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TRAF3 and NBR1 both influence the effect of the disease-causing CYLD(Arg936X) mutation on NF-κB activity.

Experimental dermatology

Danis J, Kelemen E, Rajan N, Nagy N, Széll M, Ádám É.
PMID: 33999445
Exp Dermatol. 2021 Nov;30(11):1705-1710. doi: 10.1111/exd.14365. Epub 2021 May 17.

Recently described Hungarian and Anglo-Saxon pedigrees that are affected by CYLD cutaneous syndrome (syn: Brooke-Spiegler syndrome (BSS)) carry the same disease-causing mutation (c.2806C>T, p.Arg936X) of the cylindromatosis (CYLD) gene but exhibit striking phenotypic differences. Using whole exome sequencing, missense...

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Danis J, Kelemen E, Rajan N, et al. TRAF3 and NBR1 both influence the effect of the disease-causing CYLD(Arg936X) mutation on NF-κB activity. Exp Dermatol. 2021;30(11):1705-1710doi: 10.1111/exd.14365.
Danis, J., Kelemen, E., Rajan, N., Nagy, N., Széll, M., & Ádám, �. �. (2021). TRAF3 and NBR1 both influence the effect of the disease-causing CYLD(Arg936X) mutation on NF-κB activity. Experimental dermatology, 30(11), 1705-1710. https://doi.org/10.1111/exd.14365
Danis, Judit, et al. "TRAF3 and NBR1 both influence the effect of the disease-causing CYLD(Arg936X) mutation on NF-κB activity." Experimental dermatology vol. 30,11 (2021): 1705-1710. doi: https://doi.org/10.1111/exd.14365
Danis J, Kelemen E, Rajan N, Nagy N, Széll M, Ádám É. TRAF3 and NBR1 both influence the effect of the disease-causing CYLD(Arg936X) mutation on NF-κB activity. Exp Dermatol. 2021 Nov;30(11):1705-1710. doi: 10.1111/exd.14365. Epub 2021 May 17. PMID: 33999445.
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Non-Surgical Correction of Congenital Ear Anomalies: A Critical Assessment of Caretaker Burdens and Aesthetic Outcomes.

Aesthetic plastic surgery

Dinis J, Junn A, Long A, Phillips S, Reategui A, Kaplan A, Alperovich M.
PMID: 34608514
Aesthetic Plast Surg. 2021 Oct 04; doi: 10.1007/s00266-021-02610-y. Epub 2021 Oct 04.

INTRODUCTION: Congenital ear anomalies result from cartilage and skin compression in utero. They can be corrected in infancy before the cartilage hardens and loses its malleability. Caretaker burden of ear molding and its impact on esthetic outcomes has not...

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Dinis J, Junn A, Long A, et al. Non-Surgical Correction of Congenital Ear Anomalies: A Critical Assessment of Caretaker Burdens and Aesthetic Outcomes. Aesthetic Plast Surg. 2021;doi: 10.1007/s00266-021-02610-y.
Dinis, J., Junn, A., Long, A., Phillips, S., Reategui, A., Kaplan, A., & Alperovich, M. (2021). Non-Surgical Correction of Congenital Ear Anomalies: A Critical Assessment of Caretaker Burdens and Aesthetic Outcomes. Aesthetic plastic surgery, . https://doi.org/10.1007/s00266-021-02610-y
Dinis, Jacob, et al. "Non-Surgical Correction of Congenital Ear Anomalies: A Critical Assessment of Caretaker Burdens and Aesthetic Outcomes." Aesthetic plastic surgery vol. (2021). doi: https://doi.org/10.1007/s00266-021-02610-y
Dinis J, Junn A, Long A, Phillips S, Reategui A, Kaplan A, Alperovich M. Non-Surgical Correction of Congenital Ear Anomalies: A Critical Assessment of Caretaker Burdens and Aesthetic Outcomes. Aesthetic Plast Surg. 2021 Oct 04; doi: 10.1007/s00266-021-02610-y. Epub 2021 Oct 04. PMID: 34608514.
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Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes.

International journal of molecular sciences

Kelemen E, Ádám É, Sági SM, Göblös A, Kemény L, Bata-Csörgő Z, Széll M, Danis J.
PMID: 35008970
Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010540.

Psoriasis is a multifactorial, chronic inflammatory skin disease, the development of which is affected by both genetic and environmental factors. Cytosolic nucleic acid fragments, recognized as pathogen- and danger-associated molecular patterns, are highly abundant in psoriatic skin. It is...

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Kelemen E, Ádám É, Sági SM, et al. Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. Int J Mol Sci. 2022;23(1)doi: 10.3390/ijms23010540.
Kelemen, E., Ádám, �. �., Sági, S. M., Göblös, A., Kemény, L., Bata-Csörgő, Z., Széll, M., & Danis, J. (2022). Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. International journal of molecular sciences, 23(1), . https://doi.org/10.3390/ijms23010540
Kelemen, Evelyn, et al. "Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes." International journal of molecular sciences vol. 23,1 (2022). doi: https://doi.org/10.3390/ijms23010540
Kelemen E, Ádám É, Sági SM, Göblös A, Kemény L, Bata-Csörgő Z, Széll M, Danis J. Psoriasis-Associated Inflammatory Conditions Induce IL-23 mRNA Expression in Normal Human Epidermal Keratinocytes. Int J Mol Sci. 2022 Jan 04;23(1). doi: 10.3390/ijms23010540. PMID: 35008970.
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Showing 265 to 267 of 267 entries