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Showing 13 to 24 of 177 entries
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The role of FLT3 inhibitors as maintenance therapy following hematopoietic stem cell transplant.

Leukemia research reports

Li GX, Wang L, Yaghmour B, Ramsingh G, Yaghmour G.
PMID: 30112274
Leuk Res Rep. 2018 Jul 19;10:26-36. doi: 10.1016/j.lrr.2018.06.003. eCollection 2018.

Activating mutations in FLT3 in acute myeloid leukemia (AML) portend a poor prognosis, and targeting FLT3 with a tyrosine kinase inhibitor has been an area of intense research recently. Most FLT3 mutated AML patients undergo hematopoietic stem cell transplantation...

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Li GX, Wang L, Yaghmour B, et al. The role of FLT3 inhibitors as maintenance therapy following hematopoietic stem cell transplant. Leuk Res Rep. 2018;10:26-36doi: 10.1016/j.lrr.2018.06.003.
Li, G. X., Wang, L., Yaghmour, B., Ramsingh, G., & Yaghmour, G. (2018). The role of FLT3 inhibitors as maintenance therapy following hematopoietic stem cell transplant. Leukemia research reports, 1026-36. https://doi.org/10.1016/j.lrr.2018.06.003
Li, Grace Xiuqing, et al. "The role of FLT3 inhibitors as maintenance therapy following hematopoietic stem cell transplant." Leukemia research reports vol. 10 (2018): 26-36. doi: https://doi.org/10.1016/j.lrr.2018.06.003
Li GX, Wang L, Yaghmour B, Ramsingh G, Yaghmour G. The role of FLT3 inhibitors as maintenance therapy following hematopoietic stem cell transplant. Leuk Res Rep. 2018 Jul 19;10:26-36. doi: 10.1016/j.lrr.2018.06.003. eCollection 2018. PMID: 30112274; PMCID: PMC6092446.
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Mechanisms of NF-κB p65 and strategies for therapeutic manipulation.

Journal of inflammation research

Giridharan S, Srinivasan M.
PMID: 30464573
J Inflamm Res. 2018 Oct 30;11:407-419. doi: 10.2147/JIR.S140188. eCollection 2018.

The transcription factor NF-κB is a critical regulator of immune and inflammatory responses. In mammals, the NF-κB/Rel family comprises five members: p50, p52, p65 (Rel-A), c-Rel, and Rel-B proteins, which form homo- or heterodimers and remain as an inactive...

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Giridharan S, Srinivasan M. Mechanisms of NF-κB p65 and strategies for therapeutic manipulation. J Inflamm Res. 2018;11:407-419doi: 10.2147/JIR.S140188.
Giridharan, S., & Srinivasan, M. (2018). Mechanisms of NF-κB p65 and strategies for therapeutic manipulation. Journal of inflammation research, 11407-419. https://doi.org/10.2147/JIR.S140188
Giridharan, Sivagami, and Srinivasan, Mythily. "Mechanisms of NF-κB p65 and strategies for therapeutic manipulation." Journal of inflammation research vol. 11 (2018): 407-419. doi: https://doi.org/10.2147/JIR.S140188
Giridharan S, Srinivasan M. Mechanisms of NF-κB p65 and strategies for therapeutic manipulation. J Inflamm Res. 2018 Oct 30;11:407-419. doi: 10.2147/JIR.S140188. eCollection 2018. PMID: 30464573; PMCID: PMC6217131.
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A multimodal computer-aided diagnostic system for precise identification of renal allograft rejection: Preliminary results.

Medical physics

Shehata M, Shalaby A, Switala AE, El-Baz M, Ghazal M, Fraiwan L, Khalil A, El-Ghar MA, Badawy M, Bakr AM, Dwyer A, Elmaghraby A, Giridharan G, Keynton R, El-Baz A.
PMID: 32130734
Med Phys. 2020 Jun;47(6):2427-2440. doi: 10.1002/mp.14109. Epub 2020 Apr 03.

PURPOSE: Early assessment of renal allograft function post-transplantation is crucial to minimize and control allograft rejection. Biopsy - the gold standard - is used only as a last resort due to its invasiveness, high cost, adverse events (e.g., bleeding,...

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Shehata M, Shalaby A, Switala AE, et al. A multimodal computer-aided diagnostic system for precise identification of renal allograft rejection: Preliminary results. Med Phys. 2020;47(6):2427-2440doi: 10.1002/mp.14109.
Shehata, M., Shalaby, A., Switala, A. E., El-Baz, M., Ghazal, M., Fraiwan, L., Khalil, A., El-Ghar, M. A., Badawy, M., Bakr, A. M., Dwyer, A., Elmaghraby, A., Giridharan, G., Keynton, R., & El-Baz, A. (2020). A multimodal computer-aided diagnostic system for precise identification of renal allograft rejection: Preliminary results. Medical physics, 47(6), 2427-2440. https://doi.org/10.1002/mp.14109
Shehata, Mohamed, et al. "A multimodal computer-aided diagnostic system for precise identification of renal allograft rejection: Preliminary results." Medical physics vol. 47,6 (2020): 2427-2440. doi: https://doi.org/10.1002/mp.14109
Shehata M, Shalaby A, Switala AE, El-Baz M, Ghazal M, Fraiwan L, Khalil A, El-Ghar MA, Badawy M, Bakr AM, Dwyer A, Elmaghraby A, Giridharan G, Keynton R, El-Baz A. A multimodal computer-aided diagnostic system for precise identification of renal allograft rejection: Preliminary results. Med Phys. 2020 Jun;47(6):2427-2440. doi: 10.1002/mp.14109. Epub 2020 Apr 03. PMID: 32130734; PMCID: PMC8524762.
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Small for gestational age babies and depressive symptoms of mothers during pregnancy: Results from a birth cohort in India.

Wellcome open research

Babu GR, Murthy GVS, Reddy Y, Deepa R, Yamuna A, Prafulla S, Krishnan A, Lobo E, Rathnaiah M, Kinra S.
PMID: 31828224
Wellcome Open Res. 2020 Feb 06;3:76. doi: 10.12688/wellcomeopenres.14618.3. eCollection 2018.

No abstract available.

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Babu GR, Murthy GVS, Reddy Y, et al. Small for gestational age babies and depressive symptoms of mothers during pregnancy: Results from a birth cohort in India. Wellcome Open Res. 2020;3:76doi: 10.12688/wellcomeopenres.14618.3.
Babu, G. R., Murthy, G. V. S., Reddy, Y., Deepa, R., Yamuna, A., Prafulla, S., Krishnan, A., Lobo, E., Rathnaiah, M., & Kinra, S. (2018). Small for gestational age babies and depressive symptoms of mothers during pregnancy: Results from a birth cohort in India. Wellcome open research, 376. https://doi.org/10.12688/wellcomeopenres.14618.3
Babu, Giridhara R, et al. "Small for gestational age babies and depressive symptoms of mothers during pregnancy: Results from a birth cohort in India." Wellcome open research vol. 3 (2018): 76. doi: https://doi.org/10.12688/wellcomeopenres.14618.3
Babu GR, Murthy GVS, Reddy Y, Deepa R, Yamuna A, Prafulla S, Krishnan A, Lobo E, Rathnaiah M, Kinra S. Small for gestational age babies and depressive symptoms of mothers during pregnancy: Results from a birth cohort in India. Wellcome Open Res. 2020 Feb 06;3:76. doi: 10.12688/wellcomeopenres.14618.3. eCollection 2018. PMID: 31828224; PMCID: PMC6892423.
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Characteristics of late transplant-associated thrombotic microangiopathy in patients who underwent allogeneic hematopoietic stem cell transplantation.

American journal of hematology

Heybeli C, Sridharan M, Alkhateeb HB, Villasboas Bisneto JC, Buadi FK, Chen D, Dingli D, Dispenzieri A, Gertz MA, Go RS, Hashmi SK, Hayman SR, Hogan WJ, Inwards DJ, Kenderian SS, Kumar SK, Litzow MR, Porrata LF, Lacy MQ, Micallef IN, Patnaik MM, Shah MV, Leung N.
PMID: 32618000
Am J Hematol. 2020 Jul 02; doi: 10.1002/ajh.25922. Epub 2020 Jul 02.

Transplant-associated thrombotic microangiopathy (TA-TMA) has a wide range of presentations after hematopoietic stem-cell transplantation (HSCT). We retrospectively studied the risk factors and outcomes of patients with early (≤day 100) and late (>day 100) TA-TMA. Among the 1451 HSCT recipients,...

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Heybeli C, Sridharan M, Alkhateeb HB, et al. Characteristics of late transplant-associated thrombotic microangiopathy in patients who underwent allogeneic hematopoietic stem cell transplantation. Am J Hematol. 2020;doi: 10.1002/ajh.25922.
Heybeli, C., Sridharan, M., Alkhateeb, H. B., Villasboas Bisneto, J. C., Buadi, F. K., Chen, D., Dingli, D., Dispenzieri, A., Gertz, M. A., Go, R. S., Hashmi, S. K., Hayman, S. R., Hogan, W. J., Inwards, D. J., Kenderian, S. S., Kumar, S. K., Litzow, M. R., Porrata, L. F., Lacy, M. Q., Micallef, I. N., Patnaik, M. M., Shah, M. V., & Leung, N. (2020). Characteristics of late transplant-associated thrombotic microangiopathy in patients who underwent allogeneic hematopoietic stem cell transplantation. American journal of hematology, . https://doi.org/10.1002/ajh.25922
Heybeli, Cihan, et al. "Characteristics of late transplant-associated thrombotic microangiopathy in patients who underwent allogeneic hematopoietic stem cell transplantation." American journal of hematology vol. (2020). doi: https://doi.org/10.1002/ajh.25922
Heybeli C, Sridharan M, Alkhateeb HB, Villasboas Bisneto JC, Buadi FK, Chen D, Dingli D, Dispenzieri A, Gertz MA, Go RS, Hashmi SK, Hayman SR, Hogan WJ, Inwards DJ, Kenderian SS, Kumar SK, Litzow MR, Porrata LF, Lacy MQ, Micallef IN, Patnaik MM, Shah MV, Leung N. Characteristics of late transplant-associated thrombotic microangiopathy in patients who underwent allogeneic hematopoietic stem cell transplantation. Am J Hematol. 2020 Jul 02; doi: 10.1002/ajh.25922. Epub 2020 Jul 02. PMID: 32618000.
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Molecular docking analysis of a secondary metabolite with the glycoprotein receptors of HSV 1 and HSV 2.

Bioinformation

Saran N, Anandharaj B, Bupesh G, Vasanth S, Beulah JPJ, Balachandar V.
PMID: 32256009
Bioinformation. 2019 Dec 31;15(12):887-895. doi: 10.6026/97320630015887. eCollection 2019.

Herpes simplex viruses (HSV) are alpha herpes viruses, which causes life-threatening illness. Therefore, it is of interest to design and develop potential drugs to treat HSV infections. We show the optimal molecular docking properties of a secondary metabolite (3,...

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Saran N, Anandharaj B, Bupesh G, et al. Molecular docking analysis of a secondary metabolite with the glycoprotein receptors of HSV 1 and HSV 2. Bioinformation. 2019;15(12):887-895doi: 10.6026/97320630015887.
Saran, N., Anandharaj, B., Bupesh, G., Vasanth, S., Beulah, J. P. J., & Balachandar, V. (2019). Molecular docking analysis of a secondary metabolite with the glycoprotein receptors of HSV 1 and HSV 2. Bioinformation, 15(12), 887-895. https://doi.org/10.6026/97320630015887
Saran, Natarajan, et al. "Molecular docking analysis of a secondary metabolite with the glycoprotein receptors of HSV 1 and HSV 2." Bioinformation vol. 15,12 (2019): 887-895. doi: https://doi.org/10.6026/97320630015887
Saran N, Anandharaj B, Bupesh G, Vasanth S, Beulah JPJ, Balachandar V. Molecular docking analysis of a secondary metabolite with the glycoprotein receptors of HSV 1 and HSV 2. Bioinformation. 2019 Dec 31;15(12):887-895. doi: 10.6026/97320630015887. eCollection 2019. PMID: 32256009; PMCID: PMC7088423.
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The impact of early life stress and immune challenge on behavior and glia cells alteration in late adolescent rats.

International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience

Réus GZ, Giridharan VV, de Moura AB, Borba LA, Botelho MEM, Behenck JP, Generoso JS, Selvaraj S, Bhatti G, Barichello T, Quevedo J.
PMID: 33788296
Int J Dev Neurosci. 2021 Aug;81(5):407-415. doi: 10.1002/jdn.10108. Epub 2021 Jun 22.

Maternal deprivation (MD) is known to be related to long-term changes that could influence the onset of psychiatric disorders. Studies have demonstrated that early life stress makes the cells in the brain more susceptible to subsequent stressors. To test...

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Réus GZ, Giridharan VV, de Moura AB, et al. The impact of early life stress and immune challenge on behavior and glia cells alteration in late adolescent rats. Int J Dev Neurosci. 2021;81(5):407-415doi: 10.1002/jdn.10108.
Réus, G. Z., Giridharan, V. V., de Moura, A. B., Borba, L. A., Botelho, M. E. M., Behenck, J. P., Generoso, J. S., Selvaraj, S., Bhatti, G., Barichello, T., & Quevedo, J. (2021). The impact of early life stress and immune challenge on behavior and glia cells alteration in late adolescent rats. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 81(5), 407-415. https://doi.org/10.1002/jdn.10108
Réus, Gislaine Z, et al. "The impact of early life stress and immune challenge on behavior and glia cells alteration in late adolescent rats." International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience vol. 81,5 (2021): 407-415. doi: https://doi.org/10.1002/jdn.10108
Réus GZ, Giridharan VV, de Moura AB, Borba LA, Botelho MEM, Behenck JP, Generoso JS, Selvaraj S, Bhatti G, Barichello T, Quevedo J. The impact of early life stress and immune challenge on behavior and glia cells alteration in late adolescent rats. Int J Dev Neurosci. 2021 Aug;81(5):407-415. doi: 10.1002/jdn.10108. Epub 2021 Jun 22. PMID: 33788296.
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Evaluation of soluble fibrin monomer complex in patients in SARS-CoV-2 COVID-19 infection-associated coagulopathy.

European journal of haematology

Sridharan M, D Navitskas S, M Kock E, E Houghton D, M Heikal N, Chen D, K Pruthi R.
PMID: 34921683
Eur J Haematol. 2021 Dec 18; doi: 10.1111/ejh.13738. Epub 2021 Dec 18.

Hospitalized patients with COVID-19 infection frequently have coagulopathy resembling disseminated intravascular coagulation (DIC). An elevation of D-dimer level is associated with a poor prognosis; however, the role of other fibrin degradation products, such as soluble fibrin monomers (SFMC), is...

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Sridharan M, D Navitskas S, M Kock E, et al. Evaluation of soluble fibrin monomer complex in patients in SARS-CoV-2 COVID-19 infection-associated coagulopathy. Eur J Haematol. 2021;doi: 10.1111/ejh.13738.
Sridharan, M., D Navitskas, S., M Kock, E., E Houghton, D., M Heikal, N., Chen, D., & K Pruthi, R. (2021). Evaluation of soluble fibrin monomer complex in patients in SARS-CoV-2 COVID-19 infection-associated coagulopathy. European journal of haematology, . https://doi.org/10.1111/ejh.13738
Sridharan, Meera, et al. "Evaluation of soluble fibrin monomer complex in patients in SARS-CoV-2 COVID-19 infection-associated coagulopathy." European journal of haematology vol. (2021). doi: https://doi.org/10.1111/ejh.13738
Sridharan M, D Navitskas S, M Kock E, E Houghton D, M Heikal N, Chen D, K Pruthi R. Evaluation of soluble fibrin monomer complex in patients in SARS-CoV-2 COVID-19 infection-associated coagulopathy. Eur J Haematol. 2021 Dec 18; doi: 10.1111/ejh.13738. Epub 2021 Dec 18. PMID: 34921683.
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Molecular docking analysis of zanamavir with haem agglutinin neuraminidase of human para influenza virus type 3.

Bioinformation

Indumathi CP, Bupesh G, Vasanth S, Senthilkumar V, Anandh AV, Pandian K.
PMID: 31831955
Bioinformation. 2019 Oct 31;15(10):730-734. doi: 10.6026/97320630015730. eCollection 2019.

The human para influenza virus (HPIV) type 3 is an imperative respiratory virus which cause upper and lower respiratory infections. The receptor involved in the viral infection is haem agglutinin neuraminidase. It is of interest to study the interaction...

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Indumathi CP, Bupesh G, Vasanth S, et al. Molecular docking analysis of zanamavir with haem agglutinin neuraminidase of human para influenza virus type 3. Bioinformation. 2019;15(10):730-734doi: 10.6026/97320630015730.
Indumathi, C. P., Bupesh, G., Vasanth, S., Senthilkumar, V., Anandh, A. V., & Pandian, K. (2019). Molecular docking analysis of zanamavir with haem agglutinin neuraminidase of human para influenza virus type 3. Bioinformation, 15(10), 730-734. https://doi.org/10.6026/97320630015730
Indumathi, Chinayan Palani, et al. "Molecular docking analysis of zanamavir with haem agglutinin neuraminidase of human para influenza virus type 3." Bioinformation vol. 15,10 (2019): 730-734. doi: https://doi.org/10.6026/97320630015730
Indumathi CP, Bupesh G, Vasanth S, Senthilkumar V, Anandh AV, Pandian K. Molecular docking analysis of zanamavir with haem agglutinin neuraminidase of human para influenza virus type 3. Bioinformation. 2019 Oct 31;15(10):730-734. doi: 10.6026/97320630015730. eCollection 2019. PMID: 31831955; PMCID: PMC6900320.
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Do Gestational Obesity and Gestational Diabetes Have an Independent Effect on Neonatal Adiposity? Results of Mediation Analysis from a Cohort Study in South India.

Clinical epidemiology

Babu GR, Deepa R, Lewis MG, Lobo E, Krishnan A, Ana Y, Katon JG, Enquobahrie DA, Arah OA, Kinra S, Murthy G.
PMID: 31920399
Clin Epidemiol. 2019 Dec 27;11:1067-1080. doi: 10.2147/CLEP.S222726. eCollection 2019.

PURPOSE: Neonates born to mothers with obesity or gestational diabetes mellitus (GDM) have an increased chance of various metabolic disorders later in life. In India, it is unclear whether maternal obesity or GDM is related to offspring adiposity. We...

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Babu GR, Deepa R, Lewis MG, et al. Do Gestational Obesity and Gestational Diabetes Have an Independent Effect on Neonatal Adiposity? Results of Mediation Analysis from a Cohort Study in South India. Clin Epidemiol. 2019;11:1067-1080doi: 10.2147/CLEP.S222726.
Babu, G. R., Deepa, R., Lewis, M. G., Lobo, E., Krishnan, A., Ana, Y., Katon, J. G., Enquobahrie, D. A., Arah, O. A., Kinra, S., & Murthy, G. (2019). Do Gestational Obesity and Gestational Diabetes Have an Independent Effect on Neonatal Adiposity? Results of Mediation Analysis from a Cohort Study in South India. Clinical epidemiology, 111067-1080. https://doi.org/10.2147/CLEP.S222726
Babu, Giridhara R, et al. "Do Gestational Obesity and Gestational Diabetes Have an Independent Effect on Neonatal Adiposity? Results of Mediation Analysis from a Cohort Study in South India." Clinical epidemiology vol. 11 (2019): 1067-1080. doi: https://doi.org/10.2147/CLEP.S222726
Babu GR, Deepa R, Lewis MG, Lobo E, Krishnan A, Ana Y, Katon JG, Enquobahrie DA, Arah OA, Kinra S, Murthy G. Do Gestational Obesity and Gestational Diabetes Have an Independent Effect on Neonatal Adiposity? Results of Mediation Analysis from a Cohort Study in South India. Clin Epidemiol. 2019 Dec 27;11:1067-1080. doi: 10.2147/CLEP.S222726. eCollection 2019. PMID: 31920399; PMCID: PMC6938184.
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Stanniocalcin 1 Inhibits the Inflammatory Response in Microglia and Protects Against Sepsis-Associated Encephalopathy.

Neurotoxicity research

Bonfante S, Joaquim L, Fileti ME, Giustina AD, de Souza Goldim MP, Danielski LG, Cittadin E, De Carli RJ, de Farias BX, Engel NA, da Rosa N, Fortunato JJ, Giridharan V, Scaini G, Rezin GT, Generoso J, de Bitencourt RM, Terra S, Barichello T, Petronilho F.
PMID: 33025358
Neurotox Res. 2021 Apr;39(2):119-132. doi: 10.1007/s12640-020-00293-y. Epub 2020 Oct 06.

Sepsis-associated encephalopathy is a serious consequence of sepsis, triggered by the host response against an infectious agent, that can lead to brain damage and cognitive impairment. Several mechanisms have been proposed in this bidirectional communication between the immune system...

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Bonfante S, Joaquim L, Fileti ME, et al. Stanniocalcin 1 Inhibits the Inflammatory Response in Microglia and Protects Against Sepsis-Associated Encephalopathy. Neurotox Res. 2020;39(2):119-132doi: 10.1007/s12640-020-00293-y.
Bonfante, S., Joaquim, L., Fileti, M. E., Giustina, A. D., de Souza Goldim, M. P., Danielski, L. G., Cittadin, E., De Carli, R. J., de Farias, B. X., Engel, N. A., da Rosa, N., Fortunato, J. J., Giridharan, V., Scaini, G., Rezin, G. T., Generoso, J., de Bitencourt, R. M., Terra, S., Barichello, T., & Petronilho, F. (2021). Stanniocalcin 1 Inhibits the Inflammatory Response in Microglia and Protects Against Sepsis-Associated Encephalopathy. Neurotoxicity research, 39(2), 119-132. https://doi.org/10.1007/s12640-020-00293-y
Bonfante, Sandra, et al. "Stanniocalcin 1 Inhibits the Inflammatory Response in Microglia and Protects Against Sepsis-Associated Encephalopathy." Neurotoxicity research vol. 39,2 (2021): 119-132. doi: https://doi.org/10.1007/s12640-020-00293-y
Bonfante S, Joaquim L, Fileti ME, Giustina AD, de Souza Goldim MP, Danielski LG, Cittadin E, De Carli RJ, de Farias BX, Engel NA, da Rosa N, Fortunato JJ, Giridharan V, Scaini G, Rezin GT, Generoso J, de Bitencourt RM, Terra S, Barichello T, Petronilho F. Stanniocalcin 1 Inhibits the Inflammatory Response in Microglia and Protects Against Sepsis-Associated Encephalopathy. Neurotox Res. 2021 Apr;39(2):119-132. doi: 10.1007/s12640-020-00293-y. Epub 2020 Oct 06. PMID: 33025358.
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An association between mitochondria and microglia effector function. What do we think we know?.

Neuroimmunology and neuroinflammation

Harry GJ, Childers G, Giridharan S, Hernandes IL.
PMID: 32934971
Neuroimmunol Neuroinflamm. 2020;7:150-165. doi: 10.20517/2347-8659.2020.07. Epub 2020 Jun 16.

While resident innate immune cells of the central nervous system, the microglia, represent a cell population unique in origin, microenvironment, and longevity, they assume many properties displayed by peripheral macrophages. One prominent shared property is the ability to undergo...

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Harry GJ, Childers G, Giridharan S, et al. An association between mitochondria and microglia effector function. What do we think we know?. Neuroimmunol Neuroinflamm. 2020;7:150-165doi: 10.20517/2347-8659.2020.07.
Harry, G. J., Childers, G., Giridharan, S., & Hernandes, I. L. (2020). An association between mitochondria and microglia effector function. What do we think we know?. Neuroimmunology and neuroinflammation, 7150-165. https://doi.org/10.20517/2347-8659.2020.07
Harry, G Jean, et al. "An association between mitochondria and microglia effector function. What do we think we know?." Neuroimmunology and neuroinflammation vol. 7 (2020): 150-165. doi: https://doi.org/10.20517/2347-8659.2020.07
Harry GJ, Childers G, Giridharan S, Hernandes IL. An association between mitochondria and microglia effector function. What do we think we know?. Neuroimmunol Neuroinflamm. 2020;7:150-165. doi: 10.20517/2347-8659.2020.07. Epub 2020 Jun 16. PMID: 32934971; PMCID: PMC7489447.
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