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The Role of Oxidative Stress and Hormones in Controlling Obesity.

Frontiers in endocrinology

Di Domenico M, Pinto F, Quagliuolo L, Contaldo M, Settembre G, Romano A, Coppola M, Ferati K, Bexheti-Ferati A, Sciarra A, Nicoletti GF, Ferraro GA, Boccellino M.
PMID: 31456748
Front Endocrinol (Lausanne). 2019 Aug 13;10:540. doi: 10.3389/fendo.2019.00540. eCollection 2019.

The accumulation of adipose tissue in the body occurs because the energy introduced with food and drink exceeds that expense, but to understand why this imbalance is established and why it is maintained over time, it is important to...

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Di Domenico M, Pinto F, Quagliuolo L, et al. The Role of Oxidative Stress and Hormones in Controlling Obesity. Front Endocrinol (Lausanne). 2019;10:540doi: 10.3389/fendo.2019.00540.
Di Domenico, M., Pinto, F., Quagliuolo, L., Contaldo, M., Settembre, G., Romano, A., Coppola, M., Ferati, K., Bexheti-Ferati, A., Sciarra, A., Nicoletti, G. F., Ferraro, G. A., & Boccellino, M. (2019). The Role of Oxidative Stress and Hormones in Controlling Obesity. Frontiers in endocrinology, 10540. https://doi.org/10.3389/fendo.2019.00540
Di Domenico, Marina, et al. "The Role of Oxidative Stress and Hormones in Controlling Obesity." Frontiers in endocrinology vol. 10 (2019): 540. doi: https://doi.org/10.3389/fendo.2019.00540
Di Domenico M, Pinto F, Quagliuolo L, Contaldo M, Settembre G, Romano A, Coppola M, Ferati K, Bexheti-Ferati A, Sciarra A, Nicoletti GF, Ferraro GA, Boccellino M. The Role of Oxidative Stress and Hormones in Controlling Obesity. Front Endocrinol (Lausanne). 2019 Aug 13;10:540. doi: 10.3389/fendo.2019.00540. eCollection 2019. PMID: 31456748; PMCID: PMC6701166.
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Corrigendum: SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome.

Nature genetics

Shaw ND, Brand H, Kupchinsky ZA, Bengani H, Plummer L, Jones TI, Erdin S, Williamson KA, Rainger J, Stortchevoi A, Samocha K, Currall BB, Dunican DS, Collins RL, Willer JR, Lek A, Lek M, Nassan M, Pereira S, Kammin T, Lucente D, Silva A, Seabra CM, Chiang C, An Y, Ansari M, Rainger JK, Joss S, Smith JC, Lippincott MF, Singh SS, Patel N, Jing JW, Law JR, Ferraro N, Verloes A, Rauch A, Steindl K, Zweier M, Scheer I, Sato D, Okamoto N, Jacobsen C, Tryggestad J, Chernausek S, Schimmenti LA, Brasseur B, Cesaretti C, García-Ortiz JE, Buitrago TP, Silva OP, Hoffman JD, Mühlbauer W, Ruprecht KW, Loeys BL, Shino M, Kaindl AM, Cho CH, Morton CC, Meehan RR, van Heyningen V, Liao EC, Balasubramanian R, Hall JE, Seminara SB, Macarthur D, Moore SA, Yoshiura KI, Gusella JF, Marsh JA, Graham JM, Lin AE, Katsanis N, Jones PL, Crowley WF, Davis EE, FitzPatrick DR, Talkowski ME.
PMID: 28546579
Nat Genet. 2017 May 26;49(6):969. doi: 10.1038/ng0617-969c.

No abstract available.

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Shaw ND, Brand H, Kupchinsky ZA, et al. Corrigendum: SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome. Nat Genet. 2017;49(6):969doi: 10.1038/ng0617-969c.
Shaw, N. D., Brand, H., Kupchinsky, Z. A., Bengani, H., Plummer, L., Jones, T. I., Erdin, S., Williamson, K. A., Rainger, J., Stortchevoi, A., Samocha, K., Currall, B. B., Dunican, D. S., Collins, R. L., Willer, J. R., Lek, A., Lek, M., Nassan, M., Pereira, S., Kammin, T., Lucente, D., Silva, A., Seabra, C. M., Chiang, C., An, Y., Ansari, M., Rainger, J. K., Joss, S., Smith, J. C., Lippincott, M. F., Singh, S. S., Patel, N., Jing, J. W., Law, J. R., Ferraro, N., Verloes, A., Rauch, A., Steindl, K., Zweier, M., Scheer, I., Sato, D., Okamoto, N., Jacobsen, C., Tryggestad, J., Chernausek, S., Schimmenti, L. A., Brasseur, B., Cesaretti, C., García-Ortiz, J. E., Buitrago, T. P., Silva, O. P., Hoffman, J. D., Mühlbauer, W., Ruprecht, K. W., Loeys, B. L., Shino, M., Kaindl, A. M., Cho, C. H., Morton, C. C., Meehan, R. R., van Heyningen, V., Liao, E. C., Balasubramanian, R., Hall, J. E., Seminara, S. B., Macarthur, D., Moore, S. A., Yoshiura, K. I., Gusella, J. F., Marsh, J. A., Graham, J. M., Lin, A. E., Katsanis, N., Jones, P. L., Crowley, W. F., Davis, E. E., FitzPatrick, D. R., & Talkowski, M. E. (2017). Corrigendum: SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome. Nature genetics, 49(6), 969. https://doi.org/10.1038/ng0617-969c
Shaw, Natalie D, et al. "Corrigendum: SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome." Nature genetics vol. 49,6 (2017): 969. doi: https://doi.org/10.1038/ng0617-969c
Shaw ND, Brand H, Kupchinsky ZA, Bengani H, Plummer L, Jones TI, Erdin S, Williamson KA, Rainger J, Stortchevoi A, Samocha K, Currall BB, Dunican DS, Collins RL, Willer JR, Lek A, Lek M, Nassan M, Pereira S, Kammin T, Lucente D, Silva A, Seabra CM, Chiang C, An Y, Ansari M, Rainger JK, Joss S, Smith JC, Lippincott MF, Singh SS, Patel N, Jing JW, Law JR, Ferraro N, Verloes A, Rauch A, Steindl K, Zweier M, Scheer I, Sato D, Okamoto N, Jacobsen C, Tryggestad J, Chernausek S, Schimmenti LA, Brasseur B, Cesaretti C, García-Ortiz JE, Buitrago TP, Silva OP, Hoffman JD, Mühlbauer W, Ruprecht KW, Loeys BL, Shino M, Kaindl AM, Cho CH, Morton CC, Meehan RR, van Heyningen V, Liao EC, Balasubramanian R, Hall JE, Seminara SB, Macarthur D, Moore SA, Yoshiura KI, Gusella JF, Marsh JA, Graham JM, Lin AE, Katsanis N, Jones PL, Crowley WF, Davis EE, FitzPatrick DR, Talkowski ME. Corrigendum: SMCHD1 mutations associated with a rare muscular dystrophy can also cause isolated arhinia and Bosma arhinia microphthalmia syndrome. Nat Genet. 2017 May 26;49(6):969. doi: 10.1038/ng0617-969c. PMID: 28546579.
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Bone marrow uptake of .

Radiologia brasileira

do Vale RHB, Ferraro DA, Duarte PS, Carvalho G, Lima MS, Coura Filho GB, Sapienza MT, Buchpiguel CA.
PMID: 29743733
Radiol Bras. 2018 Mar-Apr;51(2):76-80. doi: 10.1590/0100-3984.2016.0201.

OBJECTIVE: To compare the degree of benign bone marrow uptake of MATERIALS AND METHODS: We analyzed the medical charts of 74 Hodgkin lymphoma patients who underwent RESULTS: For six of the ROIs, the SUVs were higher in the patients...

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do Vale RHB, Ferraro DA, Duarte PS, et al. Bone marrow uptake of . Radiol Bras. 2018;51(2):76-80doi: 10.1590/0100-3984.2016.0201.
do Vale, R. H. B., Ferraro, D. A., Duarte, P. S., Carvalho, G., Lima, M. S., Coura Filho, G. B., Sapienza, M. T., & Buchpiguel, C. A. (2018). Bone marrow uptake of . Radiologia brasileira, 51(2), 76-80. https://doi.org/10.1590/0100-3984.2016.0201
do Vale, Rômulo Hermeto Bueno, et al. "Bone marrow uptake of ." Radiologia brasileira vol. 51,2 (2018): 76-80. doi: https://doi.org/10.1590/0100-3984.2016.0201
do Vale RHB, Ferraro DA, Duarte PS, Carvalho G, Lima MS, Coura Filho GB, Sapienza MT, Buchpiguel CA. Bone marrow uptake of . Radiol Bras. 2018 Mar-Apr;51(2):76-80. doi: 10.1590/0100-3984.2016.0201. PMID: 29743733; PMCID: PMC5935399.
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GSK-3β: A Bifunctional Role in Cell Death Pathways.

International journal of cell biology

Jacobs KM, Bhave SR, Ferraro DJ, Jaboin JJ, Hallahan DE, Thotala D.
PMID: 22675363
Int J Cell Biol. 2012;2012:930710. doi: 10.1155/2012/930710. Epub 2012 May 21.

Although glycogen synthase kinase-3 beta (GSK-3β) was originally named for its ability to phosphorylate glycogen synthase and regulate glucose metabolism, this multifunctional kinase is presently known to be a key regulator of a wide range of cellular functions. GSK-3β...

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Jacobs KM, Bhave SR, Ferraro DJ, et al. GSK-3β: A Bifunctional Role in Cell Death Pathways. Int J Cell Biol. 2012;2012:930710doi: 10.1155/2012/930710.
Jacobs, K. M., Bhave, S. R., Ferraro, D. J., Jaboin, J. J., Hallahan, D. E., & Thotala, D. (2012). GSK-3β: A Bifunctional Role in Cell Death Pathways. International journal of cell biology, 2012930710. https://doi.org/10.1155/2012/930710
Jacobs, Keith M, et al. "GSK-3β: A Bifunctional Role in Cell Death Pathways." International journal of cell biology vol. 2012 (2012): 930710. doi: https://doi.org/10.1155/2012/930710
Jacobs KM, Bhave SR, Ferraro DJ, Jaboin JJ, Hallahan DE, Thotala D. GSK-3β: A Bifunctional Role in Cell Death Pathways. Int J Cell Biol. 2012;2012:930710. doi: 10.1155/2012/930710. Epub 2012 May 21. PMID: 22675363; PMCID: PMC3364548.
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Anaerobic nitrite production by plant cells and tissues: evidence for two nitrate pools.

Plant physiology

Ferrari TE, Yoder OC, Filner P.
PMID: 16658345
Plant Physiol. 1973 Mar;51(3):423-31. doi: 10.1104/pp.51.3.423.

Tobacco (Nicotiana tabacum L. cv. Xanthi) XD cells containing nitrate and nitrate reductase stopped producing nitrite after approximately 1 hour when incubated under anaerobic conditions. The cessation of nitrite production was not due to an inactivation of the nitrate...

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Ferrari TE, Yoder OC, Filner P. Anaerobic nitrite production by plant cells and tissues: evidence for two nitrate pools. Plant Physiol. 1973;51(3):423-31doi: 10.1104/pp.51.3.423.
Ferrari, T. E., Yoder, O. C., & Filner, P. (1973). Anaerobic nitrite production by plant cells and tissues: evidence for two nitrate pools. Plant physiology, 51(3), 423-31. https://doi.org/10.1104/pp.51.3.423
Ferrari, T E, et al. "Anaerobic nitrite production by plant cells and tissues: evidence for two nitrate pools." Plant physiology vol. 51,3 (1973): 423-31. doi: https://doi.org/10.1104/pp.51.3.423
Ferrari TE, Yoder OC, Filner P. Anaerobic nitrite production by plant cells and tissues: evidence for two nitrate pools. Plant Physiol. 1973 Mar;51(3):423-31. doi: 10.1104/pp.51.3.423. PMID: 16658345; PMCID: PMC366281.
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4', 4'''-Dimethoxy Cupressuflavanone, a New Biflavanone from Eupatorium subhastatum.

Planta medica

Ferraro GE, Martino VS, Coussio JD.
PMID: 17345467
Planta Med. 1986 Dec;(6):526. doi: 10.1055/s-2007-969313.

No abstract available.

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Ferraro GE, Martino VS, Coussio JD. 4', 4'''-Dimethoxy Cupressuflavanone, a New Biflavanone from Eupatorium subhastatum. Planta Med. 1986;(6):526doi: 10.1055/s-2007-969313.
Ferraro, G. E., Martino, V. S., & Coussio, J. D. (1986). 4', 4'''-Dimethoxy Cupressuflavanone, a New Biflavanone from Eupatorium subhastatum. Planta medica, (6), 526. https://doi.org/10.1055/s-2007-969313
Ferraro, G E, et al. "4', 4'''-Dimethoxy Cupressuflavanone, a New Biflavanone from Eupatorium subhastatum." Planta medica vol. ,6 (1986): 526. doi: https://doi.org/10.1055/s-2007-969313
Ferraro GE, Martino VS, Coussio JD. 4', 4'''-Dimethoxy Cupressuflavanone, a New Biflavanone from Eupatorium subhastatum. Planta Med. 1986 Dec;(6):526. doi: 10.1055/s-2007-969313. PMID: 17345467.
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Dynamical age differences among coeval star clusters as revealed by blue stragglers.

Nature

Ferraro FR, Lanzoni B, Dalessandro E, Beccari G, Pasquato M, Miocchi P, Rood RT, Sigurdsson S, Sills A, Vesperini E, Mapelli M, Contreras R, Sanna N, Mucciarelli A.
PMID: 23257880
Nature. 2012 Dec 20;492(7429):393-5. doi: 10.1038/nature11686.

Globular star clusters that formed at the same cosmic time may have evolved rather differently from the dynamical point of view (because that evolution depends on the internal environment) through a variety of processes that tend progressively to segregate...

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Ferraro FR, Lanzoni B, Dalessandro E, et al. Dynamical age differences among coeval star clusters as revealed by blue stragglers. Nature. 2012;492(7429):393-5doi: 10.1038/nature11686.
Ferraro, F. R., Lanzoni, B., Dalessandro, E., Beccari, G., Pasquato, M., Miocchi, P., Rood, R. T., Sigurdsson, S., Sills, A., Vesperini, E., Mapelli, M., Contreras, R., Sanna, N., & Mucciarelli, A. (2012). Dynamical age differences among coeval star clusters as revealed by blue stragglers. Nature, 492(7429), 393-5. https://doi.org/10.1038/nature11686
Ferraro, F R, et al. "Dynamical age differences among coeval star clusters as revealed by blue stragglers." Nature vol. 492,7429 (2012): 393-5. doi: https://doi.org/10.1038/nature11686
Ferraro FR, Lanzoni B, Dalessandro E, Beccari G, Pasquato M, Miocchi P, Rood RT, Sigurdsson S, Sills A, Vesperini E, Mapelli M, Contreras R, Sanna N, Mucciarelli A. Dynamical age differences among coeval star clusters as revealed by blue stragglers. Nature. 2012 Dec 20;492(7429):393-5. doi: 10.1038/nature11686. PMID: 23257880.
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Two distinct sequences of blue straggler stars in the globular cluster M 30.

Nature

Ferraro FR, Beccari G, Dalessandro E, Lanzoni B, Sills A, Rood RT, Pecci FF, Karakas AI, Miocchi P, Bovinelli S.
PMID: 20033041
Nature. 2009 Dec 24;462(7276):1028-31. doi: 10.1038/nature08607.

Stars in globular clusters are generally believed to have all formed at the same time, early in the Galaxy's history. 'Blue stragglers' are stars massive enough that they should have evolved into white dwarfs long ago. Two possible mechanisms...

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Ferraro FR, Beccari G, Dalessandro E, et al. Two distinct sequences of blue straggler stars in the globular cluster M 30. Nature. 2009;462(7276):1028-31doi: 10.1038/nature08607.
Ferraro, F. R., Beccari, G., Dalessandro, E., Lanzoni, B., Sills, A., Rood, R. T., Pecci, F. F., Karakas, A. I., Miocchi, P., & Bovinelli, S. (2009). Two distinct sequences of blue straggler stars in the globular cluster M 30. Nature, 462(7276), 1028-31. https://doi.org/10.1038/nature08607
Ferraro, F R, et al. "Two distinct sequences of blue straggler stars in the globular cluster M 30." Nature vol. 462,7276 (2009): 1028-31. doi: https://doi.org/10.1038/nature08607
Ferraro FR, Beccari G, Dalessandro E, Lanzoni B, Sills A, Rood RT, Pecci FF, Karakas AI, Miocchi P, Bovinelli S. Two distinct sequences of blue straggler stars in the globular cluster M 30. Nature. 2009 Dec 24;462(7276):1028-31. doi: 10.1038/nature08607. PMID: 20033041.
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Pathogenic mechanisms of anti-neutrophil cytoplasm antibody-associated vasculitis.

Expert review of clinical immunology

Ferraro AJ, Hassan B, Savage CO.
PMID: 20477159
Expert Rev Clin Immunol. 2007 Jul;3(4):543-55. doi: 10.1586/1744666X.3.4.543.

Anti-neutrophil cytoplasm antibody (ANCA)-associated vasculitides are rare autoimmune diseases that can often be life threatening. They particularly affect the kidneys and lungs but can also affect many other organs. Various hypotheses have been proposed to explain the loss of...

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Ferraro AJ, Hassan B, Savage CO. Pathogenic mechanisms of anti-neutrophil cytoplasm antibody-associated vasculitis. Expert Rev Clin Immunol. 2007;3(4):543-55doi: 10.1586/1744666X.3.4.543.
Ferraro, A. J., Hassan, B., & Savage, C. O. (2007). Pathogenic mechanisms of anti-neutrophil cytoplasm antibody-associated vasculitis. Expert review of clinical immunology, 3(4), 543-55. https://doi.org/10.1586/1744666X.3.4.543
Ferraro, Alastair J, et al. "Pathogenic mechanisms of anti-neutrophil cytoplasm antibody-associated vasculitis." Expert review of clinical immunology vol. 3,4 (2007): 543-55. doi: https://doi.org/10.1586/1744666X.3.4.543
Ferraro AJ, Hassan B, Savage CO. Pathogenic mechanisms of anti-neutrophil cytoplasm antibody-associated vasculitis. Expert Rev Clin Immunol. 2007 Jul;3(4):543-55. doi: 10.1586/1744666X.3.4.543. PMID: 20477159.
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A model for self-recognition and regulation of the incompatibility response of pollen.

TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik

Ferrari TE, Wallace DH.
PMID: 24407838
Theor Appl Genet. 1977 Sep;50(5):211-25. doi: 10.1007/BF00273755.

Recent biochemical studies with Brassica indicate that the pollen grain has a primary role in the control of self incompatibility. Combining this new evidence with that from prior genetic, biochemical, and ultrastructural studies, a working model is hypothesized for...

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Ferrari TE, Wallace DH. A model for self-recognition and regulation of the incompatibility response of pollen. Theor Appl Genet. 1977;50(5):211-25doi: 10.1007/BF00273755.
Ferrari, T. E., & Wallace, D. H. (1977). A model for self-recognition and regulation of the incompatibility response of pollen. TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 50(5), 211-25. https://doi.org/10.1007/BF00273755
Ferrari, T E, and Wallace, D H. "A model for self-recognition and regulation of the incompatibility response of pollen." TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik vol. 50,5 (1977): 211-25. doi: https://doi.org/10.1007/BF00273755
Ferrari TE, Wallace DH. A model for self-recognition and regulation of the incompatibility response of pollen. Theor Appl Genet. 1977 Sep;50(5):211-25. doi: 10.1007/BF00273755. PMID: 24407838.
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Human mu opioid receptor gene polymorphisms and vulnerability to substance abuse.

Addiction biology

Berrettini WH, Hoehe MR, Ferraro TN, Demaria PA, Gottheil E.
PMID: 26735786
Addict Biol. 1997 Jul;2(3):303-8. doi: 10.1080/13556219772598.

Two polymorphisms of the human mu opioid receptor gene are described. A non-coding region polymorphism (G to T) occurs at nucleotide 175 preceding the initiation of translation. A coding polymorphism in exon 1 (C to T) at nucleotide 229...

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Berrettini WH, Hoehe MR, Ferraro TN, et al. Human mu opioid receptor gene polymorphisms and vulnerability to substance abuse. Addict Biol. 1997;2(3):303-8doi: 10.1080/13556219772598.
Berrettini, W. H., Hoehe, M. R., Ferraro, T. N., Demaria, P. A., & Gottheil, E. (1997). Human mu opioid receptor gene polymorphisms and vulnerability to substance abuse. Addiction biology, 2(3), 303-8. https://doi.org/10.1080/13556219772598
Berrettini, W H, et al. "Human mu opioid receptor gene polymorphisms and vulnerability to substance abuse." Addiction biology vol. 2,3 (1997): 303-8. doi: https://doi.org/10.1080/13556219772598
Berrettini WH, Hoehe MR, Ferraro TN, Demaria PA, Gottheil E. Human mu opioid receptor gene polymorphisms and vulnerability to substance abuse. Addict Biol. 1997 Jul;2(3):303-8. doi: 10.1080/13556219772598. PMID: 26735786.
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Probiotics as a complementary therapeutic approach in nonalcoholic fatty liver disease.

World journal of hepatology

Ferolla SM, Armiliato GN, Couto CA, Ferrari TC.
PMID: 25848479
World J Hepatol. 2015 Mar 27;7(3):559-65. doi: 10.4254/wjh.v7.i3.559.

Nonalcoholic fatty liver disease (NAFLD) is currently recognized as one of the most common causes of chronic liver disease. It involves a spectrum of conditions that include pure steatosis without inflammation, steatohepatitis, fibrosis and cirrhosis. The key factor in...

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Ferolla SM, Armiliato GN, Couto CA, et al. Probiotics as a complementary therapeutic approach in nonalcoholic fatty liver disease. World J Hepatol. 2015;7(3):559-65doi: 10.4254/wjh.v7.i3.559.
Ferolla, S. M., Armiliato, G. N., Couto, C. A., & Ferrari, T. C. (2015). Probiotics as a complementary therapeutic approach in nonalcoholic fatty liver disease. World journal of hepatology, 7(3), 559-65. https://doi.org/10.4254/wjh.v7.i3.559
Ferolla, Silvia Marinho, et al. "Probiotics as a complementary therapeutic approach in nonalcoholic fatty liver disease." World journal of hepatology vol. 7,3 (2015): 559-65. doi: https://doi.org/10.4254/wjh.v7.i3.559
Ferolla SM, Armiliato GN, Couto CA, Ferrari TC. Probiotics as a complementary therapeutic approach in nonalcoholic fatty liver disease. World J Hepatol. 2015 Mar 27;7(3):559-65. doi: 10.4254/wjh.v7.i3.559. PMID: 25848479; PMCID: PMC4381178.
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