Vainchenker, W., Raslova, H., Plo, I., & Godin, I. (2021). Lyl-1 regulates primitive macrophages and microglia development. Communications biology, 4(1), 1382. https://doi.org/10.1038/s42003-021-02886-5
Wang, Shoutang, et al. "Lyl-1 regulates primitive macrophages and microglia development." Communications biology vol. 4,1 (2021): 1382. doi: https://doi.org/10.1038/s42003-021-02886-5
Wang S, Ren D, Arkoun B, Kaushik AL, Matherat G, Lécluse Y, Filipp D, Vainchenker W, Raslova H, Plo I, Godin I. Lyl-1 regulates primitive macrophages and microglia development. Commun Biol. 2021 Dec 09;4(1):1382. doi: 10.1038/s42003-021-02886-5. PMID: 34887504.
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Genome-Wide Identification and Expression Analysis of AP2/ERF Transcription Factor Related to Drought Stress in Cultivated Peanut (.

Frontiers in genetics

Cui M, Haider MS, Chai P, Guo J, Du P, Li H, Dong W, Huang B, Zheng Z, Shi L, Zhang X, Han S.
PMID: 34721538
Front Genet. 2021 Oct 13;12:750761. doi: 10.3389/fgene.2021.750761. eCollection 2021.

APETALA2/ethylene response element-binding factor (AP2/ERF) transcription factors (TFs) have been found to regulate plant growth and development and response to various abiotic stresses. However, detailed information of

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Cui M, Haider MS, Chai P, et al. Genome-Wide Identification and Expression Analysis of AP2/ERF Transcription Factor Related to Drought Stress in Cultivated Peanut (. Front Genet. 2021;12:750761doi: 10.3389/fgene.2021.750761.
Cui, M., Haider, M. S., Chai, P., Guo, J., Du, P., Li, H., Dong, W., Huang, B., Zheng, Z., Shi, L., Zhang, X., & Han, S. (2021). Genome-Wide Identification and Expression Analysis of AP2/ERF Transcription Factor Related to Drought Stress in Cultivated Peanut (. Frontiers in genetics, 12750761. https://doi.org/10.3389/fgene.2021.750761
Cui, Mengjie, et al. "Genome-Wide Identification and Expression Analysis of AP2/ERF Transcription Factor Related to Drought Stress in Cultivated Peanut (." Frontiers in genetics vol. 12 (2021): 750761. doi: https://doi.org/10.3389/fgene.2021.750761
Cui M, Haider MS, Chai P, Guo J, Du P, Li H, Dong W, Huang B, Zheng Z, Shi L, Zhang X, Han S. Genome-Wide Identification and Expression Analysis of AP2/ERF Transcription Factor Related to Drought Stress in Cultivated Peanut (. Front Genet. 2021 Oct 13;12:750761. doi: 10.3389/fgene.2021.750761. eCollection 2021. PMID: 34721538; PMCID: PMC8548641.
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Methylation in the promoter regions of WT1, NKX6-1 and DBC1 genes in cervical cancer tissues of Uygur women in Xinjiang.

Genetics and molecular biology

Wu D, Zhang J, Fan P, Li H, Li D, Pan H, He H, Ren X, Pan Z, Shao R, Pan Z.
PMID: 29658966
Genet Mol Biol. 2018 Jan-Mar;41(1):9-17. doi: 10.1590/1678-4685-GMB-2016-0146.

This study aimed to explore: 1) DNA methylation in the promoter regions of Wilms tumor gene 1 (WT1), NK6 transcription factor related locus 1 gene (NKX6-1) and Deleted in bladder cancer 1 (DBC1) gene in cervical cancer tissues of...

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Wu D, Zhang J, Fan P, et al. Methylation in the promoter regions of WT1, NKX6-1 and DBC1 genes in cervical cancer tissues of Uygur women in Xinjiang. Genet Mol Biol. 2018;41(1):9-17doi: 10.1590/1678-4685-GMB-2016-0146.
Wu, D., Zhang, J., Fan, P., Li, H., Li, D., Pan, H., He, H., Ren, X., Pan, Z., Shao, R., & Pan, Z. (2018). Methylation in the promoter regions of WT1, NKX6-1 and DBC1 genes in cervical cancer tissues of Uygur women in Xinjiang. Genetics and molecular biology, 41(1), 9-17. https://doi.org/10.1590/1678-4685-GMB-2016-0146
Wu, Dan, et al. "Methylation in the promoter regions of WT1, NKX6-1 and DBC1 genes in cervical cancer tissues of Uygur women in Xinjiang." Genetics and molecular biology vol. 41,1 (2018): 9-17. doi: https://doi.org/10.1590/1678-4685-GMB-2016-0146
Wu D, Zhang J, Fan P, Li H, Li D, Pan H, He H, Ren X, Pan Z, Shao R, Pan Z. Methylation in the promoter regions of WT1, NKX6-1 and DBC1 genes in cervical cancer tissues of Uygur women in Xinjiang. Genet Mol Biol. 2018 Jan-Mar;41(1):9-17. doi: 10.1590/1678-4685-GMB-2016-0146. PMID: 29658966; PMCID: PMC5901490.
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Identification of Three Novel Susceptibility Loci for Inflammatory Bowel Disease in Koreans in an Extended Genome-Wide Association Study.

Journal of Crohn's & colitis

Jung S, Ye BD, Lee HS, Baek J, Kim G, Park D, Park SH, Yang SK, Han B, Liu J, Song K.
PMID: 33853113
J Crohns Colitis. 2021 Nov 08;15(11):1898-1907. doi: 10.1093/ecco-jcc/jjab060.

BACKGROUND AND AIMS: Genome-wide association studies [GWAS] of inflammatory bowel disease [IBD] in multiple populations have identified over 240 susceptibility loci. We previously performed a largest-to-date Asian-specific IBD GWAS to identify two new IBD risk loci and confirm associations...

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Jung S, Ye BD, Lee HS, et al. Identification of Three Novel Susceptibility Loci for Inflammatory Bowel Disease in Koreans in an Extended Genome-Wide Association Study. J Crohns Colitis. 2021;15(11):1898-1907doi: 10.1093/ecco-jcc/jjab060.
Jung, S., Ye, B. D., Lee, H. S., Baek, J., Kim, G., Park, D., Park, S. H., Yang, S. K., Han, B., Liu, J., & Song, K. (2021). Identification of Three Novel Susceptibility Loci for Inflammatory Bowel Disease in Koreans in an Extended Genome-Wide Association Study. Journal of Crohn's & colitis, 15(11), 1898-1907. https://doi.org/10.1093/ecco-jcc/jjab060
Jung, Seulgi, et al. "Identification of Three Novel Susceptibility Loci for Inflammatory Bowel Disease in Koreans in an Extended Genome-Wide Association Study." Journal of Crohn's & colitis vol. 15,11 (2021): 1898-1907. doi: https://doi.org/10.1093/ecco-jcc/jjab060
Jung S, Ye BD, Lee HS, Baek J, Kim G, Park D, Park SH, Yang SK, Han B, Liu J, Song K. Identification of Three Novel Susceptibility Loci for Inflammatory Bowel Disease in Koreans in an Extended Genome-Wide Association Study. J Crohns Colitis. 2021 Nov 08;15(11):1898-1907. doi: 10.1093/ecco-jcc/jjab060. PMID: 33853113.
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Identification of the Transcription Factor Relationships Associated with Androgen Deprivation Therapy Response and Metastatic Progression in Prostate Cancer.

Cancers

Sharma NV, Pellegrini KL, Ouellet V, Giuste FO, Ramalingam S, Watanabe K, Adam-Granger E, Fossouo L, You S, Freeman MR, Vertino P, Conneely K, Osunkoya AO, Trudel D, Mes-Masson AM, Petros JA, Saad F, Moreno CS.
PMID: 30314329
Cancers (Basel). 2018 Oct 11;10(10). doi: 10.3390/cancers10100379.

BACKGROUND: Patients with locally advanced or recurrent prostate cancer typically undergo androgen deprivation therapy (ADT), but the benefits are often short-lived and the responses variable. ADT failure results in castration-resistant prostate cancer (CRPC), which inevitably leads to metastasis. We...

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Sharma NV, Pellegrini KL, Ouellet V, et al. Identification of the Transcription Factor Relationships Associated with Androgen Deprivation Therapy Response and Metastatic Progression in Prostate Cancer. Cancers (Basel). 2018;10(10)doi: 10.3390/cancers10100379.
Sharma, N. V., Pellegrini, K. L., Ouellet, V., Giuste, F. O., Ramalingam, S., Watanabe, K., Adam-Granger, E., Fossouo, L., You, S., Freeman, M. R., Vertino, P., Conneely, K., Osunkoya, A. O., Trudel, D., Mes-Masson, A. M., Petros, J. A., Saad, F., & Moreno, C. S. (2018). Identification of the Transcription Factor Relationships Associated with Androgen Deprivation Therapy Response and Metastatic Progression in Prostate Cancer. Cancers, 10(10), . https://doi.org/10.3390/cancers10100379
Sharma, Nitya V, et al. "Identification of the Transcription Factor Relationships Associated with Androgen Deprivation Therapy Response and Metastatic Progression in Prostate Cancer." Cancers vol. 10,10 (2018). doi: https://doi.org/10.3390/cancers10100379
Sharma NV, Pellegrini KL, Ouellet V, Giuste FO, Ramalingam S, Watanabe K, Adam-Granger E, Fossouo L, You S, Freeman MR, Vertino P, Conneely K, Osunkoya AO, Trudel D, Mes-Masson AM, Petros JA, Saad F, Moreno CS. Identification of the Transcription Factor Relationships Associated with Androgen Deprivation Therapy Response and Metastatic Progression in Prostate Cancer. Cancers (Basel). 2018 Oct 11;10(10). doi: 10.3390/cancers10100379. PMID: 30314329; PMCID: PMC6210624.
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Cardiomyocyte-Specific COMMD1 Deletion Suppresses Ischemia-Induced Myocardial Apoptosis.

Cardiovascular toxicology

Li C, Peng H, Kang YJ.
PMID: 33900545
Cardiovasc Toxicol. 2021 Jul;21(7):572-581. doi: 10.1007/s12012-021-09650-5. Epub 2021 Apr 26.

Copper metabolism MURR domain 1 (COMMD1) increases in ischemic myocardium along with suppressed contractility. Cardiomyocyte-specific deletion of COMMD1 preserved myocardial contractile function in response to the same ischemic insult. This study was undertaken to test the hypothesis that cardiomyocyte...

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Li C, Peng H, Kang YJ. Cardiomyocyte-Specific COMMD1 Deletion Suppresses Ischemia-Induced Myocardial Apoptosis. Cardiovasc Toxicol. 2021;21(7):572-581doi: 10.1007/s12012-021-09650-5.
Li, C., Peng, H., & Kang, Y. J. (2021). Cardiomyocyte-Specific COMMD1 Deletion Suppresses Ischemia-Induced Myocardial Apoptosis. Cardiovascular toxicology, 21(7), 572-581. https://doi.org/10.1007/s12012-021-09650-5
Li, Chen, et al. "Cardiomyocyte-Specific COMMD1 Deletion Suppresses Ischemia-Induced Myocardial Apoptosis." Cardiovascular toxicology vol. 21,7 (2021): 572-581. doi: https://doi.org/10.1007/s12012-021-09650-5
Li C, Peng H, Kang YJ. Cardiomyocyte-Specific COMMD1 Deletion Suppresses Ischemia-Induced Myocardial Apoptosis. Cardiovasc Toxicol. 2021 Jul;21(7):572-581. doi: 10.1007/s12012-021-09650-5. Epub 2021 Apr 26. PMID: 33900545.
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Pure Cortical Ependymoma With RELA-Fusion Positive in a Young Adult.

Brain tumor research and treatment

Lee SW, Lee JH, Lee MS, Suh SJ, Lee YS, Kim JW.
PMID: 34725994
Brain Tumor Res Treat. 2021 Oct;9(2):111-116. doi: 10.14791/btrt.2021.9.e23.

A 25-year-old female presented with a generalized tonic-clonic seizure. She had no previous history of seizures. A brain magnetic resonance imaging scan revealed a solitary enhancing mass in the right fronto-parietal cortex. During surgery, the mass was noted to...

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Lee SW, Lee JH, Lee MS, et al. Pure Cortical Ependymoma With RELA-Fusion Positive in a Young Adult. Brain Tumor Res Treat. 2021;9(2):111-116doi: 10.14791/btrt.2021.9.e23.
Lee, S. W., Lee, J. H., Lee, M. S., Suh, S. J., Lee, Y. S., & Kim, J. W. (2021). Pure Cortical Ependymoma With RELA-Fusion Positive in a Young Adult. Brain tumor research and treatment, 9(2), 111-116. https://doi.org/10.14791/btrt.2021.9.e23
Lee, Sang-Woo, et al. "Pure Cortical Ependymoma With RELA-Fusion Positive in a Young Adult." Brain tumor research and treatment vol. 9,2 (2021): 111-116. doi: https://doi.org/10.14791/btrt.2021.9.e23
Lee SW, Lee JH, Lee MS, Suh SJ, Lee YS, Kim JW. Pure Cortical Ependymoma With RELA-Fusion Positive in a Young Adult. Brain Tumor Res Treat. 2021 Oct;9(2):111-116. doi: 10.14791/btrt.2021.9.e23. PMID: 34725994; PMCID: PMC8561227.
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A proteomic approach for the identification of novel lysine methyltransferase substrates.

Epigenetics & chromatin

Levy D, Liu CL, Yang Z, Newman AM, Alizadeh AA, Utz PJ, Gozani O.
PMID: 22024134
Epigenetics Chromatin. 2011 Oct 24;4:19. doi: 10.1186/1756-8935-4-19.

BACKGROUND: Signaling via protein lysine methylation has been proposed to play a central role in the regulation of many physiologic and pathologic programs. In contrast to other post-translational modifications such as phosphorylation, proteome-wide approaches to investigate lysine methylation networks...

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Levy D, Liu CL, Yang Z, et al. A proteomic approach for the identification of novel lysine methyltransferase substrates. Epigenetics Chromatin. 2011;4:19doi: 10.1186/1756-8935-4-19.
Levy, D., Liu, C. L., Yang, Z., Newman, A. M., Alizadeh, A. A., Utz, P. J., & Gozani, O. (2011). A proteomic approach for the identification of novel lysine methyltransferase substrates. Epigenetics & chromatin, 419. https://doi.org/10.1186/1756-8935-4-19
Levy, Dan, et al. "A proteomic approach for the identification of novel lysine methyltransferase substrates." Epigenetics & chromatin vol. 4 (2011): 19. doi: https://doi.org/10.1186/1756-8935-4-19
Levy D, Liu CL, Yang Z, Newman AM, Alizadeh AA, Utz PJ, Gozani O. A proteomic approach for the identification of novel lysine methyltransferase substrates. Epigenetics Chromatin. 2011 Oct 24;4:19. doi: 10.1186/1756-8935-4-19. PMID: 22024134; PMCID: PMC3212905.
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MiR-519d-5p modulates the sensitivity of breast cancer to chemotherapy by forming a negative feedback loop with RELA.

Annals of translational medicine

Li D, Wang T, Yu Z, Zhang Y, Wu X, Zheng N, Zhang W, Wu L.
PMID: 34430612
Ann Transl Med. 2021 Jul;9(14):1171. doi: 10.21037/atm-21-3241.

BACKGROUND: The chemoresistance of breast cancer (BC) has become the main cause of treatment failure. MicroRNAs (miRNAs) play a critical role in tumorigenesis, development, and chemoresistance, but the underlying mechanism of miR-519d in BC development and chemotherapy sensitivity remains...

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Li D, Wang T, Yu Z, et al. MiR-519d-5p modulates the sensitivity of breast cancer to chemotherapy by forming a negative feedback loop with RELA. Ann Transl Med. 2021;9(14):1171doi: 10.21037/atm-21-3241.
Li, D., Wang, T., Yu, Z., Zhang, Y., Wu, X., Zheng, N., Zhang, W., & Wu, L. (2021). MiR-519d-5p modulates the sensitivity of breast cancer to chemotherapy by forming a negative feedback loop with RELA. Annals of translational medicine, 9(14), 1171. https://doi.org/10.21037/atm-21-3241
Li, Ding, et al. "MiR-519d-5p modulates the sensitivity of breast cancer to chemotherapy by forming a negative feedback loop with RELA." Annals of translational medicine vol. 9,14 (2021): 1171. doi: https://doi.org/10.21037/atm-21-3241
Li D, Wang T, Yu Z, Zhang Y, Wu X, Zheng N, Zhang W, Wu L. MiR-519d-5p modulates the sensitivity of breast cancer to chemotherapy by forming a negative feedback loop with RELA. Ann Transl Med. 2021 Jul;9(14):1171. doi: 10.21037/atm-21-3241. PMID: 34430612; PMCID: PMC8350717.
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Aggressive Supratentorial Ependymoma, RELA Fusion-Positive with Extracranial Metastasis: A Case Report.

Journal of pathology and translational medicine

Kim SI, Lee Y, Kim SK, Kang HJ, Park SH.
PMID: 29161788
J Pathol Transl Med. 2017 Nov;51(6):588-593. doi: 10.4132/jptm.2017.08.10. Epub 2017 Nov 15.

Ependymoma is the third most common pediatric primary brain tumor. Ependymomas are categorized according to their locations and genetic abnormalities, and these two parameters are important prognostic factors for patient outcome. For supratentorial (ST) ependymomas, RELA fusion-positive ependymomas show...

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Kim SI, Lee Y, Kim SK, et al. Aggressive Supratentorial Ependymoma, RELA Fusion-Positive with Extracranial Metastasis: A Case Report. J Pathol Transl Med. 2017;51(6):588-593doi: 10.4132/jptm.2017.08.10.
Kim, S. I., Lee, Y., Kim, S. K., Kang, H. J., & Park, S. H. (2017). Aggressive Supratentorial Ependymoma, RELA Fusion-Positive with Extracranial Metastasis: A Case Report. Journal of pathology and translational medicine, 51(6), 588-593. https://doi.org/10.4132/jptm.2017.08.10
Kim, Seong-Ik, et al. "Aggressive Supratentorial Ependymoma, RELA Fusion-Positive with Extracranial Metastasis: A Case Report." Journal of pathology and translational medicine vol. 51,6 (2017): 588-593. doi: https://doi.org/10.4132/jptm.2017.08.10
Kim SI, Lee Y, Kim SK, Kang HJ, Park SH. Aggressive Supratentorial Ependymoma, RELA Fusion-Positive with Extracranial Metastasis: A Case Report. J Pathol Transl Med. 2017 Nov;51(6):588-593. doi: 10.4132/jptm.2017.08.10. Epub 2017 Nov 15. PMID: 29161788; PMCID: PMC5700879.
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Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma.

Journal of oncology

Wang R, Wang Q.
PMID: 34992653
J Oncol. 2021 Dec 28;2021:1370451. doi: 10.1155/2021/1370451. eCollection 2021.

BACKGROUND: Neuroblastoma is a common solid tumor originating from the sympathetic nervous system, commonly found in children, and it is one of the leading causes of tumor-related deaths in children. In addition to pathological features, molecular-level features, such as...

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Wang R, Wang Q. Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. J Oncol. 2021;2021:1370451doi: 10.1155/2021/1370451.
Wang, R., & Wang, Q. (2021). Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. Journal of oncology, 20211370451. https://doi.org/10.1155/2021/1370451
Wang, Rujia, and Wang, Qian. "Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma." Journal of oncology vol. 2021 (2021): 1370451. doi: https://doi.org/10.1155/2021/1370451
Wang R, Wang Q. Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. J Oncol. 2021 Dec 28;2021:1370451. doi: 10.1155/2021/1370451. eCollection 2021. PMID: 34992653; PMCID: PMC8727167.
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Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma.

Journal of oncology

Wang R, Wang Q.
PMID: 34992653
J Oncol. 2021 Dec 28;2021:1370451. doi: 10.1155/2021/1370451. eCollection 2021.

BACKGROUND: Neuroblastoma is a common solid tumor originating from the sympathetic nervous system, commonly found in children, and it is one of the leading causes of tumor-related deaths in children. In addition to pathological features, molecular-level features, such as...

Cite
Wang R, Wang Q. Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. J Oncol. 2021;2021:1370451doi: 10.1155/2021/1370451.
Wang, R., & Wang, Q. (2021). Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. Journal of oncology, 20211370451. https://doi.org/10.1155/2021/1370451
Wang, Rujia, and Wang, Qian. "Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma." Journal of oncology vol. 2021 (2021): 1370451. doi: https://doi.org/10.1155/2021/1370451
Wang R, Wang Q. Identification and External Validation of a Transcription Factor-Related Prognostic Signature in Pediatric Neuroblastoma. J Oncol. 2021 Dec 28;2021:1370451. doi: 10.1155/2021/1370451. eCollection 2021. PMID: 34992653; PMCID: PMC8727167.
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Showing 13 to 24 of 24 entries