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Cancer cell growth and survival as a system-level property sustained by enhanced glycolysis and mitochondrial metabolic remodeling.

Frontiers in physiology

Alberghina L, Gaglio D, Gelfi C, Moresco RM, Mauri G, Bertolazzi P, Messa C, Gilardi MC, Chiaradonna F, Vanoni M.
PMID: 22988443
Front Physiol. 2012 Sep 12;3:362. doi: 10.3389/fphys.2012.00362. eCollection 2012.

Systems Biology holds that complex cellular functions are generated as system-level properties endowed with robustness, each involving large networks of molecular determinants, generally identified by "omics" analyses. In this paper we describe four basic cancer cell properties that can...

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Alberghina L, Gaglio D, Gelfi C, et al. Cancer cell growth and survival as a system-level property sustained by enhanced glycolysis and mitochondrial metabolic remodeling. Front Physiol. 2012;3:362doi: 10.3389/fphys.2012.00362.
Alberghina, L., Gaglio, D., Gelfi, C., Moresco, R. M., Mauri, G., Bertolazzi, P., Messa, C., Gilardi, M. C., Chiaradonna, F., & Vanoni, M. (2012). Cancer cell growth and survival as a system-level property sustained by enhanced glycolysis and mitochondrial metabolic remodeling. Frontiers in physiology, 3362. https://doi.org/10.3389/fphys.2012.00362
Alberghina, Lilia, et al. "Cancer cell growth and survival as a system-level property sustained by enhanced glycolysis and mitochondrial metabolic remodeling." Frontiers in physiology vol. 3 (2012): 362. doi: https://doi.org/10.3389/fphys.2012.00362
Alberghina L, Gaglio D, Gelfi C, Moresco RM, Mauri G, Bertolazzi P, Messa C, Gilardi MC, Chiaradonna F, Vanoni M. Cancer cell growth and survival as a system-level property sustained by enhanced glycolysis and mitochondrial metabolic remodeling. Front Physiol. 2012 Sep 12;3:362. doi: 10.3389/fphys.2012.00362. eCollection 2012. PMID: 22988443; PMCID: PMC3440026.
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Terpenoid treatment in osteoporosis: this is where we have come in research.

Trends in endocrinology and metabolism: TEM

Bellavia D, Caradonna F, Dimarco E, Costa V, Carina V, De Luca A, Raimondi L, Gentile C, Alessandro R, Fini M, Giavaresi G.
PMID: 34481733
Trends Endocrinol Metab. 2021 Nov;32(11):846-861. doi: 10.1016/j.tem.2021.07.011. Epub 2021 Sep 01.

Lower bone resistance to load is due to the imbalance of bone homeostasis, where excessive bone resorption, compared with bone formation, determines a progressive osteopenia, leading to a high risk of fractures and consequent pain and functional limitations. Terpenoids,...

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Bellavia D, Caradonna F, Dimarco E, et al. Terpenoid treatment in osteoporosis: this is where we have come in research. Trends Endocrinol Metab. 2021;32(11):846-861doi: 10.1016/j.tem.2021.07.011.
Bellavia, D., Caradonna, F., Dimarco, E., Costa, V., Carina, V., De Luca, A., Raimondi, L., Gentile, C., Alessandro, R., Fini, M., & Giavaresi, G. (2021). Terpenoid treatment in osteoporosis: this is where we have come in research. Trends in endocrinology and metabolism: TEM, 32(11), 846-861. https://doi.org/10.1016/j.tem.2021.07.011
Bellavia, Daniele, et al. "Terpenoid treatment in osteoporosis: this is where we have come in research." Trends in endocrinology and metabolism: TEM vol. 32,11 (2021): 846-861. doi: https://doi.org/10.1016/j.tem.2021.07.011
Bellavia D, Caradonna F, Dimarco E, Costa V, Carina V, De Luca A, Raimondi L, Gentile C, Alessandro R, Fini M, Giavaresi G. Terpenoid treatment in osteoporosis: this is where we have come in research. Trends Endocrinol Metab. 2021 Nov;32(11):846-861. doi: 10.1016/j.tem.2021.07.011. Epub 2021 Sep 01. PMID: 34481733.
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Intergenerational Effect of Early Life Exposure to Permethrin: Changes in Global DNA Methylation and in .

Toxics

Bordoni L, Nasuti C, Mirto M, Caradonna F, Gabbianelli R.
PMID: 29051472
Toxics. 2015 Nov 19;3(4):451-461. doi: 10.3390/toxics3040451.

Environmental exposure to pesticides during the early stages of development represents an important risk factor for the onset of neurodegenerative diseases in adult age. Neonatal exposure to Permethrin (PERM), a member of the family of synthetic pyrethroids, can induce...

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Bordoni L, Nasuti C, Mirto M, et al. Intergenerational Effect of Early Life Exposure to Permethrin: Changes in Global DNA Methylation and in . Toxics. 2015;3(4):451-461doi: 10.3390/toxics3040451.
Bordoni, L., Nasuti, C., Mirto, M., Caradonna, F., & Gabbianelli, R. (2015). Intergenerational Effect of Early Life Exposure to Permethrin: Changes in Global DNA Methylation and in . Toxics, 3(4), 451-461. https://doi.org/10.3390/toxics3040451
Bordoni, Laura, et al. "Intergenerational Effect of Early Life Exposure to Permethrin: Changes in Global DNA Methylation and in ." Toxics vol. 3,4 (2015): 451-461. doi: https://doi.org/10.3390/toxics3040451
Bordoni L, Nasuti C, Mirto M, Caradonna F, Gabbianelli R. Intergenerational Effect of Early Life Exposure to Permethrin: Changes in Global DNA Methylation and in . Toxics. 2015 Nov 19;3(4):451-461. doi: 10.3390/toxics3040451. PMID: 29051472; PMCID: PMC5606645.
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Transcriptional profiling of immortalized and K-ras-transformed mouse fibroblasts upon PKA stimulation by forskolin in low glucose availability.

Genomics data

Chiaradonna F, Pirola Y, Ricciardiello F, Palorini R.
PMID: 27486565
Genom Data. 2016 Jul 07;9:100-4. doi: 10.1016/j.gdata.2016.07.004. eCollection 2016 Sep.

Forskolin (FSK) induces activation of protein kinase A (PKA). This activation protects specifically some cancer cells from death induced by glucose starvation. Cell effects upon FSK treatment prompted us to investigate in detail the physiological role of PKA in...

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Chiaradonna F, Pirola Y, Ricciardiello F, et al. Transcriptional profiling of immortalized and K-ras-transformed mouse fibroblasts upon PKA stimulation by forskolin in low glucose availability. Genom Data. 2016;9:100-4doi: 10.1016/j.gdata.2016.07.004.
Chiaradonna, F., Pirola, Y., Ricciardiello, F., & Palorini, R. (2016). Transcriptional profiling of immortalized and K-ras-transformed mouse fibroblasts upon PKA stimulation by forskolin in low glucose availability. Genomics data, 9100-4. https://doi.org/10.1016/j.gdata.2016.07.004
Chiaradonna, Ferdinando, et al. "Transcriptional profiling of immortalized and K-ras-transformed mouse fibroblasts upon PKA stimulation by forskolin in low glucose availability." Genomics data vol. 9 (2016): 100-4. doi: https://doi.org/10.1016/j.gdata.2016.07.004
Chiaradonna F, Pirola Y, Ricciardiello F, Palorini R. Transcriptional profiling of immortalized and K-ras-transformed mouse fibroblasts upon PKA stimulation by forskolin in low glucose availability. Genom Data. 2016 Jul 07;9:100-4. doi: 10.1016/j.gdata.2016.07.004. eCollection 2016 Sep. PMID: 27486565; PMCID: PMC4957573.
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Showing 13 to 16 of 16 entries