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Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia.

Blood cancer discovery

Fischer U, Yang JJ, Ikawa T, Hein D, Vicente-Dueñas C, Borkhardt A, Sánchez-García I.
PMID: 33392513
Blood Cancer Discov. 2020 Nov;1(3):224-233. doi: 10.1158/2643-3230.BCD-20-0011. Epub 2020 Sep 14.

B-cells are an integral part of the adaptive immune system and regulate innate immunity. Derived from hematopoietic stem cells they mature through a series of cell fate decisions. Complex transcriptional circuits form and dissipate dynamically during these lineage restrictions....

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Fischer U, Yang JJ, Ikawa T, et al. Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood Cancer Discov. 2020;1(3):224-233doi: 10.1158/2643-3230.BCD-20-0011.
Fischer, U., Yang, J. J., Ikawa, T., Hein, D., Vicente-Dueñas, C., Borkhardt, A., & Sánchez-García, I. (2020). Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood cancer discovery, 1(3), 224-233. https://doi.org/10.1158/2643-3230.BCD-20-0011
Fischer, Ute, et al. "Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia." Blood cancer discovery vol. 1,3 (2020): 224-233. doi: https://doi.org/10.1158/2643-3230.BCD-20-0011
Fischer U, Yang JJ, Ikawa T, Hein D, Vicente-Dueñas C, Borkhardt A, Sánchez-García I. Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood Cancer Discov. 2020 Nov;1(3):224-233. doi: 10.1158/2643-3230.BCD-20-0011. Epub 2020 Sep 14. PMID: 33392513; PMCID: PMC7774874.
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Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia.

Blood cancer discovery

Fischer U, Yang JJ, Ikawa T, Hein D, Vicente-Dueñas C, Borkhardt A, Sánchez-García I.
PMID: 33392513
Blood Cancer Discov. 2020 Nov;1(3):224-233. doi: 10.1158/2643-3230.BCD-20-0011. Epub 2020 Sep 14.

B-cells are an integral part of the adaptive immune system and regulate innate immunity. Derived from hematopoietic stem cells they mature through a series of cell fate decisions. Complex transcriptional circuits form and dissipate dynamically during these lineage restrictions....

Cite
Fischer U, Yang JJ, Ikawa T, et al. Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood Cancer Discov. 2020;1(3):224-233doi: 10.1158/2643-3230.BCD-20-0011.
Fischer, U., Yang, J. J., Ikawa, T., Hein, D., Vicente-Dueñas, C., Borkhardt, A., & Sánchez-García, I. (2020). Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood cancer discovery, 1(3), 224-233. https://doi.org/10.1158/2643-3230.BCD-20-0011
Fischer, Ute, et al. "Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia." Blood cancer discovery vol. 1,3 (2020): 224-233. doi: https://doi.org/10.1158/2643-3230.BCD-20-0011
Fischer U, Yang JJ, Ikawa T, Hein D, Vicente-Dueñas C, Borkhardt A, Sánchez-García I. Cell Fate Decisions: The Role of Transcription Factors in Early B-cell Development and Leukemia. Blood Cancer Discov. 2020 Nov;1(3):224-233. doi: 10.1158/2643-3230.BCD-20-0011. Epub 2020 Sep 14. PMID: 33392513; PMCID: PMC7774874.
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Severe encephalopathy, lactic acidosis, vegetative instability and neuropathy with 5-Fluorouracil treatment - pyrimidine degradation defect or beriberi?.

Case reports in oncology

Rosen A, van Kuilenburg A, Assmann B, Kuhlen M, Borkhardt A.
PMID: 21941485
Case Rep Oncol. 2011 May;4(2):371-6. doi: 10.1159/000328803. Epub 2011 Aug 10.

We present the case of a 19-year-old female with nasopharyngeal carcinoma, who received two courses of chemotherapy with 5-fluorouracil (5-FU) in combination with folic acid and cisplatin. Upon developing esophageal strictures in the course of her radiotherapy, she required...

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Rosen A, van Kuilenburg A, Assmann B, et al. Severe encephalopathy, lactic acidosis, vegetative instability and neuropathy with 5-Fluorouracil treatment - pyrimidine degradation defect or beriberi?. Case Rep Oncol. 2011;4(2):371-6doi: 10.1159/000328803.
Rosen, A., van Kuilenburg, A., Assmann, B., Kuhlen, M., & Borkhardt, A. (2011). Severe encephalopathy, lactic acidosis, vegetative instability and neuropathy with 5-Fluorouracil treatment - pyrimidine degradation defect or beriberi?. Case reports in oncology, 4(2), 371-6. https://doi.org/10.1159/000328803
Rosen, A, et al. "Severe encephalopathy, lactic acidosis, vegetative instability and neuropathy with 5-Fluorouracil treatment - pyrimidine degradation defect or beriberi?." Case reports in oncology vol. 4,2 (2011): 371-6. doi: https://doi.org/10.1159/000328803
Rosen A, van Kuilenburg A, Assmann B, Kuhlen M, Borkhardt A. Severe encephalopathy, lactic acidosis, vegetative instability and neuropathy with 5-Fluorouracil treatment - pyrimidine degradation defect or beriberi?. Case Rep Oncol. 2011 May;4(2):371-6. doi: 10.1159/000328803. Epub 2011 Aug 10. PMID: 21941485; PMCID: PMC3177792.
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Germline POT1 Deregulation Can Predispose to Myeloid Malignancies in Childhood.

International journal of molecular sciences

Michler P, Schedel A, Witschas M, Friedrich UA, Wagener R, Mehtonen J, Brozou T, Menzel M, Walter C, Nabi D, Pearce G, Erlacher M, Göhring G, Dugas M, Heinäniemi M, Borkhardt A, Stölzel F, Hauer J, Auer F.
PMID: 34769003
Int J Mol Sci. 2021 Oct 26;22(21). doi: 10.3390/ijms222111572.

While the shelterin complex guards and coordinates the mechanism of telomere regulation, deregulation of this process is tightly linked to malignant transformation and cancer. Here, we present the novel finding of a germline stop-gain variant (p.Q199*) in the shelterin...

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Michler P, Schedel A, Witschas M, et al. Germline POT1 Deregulation Can Predispose to Myeloid Malignancies in Childhood. Int J Mol Sci. 2021;22(21)doi: 10.3390/ijms222111572.
Michler, P., Schedel, A., Witschas, M., Friedrich, U. A., Wagener, R., Mehtonen, J., Brozou, T., Menzel, M., Walter, C., Nabi, D., Pearce, G., Erlacher, M., Göhring, G., Dugas, M., Heinäniemi, M., Borkhardt, A., Stölzel, F., Hauer, J., & Auer, F. (2021). Germline POT1 Deregulation Can Predispose to Myeloid Malignancies in Childhood. International journal of molecular sciences, 22(21), . https://doi.org/10.3390/ijms222111572
Michler, Pia, et al. "Germline POT1 Deregulation Can Predispose to Myeloid Malignancies in Childhood." International journal of molecular sciences vol. 22,21 (2021). doi: https://doi.org/10.3390/ijms222111572
Michler P, Schedel A, Witschas M, Friedrich UA, Wagener R, Mehtonen J, Brozou T, Menzel M, Walter C, Nabi D, Pearce G, Erlacher M, Göhring G, Dugas M, Heinäniemi M, Borkhardt A, Stölzel F, Hauer J, Auer F. Germline POT1 Deregulation Can Predispose to Myeloid Malignancies in Childhood. Int J Mol Sci. 2021 Oct 26;22(21). doi: 10.3390/ijms222111572. PMID: 34769003; PMCID: PMC8583981.
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Cyclic Crack Growth in Chemically Tailored Isotropic Austenitic Steel Processed by Electron Beam Powder Bed Fusion.

Materials (Basel, Switzerland)

Droste M, Wagner R, Günther J, Burkhardt C, Henkel S, Niendorf T, Biermann H.
PMID: 34772069
Materials (Basel). 2021 Nov 01;14(21). doi: 10.3390/ma14216544.

The present study analyzes the cyclic crack propagation behavior in an austenitic steel processed by electron beam powder bed fusion (PBF-EB). The threshold value of crack growth as well as the crack growth behavior in the Paris regime were...

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Droste M, Wagner R, Günther J, et al. Cyclic Crack Growth in Chemically Tailored Isotropic Austenitic Steel Processed by Electron Beam Powder Bed Fusion. Materials (Basel). 2021;14(21)doi: 10.3390/ma14216544.
Droste, M., Wagner, R., Günther, J., Burkhardt, C., Henkel, S., Niendorf, T., & Biermann, H. (2021). Cyclic Crack Growth in Chemically Tailored Isotropic Austenitic Steel Processed by Electron Beam Powder Bed Fusion. Materials (Basel, Switzerland), 14(21), . https://doi.org/10.3390/ma14216544
Droste, Matthias, et al. "Cyclic Crack Growth in Chemically Tailored Isotropic Austenitic Steel Processed by Electron Beam Powder Bed Fusion." Materials (Basel, Switzerland) vol. 14,21 (2021). doi: https://doi.org/10.3390/ma14216544
Droste M, Wagner R, Günther J, Burkhardt C, Henkel S, Niendorf T, Biermann H. Cyclic Crack Growth in Chemically Tailored Isotropic Austenitic Steel Processed by Electron Beam Powder Bed Fusion. Materials (Basel). 2021 Nov 01;14(21). doi: 10.3390/ma14216544. PMID: 34772069; PMCID: PMC8585378.
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Chromosomal risk classification in high hyperdiploid acute lymphocytic leukaemia: the beginning of a new chapter.

The Lancet. Haematology

Borkhardt A, Haas OA.
PMID: 34971585
Lancet Haematol. 2022 Jan;9(1):e9. doi: 10.1016/S2352-3026(21)00348-3.

No abstract available.

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Borkhardt A, Haas OA. Chromosomal risk classification in high hyperdiploid acute lymphocytic leukaemia: the beginning of a new chapter. Lancet Haematol. 2022;9(1):e9doi: 10.1016/S2352-3026(21)00348-3.
Borkhardt, A., & Haas, O. A. (2022). Chromosomal risk classification in high hyperdiploid acute lymphocytic leukaemia: the beginning of a new chapter. The Lancet. Haematology, 9(1), e9. https://doi.org/10.1016/S2352-3026(21)00348-3
Borkhardt, Arndt, and Haas, Oskar A. "Chromosomal risk classification in high hyperdiploid acute lymphocytic leukaemia: the beginning of a new chapter." The Lancet. Haematology vol. 9,1 (2022): e9. doi: https://doi.org/10.1016/S2352-3026(21)00348-3
Borkhardt A, Haas OA. Chromosomal risk classification in high hyperdiploid acute lymphocytic leukaemia: the beginning of a new chapter. Lancet Haematol. 2022 Jan;9(1):e9. doi: 10.1016/S2352-3026(21)00348-3. PMID: 34971585.
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Core-shell bioprinting as a strategy to apply differentiation factors in a spatially defined manner inside osteochondral tissue substitutes.

Biofabrication

Kilian D, Cometta S, Bernhardt A, Taymour R, Golde J, Ahlfeld T, Emmermacher J, Gelinsky M, Lode A.
PMID: 34933296
Biofabrication. 2022 Jan 06;14(1). doi: 10.1088/1758-5090/ac457b.

One of the key challenges in osteochondral tissue engineering is to define specified zones with varying material properties, cell types and biochemical factors supporting locally adjusted differentiation into the osteogenic and chondrogenic lineage, respectively. Herein, extrusion-based core-shell bioprinting is...

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Kilian D, Cometta S, Bernhardt A, et al. Core-shell bioprinting as a strategy to apply differentiation factors in a spatially defined manner inside osteochondral tissue substitutes. Biofabrication. 2022;14(1)doi: 10.1088/1758-5090/ac457b.
Kilian, D., Cometta, S., Bernhardt, A., Taymour, R., Golde, J., Ahlfeld, T., Emmermacher, J., Gelinsky, M., & Lode, A. (2022). Core-shell bioprinting as a strategy to apply differentiation factors in a spatially defined manner inside osteochondral tissue substitutes. Biofabrication, 14(1), . https://doi.org/10.1088/1758-5090/ac457b
Kilian, David, et al. "Core-shell bioprinting as a strategy to apply differentiation factors in a spatially defined manner inside osteochondral tissue substitutes." Biofabrication vol. 14,1 (2022). doi: https://doi.org/10.1088/1758-5090/ac457b
Kilian D, Cometta S, Bernhardt A, Taymour R, Golde J, Ahlfeld T, Emmermacher J, Gelinsky M, Lode A. Core-shell bioprinting as a strategy to apply differentiation factors in a spatially defined manner inside osteochondral tissue substitutes. Biofabrication. 2022 Jan 06;14(1). doi: 10.1088/1758-5090/ac457b. PMID: 34933296.
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Showing 301 to 307 of 307 entries