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Crush cytology: an expeditious diagnostic tool for gastrointestinal tract malignancy.

Endoscopy international open

Desai P, Kabrawala M, Patel C, Arora P, Mehta R, Nandwani S, Kalra P, Prajapati R, Patel N, Parekh K.
PMID: 33937515
Endosc Int Open. 2021 May;9(5):E735-E740. doi: 10.1055/a-1388-6479. Epub 2021 Apr 22.

No abstract available.

Cite
Desai P, Kabrawala M, Patel C, et al. Crush cytology: an expeditious diagnostic tool for gastrointestinal tract malignancy. Endosc Int Open. 2021;9(5):E735-E740doi: 10.1055/a-1388-6479.
Desai, P., Kabrawala, M., Patel, C., Arora, P., Mehta, R., Nandwani, S., Kalra, P., Prajapati, R., Patel, N., & Parekh, K. (2021). Crush cytology: an expeditious diagnostic tool for gastrointestinal tract malignancy. Endoscopy international open, 9(5), E735-E740. https://doi.org/10.1055/a-1388-6479
Desai, Pankaj, et al. "Crush cytology: an expeditious diagnostic tool for gastrointestinal tract malignancy." Endoscopy international open vol. 9,5 (2021): E735-E740. doi: https://doi.org/10.1055/a-1388-6479
Desai P, Kabrawala M, Patel C, Arora P, Mehta R, Nandwani S, Kalra P, Prajapati R, Patel N, Parekh K. Crush cytology: an expeditious diagnostic tool for gastrointestinal tract malignancy. Endosc Int Open. 2021 May;9(5):E735-E740. doi: 10.1055/a-1388-6479. Epub 2021 Apr 22. PMID: 33937515; PMCID: PMC8062225.
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Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice.

Genome medicine

Haigh JL, Adhikari A, Copping NA, Stradleigh T, Wade AA, Catta-Preta R, Su-Feher L, Zdilar I, Morse S, Fenton TA, Nguyen A, Quintero D, Agezew S, Sramek M, Kreun EJ, Carter J, Gompers A, Lambert JT, Canales CP, Pennacchio LA, Visel A, Dickel DE, Silverman JL, Nord AS.
PMID: 33910599
Genome Med. 2021 Apr 26;13(1):69. doi: 10.1186/s13073-021-00884-0.

BACKGROUND: Genes with multiple co-active promoters appear common in brain, yet little is known about functional requirements for these potentially redundant genomic regulatory elements. SCN1A, which encodes the NaMETHODS: To determine the functional role of one of these potentially...

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Haigh JL, Adhikari A, Copping NA, et al. Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice. Genome Med. 2021;13(1):69doi: 10.1186/s13073-021-00884-0.
Haigh, J. L., Adhikari, A., Copping, N. A., Stradleigh, T., Wade, A. A., Catta-Preta, R., Su-Feher, L., Zdilar, I., Morse, S., Fenton, T. A., Nguyen, A., Quintero, D., Agezew, S., Sramek, M., Kreun, E. J., Carter, J., Gompers, A., Lambert, J. T., Canales, C. P., Pennacchio, L. A., Visel, A., Dickel, D. E., Silverman, J. L., & Nord, A. S. (2021). Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice. Genome medicine, 13(1), 69. https://doi.org/10.1186/s13073-021-00884-0
Haigh, Jessica L, et al. "Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice." Genome medicine vol. 13,1 (2021): 69. doi: https://doi.org/10.1186/s13073-021-00884-0
Haigh JL, Adhikari A, Copping NA, Stradleigh T, Wade AA, Catta-Preta R, Su-Feher L, Zdilar I, Morse S, Fenton TA, Nguyen A, Quintero D, Agezew S, Sramek M, Kreun EJ, Carter J, Gompers A, Lambert JT, Canales CP, Pennacchio LA, Visel A, Dickel DE, Silverman JL, Nord AS. Deletion of a non-canonical regulatory sequence causes loss of Scn1a expression and epileptic phenotypes in mice. Genome Med. 2021 Apr 26;13(1):69. doi: 10.1186/s13073-021-00884-0. PMID: 33910599; PMCID: PMC8080386.
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Overlap of Granulomatosis With Polyangiitis and IgA Nephropathy.

Cureus

Zeidan BS, Hernandez A, Desai P.
PMID: 34786253
Cureus. 2021 Oct 11;13(10):e18672. doi: 10.7759/cureus.18672. eCollection 2021 Oct.

Acute glomerulonephritis is a constellation of renal disorders which are precipitated by an immunologic mechanism triggering inflammation and proliferation of glomerular tissue resulting in damage to the basement membrane, mesangium, or capillary endothelium. Two of the most well-known manifestations...

Cite
Zeidan BS, Hernandez A, Desai P. Overlap of Granulomatosis With Polyangiitis and IgA Nephropathy. Cureus. 2021;13(10):e18672doi: 10.7759/cureus.18672.
Zeidan, B. S., Hernandez, A., & Desai, P. (2021). Overlap of Granulomatosis With Polyangiitis and IgA Nephropathy. Cureus, 13(10), e18672. https://doi.org/10.7759/cureus.18672
Zeidan, Bassem S, et al. "Overlap of Granulomatosis With Polyangiitis and IgA Nephropathy." Cureus vol. 13,10 (2021): e18672. doi: https://doi.org/10.7759/cureus.18672
Zeidan BS, Hernandez A, Desai P. Overlap of Granulomatosis With Polyangiitis and IgA Nephropathy. Cureus. 2021 Oct 11;13(10):e18672. doi: 10.7759/cureus.18672. eCollection 2021 Oct. PMID: 34786253; PMCID: PMC8579883.
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Showing 517 to 519 of 519 entries