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Dietary Lecithin Supplementation Can Improve the Quality of the M. Longissimus thoracis.

Animals : an open access journal from MDPI

D'Souza DN, Blake BL, Williams IH, Mullan BP, Pethick DW, Dunshea FR.
PMID: 26610579
Animals (Basel). 2015 Nov 25;5(4):1180-91. doi: 10.3390/ani5040405.

Forty crossbred (Large White × Landrace × Duroc) female pigs (16.4 kg ± 0.94 kg) were used to investigate the effect of dietary lecithin supplementation on growth performance and pork quality. Pigs were randomly allocated to a commercial diet...

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D'Souza DN, Blake BL, Williams IH, et al. Dietary Lecithin Supplementation Can Improve the Quality of the M. Longissimus thoracis. Animals (Basel). 2015;5(4):1180-91doi: 10.3390/ani5040405.
D'Souza, D. N., Blake, B. L., Williams, I. H., Mullan, B. P., Pethick, D. W., & Dunshea, F. R. (2015). Dietary Lecithin Supplementation Can Improve the Quality of the M. Longissimus thoracis. Animals : an open access journal from MDPI, 5(4), 1180-91. https://doi.org/10.3390/ani5040405
D'Souza, Darryl N, et al. "Dietary Lecithin Supplementation Can Improve the Quality of the M. Longissimus thoracis." Animals : an open access journal from MDPI vol. 5,4 (2015): 1180-91. doi: https://doi.org/10.3390/ani5040405
D'Souza DN, Blake BL, Williams IH, Mullan BP, Pethick DW, Dunshea FR. Dietary Lecithin Supplementation Can Improve the Quality of the M. Longissimus thoracis. Animals (Basel). 2015 Nov 25;5(4):1180-91. doi: 10.3390/ani5040405. PMID: 26610579; PMCID: PMC4693209.
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Survival and severity in dominant cerebellar ataxias.

Annals of clinical and translational neurology

Monin ML, Tezenas du Montcel S, Marelli C, Cazeneuve C, Charles P, Tallaksen C, Forlani S, Stevanin G, Brice A, Durr A.
PMID: 25750924
Ann Clin Transl Neurol. 2015 Feb;2(2):202-7. doi: 10.1002/acn3.156. Epub 2015 Jan 07.

Inherited spinocerebellar ataxias (SCAs) are known to be genetically and clinically heterogeneous. Whether severity and survival are variable, however, is not known. We, therefore, studied survival and severity in 446 cases and 509 relatives with known mutations. Survival was...

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Monin ML, Tezenas du Montcel S, Marelli C, et al. Survival and severity in dominant cerebellar ataxias. Ann Clin Transl Neurol. 2015;2(2):202-7doi: 10.1002/acn3.156.
Monin, M. L., Tezenas du Montcel, S., Marelli, C., Cazeneuve, C., Charles, P., Tallaksen, C., Forlani, S., Stevanin, G., Brice, A., & Durr, A. (2015). Survival and severity in dominant cerebellar ataxias. Annals of clinical and translational neurology, 2(2), 202-7. https://doi.org/10.1002/acn3.156
Monin, Marie-Lorraine, et al. "Survival and severity in dominant cerebellar ataxias." Annals of clinical and translational neurology vol. 2,2 (2015): 202-7. doi: https://doi.org/10.1002/acn3.156
Monin ML, Tezenas du Montcel S, Marelli C, Cazeneuve C, Charles P, Tallaksen C, Forlani S, Stevanin G, Brice A, Durr A. Survival and severity in dominant cerebellar ataxias. Ann Clin Transl Neurol. 2015 Feb;2(2):202-7. doi: 10.1002/acn3.156. Epub 2015 Jan 07. PMID: 25750924; PMCID: PMC4338960.
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Identification of Social Anxiety in Schools: The Utility of a Two-Step Screening Process.

Contemporary school psychology

Sweeney C, Warner CM, Brice C, Stewart C, Ryan J, Loeb KL, McGrath RE.
PMID: 26609497
Contemp Sch Psychol. 2015 Dec 01;19(4):268-275. doi: 10.1007/s40688-015-0055-9. Epub 2015 May 28.

Social anxiety disorder (SAD) is highly prevalent yet largely undetected and untreated in adolescents despite the availability of effective treatments. Implementing interventions in schools enhances recognition and access to treatment for SAD. However, without reliable means to accurately identify...

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Sweeney C, Warner CM, Brice C, et al. Identification of Social Anxiety in Schools: The Utility of a Two-Step Screening Process. Contemp Sch Psychol. 2015;19(4):268-275doi: 10.1007/s40688-015-0055-9.
Sweeney, C., Warner, C. M., Brice, C., Stewart, C., Ryan, J., Loeb, K. L., & McGrath, R. E. (2015). Identification of Social Anxiety in Schools: The Utility of a Two-Step Screening Process. Contemporary school psychology, 19(4), 268-275. https://doi.org/10.1007/s40688-015-0055-9
Sweeney, Corinne, et al. "Identification of Social Anxiety in Schools: The Utility of a Two-Step Screening Process." Contemporary school psychology vol. 19,4 (2015): 268-275. doi: https://doi.org/10.1007/s40688-015-0055-9
Sweeney C, Warner CM, Brice C, Stewart C, Ryan J, Loeb KL, McGrath RE. Identification of Social Anxiety in Schools: The Utility of a Two-Step Screening Process. Contemp Sch Psychol. 2015 Dec 01;19(4):268-275. doi: 10.1007/s40688-015-0055-9. Epub 2015 May 28. PMID: 26609497; PMCID: PMC4654999.
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EFFECTS OF AVIAN SEED DISPERSAL ON THE GENETIC STRUCTURE OF WHITEBARK PINE POPULATIONS.

Evolution; international journal of organic evolution

Furnier GR, Knowles P, Clyde MA, Dancik BP.
PMID: 28563807
Evolution. 1987 May;41(3):607-612. doi: 10.1111/j.1558-5646.1987.tb05831.x.

We used allozyme analysis to examine family structure, the spatial patterning of related individuals, in two populations of whitebark pine (Pinus albicaulis), a subalpine conifer that commonly displays a multistem form. The individual stems within clumps are genetically distinct...

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Furnier GR, Knowles P, Clyde MA, et al. EFFECTS OF AVIAN SEED DISPERSAL ON THE GENETIC STRUCTURE OF WHITEBARK PINE POPULATIONS. Evolution. 1987;41(3):607-612doi: 10.1111/j.1558-5646.1987.tb05831.x.
Furnier, G. R., Knowles, P., Clyde, M. A., & Dancik, B. P. (1987). EFFECTS OF AVIAN SEED DISPERSAL ON THE GENETIC STRUCTURE OF WHITEBARK PINE POPULATIONS. Evolution; international journal of organic evolution, 41(3), 607-612. https://doi.org/10.1111/j.1558-5646.1987.tb05831.x
Furnier, Glenn R, et al. "EFFECTS OF AVIAN SEED DISPERSAL ON THE GENETIC STRUCTURE OF WHITEBARK PINE POPULATIONS." Evolution; international journal of organic evolution vol. 41,3 (1987): 607-612. doi: https://doi.org/10.1111/j.1558-5646.1987.tb05831.x
Furnier GR, Knowles P, Clyde MA, Dancik BP. EFFECTS OF AVIAN SEED DISPERSAL ON THE GENETIC STRUCTURE OF WHITEBARK PINE POPULATIONS. Evolution. 1987 May;41(3):607-612. doi: 10.1111/j.1558-5646.1987.tb05831.x. PMID: 28563807.
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Loss-of-function mutations in RAB39B are associated with typical early-onset Parkinson disease.

Neurology. Genetics

Lesage S, Bras J, Cormier-Dequaire F, Condroyer C, Nicolas A, Darwent L, Guerreiro R, Majounie E, Federoff M, Heutink P, Wood NW, Gasser T, Hardy J, Tison F, Singleton A, Brice A.
PMID: 27066548
Neurol Genet. 2015 Jun 18;1(1):e9. doi: 10.1212/NXG.0000000000000009. eCollection 2015 Jun.

Rab proteins are small molecular weight guanosine triphosphatases involved in the regulation of vesicular trafficking.(1) Three of 4 X-linked RAB genes are specific to the brain, including RAB39B. Recently, Wilson et al.(2) reported that mutations in RAB39B cause X-linked...

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Lesage S, Bras J, Cormier-Dequaire F, et al. Loss-of-function mutations in RAB39B are associated with typical early-onset Parkinson disease. Neurol Genet. 2015;1(1):e9doi: 10.1212/NXG.0000000000000009.
Lesage, S., Bras, J., Cormier-Dequaire, F., Condroyer, C., Nicolas, A., Darwent, L., Guerreiro, R., Majounie, E., Federoff, M., Heutink, P., Wood, N. W., Gasser, T., Hardy, J., Tison, F., Singleton, A., Brice, A. (2015). Loss-of-function mutations in RAB39B are associated with typical early-onset Parkinson disease. Neurology. Genetics, 1(1), e9. https://doi.org/10.1212/NXG.0000000000000009
Lesage, Suzanne, et al. "Loss-of-function mutations in RAB39B are associated with typical early-onset Parkinson disease." Neurology. Genetics vol. 1,1 (2015): e9. doi: https://doi.org/10.1212/NXG.0000000000000009
Lesage S, Bras J, Cormier-Dequaire F, Condroyer C, Nicolas A, Darwent L, Guerreiro R, Majounie E, Federoff M, Heutink P, Wood NW, Gasser T, Hardy J, Tison F, Singleton A, Brice A. Loss-of-function mutations in RAB39B are associated with typical early-onset Parkinson disease. Neurol Genet. 2015 Jun 18;1(1):e9. doi: 10.1212/NXG.0000000000000009. eCollection 2015 Jun. PMID: 27066548; PMCID: PMC4821081.
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Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention.

Alzheimer's & dementia (Amsterdam, Netherlands)

Doherty BM, Schultz SA, Oh JM, Koscik RL, Dowling NM, Barnhart TE, Murali D, Gallagher CL, Carlsson CM, Bendlin BB, LaRue A, Hermann BP, Rowley HA, Asthana S, Sager MA, Christian BT, Johnson SC, Okonkwo OC.
PMID: 26161436
Alzheimers Dement (Amst). 2015 Jun 01;1(2):160-169. doi: 10.1016/j.dadm.2015.01.003.

There is a growing interest in understanding how amyloid-β (Aβ) accumulation in preclinical Alzheimer's disease relates to brain morphometric measures and cognition. Existing investigations in this area have been primarily conducted in older cognitively-normal (CN) individuals. Therefore, not much...

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Doherty BM, Schultz SA, Oh JM, et al. Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimers Dement (Amst). 2015;1(2):160-169doi: 10.1016/j.dadm.2015.01.003.
Doherty, B. M., Schultz, S. A., Oh, J. M., Koscik, R. L., Dowling, N. M., Barnhart, T. E., Murali, D., Gallagher, C. L., Carlsson, C. M., Bendlin, B. B., LaRue, A., Hermann, B. P., Rowley, H. A., Asthana, S., Sager, M. A., Christian, B. T., Johnson, S. C., & Okonkwo, O. C. (2015). Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimer's & dementia (Amsterdam, Netherlands), 1(2), 160-169. https://doi.org/10.1016/j.dadm.2015.01.003
Doherty, Benjamin M, et al. "Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 1,2 (2015): 160-169. doi: https://doi.org/10.1016/j.dadm.2015.01.003
Doherty BM, Schultz SA, Oh JM, Koscik RL, Dowling NM, Barnhart TE, Murali D, Gallagher CL, Carlsson CM, Bendlin BB, LaRue A, Hermann BP, Rowley HA, Asthana S, Sager MA, Christian BT, Johnson SC, Okonkwo OC. Amyloid burden, cortical thickness, and cognitive function in the Wisconsin Registry for Alzheimer's Prevention. Alzheimers Dement (Amst). 2015 Jun 01;1(2):160-169. doi: 10.1016/j.dadm.2015.01.003. PMID: 26161436; PMCID: PMC4492165.
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Evolution of antimicrobial peptides to self-assembled peptides for biomaterial applications.

Pathogens (Basel, Switzerland)

McCloskey AP, Gilmore BF, Laverty G.
PMID: 25436505
Pathogens. 2014 Oct 03;3(4):791-821. doi: 10.3390/pathogens3040791.

Biomaterial-related infections are a persistent burden on patient health, recovery, mortality and healthcare budgets. Self-assembled antimicrobial peptides have evolved from the area of antimicrobial peptides. Peptides serve as important weapons in nature, and increasingly medicine, for combating microbial infection...

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McCloskey AP, Gilmore BF, Laverty G. Evolution of antimicrobial peptides to self-assembled peptides for biomaterial applications. Pathogens. 2014;3(4):791-821doi: 10.3390/pathogens3040791.
McCloskey, A. P., Gilmore, B. F., & Laverty, G. (2014). Evolution of antimicrobial peptides to self-assembled peptides for biomaterial applications. Pathogens (Basel, Switzerland), 3(4), 791-821. https://doi.org/10.3390/pathogens3040791
McCloskey, Alice P, et al. "Evolution of antimicrobial peptides to self-assembled peptides for biomaterial applications." Pathogens (Basel, Switzerland) vol. 3,4 (2014): 791-821. doi: https://doi.org/10.3390/pathogens3040791
McCloskey AP, Gilmore BF, Laverty G. Evolution of antimicrobial peptides to self-assembled peptides for biomaterial applications. Pathogens. 2014 Oct 03;3(4):791-821. doi: 10.3390/pathogens3040791. PMID: 25436505; PMCID: PMC4282886.
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Monocyte-derived macrophages do not explain susceptibility to pulmonary non-tuberculous mycobacterial disease.

Clinical & translational immunology

de Jong E, Lim A, Waterer G, Price P.
PMID: 25505947
Clin Transl Immunology. 2012 Nov 14;1(11):e2. doi: 10.1038/cti.2012.1. eCollection 2012 Nov.

Pulmonary infections with non-tuberculous mycobacteria (NTM) affect a subset of older individuals (mostly women) with no known immunological defects. As NTMs are intracellular pathogens, it is important to establish whether NTM disease is associated with defective production of Th1...

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de Jong E, Lim A, Waterer G, et al. Monocyte-derived macrophages do not explain susceptibility to pulmonary non-tuberculous mycobacterial disease. Clin Transl Immunology. 2012;1(11):e2doi: 10.1038/cti.2012.1.
de Jong, E., Lim, A., Waterer, G., & Price, P. (2012). Monocyte-derived macrophages do not explain susceptibility to pulmonary non-tuberculous mycobacterial disease. Clinical & translational immunology, 1(11), e2. https://doi.org/10.1038/cti.2012.1
de Jong, Emma, et al. "Monocyte-derived macrophages do not explain susceptibility to pulmonary non-tuberculous mycobacterial disease." Clinical & translational immunology vol. 1,11 (2012): e2. doi: https://doi.org/10.1038/cti.2012.1
de Jong E, Lim A, Waterer G, Price P. Monocyte-derived macrophages do not explain susceptibility to pulmonary non-tuberculous mycobacterial disease. Clin Transl Immunology. 2012 Nov 14;1(11):e2. doi: 10.1038/cti.2012.1. eCollection 2012 Nov. PMID: 25505947; PMCID: PMC4256549.
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HIV patients stable on ART retain evidence of a high CMV load but changes to Natural Killer cell phenotypes reflect both HIV and CMV.

AIDS research and therapy

Affandi JS, Montgomery J, Lee S, Price P.
PMID: 26664457
AIDS Res Ther. 2015 Dec 09;12:41. doi: 10.1186/s12981-015-0080-9. eCollection 2015.

BACKGROUND: Whilst ART corrects many effects of HIV disease, T cell populations retain features of accelerated immunological aging.METHODS: Here we analyse phenotypic changes to natural killer (NK) cells in HIV patients who began ART with

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Affandi JS, Montgomery J, Lee S, et al. HIV patients stable on ART retain evidence of a high CMV load but changes to Natural Killer cell phenotypes reflect both HIV and CMV. AIDS Res Ther. 2015;12:41doi: 10.1186/s12981-015-0080-9.
Affandi, J. S., Montgomery, J., Lee, S., & Price, P. (2015). HIV patients stable on ART retain evidence of a high CMV load but changes to Natural Killer cell phenotypes reflect both HIV and CMV. AIDS research and therapy, 1241. https://doi.org/10.1186/s12981-015-0080-9
Affandi, Jacquita S, et al. "HIV patients stable on ART retain evidence of a high CMV load but changes to Natural Killer cell phenotypes reflect both HIV and CMV." AIDS research and therapy vol. 12 (2015): 41. doi: https://doi.org/10.1186/s12981-015-0080-9
Affandi JS, Montgomery J, Lee S, Price P. HIV patients stable on ART retain evidence of a high CMV load but changes to Natural Killer cell phenotypes reflect both HIV and CMV. AIDS Res Ther. 2015 Dec 09;12:41. doi: 10.1186/s12981-015-0080-9. eCollection 2015. PMID: 26664457; PMCID: PMC4673723.
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[New discoveries in pediatrics to be used by the practitioner].

Bulletin medical

BAIZE P.
PMID: 13066851
Bull Med. 1953 May 15;67(10):261-70.

No abstract available.

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BAIZE P. Acquisitions nouvelles en pédiatrie à l'usage des praticiens. [New discoveries in pediatrics to be used by the practitioner]. Bull Med. 1953;67(10):261-70
BAIZE, P. (1953). Acquisitions nouvelles en pédiatrie à l'usage des praticiens. [New discoveries in pediatrics to be used by the practitioner]. Bulletin medical, 67(10), 261-70.
BAIZE, P. "Acquisitions nouvelles en pédiatrie à l'usage des praticiens." [New discoveries in pediatrics to be used by the practitioner]. Bulletin medical vol. 67,10 (1953): 261-70.
BAIZE P. Acquisitions nouvelles en pédiatrie à l'usage des praticiens. [New discoveries in pediatrics to be used by the practitioner]. Bull Med. 1953 May 15;67(10):261-70. Undetermined. PMID: 13066851.
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[New advances in pediatrics for the use of the practitioner].

Bulletin medical

BAIZE P.
PMID: 13182394
Bull Med. 1954 May 01;68(9):229-35.

No abstract available.

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BAIZE P. Acquistions nouvelles en pédiatrie à l'usage des practiciens. [New advances in pediatrics for the use of the practitioner]. Bull Med. 1954;68(9):229-35
BAIZE, P. (1954). Acquistions nouvelles en pédiatrie à l'usage des practiciens. [New advances in pediatrics for the use of the practitioner]. Bulletin medical, 68(9), 229-35.
BAIZE, P. "Acquistions nouvelles en pédiatrie à l'usage des practiciens." [New advances in pediatrics for the use of the practitioner]. Bulletin medical vol. 68,9 (1954): 229-35.
BAIZE P. Acquistions nouvelles en pédiatrie à l'usage des practiciens. [New advances in pediatrics for the use of the practitioner]. Bull Med. 1954 May 01;68(9):229-35. Undetermined. PMID: 13182394.
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The effects of caffeine on simulated driving, subjective alertness and sustained attention.

Human psychopharmacology

Brice C, Smith A.
PMID: 12404548
Hum Psychopharmacol. 2001 Oct;16(7):523-531. doi: 10.1002/hup.327.

There is evidence that caffeine increases alertness and reduces fatigue. This may be especially so in low arousal situations (e.g. working at night or for prolonged hours). Caffeine has also been found to improve performance on vigilance tasks and...

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Brice C, Smith A. The effects of caffeine on simulated driving, subjective alertness and sustained attention. Hum Psychopharmacol. 2001;16(7):523-531doi: 10.1002/hup.327.
Brice, C., & Smith, A. (2001). The effects of caffeine on simulated driving, subjective alertness and sustained attention. Human psychopharmacology, 16(7), 523-531. https://doi.org/10.1002/hup.327
Brice, Carolyn, and Smith, Andrew. "The effects of caffeine on simulated driving, subjective alertness and sustained attention." Human psychopharmacology vol. 16,7 (2001): 523-531. doi: https://doi.org/10.1002/hup.327
Brice C, Smith A. The effects of caffeine on simulated driving, subjective alertness and sustained attention. Hum Psychopharmacol. 2001 Oct;16(7):523-531. doi: 10.1002/hup.327. PMID: 12404548.
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Showing 13 to 24 of 645 entries