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Showing 157 to 166 of 166 entries
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Myelin and non-myelin debris contribute to foamy macrophage formation after spinal cord injury.

Neurobiology of disease

Ryan CB, Choi JS, Al-Ali H, Lee JK.
PMID: 34979258
Neurobiol Dis. 2021 Dec 31;105608. doi: 10.1016/j.nbd.2021.105608. Epub 2021 Dec 31.

Tissue damage after spinal cord injury (SCI) elicits a robust inflammatory cascade that fails to resolve in a timely manner, resulting in impaired wound healing and cellular regeneration. This inflammatory response is partly mediated by infiltrating immune cells, including...

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Ryan CB, Choi JS, Al-Ali H, et al. Myelin and non-myelin debris contribute to foamy macrophage formation after spinal cord injury. Neurobiol Dis. 2021;105608doi: 10.1016/j.nbd.2021.105608.
Ryan, C. B., Choi, J. S., Al-Ali, H., & Lee, J. K. (2021). Myelin and non-myelin debris contribute to foamy macrophage formation after spinal cord injury. Neurobiology of disease, 105608. https://doi.org/10.1016/j.nbd.2021.105608
Ryan, Christine B, et al. "Myelin and non-myelin debris contribute to foamy macrophage formation after spinal cord injury." Neurobiology of disease vol. (2021): 105608. doi: https://doi.org/10.1016/j.nbd.2021.105608
Ryan CB, Choi JS, Al-Ali H, Lee JK. Myelin and non-myelin debris contribute to foamy macrophage formation after spinal cord injury. Neurobiol Dis. 2021 Dec 31;105608. doi: 10.1016/j.nbd.2021.105608. Epub 2021 Dec 31. PMID: 34979258.
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Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities.

Clinical genetics

Alawbathani S, Westenberger A, Ordonez-Herrera N, Al-Hilali M, Al Hebby H, Alabbas F, Alhashem AM, Elyamany G, Megarbane A, Kose M, Alhashmi N, Al Sukaiti N, Al-Raqad M, Al-Tawalbeh S, Abu Adas Blanco O, Alkhattabi F, Sng D, Al-Ali R, Khan S, Tawamie H, Tripolszki K, Karageorgou V, Trunzo R, Al Mutairi F, Reversade B, Bauer P, Bertoli-Avella AM.
PMID: 34708404
Clin Genet. 2022 Feb;101(2):247-254. doi: 10.1111/cge.14081. Epub 2021 Nov 06.

Biallelic changes in the ZNFX1 gene have been recently reported to cause severe familial immunodeficiency. Through a search of our bio/databank with information from genetic testing of >55 000 individuals, we identified nine additional patients from seven families with...

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Alawbathani S, Westenberger A, Ordonez-Herrera N, et al. Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clin Genet. 2021;101(2):247-254doi: 10.1111/cge.14081.
Alawbathani, S., Westenberger, A., Ordonez-Herrera, N., Al-Hilali, M., Al Hebby, H., Alabbas, F., Alhashem, A. M., Elyamany, G., Megarbane, A., Kose, M., Alhashmi, N., Al Sukaiti, N., Al-Raqad, M., Al-Tawalbeh, S., Abu Adas Blanco, O., Alkhattabi, F., Sng, D., Al-Ali, R., Khan, S., Tawamie, H., Tripolszki, K., Karageorgou, V., Trunzo, R., Al Mutairi, F., Reversade, B., Bauer, P., & Bertoli-Avella, A. M. (2022). Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clinical genetics, 101(2), 247-254. https://doi.org/10.1111/cge.14081
Alawbathani, Salem, et al. "Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities." Clinical genetics vol. 101,2 (2022): 247-254. doi: https://doi.org/10.1111/cge.14081
Alawbathani S, Westenberger A, Ordonez-Herrera N, Al-Hilali M, Al Hebby H, Alabbas F, Alhashem AM, Elyamany G, Megarbane A, Kose M, Alhashmi N, Al Sukaiti N, Al-Raqad M, Al-Tawalbeh S, Abu Adas Blanco O, Alkhattabi F, Sng D, Al-Ali R, Khan S, Tawamie H, Tripolszki K, Karageorgou V, Trunzo R, Al Mutairi F, Reversade B, Bauer P, Bertoli-Avella AM. Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clin Genet. 2022 Feb;101(2):247-254. doi: 10.1111/cge.14081. Epub 2021 Nov 06. PMID: 34708404.
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Association of Ambient Fine Particulate Matter Air Pollution With Kidney Transplant Outcomes.

JAMA network open

Chang SH, Merzkani M, Murad H, Wang M, Bowe B, Lentine KL, Al-Aly Z, Alhamad T.
PMID: 34618038
JAMA Netw Open. 2021 Oct 01;4(10):e2128190. doi: 10.1001/jamanetworkopen.2021.28190.

IMPORTANCE: Increased levels of ambient fine particulate matter (PM2.5) air pollution are associated with increased risks for detrimental health outcomes, but risks for patients with kidney transplants (KTs) remain unknown.OBJECTIVE: To investigate the association of PM2.5 exposure with KT...

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Chang SH, Merzkani M, Murad H, et al. Association of Ambient Fine Particulate Matter Air Pollution With Kidney Transplant Outcomes. JAMA Netw Open. 2021;4(10):e2128190doi: 10.1001/jamanetworkopen.2021.28190.
Chang, S. H., Merzkani, M., Murad, H., Wang, M., Bowe, B., Lentine, K. L., Al-Aly, Z., & Alhamad, T. (2021). Association of Ambient Fine Particulate Matter Air Pollution With Kidney Transplant Outcomes. JAMA network open, 4(10), e2128190. https://doi.org/10.1001/jamanetworkopen.2021.28190
Chang, Su-Hsin, et al. "Association of Ambient Fine Particulate Matter Air Pollution With Kidney Transplant Outcomes." JAMA network open vol. 4,10 (2021): e2128190. doi: https://doi.org/10.1001/jamanetworkopen.2021.28190
Chang SH, Merzkani M, Murad H, Wang M, Bowe B, Lentine KL, Al-Aly Z, Alhamad T. Association of Ambient Fine Particulate Matter Air Pollution With Kidney Transplant Outcomes. JAMA Netw Open. 2021 Oct 01;4(10):e2128190. doi: 10.1001/jamanetworkopen.2021.28190. PMID: 34618038; PMCID: PMC8498852.
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Phenotypic Screening Following Transcriptomic Deconvolution to Identify Transcription Factors Mediating Axon Growth Induced by a Kinase Inhibitor.

SLAS discovery : advancing life sciences R & D

Lowell JA, O'Neill N, Danzi MC, Al-Ali H, Bixby JL, Lemmon VP.
PMID: 34218704
SLAS Discov. 2021 Dec;26(10):1337-1354. doi: 10.1177/24725552211026270. Epub 2021 Jul 03.

After injury to the central nervous system (CNS), both neuron-intrinsic limitations on regenerative responses and inhibitory factors in the injured CNS environment restrict regenerative axon growth. Instances of successful axon regrowth offer opportunities to identify features that differentiate these...

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Lowell JA, O'Neill N, Danzi MC, et al. Phenotypic Screening Following Transcriptomic Deconvolution to Identify Transcription Factors Mediating Axon Growth Induced by a Kinase Inhibitor. SLAS Discov. 2021;26(10):1337-1354doi: 10.1177/24725552211026270.
Lowell, J. A., O'Neill, N., Danzi, M. C., Al-Ali, H., Bixby, J. L., & Lemmon, V. P. (2021). Phenotypic Screening Following Transcriptomic Deconvolution to Identify Transcription Factors Mediating Axon Growth Induced by a Kinase Inhibitor. SLAS discovery : advancing life sciences R & D, 26(10), 1337-1354. https://doi.org/10.1177/24725552211026270
Lowell, Jeffrey A, et al. "Phenotypic Screening Following Transcriptomic Deconvolution to Identify Transcription Factors Mediating Axon Growth Induced by a Kinase Inhibitor." SLAS discovery : advancing life sciences R & D vol. 26,10 (2021): 1337-1354. doi: https://doi.org/10.1177/24725552211026270
Lowell JA, O'Neill N, Danzi MC, Al-Ali H, Bixby JL, Lemmon VP. Phenotypic Screening Following Transcriptomic Deconvolution to Identify Transcription Factors Mediating Axon Growth Induced by a Kinase Inhibitor. SLAS Discov. 2021 Dec;26(10):1337-1354. doi: 10.1177/24725552211026270. Epub 2021 Jul 03. PMID: 34218704.
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Microarray analysis reveals ONC201 mediated differential mechanisms of CHOP gene regulation in metastatic and nonmetastatic colorectal cancer cells.

Scientific reports

Al Madhoun A, Haddad D, Al Tarrah M, Jacob S, Al-Ali W, Nizam R, Miranda L, Al-Rashed F, Sindhu S, Ahmad R, Bitar MS, Al-Mulla F.
PMID: 34088951
Sci Rep. 2021 Jun 04;11(1):11893. doi: 10.1038/s41598-021-91092-8.

The imipramine ONC201 has antiproliferative effects in several cancer cell types and activates integrated stress response pathway associated with the induction of Damage Inducible Transcript 3 (DDIT3, also known as C/EBP homologous protein or CHOP). We investigated the signaling...

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Al Madhoun A, Haddad D, Al Tarrah M, et al. Microarray analysis reveals ONC201 mediated differential mechanisms of CHOP gene regulation in metastatic and nonmetastatic colorectal cancer cells. Sci Rep. 2021;11(1):11893doi: 10.1038/s41598-021-91092-8.
Al Madhoun, A., Haddad, D., Al Tarrah, M., Jacob, S., Al-Ali, W., Nizam, R., Miranda, L., Al-Rashed, F., Sindhu, S., Ahmad, R., Bitar, M. S., & Al-Mulla, F. (2021). Microarray analysis reveals ONC201 mediated differential mechanisms of CHOP gene regulation in metastatic and nonmetastatic colorectal cancer cells. Scientific reports, 11(1), 11893. https://doi.org/10.1038/s41598-021-91092-8
Al Madhoun, Ashraf, et al. "Microarray analysis reveals ONC201 mediated differential mechanisms of CHOP gene regulation in metastatic and nonmetastatic colorectal cancer cells." Scientific reports vol. 11,1 (2021): 11893. doi: https://doi.org/10.1038/s41598-021-91092-8
Al Madhoun A, Haddad D, Al Tarrah M, Jacob S, Al-Ali W, Nizam R, Miranda L, Al-Rashed F, Sindhu S, Ahmad R, Bitar MS, Al-Mulla F. Microarray analysis reveals ONC201 mediated differential mechanisms of CHOP gene regulation in metastatic and nonmetastatic colorectal cancer cells. Sci Rep. 2021 Jun 04;11(1):11893. doi: 10.1038/s41598-021-91092-8. PMID: 34088951; PMCID: PMC8178367.
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Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities.

Clinical genetics

Alawbathani S, Westenberger A, Ordonez-Herrera N, Al-Hilali M, Al Hebby H, Alabbas F, Alhashem AM, Elyamany G, Megarbane A, Kose M, Alhashmi N, Al Sukaiti N, Al-Raqad M, Al-Tawalbeh S, Abu Adas Blanco O, Alkhattabi F, Sng D, Al-Ali R, Khan S, Tawamie H, Tripolszki K, Karageorgou V, Trunzo R, Al Mutairi F, Reversade B, Bauer P, Bertoli-Avella AM.
PMID: 34708404
Clin Genet. 2022 Feb;101(2):247-254. doi: 10.1111/cge.14081. Epub 2021 Nov 06.

Biallelic changes in the ZNFX1 gene have been recently reported to cause severe familial immunodeficiency. Through a search of our bio/databank with information from genetic testing of >55 000 individuals, we identified nine additional patients from seven families with...

Cite
Alawbathani S, Westenberger A, Ordonez-Herrera N, et al. Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clin Genet. 2021;101(2):247-254doi: 10.1111/cge.14081.
Alawbathani, S., Westenberger, A., Ordonez-Herrera, N., Al-Hilali, M., Al Hebby, H., Alabbas, F., Alhashem, A. M., Elyamany, G., Megarbane, A., Kose, M., Alhashmi, N., Al Sukaiti, N., Al-Raqad, M., Al-Tawalbeh, S., Abu Adas Blanco, O., Alkhattabi, F., Sng, D., Al-Ali, R., Khan, S., Tawamie, H., Tripolszki, K., Karageorgou, V., Trunzo, R., Al Mutairi, F., Reversade, B., Bauer, P., & Bertoli-Avella, A. M. (2022). Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clinical genetics, 101(2), 247-254. https://doi.org/10.1111/cge.14081
Alawbathani, Salem, et al. "Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities." Clinical genetics vol. 101,2 (2022): 247-254. doi: https://doi.org/10.1111/cge.14081
Alawbathani S, Westenberger A, Ordonez-Herrera N, Al-Hilali M, Al Hebby H, Alabbas F, Alhashem AM, Elyamany G, Megarbane A, Kose M, Alhashmi N, Al Sukaiti N, Al-Raqad M, Al-Tawalbeh S, Abu Adas Blanco O, Alkhattabi F, Sng D, Al-Ali R, Khan S, Tawamie H, Tripolszki K, Karageorgou V, Trunzo R, Al Mutairi F, Reversade B, Bauer P, Bertoli-Avella AM. Biallelic ZNFX1 variants are associated with a spectrum of immuno-hematological abnormalities. Clin Genet. 2022 Feb;101(2):247-254. doi: 10.1111/cge.14081. Epub 2021 Nov 06. PMID: 34708404.
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Histone Modification in NSCLC: Molecular Mechanisms and Therapeutic Targets.

International journal of molecular sciences

Bajbouj K, Al-Ali A, Ramakrishnan RK, Saber-Ayad M, Hamid Q.
PMID: 34769131
Int J Mol Sci. 2021 Oct 28;22(21). doi: 10.3390/ijms222111701.

Lung cancer is the leading cause of cancer mortality in both genders, with non-small cell lung cancer (NSCLC) accounting for about 85% of all lung cancers. At the time of diagnosis, the tumour is usually locally advanced or metastatic,...

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Bajbouj K, Al-Ali A, Ramakrishnan RK, et al. Histone Modification in NSCLC: Molecular Mechanisms and Therapeutic Targets. Int J Mol Sci. 2021;22(21)doi: 10.3390/ijms222111701.
Bajbouj, K., Al-Ali, A., Ramakrishnan, R. K., Saber-Ayad, M., & Hamid, Q. (2021). Histone Modification in NSCLC: Molecular Mechanisms and Therapeutic Targets. International journal of molecular sciences, 22(21), . https://doi.org/10.3390/ijms222111701
Bajbouj, Khuloud, et al. "Histone Modification in NSCLC: Molecular Mechanisms and Therapeutic Targets." International journal of molecular sciences vol. 22,21 (2021). doi: https://doi.org/10.3390/ijms222111701
Bajbouj K, Al-Ali A, Ramakrishnan RK, Saber-Ayad M, Hamid Q. Histone Modification in NSCLC: Molecular Mechanisms and Therapeutic Targets. Int J Mol Sci. 2021 Oct 28;22(21). doi: 10.3390/ijms222111701. PMID: 34769131; PMCID: PMC8584007.
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Recurrent terminal ventricle cyst: a case report.

Journal of surgical case reports

Baig Mirza A, Visagan R, Boardman T, Murphy C, Al-Ali B, Kellett C, Grahovac G.
PMID: 34804483
J Surg Case Rep. 2021 Nov 11;2021(11):rjab498. doi: 10.1093/jscr/rjab498. eCollection 2021 Nov.

The terminal ventricle (TV) of Krause is a rare cystic dilation of the conus' central canal. Due to limited understanding surrounding its pathophysiology, optimal management remains controversial. We report a 25-year-old female presenting with acute paraparesis. Magnetic resonance imaging...

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Baig Mirza A, Visagan R, Boardman T, et al. Recurrent terminal ventricle cyst: a case report. J Surg Case Rep. 2021;2021(11):rjab498doi: 10.1093/jscr/rjab498.
Baig Mirza, A., Visagan, R., Boardman, T., Murphy, C., Al-Ali, B., Kellett, C., & Grahovac, G. (2021). Recurrent terminal ventricle cyst: a case report. Journal of surgical case reports, 2021(11), rjab498. https://doi.org/10.1093/jscr/rjab498
Baig Mirza, Asfand, et al. "Recurrent terminal ventricle cyst: a case report." Journal of surgical case reports vol. 2021,11 (2021): rjab498. doi: https://doi.org/10.1093/jscr/rjab498
Baig Mirza A, Visagan R, Boardman T, Murphy C, Al-Ali B, Kellett C, Grahovac G. Recurrent terminal ventricle cyst: a case report. J Surg Case Rep. 2021 Nov 11;2021(11):rjab498. doi: 10.1093/jscr/rjab498. eCollection 2021 Nov. PMID: 34804483; PMCID: PMC8598984.
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Mechanical Thrombectomy for Treatment of Cerebral Venous Sinus Thrombosis in Vaccine-Induced Immune Thrombotic Thrombocytopenia.

AJNR. American journal of neuroradiology

Chew HS, Al-Ali S, Butler B, Rajapakse D, Nader K, Chavda S, Lamin S.
PMID: 34949591
AJNR Am J Neuroradiol. 2022 Jan;43(1):98-101. doi: 10.3174/ajnr.A7381. Epub 2021 Dec 23.

Reports of a rare form of cerebral venous sinus thrombosis with profound thrombocytopenia have emerged following introduction of the adenovirus-vectored coronavirus disease 2019 (COVID-19) vaccines. Between March and June 2021, seven cases of refractory vaccine-induced immune thrombotic thrombocytopenia were...

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Chew HS, Al-Ali S, Butler B, et al. Mechanical Thrombectomy for Treatment of Cerebral Venous Sinus Thrombosis in Vaccine-Induced Immune Thrombotic Thrombocytopenia. AJNR Am J Neuroradiol. 2021;43(1):98-101doi: 10.3174/ajnr.A7381.
Chew, H. S., Al-Ali, S., Butler, B., Rajapakse, D., Nader, K., Chavda, S., & Lamin, S. (2022). Mechanical Thrombectomy for Treatment of Cerebral Venous Sinus Thrombosis in Vaccine-Induced Immune Thrombotic Thrombocytopenia. AJNR. American journal of neuroradiology, 43(1), 98-101. https://doi.org/10.3174/ajnr.A7381
Chew, H S, et al. "Mechanical Thrombectomy for Treatment of Cerebral Venous Sinus Thrombosis in Vaccine-Induced Immune Thrombotic Thrombocytopenia." AJNR. American journal of neuroradiology vol. 43,1 (2022): 98-101. doi: https://doi.org/10.3174/ajnr.A7381
Chew HS, Al-Ali S, Butler B, Rajapakse D, Nader K, Chavda S, Lamin S. Mechanical Thrombectomy for Treatment of Cerebral Venous Sinus Thrombosis in Vaccine-Induced Immune Thrombotic Thrombocytopenia. AJNR Am J Neuroradiol. 2022 Jan;43(1):98-101. doi: 10.3174/ajnr.A7381. Epub 2021 Dec 23. PMID: 34949591.
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Early Diagnosis of Classic Homocystinuria in Kuwait through Newborn Screening: A 6-Year Experience.

International journal of neonatal screening

Alsharhan H, Ahmed AA, Ali NM, Alahmad A, Albash B, Elshafie RM, Alkanderi S, Elkazzaz UM, Cyril PX, Abdelrahman RM, Elmonairy AA, Ibrahim SM, Elfeky YME, Sadik DI, Al-Enezi SD, Salloum AM, Girish Y, Al-Ali M, Ramadan DG, Alsafi R, Al-Rushood M, Bastaki L.
PMID: 34449519
Int J Neonatal Screen. 2021 Aug 17;7(3). doi: 10.3390/ijns7030056.

Kuwait is a small Arabian Gulf country with a high rate of consanguinity and where a national newborn screening program was expanded in October 2014 to include a wide range of endocrine and metabolic disorders. A retrospective study conducted...

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Alsharhan H, Ahmed AA, Ali NM, et al. Early Diagnosis of Classic Homocystinuria in Kuwait through Newborn Screening: A 6-Year Experience. Int J Neonatal Screen. 2021;7(3)doi: 10.3390/ijns7030056.
Alsharhan, H., Ahmed, A. A., Ali, N. M., Alahmad, A., Albash, B., Elshafie, R. M., Alkanderi, S., Elkazzaz, U. M., Cyril, P. X., Abdelrahman, R. M., Elmonairy, A. A., Ibrahim, S. M., Elfeky, Y. M. E., Sadik, D. I., Al-Enezi, S. D., Salloum, A. M., Girish, Y., Al-Ali, M., Ramadan, D. G., Alsafi, R., Al-Rushood, M., & Bastaki, L. (2021). Early Diagnosis of Classic Homocystinuria in Kuwait through Newborn Screening: A 6-Year Experience. International journal of neonatal screening, 7(3), . https://doi.org/10.3390/ijns7030056
Alsharhan, Hind, et al. "Early Diagnosis of Classic Homocystinuria in Kuwait through Newborn Screening: A 6-Year Experience." International journal of neonatal screening vol. 7,3 (2021). doi: https://doi.org/10.3390/ijns7030056
Alsharhan H, Ahmed AA, Ali NM, Alahmad A, Albash B, Elshafie RM, Alkanderi S, Elkazzaz UM, Cyril PX, Abdelrahman RM, Elmonairy AA, Ibrahim SM, Elfeky YME, Sadik DI, Al-Enezi SD, Salloum AM, Girish Y, Al-Ali M, Ramadan DG, Alsafi R, Al-Rushood M, Bastaki L. Early Diagnosis of Classic Homocystinuria in Kuwait through Newborn Screening: A 6-Year Experience. Int J Neonatal Screen. 2021 Aug 17;7(3). doi: 10.3390/ijns7030056. PMID: 34449519; PMCID: PMC8395821.
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