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Showing 25 to 36 of 1912 entries
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Molecular Genetic Diagnosis of a Bethlem Myopathy Family with an Autosomal-Dominant COL6A1 Mutation, as Evidenced by Exome Sequencing.

Journal of clinical neurology (Seoul, Korea)

Park HJ, Choi YC, Kim SM, Kim SH, Hong YB, Yoon BR, Chung KW, Choi BO.
PMID: 25749816
J Clin Neurol. 2015 Apr;11(2):183-7. doi: 10.3988/jcn.2015.11.2.183. Epub 2014 Nov 11.

BACKGROUND: We describe herein the application of whole exome sequencing (WES) for the molecular genetic diagnosis of a large Korean family with dominantly inherited myopathy.CASE REPORT: The affected individuals presented with slowly progressive proximal weakness and ankle contracture. They...

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Park HJ, Choi YC, Kim SM, et al. Molecular Genetic Diagnosis of a Bethlem Myopathy Family with an Autosomal-Dominant COL6A1 Mutation, as Evidenced by Exome Sequencing. J Clin Neurol. 2014;11(2):183-7doi: 10.3988/jcn.2015.11.2.183.
Park, H. J., Choi, Y. C., Kim, S. M., Kim, S. H., Hong, Y. B., Yoon, B. R., Chung, K. W., & Choi, B. O. (2015). Molecular Genetic Diagnosis of a Bethlem Myopathy Family with an Autosomal-Dominant COL6A1 Mutation, as Evidenced by Exome Sequencing. Journal of clinical neurology (Seoul, Korea), 11(2), 183-7. https://doi.org/10.3988/jcn.2015.11.2.183
Park, Hyung Jun, et al. "Molecular Genetic Diagnosis of a Bethlem Myopathy Family with an Autosomal-Dominant COL6A1 Mutation, as Evidenced by Exome Sequencing." Journal of clinical neurology (Seoul, Korea) vol. 11,2 (2015): 183-7. doi: https://doi.org/10.3988/jcn.2015.11.2.183
Park HJ, Choi YC, Kim SM, Kim SH, Hong YB, Yoon BR, Chung KW, Choi BO. Molecular Genetic Diagnosis of a Bethlem Myopathy Family with an Autosomal-Dominant COL6A1 Mutation, as Evidenced by Exome Sequencing. J Clin Neurol. 2015 Apr;11(2):183-7. doi: 10.3988/jcn.2015.11.2.183. Epub 2014 Nov 11. PMID: 25749816; PMCID: PMC4387485.
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Clinical Features of Pulmonary Large Cell Neuroendocrine Carcinoma.

Cancer research and treatment

Park MS, Kim KD, Chung JH, Shin DH, Chung KY, Kim JH, Lee CY, Kim YS, Kim HJ, Kim SK, Ahn CM, Kim SK, Chang J.
PMID: 26680943
Cancer Res Treat. 2003 Jun;35(3):245-53. doi: 10.4143/crt.2003.35.3.245.

PURPOSE: This study was performed to investigate the clinical features of large cell neuroendocrine carcinomas (LCNEC).MATERIALS AND METHODS: We retrospectively reviewed the histopathology and clinical information of 37 patients with LCNEC, diagnosed between June 1992 and May 2002 at...

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Park MS, Kim KD, Chung JH, et al. Clinical Features of Pulmonary Large Cell Neuroendocrine Carcinoma. Cancer Res Treat. 2003;35(3):245-53doi: 10.4143/crt.2003.35.3.245.
Park, M. S., Kim, K. D., Chung, J. H., Shin, D. H., Chung, K. Y., Kim, J. H., Lee, C. Y., Kim, Y. S., Kim, H. J., Kim, S. K., Ahn, C. M., Kim, S. K., & Chang, J. (2003). Clinical Features of Pulmonary Large Cell Neuroendocrine Carcinoma. Cancer research and treatment, 35(3), 245-53. https://doi.org/10.4143/crt.2003.35.3.245
Park, Moo Suk, et al. "Clinical Features of Pulmonary Large Cell Neuroendocrine Carcinoma." Cancer research and treatment vol. 35,3 (2003): 245-53. doi: https://doi.org/10.4143/crt.2003.35.3.245
Park MS, Kim KD, Chung JH, Shin DH, Chung KY, Kim JH, Lee CY, Kim YS, Kim HJ, Kim SK, Ahn CM, Kim SK, Chang J. Clinical Features of Pulmonary Large Cell Neuroendocrine Carcinoma. Cancer Res Treat. 2003 Jun;35(3):245-53. doi: 10.4143/crt.2003.35.3.245. PMID: 26680943.
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Intramural Reentrant Ventricular Tachycardia in a Patient with Severe Hypertensive Left Ventricular Hypertrophy.

Korean circulation journal

Lin CY, Chung FP, Lin YJ, Chen SA.
PMID: 26617657
Korean Circ J. 2015 Nov;45(6):526-30. doi: 10.4070/kcj.2015.45.6.526. Epub 2015 Nov 25.

We describe the case of a patient with severe hypertensive left ventricular hypertrophy and sustained hemodynamically unstable ventricular tachycardia (VT). Entrainment was demonstrated in the electrophysiological study. Activation mapping and pacemapping identified the location of the intramural reentrant VT...

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Lin CY, Chung FP, Lin YJ, et al. Intramural Reentrant Ventricular Tachycardia in a Patient with Severe Hypertensive Left Ventricular Hypertrophy. Korean Circ J. 2015;45(6):526-30doi: 10.4070/kcj.2015.45.6.526.
Lin, C. Y., Chung, F. P., Lin, Y. J., & Chen, S. A. (2015). Intramural Reentrant Ventricular Tachycardia in a Patient with Severe Hypertensive Left Ventricular Hypertrophy. Korean circulation journal, 45(6), 526-30. https://doi.org/10.4070/kcj.2015.45.6.526
Lin, Chin-Yu, et al. "Intramural Reentrant Ventricular Tachycardia in a Patient with Severe Hypertensive Left Ventricular Hypertrophy." Korean circulation journal vol. 45,6 (2015): 526-30. doi: https://doi.org/10.4070/kcj.2015.45.6.526
Lin CY, Chung FP, Lin YJ, Chen SA. Intramural Reentrant Ventricular Tachycardia in a Patient with Severe Hypertensive Left Ventricular Hypertrophy. Korean Circ J. 2015 Nov;45(6):526-30. doi: 10.4070/kcj.2015.45.6.526. Epub 2015 Nov 25. PMID: 26617657; PMCID: PMC4661370.
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Structural Aspects of GPCR-G Protein Coupling.

Toxicological research

Chung KY.
PMID: 24386514
Toxicol Res. 2013 Sep;29(3):149-55. doi: 10.5487/TR.2013.29.3.149.

G protein-coupled receptors (GPCRs) are membrane receptors; approximately 40% of drugs on the market target GPCRs. A precise understanding of the activation mechanism of GPCRs would facilitate the development of more effective and less toxic drugs. Heterotrimeric G proteins...

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Chung KY. Structural Aspects of GPCR-G Protein Coupling. Toxicol Res. 2013;29(3):149-55doi: 10.5487/TR.2013.29.3.149.
Chung, K. Y. (2013). Structural Aspects of GPCR-G Protein Coupling. Toxicological research, 29(3), 149-55. https://doi.org/10.5487/TR.2013.29.3.149
Chung, Ka Young. "Structural Aspects of GPCR-G Protein Coupling." Toxicological research vol. 29,3 (2013): 149-55. doi: https://doi.org/10.5487/TR.2013.29.3.149
Chung KY. Structural Aspects of GPCR-G Protein Coupling. Toxicol Res. 2013 Sep;29(3):149-55. doi: 10.5487/TR.2013.29.3.149. PMID: 24386514; PMCID: PMC3877993.
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Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production.

Asian-Australasian journal of animal sciences

Johnson BJ, Smith SB, Chung KY.
PMID: 25050012
Asian-Australas J Anim Sci. 2014 May;27(5):757-66. doi: 10.5713/ajas.2012.12524.

Postnatal muscle hypertrophy of beef cattle is the result of enhanced myofibrillar protein synthesis and reduced protein turnover. Skeletal muscle hypertrophy has been studied in cattle fed β-adrenergic agonists (β-AA), which are receptor-mediated enhancers of protein synthesis and inhibitors...

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Johnson BJ, Smith SB, Chung KY. Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australas J Anim Sci. 2014;27(5):757-66doi: 10.5713/ajas.2012.12524.
Johnson, B. J., Smith, S. B., & Chung, K. Y. (2014). Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australasian journal of animal sciences, 27(5), 757-66. https://doi.org/10.5713/ajas.2012.12524
Johnson, Bradley J, et al. "Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production." Asian-Australasian journal of animal sciences vol. 27,5 (2014): 757-66. doi: https://doi.org/10.5713/ajas.2012.12524
Johnson BJ, Smith SB, Chung KY. Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australas J Anim Sci. 2014 May;27(5):757-66. doi: 10.5713/ajas.2012.12524. PMID: 25050012; PMCID: PMC4093185.
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Surgical Treatment of Postpneumonectomy Syndrome with Tissue Expanders in Children.

The Korean journal of thoracic and cardiovascular surgery

Jung HS, Suh JW, Kim TH, Lee CY, Chung KY.
PMID: 26078932
Korean J Thorac Cardiovasc Surg. 2015 Jun;48(3):217-9. doi: 10.5090/kjtcs.2015.48.3.217. Epub 2015 Jun 05.

Postpneumonectomy syndrome (PPS) is a rare late complication of pneumonectomy. It occurs more often in children than in adults, and is characterized by respiratory failure resulting from bronchial compression caused by severe mediastinal shift. Various methods have been used...

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Jung HS, Suh JW, Kim TH, et al. Surgical Treatment of Postpneumonectomy Syndrome with Tissue Expanders in Children. Korean J Thorac Cardiovasc Surg. 2015;48(3):217-9doi: 10.5090/kjtcs.2015.48.3.217.
Jung, H. S., Suh, J. W., Kim, T. H., Lee, C. Y., & Chung, K. Y. (2015). Surgical Treatment of Postpneumonectomy Syndrome with Tissue Expanders in Children. The Korean journal of thoracic and cardiovascular surgery, 48(3), 217-9. https://doi.org/10.5090/kjtcs.2015.48.3.217
Jung, Hee Suk, et al. "Surgical Treatment of Postpneumonectomy Syndrome with Tissue Expanders in Children." The Korean journal of thoracic and cardiovascular surgery vol. 48,3 (2015): 217-9. doi: https://doi.org/10.5090/kjtcs.2015.48.3.217
Jung HS, Suh JW, Kim TH, Lee CY, Chung KY. Surgical Treatment of Postpneumonectomy Syndrome with Tissue Expanders in Children. Korean J Thorac Cardiovasc Surg. 2015 Jun;48(3):217-9. doi: 10.5090/kjtcs.2015.48.3.217. Epub 2015 Jun 05. PMID: 26078932; PMCID: PMC4463230.
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The effect of imiquimod on matrix metalloproteinases and tissue inhibitors of metalloproteinases in malignant melanoma cell invasion.

Annals of dermatology

Jung JY, Kim HS, Roh MR, Roh HJ, Lee SY, Chung KY.
PMID: 24966637
Ann Dermatol. 2014 Jun;26(3):363-73. doi: 10.5021/ad.2014.26.3.363. Epub 2014 Jun 12.

BACKGROUND: A number of reports have been published regarding the use of imiquimod for the treatment of melanoma in situ and metastatic melanoma. Essential steps in the process of melanoma invasion and metastasis include degradation of basement membranes and...

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Jung JY, Kim HS, Roh MR, et al. The effect of imiquimod on matrix metalloproteinases and tissue inhibitors of metalloproteinases in malignant melanoma cell invasion. Ann Dermatol. 2014;26(3):363-73doi: 10.5021/ad.2014.26.3.363.
Jung, J. Y., Kim, H. S., Roh, M. R., Roh, H. J., Lee, S. Y., & Chung, K. Y. (2014). The effect of imiquimod on matrix metalloproteinases and tissue inhibitors of metalloproteinases in malignant melanoma cell invasion. Annals of dermatology, 26(3), 363-73. https://doi.org/10.5021/ad.2014.26.3.363
Jung, Jin Young, et al. "The effect of imiquimod on matrix metalloproteinases and tissue inhibitors of metalloproteinases in malignant melanoma cell invasion." Annals of dermatology vol. 26,3 (2014): 363-73. doi: https://doi.org/10.5021/ad.2014.26.3.363
Jung JY, Kim HS, Roh MR, Roh HJ, Lee SY, Chung KY. The effect of imiquimod on matrix metalloproteinases and tissue inhibitors of metalloproteinases in malignant melanoma cell invasion. Ann Dermatol. 2014 Jun;26(3):363-73. doi: 10.5021/ad.2014.26.3.363. Epub 2014 Jun 12. PMID: 24966637; PMCID: PMC4069648.
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Synergetic Resonance Matching of a Microphone and a Photoacoustic Cell.

Sensors (Basel, Switzerland)

Sim JY, Ahn CG, Huh C, Chung KH, Jeong EJ, Kim BK.
PMID: 28397761
Sensors (Basel). 2017 Apr 08;17(4). doi: 10.3390/s17040804.

We propose an approach to match the resonant characteristics of a photoacoustic cell with that of a microphone in order to enhance the signal-to-noise ratio in the photoacoustic sensor system. The synergetic resonance matching of a photoacoustic cell and...

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Sim JY, Ahn CG, Huh C, et al. Synergetic Resonance Matching of a Microphone and a Photoacoustic Cell. Sensors (Basel). 2017;17(4)doi: 10.3390/s17040804.
Sim, J. Y., Ahn, C. G., Huh, C., Chung, K. H., Jeong, E. J., & Kim, B. K. (2017). Synergetic Resonance Matching of a Microphone and a Photoacoustic Cell. Sensors (Basel, Switzerland), 17(4), . https://doi.org/10.3390/s17040804
Sim, Joo Yong, et al. "Synergetic Resonance Matching of a Microphone and a Photoacoustic Cell." Sensors (Basel, Switzerland) vol. 17,4 (2017). doi: https://doi.org/10.3390/s17040804
Sim JY, Ahn CG, Huh C, Chung KH, Jeong EJ, Kim BK. Synergetic Resonance Matching of a Microphone and a Photoacoustic Cell. Sensors (Basel). 2017 Apr 08;17(4). doi: 10.3390/s17040804. PMID: 28397761; PMCID: PMC5422165.
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Focal atrial fibrillation presenting in the origin of atrial tachycardia.

HeartRhythm case reports

Lin CY, Lin YJ, Chung FP, Chen SA.
PMID: 28491502
HeartRhythm Case Rep. 2015 Jan 02;1(1):18-21. doi: 10.1016/j.hrcr.2014.10.003. eCollection 2015 Jan.

No abstract available.

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Lin CY, Lin YJ, Chung FP, et al. Focal atrial fibrillation presenting in the origin of atrial tachycardia. HeartRhythm Case Rep. 2015;1(1):18-21doi: 10.1016/j.hrcr.2014.10.003.
Lin, C. Y., Lin, Y. J., Chung, F. P., & Chen, S. A. (2015). Focal atrial fibrillation presenting in the origin of atrial tachycardia. HeartRhythm case reports, 1(1), 18-21. https://doi.org/10.1016/j.hrcr.2014.10.003
Lin, Chin-Yu, et al. "Focal atrial fibrillation presenting in the origin of atrial tachycardia." HeartRhythm case reports vol. 1,1 (2015): 18-21. doi: https://doi.org/10.1016/j.hrcr.2014.10.003
Lin CY, Lin YJ, Chung FP, Chen SA. Focal atrial fibrillation presenting in the origin of atrial tachycardia. HeartRhythm Case Rep. 2015 Jan 02;1(1):18-21. doi: 10.1016/j.hrcr.2014.10.003. eCollection 2015 Jan. PMID: 28491502; PMCID: PMC5420053.
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Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?.

HeartRhythm case reports

Te ALD, Chung FP, Lin YJ, Chen SA.
PMID: 28491634
HeartRhythm Case Rep. 2015 Oct 20;2(1):63-66. doi: 10.1016/j.hrcr.2015.10.003. eCollection 2016 Jan.

No abstract available.

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Te ALD, Chung FP, Lin YJ, et al. Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?. HeartRhythm Case Rep. 2015;2(1):63-66doi: 10.1016/j.hrcr.2015.10.003.
Te, A. L. D., Chung, F. P., Lin, Y. J., & Chen, S. A. (2016). Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?. HeartRhythm case reports, 2(1), 63-66. https://doi.org/10.1016/j.hrcr.2015.10.003
Te, Abigail Louise D, et al. "Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?." HeartRhythm case reports vol. 2,1 (2016): 63-66. doi: https://doi.org/10.1016/j.hrcr.2015.10.003
Te ALD, Chung FP, Lin YJ, Chen SA. Irregular wide QRS complex tachycardia in a patient with pulmonary hypertension: What is the mechanism?. HeartRhythm Case Rep. 2015 Oct 20;2(1):63-66. doi: 10.1016/j.hrcr.2015.10.003. eCollection 2016 Jan. PMID: 28491634; PMCID: PMC5412644.
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E2-EPF UCP Possesses E3 Ubiquitin Ligase Activity via Its Cysteine 118 Residue.

PloS one

Lim JH, Shin HW, Chung KS, Kim NS, Kim JH, Jung HR, Im DS, Jung CR.
PMID: 27685940
PLoS One. 2016 Sep 29;11(9):e0163710. doi: 10.1371/journal.pone.0163710. eCollection 2016.

Here, we show that E2-EPF ubiquitin carrier protein (UCP) elongated E3-independent polyubiquitin chains on the lysine residues of von Hippel-Lindau protein (pVHL) and its own lysine residues both in vitro and in vivo. The initiation of the ubiquitin reaction...

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Lim JH, Shin HW, Chung KS, et al. E2-EPF UCP Possesses E3 Ubiquitin Ligase Activity via Its Cysteine 118 Residue. PLoS One. 2016;11(9):e0163710doi: 10.1371/journal.pone.0163710.
Lim, J. H., Shin, H. W., Chung, K. S., Kim, N. S., Kim, J. H., Jung, H. R., Im, D. S., & Jung, C. R. (2016). E2-EPF UCP Possesses E3 Ubiquitin Ligase Activity via Its Cysteine 118 Residue. PloS one, 11(9), e0163710. https://doi.org/10.1371/journal.pone.0163710
Lim, Jung Hwa, et al. "E2-EPF UCP Possesses E3 Ubiquitin Ligase Activity via Its Cysteine 118 Residue." PloS one vol. 11,9 (2016): e0163710. doi: https://doi.org/10.1371/journal.pone.0163710
Lim JH, Shin HW, Chung KS, Kim NS, Kim JH, Jung HR, Im DS, Jung CR. E2-EPF UCP Possesses E3 Ubiquitin Ligase Activity via Its Cysteine 118 Residue. PLoS One. 2016 Sep 29;11(9):e0163710. doi: 10.1371/journal.pone.0163710. eCollection 2016. PMID: 27685940; PMCID: PMC5042379.
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Ablation of Ventricular Tachycardia Arising from the Great Cardiac Vein - A Rare Cause of Coronary Artery Injury.

Acta Cardiologica Sinica

Suresh A, Chang SL, Lin YJ, Lo LW, Chung FP, Chen SA.
PMID: 28959111
Acta Cardiol Sin. 2017 Sep;33(5):553-555. doi: 10.6515/acs20170111a.

We herein report a case of coronary artery injury during epicardial ablation of ventricular tachycardia (VT), and describe an effective method to combat such an injury. A male patient presented with recurrent palpitations which was refractory to medications, with...

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Suresh A, Chang SL, Lin YJ, et al. Ablation of Ventricular Tachycardia Arising from the Great Cardiac Vein - A Rare Cause of Coronary Artery Injury. Acta Cardiol Sin. 2017;33(5):553-555doi: 10.6515/acs20170111a.
Suresh, A., Chang, S. L., Lin, Y. J., Lo, L. W., Chung, F. P., & Chen, S. A. (2017). Ablation of Ventricular Tachycardia Arising from the Great Cardiac Vein - A Rare Cause of Coronary Artery Injury. Acta Cardiologica Sinica, 33(5), 553-555. https://doi.org/10.6515/acs20170111a
Suresh, Allamsetty, et al. "Ablation of Ventricular Tachycardia Arising from the Great Cardiac Vein - A Rare Cause of Coronary Artery Injury." Acta Cardiologica Sinica vol. 33,5 (2017): 553-555. doi: https://doi.org/10.6515/acs20170111a
Suresh A, Chang SL, Lin YJ, Lo LW, Chung FP, Chen SA. Ablation of Ventricular Tachycardia Arising from the Great Cardiac Vein - A Rare Cause of Coronary Artery Injury. Acta Cardiol Sin. 2017 Sep;33(5):553-555. doi: 10.6515/acs20170111a. PMID: 28959111; PMCID: PMC5611355.
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Showing 25 to 36 of 1912 entries