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Showing 25 to 36 of 1417 entries
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Dehydrated Human Amnion/Chorion Grafts May Accelerate the Healing of Ulcers on Free Flaps in Patients With Venous Insufficiency and/or Lymphedema.

Eplasty

Miranda EP, Friedman A.
PMID: 27648116
Eplasty. 2016 Sep 07;16:e26. eCollection 2016.

OBJECTIVE: Ulceration of free flaps in patients with venous insufficiency and/or lymphedema is an uncommon but challenging problem. We hypothesized that dehydrated human amnion/chorion membrane (Epifix) grafts would accelerate healing of these challenging ulcers.METHODS: Retrospective analysis of prospectively acquired...

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Miranda EP, Friedman A. Dehydrated Human Amnion/Chorion Grafts May Accelerate the Healing of Ulcers on Free Flaps in Patients With Venous Insufficiency and/or Lymphedema. Eplasty. 2016;16:e26
Miranda, E. P., & Friedman, A. (2016). Dehydrated Human Amnion/Chorion Grafts May Accelerate the Healing of Ulcers on Free Flaps in Patients With Venous Insufficiency and/or Lymphedema. Eplasty, 16e26.
Miranda, Edward P, and Friedman, Alex. "Dehydrated Human Amnion/Chorion Grafts May Accelerate the Healing of Ulcers on Free Flaps in Patients With Venous Insufficiency and/or Lymphedema." Eplasty vol. 16 (2016): e26.
Miranda EP, Friedman A. Dehydrated Human Amnion/Chorion Grafts May Accelerate the Healing of Ulcers on Free Flaps in Patients With Venous Insufficiency and/or Lymphedema. Eplasty. 2016 Sep 07;16:e26. eCollection 2016. PMID: 27648116; PMCID: PMC5016387.
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Competition between two desert varieties of Medicago laciniata (L.) Mill. under controlled conditions.

Oecologia

Friedman J, Elberse WT.
PMID: 28308895
Oecologia. 1976 Dec;22(4):321-339. doi: 10.1007/BF00345311.

Two varieties of Medicago laciniata common in the Negev desert of Israel reveal a distinct pattern of distribution within two different plant communities: M. laciniata var. laciniata occupies northern slopes and wadi beds within the association of Artemisia herba-alba,...

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Friedman J, Elberse WT. Competition between two desert varieties of Medicago laciniata (L.) Mill. under controlled conditions. Oecologia. 1976;22(4):321-339doi: 10.1007/BF00345311.
Friedman, J., & Elberse, W. T. (1976). Competition between two desert varieties of Medicago laciniata (L.) Mill. under controlled conditions. Oecologia, 22(4), 321-339. https://doi.org/10.1007/BF00345311
Friedman, Jacob, and Elberse, Wim Th. "Competition between two desert varieties of Medicago laciniata (L.) Mill. under controlled conditions." Oecologia vol. 22,4 (1976): 321-339. doi: https://doi.org/10.1007/BF00345311
Friedman J, Elberse WT. Competition between two desert varieties of Medicago laciniata (L.) Mill. under controlled conditions. Oecologia. 1976 Dec;22(4):321-339. doi: 10.1007/BF00345311. PMID: 28308895.
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Large-Scale Coding Sequence Change Underlies the Evolution of Postdevelopmental Novelty in Honey Bees.

Molecular biology and evolution

Jasper WC, Linksvayer TA, Atallah J, Friedman D, Chiu JC, Johnson BR.
PMID: 26912809
Mol Biol Evol. 2016 May;33(5):1379. doi: 10.1093/molbev/msw022. Epub 2016 Feb 23.

No abstract available.

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Jasper WC, Linksvayer TA, Atallah J, et al. Large-Scale Coding Sequence Change Underlies the Evolution of Postdevelopmental Novelty in Honey Bees. Mol Biol Evol. 2016;33(5):1379doi: 10.1093/molbev/msw022.
Jasper, W. C., Linksvayer, T. A., Atallah, J., Friedman, D., Chiu, J. C., & Johnson, B. R. (2016). Large-Scale Coding Sequence Change Underlies the Evolution of Postdevelopmental Novelty in Honey Bees. Molecular biology and evolution, 33(5), 1379. https://doi.org/10.1093/molbev/msw022
Jasper, W Cameron, et al. "Large-Scale Coding Sequence Change Underlies the Evolution of Postdevelopmental Novelty in Honey Bees." Molecular biology and evolution vol. 33,5 (2016): 1379. doi: https://doi.org/10.1093/molbev/msw022
Jasper WC, Linksvayer TA, Atallah J, Friedman D, Chiu JC, Johnson BR. Large-Scale Coding Sequence Change Underlies the Evolution of Postdevelopmental Novelty in Honey Bees. Mol Biol Evol. 2016 May;33(5):1379. doi: 10.1093/molbev/msw022. Epub 2016 Feb 23. PMID: 26912809.
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Effect of allyl isothiocyanate on developmental toxicity in exposed .

Toxicology reports

Williams JR, Rayburn JR, Cline GR, Sauterer R, Friedman M.
PMID: 28962355
Toxicol Rep. 2014 Dec 17;2:222-227. doi: 10.1016/j.toxrep.2014.12.005. eCollection 2015.

The pungent natural compound allyl isothiocyanate isolated from the seeds of

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Williams JR, Rayburn JR, Cline GR, et al. Effect of allyl isothiocyanate on developmental toxicity in exposed . Toxicol Rep. 2014;2:222-227doi: 10.1016/j.toxrep.2014.12.005.
Williams, J. R., Rayburn, J. R., Cline, G. R., Sauterer, R., & Friedman, M. (2015). Effect of allyl isothiocyanate on developmental toxicity in exposed . Toxicology reports, 2222-227. https://doi.org/10.1016/j.toxrep.2014.12.005
Williams, John Russell, et al. "Effect of allyl isothiocyanate on developmental toxicity in exposed ." Toxicology reports vol. 2 (2015): 222-227. doi: https://doi.org/10.1016/j.toxrep.2014.12.005
Williams JR, Rayburn JR, Cline GR, Sauterer R, Friedman M. Effect of allyl isothiocyanate on developmental toxicity in exposed . Toxicol Rep. 2014 Dec 17;2:222-227. doi: 10.1016/j.toxrep.2014.12.005. eCollection 2015. PMID: 28962355; PMCID: PMC5598435.
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Correction: Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry.

PloS one

Viollet L, Glusman G, Murphy KJ, Newcomb TM, Reyna SP, Sweney M, Nelson B, Andermann F, Andermann E, Acsadi G, Barbano RL, Brown C, Brunkow ME, Chugani HT, Cheyette SR, Collins A, DeBrosse SD, Galas D, Friedman J, Hood L, Huff C, Jorde LB, King MD, LaSalle B, Leventer RJ, Lewelt AJ, Massart MB, Mérida MR, Ptáček LJ, Roach JC, Rust RS, Renault F, Sanger TD, Sotero de Menezes MA, Tennyson R, Uldall P, Zhang Y, Zupanc M, Xin W, Silver K, Swoboda KJ.
PMID: 26322789
PLoS One. 2015 Aug 31;10(8):e0137370. doi: 10.1371/journal.pone.0137370. eCollection 2015.

No abstract available.

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Viollet L, Glusman G, Murphy KJ, et al. Correction: Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry. PLoS One. 2015;10(8):e0137370doi: 10.1371/journal.pone.0137370.
Viollet, L., Glusman, G., Murphy, K. J., Newcomb, T. M., Reyna, S. P., Sweney, M., Nelson, B., Andermann, F., Andermann, E., Acsadi, G., Barbano, R. L., Brown, C., Brunkow, M. E., Chugani, H. T., Cheyette, S. R., Collins, A., DeBrosse, S. D., Galas, D., Friedman, J., Hood, L., Huff, C., Jorde, L. B., King, M. D., LaSalle, B., Leventer, R. J., Lewelt, A. J., Massart, M. B., Mérida, M. R., Ptáček, L. J., Roach, J. C., Rust, R. S., Renault, F., Sanger, T. D., Sotero de Menezes, M. A., Tennyson, R., Uldall, P., Zhang, Y., Zupanc, M., Xin, W., Silver, K., & Swoboda, K. J. (2015). Correction: Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry. PloS one, 10(8), e0137370. https://doi.org/10.1371/journal.pone.0137370
Viollet, Louis, et al. "Correction: Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry." PloS one vol. 10,8 (2015): e0137370. doi: https://doi.org/10.1371/journal.pone.0137370
Viollet L, Glusman G, Murphy KJ, Newcomb TM, Reyna SP, Sweney M, Nelson B, Andermann F, Andermann E, Acsadi G, Barbano RL, Brown C, Brunkow ME, Chugani HT, Cheyette SR, Collins A, DeBrosse SD, Galas D, Friedman J, Hood L, Huff C, Jorde LB, King MD, LaSalle B, Leventer RJ, Lewelt AJ, Massart MB, Mérida MR, Ptáček LJ, Roach JC, Rust RS, Renault F, Sanger TD, Sotero de Menezes MA, Tennyson R, Uldall P, Zhang Y, Zupanc M, Xin W, Silver K, Swoboda KJ. Correction: Alternating Hemiplegia of Childhood: Retrospective Genetic Study and Genotype-Phenotype Correlations in 187 Subjects from the US AHCF Registry. PLoS One. 2015 Aug 31;10(8):e0137370. doi: 10.1371/journal.pone.0137370. eCollection 2015. PMID: 26322789; PMCID: PMC4554723.
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Patient Characteristics, Safety, and Tolerability with Telaprevir Treatment for HCV in the Clinic: a Retrospective, Multicenter Study.

Journal of clinical and translational hepatology

Flamm SL, Pockros PJ, Bengtsson L, Friedman M.
PMID: 26356545
J Clin Transl Hepatol. 2014 Jun;2(2):65-73. doi: 10.14218/JCTH.2014.00007. Epub 2014 Jun 15.

BACKGROUND AND AIMS: There is a paucity of information regarding similarities and differences between patients from the phase 3 studies of telaprevir and those receiving telaprevir in clinical practice.METHODS: This retrospective chart review evaluated baseline characteristics and follow-up safety...

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Flamm SL, Pockros PJ, Bengtsson L, et al. Patient Characteristics, Safety, and Tolerability with Telaprevir Treatment for HCV in the Clinic: a Retrospective, Multicenter Study. J Clin Transl Hepatol. 2014;2(2):65-73doi: 10.14218/JCTH.2014.00007.
Flamm, S. L., Pockros, P. J., Bengtsson, L., & Friedman, M. (2014). Patient Characteristics, Safety, and Tolerability with Telaprevir Treatment for HCV in the Clinic: a Retrospective, Multicenter Study. Journal of clinical and translational hepatology, 2(2), 65-73. https://doi.org/10.14218/JCTH.2014.00007
Flamm, Steven L, et al. "Patient Characteristics, Safety, and Tolerability with Telaprevir Treatment for HCV in the Clinic: a Retrospective, Multicenter Study." Journal of clinical and translational hepatology vol. 2,2 (2014): 65-73. doi: https://doi.org/10.14218/JCTH.2014.00007
Flamm SL, Pockros PJ, Bengtsson L, Friedman M. Patient Characteristics, Safety, and Tolerability with Telaprevir Treatment for HCV in the Clinic: a Retrospective, Multicenter Study. J Clin Transl Hepatol. 2014 Jun;2(2):65-73. doi: 10.14218/JCTH.2014.00007. Epub 2014 Jun 15. PMID: 26356545; PMCID: PMC4521261.
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Inflammatory Response and Barrier Dysfunction by Different e-Cigarette Flavoring Chemicals Identified by Gas Chromatography-Mass Spectrometry in e-Liquids and e-Vapors on Human Lung Epithelial Cells and Fibroblasts.

Applied in vitro toxicology

Gerloff J, Sundar IK, Freter R, Sekera ER, Friedman AE, Robinson R, Pagano T, Rahman I.
PMID: 28337465
Appl In Vitro Toxicol. 2017 Mar 01;3(1):28-40. doi: 10.1089/aivt.2016.0030.

Recent studies suggest that electronic cigarette (e-cig) flavors can be harmful to lung tissue by imposing oxidative stress and inflammatory responses. The potential inflammatory response by lung epithelial cells and fibroblasts exposed to e-cig flavoring chemicals in addition to...

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Gerloff J, Sundar IK, Freter R, et al. Inflammatory Response and Barrier Dysfunction by Different e-Cigarette Flavoring Chemicals Identified by Gas Chromatography-Mass Spectrometry in e-Liquids and e-Vapors on Human Lung Epithelial Cells and Fibroblasts. Appl In Vitro Toxicol. 2017;3(1):28-40doi: 10.1089/aivt.2016.0030.
Gerloff, J., Sundar, I. K., Freter, R., Sekera, E. R., Friedman, A. E., Robinson, R., Pagano, T., & Rahman, I. (2017). Inflammatory Response and Barrier Dysfunction by Different e-Cigarette Flavoring Chemicals Identified by Gas Chromatography-Mass Spectrometry in e-Liquids and e-Vapors on Human Lung Epithelial Cells and Fibroblasts. Applied in vitro toxicology, 3(1), 28-40. https://doi.org/10.1089/aivt.2016.0030
Gerloff, Janice, et al. "Inflammatory Response and Barrier Dysfunction by Different e-Cigarette Flavoring Chemicals Identified by Gas Chromatography-Mass Spectrometry in e-Liquids and e-Vapors on Human Lung Epithelial Cells and Fibroblasts." Applied in vitro toxicology vol. 3,1 (2017): 28-40. doi: https://doi.org/10.1089/aivt.2016.0030
Gerloff J, Sundar IK, Freter R, Sekera ER, Friedman AE, Robinson R, Pagano T, Rahman I. Inflammatory Response and Barrier Dysfunction by Different e-Cigarette Flavoring Chemicals Identified by Gas Chromatography-Mass Spectrometry in e-Liquids and e-Vapors on Human Lung Epithelial Cells and Fibroblasts. Appl In Vitro Toxicol. 2017 Mar 01;3(1):28-40. doi: 10.1089/aivt.2016.0030. PMID: 28337465; PMCID: PMC5338075.
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Mushroom Polysaccharides: Chemistry and Antiobesity, Antidiabetes, Anticancer, and Antibiotic Properties in Cells, Rodents, and Humans.

Foods (Basel, Switzerland)

Friedman M.
PMID: 28231175
Foods. 2016 Nov 29;5(4). doi: 10.3390/foods5040080.

More than 2000 species of edible and/or medicinal mushrooms have been identified to date, many of which are widely consumed, stimulating much research on their health-promoting properties. These properties are associated with bioactive compounds produced by the mushrooms, including...

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Friedman M. Mushroom Polysaccharides: Chemistry and Antiobesity, Antidiabetes, Anticancer, and Antibiotic Properties in Cells, Rodents, and Humans. Foods. 2016;5(4)doi: 10.3390/foods5040080.
Friedman, M. (2016). Mushroom Polysaccharides: Chemistry and Antiobesity, Antidiabetes, Anticancer, and Antibiotic Properties in Cells, Rodents, and Humans. Foods (Basel, Switzerland), 5(4), . https://doi.org/10.3390/foods5040080
Friedman, Mendel. "Mushroom Polysaccharides: Chemistry and Antiobesity, Antidiabetes, Anticancer, and Antibiotic Properties in Cells, Rodents, and Humans." Foods (Basel, Switzerland) vol. 5,4 (2016). doi: https://doi.org/10.3390/foods5040080
Friedman M. Mushroom Polysaccharides: Chemistry and Antiobesity, Antidiabetes, Anticancer, and Antibiotic Properties in Cells, Rodents, and Humans. Foods. 2016 Nov 29;5(4). doi: 10.3390/foods5040080. PMID: 28231175; PMCID: PMC5302426.
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Effects of body mass index-related disorders on cognition: preliminary results.

Diabetes, metabolic syndrome and obesity : targets and therapy

Yesavage JA, Kinoshita LM, Noda A, Lazzeroni LC, Fairchild JK, Taylor J, Kulick D, Friedman L, Cheng J, Zeitzer JM, O'Hara R.
PMID: 24855383
Diabetes Metab Syndr Obes. 2014 May 08;7:145-51. doi: 10.2147/DMSO.S60294. eCollection 2014.

BACKGROUND: Well-known risk factors for cognitive impairment are also associated with obesity. Research has highlighted genetic risk factors for obesity, yet the relationship of those risk factors with cognitive impairment is unknown. The objective of this study was to...

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Yesavage JA, Kinoshita LM, Noda A, et al. Effects of body mass index-related disorders on cognition: preliminary results. Diabetes Metab Syndr Obes. 2014;7:145-51doi: 10.2147/DMSO.S60294.
Yesavage, J. A., Kinoshita, L. M., Noda, A., Lazzeroni, L. C., Fairchild, J. K., Taylor, J., Kulick, D., Friedman, L., Cheng, J., Zeitzer, J. M., & O'Hara, R. (2014). Effects of body mass index-related disorders on cognition: preliminary results. Diabetes, metabolic syndrome and obesity : targets and therapy, 7145-51. https://doi.org/10.2147/DMSO.S60294
Yesavage, Jerome A, et al. "Effects of body mass index-related disorders on cognition: preliminary results." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 7 (2014): 145-51. doi: https://doi.org/10.2147/DMSO.S60294
Yesavage JA, Kinoshita LM, Noda A, Lazzeroni LC, Fairchild JK, Taylor J, Kulick D, Friedman L, Cheng J, Zeitzer JM, O'Hara R. Effects of body mass index-related disorders on cognition: preliminary results. Diabetes Metab Syndr Obes. 2014 May 08;7:145-51. doi: 10.2147/DMSO.S60294. eCollection 2014. PMID: 24855383; PMCID: PMC4020904.
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Candida albicans Shed Msb2 and Host Mucins Affect the Candidacidal Activity of Salivary Hst 5.

Pathogens (Basel, Switzerland)

Puri S, Friedman J, Saraswat D, Kumar R, Li R, Ruszaj D, Edgerton M.
PMID: 26529023
Pathogens. 2015 Oct 30;4(4):752-63. doi: 10.3390/pathogens4040752.

Salivary Histatin 5 (Hst 5) is an antimicrobial peptide that exhibits potent antifungal activity towards Candida albicans, the causative agent of oral candidiasis. However, it exhibits limited activity in vivo, largely due to inactivation by salivary components of both...

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Puri S, Friedman J, Saraswat D, et al. Candida albicans Shed Msb2 and Host Mucins Affect the Candidacidal Activity of Salivary Hst 5. Pathogens. 2015;4(4):752-63doi: 10.3390/pathogens4040752.
Puri, S., Friedman, J., Saraswat, D., Kumar, R., Li, R., Ruszaj, D., & Edgerton, M. (2015). Candida albicans Shed Msb2 and Host Mucins Affect the Candidacidal Activity of Salivary Hst 5. Pathogens (Basel, Switzerland), 4(4), 752-63. https://doi.org/10.3390/pathogens4040752
Puri, Sumant, et al. "Candida albicans Shed Msb2 and Host Mucins Affect the Candidacidal Activity of Salivary Hst 5." Pathogens (Basel, Switzerland) vol. 4,4 (2015): 752-63. doi: https://doi.org/10.3390/pathogens4040752
Puri S, Friedman J, Saraswat D, Kumar R, Li R, Ruszaj D, Edgerton M. Candida albicans Shed Msb2 and Host Mucins Affect the Candidacidal Activity of Salivary Hst 5. Pathogens. 2015 Oct 30;4(4):752-63. doi: 10.3390/pathogens4040752. PMID: 26529023; PMCID: PMC4693163.
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Novel role of hematopoietic stem cells in immunologic rejection of malignant gliomas.

Oncoimmunology

Flores C, Pham C, Snyder D, Yang S, Sanchez-Perez L, Sayour E, Cui X, Kemeny H, Friedman H, Bigner DD, Sampson J, Mitchell DA.
PMID: 25949916
Oncoimmunology. 2015 Jan 22;4(3):e994374. doi: 10.4161/2162402X.2014.994374. eCollection 2015 Mar.

Adoptive cellular therapy (ACT) after lymphodepletive conditioning can induce dramatic clinical responses, but this approach has been largely limited to melanoma due to a lack of reliable methods for expanding tumor-specific lymphocytes from the majority of other solid cancers....

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Flores C, Pham C, Snyder D, et al. Novel role of hematopoietic stem cells in immunologic rejection of malignant gliomas. Oncoimmunology. 2015;4(3):e994374doi: 10.4161/2162402X.2014.994374.
Flores, C., Pham, C., Snyder, D., Yang, S., Sanchez-Perez, L., Sayour, E., Cui, X., Kemeny, H., Friedman, H., Bigner, D. D., Sampson, J., & Mitchell, D. A. (2015). Novel role of hematopoietic stem cells in immunologic rejection of malignant gliomas. Oncoimmunology, 4(3), e994374. https://doi.org/10.4161/2162402X.2014.994374
Flores, Catherine, et al. "Novel role of hematopoietic stem cells in immunologic rejection of malignant gliomas." Oncoimmunology vol. 4,3 (2015): e994374. doi: https://doi.org/10.4161/2162402X.2014.994374
Flores C, Pham C, Snyder D, Yang S, Sanchez-Perez L, Sayour E, Cui X, Kemeny H, Friedman H, Bigner DD, Sampson J, Mitchell DA. Novel role of hematopoietic stem cells in immunologic rejection of malignant gliomas. Oncoimmunology. 2015 Jan 22;4(3):e994374. doi: 10.4161/2162402X.2014.994374. eCollection 2015 Mar. PMID: 25949916; PMCID: PMC4404923.
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The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species.

Genes & cancer

Deng X, Mercer SE, Sun CY, Friedman E.
PMID: 24955215
Genes Cancer. 2014 Apr;5(1):22-30. doi: 10.18632/genesandcancer.1.

Mirk kinase is a gene upregulated and sometimes amplified in pancreatic cancers and in ovarian cancers, but expressed at very low levels in most normal diploid cells except for skeletal muscle. The muscle cell function of Mirk kinase selected...

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Deng X, Mercer SE, Sun CY, et al. The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species. Genes Cancer. 2014;5(1):22-30doi: 10.18632/genesandcancer.1.
Deng, X., Mercer, S. E., Sun, C. Y., & Friedman, E. (2014). The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species. Genes & cancer, 5(1), 22-30. https://doi.org/10.18632/genesandcancer.1
Deng, Xiaobing, et al. "The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species." Genes & cancer vol. 5,1 (2014): 22-30. doi: https://doi.org/10.18632/genesandcancer.1
Deng X, Mercer SE, Sun CY, Friedman E. The normal function of the cancer kinase Mirk/dyrk1B is to reduce reactive oxygen species. Genes Cancer. 2014 Apr;5(1):22-30. doi: 10.18632/genesandcancer.1. PMID: 24955215; PMCID: PMC4063251.
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Showing 25 to 36 of 1417 entries