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Political issues in internal medicine in Europe. A position paper.

European journal of internal medicine

, Bauer W, Schumm-Draeger PM, Koebberling J, Gjoerup T, Garcia Alegria JJ, Ferreira F, Higgens C, Kramer M, Licata G, Mittelman M, O'hare J, Unal S.
PMID: 15967343
Eur J Intern Med. 2005 Jun;16(3):214-217. doi: 10.1016/j.ejim.2005.02.003.

What will be the future of internal medicine in Europe? Because of rapidly growing concerns regarding the position of internal medicine in many European countries, the European Federation of Internal Medicine (EFIM) has established a working group to analyze...

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, Bauer W, Schumm-Draeger PM, et al. Political issues in internal medicine in Europe. A position paper. Eur J Intern Med. 2005;16(3):214-217doi: 10.1016/j.ejim.2005.02.003.
, Bauer, W., Schumm-Draeger, P. M., Koebberling, J., Gjoerup, T., Garcia Alegria, J. J., Ferreira, F., Higgens, C., Kramer, M., Licata, G., Mittelman, M., O'hare, J., & Unal, S. (2005). Political issues in internal medicine in Europe. A position paper. European journal of internal medicine, 16(3), 214-217. https://doi.org/10.1016/j.ejim.2005.02.003
, et al. "Political issues in internal medicine in Europe. A position paper." European journal of internal medicine vol. 16,3 (2005): 214-217. doi: https://doi.org/10.1016/j.ejim.2005.02.003
, Bauer W, Schumm-Draeger PM, Koebberling J, Gjoerup T, Garcia Alegria JJ, Ferreira F, Higgens C, Kramer M, Licata G, Mittelman M, O'hare J, Unal S. Political issues in internal medicine in Europe. A position paper. Eur J Intern Med. 2005 Jun;16(3):214-217. doi: 10.1016/j.ejim.2005.02.003. PMID: 15967343.
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About the treatment of ostitis ossis temporalis chronica with simple resectio processus mastoid, sulphathiazole sealing and primary suturing.

Nordisk medicin

NORUP S.
PMID: 18874010
Nord Med. 1948 May 21;38(21):1049.

No abstract available.

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NORUP S. Om behandlingen af ostitis ossis temporalis chronica med simpel resectio processus mastoidei, sulfatiazolplombering og primaer suturering. About the treatment of ostitis ossis temporalis chronica with simple resectio processus mastoid, sulphathiazole sealing and primary suturing. Nord Med. 1948;38(21):1049
NORUP, S. (1948). Om behandlingen af ostitis ossis temporalis chronica med simpel resectio processus mastoidei, sulfatiazolplombering og primaer suturering. About the treatment of ostitis ossis temporalis chronica with simple resectio processus mastoid, sulphathiazole sealing and primary suturing. Nordisk medicin, 38(21), 1049.
NORUP, S. "Om behandlingen af ostitis ossis temporalis chronica med simpel resectio processus mastoidei, sulfatiazolplombering og primaer suturering." About the treatment of ostitis ossis temporalis chronica with simple resectio processus mastoid, sulphathiazole sealing and primary suturing. Nordisk medicin vol. 38,21 (1948): 1049.
NORUP S. Om behandlingen af ostitis ossis temporalis chronica med simpel resectio processus mastoidei, sulfatiazolplombering og primaer suturering. About the treatment of ostitis ossis temporalis chronica with simple resectio processus mastoid, sulphathiazole sealing and primary suturing. Nord Med. 1948 May 21;38(21):1049. Swedish. PMID: 18874010.
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Urodilatin reverses the detrimental influence of bradykinin in acute ischemic stroke.

Experimental neurology

Dobrivojević M, Špiranec K, Gorup D, Erjavec I, Habek N, Radmilović M, Unfirer S, Ćosić A, Drenjančević I, Gajović S, Sinđić A.
PMID: 27432758
Exp Neurol. 2016 Oct;284:1-10. doi: 10.1016/j.expneurol.2016.07.007. Epub 2016 Jul 16.

Occlusion of cerebral arteries leads to ischemic stroke accompanied by subsequent brain edema. Bradykinin (BK) is involved in the formation of cerebral edema, and natriuretic peptides (NPs) potentially have beneficial effects on brain edema formation via a still unknown...

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Dobrivojević M, Špiranec K, Gorup D, et al. Urodilatin reverses the detrimental influence of bradykinin in acute ischemic stroke. Exp Neurol. 2016;284:1-10doi: 10.1016/j.expneurol.2016.07.007.
Dobrivojević, M., Špiranec, K., Gorup, D., Erjavec, I., Habek, N., Radmilović, M., Unfirer, S., Ćosić, A., Drenjančević, I., Gajović, S., & Sinđić, A. (2016). Urodilatin reverses the detrimental influence of bradykinin in acute ischemic stroke. Experimental neurology, 2841-10. https://doi.org/10.1016/j.expneurol.2016.07.007
Dobrivojević, Marina, et al. "Urodilatin reverses the detrimental influence of bradykinin in acute ischemic stroke." Experimental neurology vol. 284 (2016): 1-10. doi: https://doi.org/10.1016/j.expneurol.2016.07.007
Dobrivojević M, Špiranec K, Gorup D, Erjavec I, Habek N, Radmilović M, Unfirer S, Ćosić A, Drenjančević I, Gajović S, Sinđić A. Urodilatin reverses the detrimental influence of bradykinin in acute ischemic stroke. Exp Neurol. 2016 Oct;284:1-10. doi: 10.1016/j.expneurol.2016.07.007. Epub 2016 Jul 16. PMID: 27432758.
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A Prospective Analysis of Plasma Phospholipid Fatty Acids and Breast Cancer Risk in 2 Provinces in Canada.

Current developments in nutrition

Newell M, Ghosh S, Goruk S, Pakseresht M, Vena JE, Dummer TJB, Field CJ.
PMID: 33889794
Curr Dev Nutr. 2021 Mar 25;5(4):nzab022. doi: 10.1093/cdn/nzab022. eCollection 2021 Apr.

BACKGROUND: Studies suggest that fatty acid status influences breast cancer etiology, yet the roles of individual fatty acids in breast cancer risk are unclear, specifically when central adiposity and menopausal status are considered.OBJECTIVES: This study examined the associations of...

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Newell M, Ghosh S, Goruk S, et al. A Prospective Analysis of Plasma Phospholipid Fatty Acids and Breast Cancer Risk in 2 Provinces in Canada. Curr Dev Nutr. 2021;5(4):nzab022doi: 10.1093/cdn/nzab022.
Newell, M., Ghosh, S., Goruk, S., Pakseresht, M., Vena, J. E., Dummer, T. J. B., & Field, C. J. (2021). A Prospective Analysis of Plasma Phospholipid Fatty Acids and Breast Cancer Risk in 2 Provinces in Canada. Current developments in nutrition, 5(4), nzab022. https://doi.org/10.1093/cdn/nzab022
Newell, Marnie, et al. "A Prospective Analysis of Plasma Phospholipid Fatty Acids and Breast Cancer Risk in 2 Provinces in Canada." Current developments in nutrition vol. 5,4 (2021): nzab022. doi: https://doi.org/10.1093/cdn/nzab022
Newell M, Ghosh S, Goruk S, Pakseresht M, Vena JE, Dummer TJB, Field CJ. A Prospective Analysis of Plasma Phospholipid Fatty Acids and Breast Cancer Risk in 2 Provinces in Canada. Curr Dev Nutr. 2021 Mar 25;5(4):nzab022. doi: 10.1093/cdn/nzab022. eCollection 2021 Apr. PMID: 33889794; PMCID: PMC8049855.
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Cerebellar radiological abnormalities in children with neurofibromatosis type 1: part 2 - a neuroimaging natural history study with clinical correlations.

Cerebellum & ataxias

Salman MS, Hossain S, Gorun S, Alqublan L, Bunge M, Rozovsky K.
PMID: 30410778
Cerebellum Ataxias. 2018 Oct 30;5:13. doi: 10.1186/s40673-018-0092-z. eCollection 2018.

BACKGROUND: Focal abnormal signal intensities (FASI) on brain MRI occur commonly in patients with neurofibromatosis type 1 (NF1). The natural history of cerebellar FASI and their correlation with clinical features have not been studied comprehensively. Our aims are to...

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Salman MS, Hossain S, Gorun S, et al. Cerebellar radiological abnormalities in children with neurofibromatosis type 1: part 2 - a neuroimaging natural history study with clinical correlations. Cerebellum Ataxias. 2018;5:13doi: 10.1186/s40673-018-0092-z.
Salman, M. S., Hossain, S., Gorun, S., Alqublan, L., Bunge, M., & Rozovsky, K. (2018). Cerebellar radiological abnormalities in children with neurofibromatosis type 1: part 2 - a neuroimaging natural history study with clinical correlations. Cerebellum & ataxias, 513. https://doi.org/10.1186/s40673-018-0092-z
Salman, Michael S, et al. "Cerebellar radiological abnormalities in children with neurofibromatosis type 1: part 2 - a neuroimaging natural history study with clinical correlations." Cerebellum & ataxias vol. 5 (2018): 13. doi: https://doi.org/10.1186/s40673-018-0092-z
Salman MS, Hossain S, Gorun S, Alqublan L, Bunge M, Rozovsky K. Cerebellar radiological abnormalities in children with neurofibromatosis type 1: part 2 - a neuroimaging natural history study with clinical correlations. Cerebellum Ataxias. 2018 Oct 30;5:13. doi: 10.1186/s40673-018-0092-z. eCollection 2018. PMID: 30410778; PMCID: PMC6208104.
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Df(h15q13)/+ Mouse Model Reveals Loss of Astrocytes and Synaptic-Related Changes of the Excitatory and Inhibitory Circuits in the Medial Prefrontal Cortex.

Cerebral cortex (New York, N.Y. : 1991)

Al-Absi AR, Qvist P, Glerup S, Sanchez C, Nyengaard JR.
PMID: 33123721
Cereb Cortex. 2021 Feb 05;31(3):1609-1621. doi: 10.1093/cercor/bhaa313.

The 15q13.3 deletion is associated with multiple neurodevelopmental disorders including epilepsy, schizophrenia, and autism. The Df(h15q13)/+ mouse model was recently generated that recapitulates several phenotypic features of the human 15q13.3 deletion syndrome (DS). However, the biological substrates underlying these...

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Al-Absi AR, Qvist P, Glerup S, et al. Df(h15q13)/+ Mouse Model Reveals Loss of Astrocytes and Synaptic-Related Changes of the Excitatory and Inhibitory Circuits in the Medial Prefrontal Cortex. Cereb Cortex. 2021;31(3):1609-1621doi: 10.1093/cercor/bhaa313.
Al-Absi, A. R., Qvist, P., Glerup, S., Sanchez, C., & Nyengaard, J. R. (2021). Df(h15q13)/+ Mouse Model Reveals Loss of Astrocytes and Synaptic-Related Changes of the Excitatory and Inhibitory Circuits in the Medial Prefrontal Cortex. Cerebral cortex (New York, N.Y. : 1991), 31(3), 1609-1621. https://doi.org/10.1093/cercor/bhaa313
Al-Absi, Abdel-Rahman, et al. "Df(h15q13)/+ Mouse Model Reveals Loss of Astrocytes and Synaptic-Related Changes of the Excitatory and Inhibitory Circuits in the Medial Prefrontal Cortex." Cerebral cortex (New York, N.Y. : 1991) vol. 31,3 (2021): 1609-1621. doi: https://doi.org/10.1093/cercor/bhaa313
Al-Absi AR, Qvist P, Glerup S, Sanchez C, Nyengaard JR. Df(h15q13)/+ Mouse Model Reveals Loss of Astrocytes and Synaptic-Related Changes of the Excitatory and Inhibitory Circuits in the Medial Prefrontal Cortex. Cereb Cortex. 2021 Feb 05;31(3):1609-1621. doi: 10.1093/cercor/bhaa313. PMID: 33123721.
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Characterization of Non-Small-Cell Lung Cancers With MET Exon 14 Skipping Alterations Detected in Tissue or Liquid: Clinicogenomics and Real-World Treatment Patterns.

JCO precision oncology

Lee JK, Madison R, Classon A, Gjoerup O, Rosenzweig M, Frampton GM, Alexander BM, Oxnard GR, Venstrom JM, Awad MM, Schrock AB.
PMID: 34476332
JCO Precis Oncol. 2021 Aug 25;5. doi: 10.1200/PO.21.00122. eCollection 2021.

METHODS: Hybrid capture-based comprehensive genomic profiling (CGP) was performed on samples from 69,219 patients with NSCLC. For treatment patterns and outcomes analysis, patients with advanced RESULTS: A total of 1,592 patients with NSCLC (2.3%) were identified with 1,599 CONCLUSION:...

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Lee JK, Madison R, Classon A, et al. Characterization of Non-Small-Cell Lung Cancers With MET Exon 14 Skipping Alterations Detected in Tissue or Liquid: Clinicogenomics and Real-World Treatment Patterns. JCO Precis Oncol. 2021;5doi: 10.1200/PO.21.00122.
Lee, J. K., Madison, R., Classon, A., Gjoerup, O., Rosenzweig, M., Frampton, G. M., Alexander, B. M., Oxnard, G. R., Venstrom, J. M., Awad, M. M., & Schrock, A. B. (2021). Characterization of Non-Small-Cell Lung Cancers With MET Exon 14 Skipping Alterations Detected in Tissue or Liquid: Clinicogenomics and Real-World Treatment Patterns. JCO precision oncology, 5. https://doi.org/10.1200/PO.21.00122
Lee, Jessica K, et al. "Characterization of Non-Small-Cell Lung Cancers With MET Exon 14 Skipping Alterations Detected in Tissue or Liquid: Clinicogenomics and Real-World Treatment Patterns." JCO precision oncology vol. 5 (2021). doi: https://doi.org/10.1200/PO.21.00122
Lee JK, Madison R, Classon A, Gjoerup O, Rosenzweig M, Frampton GM, Alexander BM, Oxnard GR, Venstrom JM, Awad MM, Schrock AB. Characterization of Non-Small-Cell Lung Cancers With MET Exon 14 Skipping Alterations Detected in Tissue or Liquid: Clinicogenomics and Real-World Treatment Patterns. JCO Precis Oncol. 2021 Aug 25;5. doi: 10.1200/PO.21.00122. eCollection 2021. PMID: 34476332; PMCID: PMC8407654.
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A novel enema method can prevent infectious complications of transrectal ultrasound-guided prostate biopsy: A single-centre experience.

International journal of clinical practice

Gokalp F, Koras O, Gursoy D, Sigva H, Porgali SB, Tamkac N, Kulak B, Ucurmak F, Gorur S.
PMID: 34580970
Int J Clin Pract. 2021 Sep 27;e14923. doi: 10.1111/ijcp.14923. Epub 2021 Sep 27.

BACKGROUND: TRUS biopsy is the preferred method for diagnosing prostate cancer, but it can cause infectious complications that arise with the contribution of fluoroquinolone resistance. We aimed to explore the potential protective effect of a second rectal enema before...

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Gokalp F, Koras O, Gursoy D, et al. A novel enema method can prevent infectious complications of transrectal ultrasound-guided prostate biopsy: A single-centre experience. Int J Clin Pract. 2021;e14923doi: 10.1111/ijcp.14923.
Gokalp, F., Koras, O., Gursoy, D., Sigva, H., Porgali, S. B., Tamkac, N., Kulak, B., Ucurmak, F., & Gorur, S. (2021). A novel enema method can prevent infectious complications of transrectal ultrasound-guided prostate biopsy: A single-centre experience. International journal of clinical practice, e14923. https://doi.org/10.1111/ijcp.14923
Gokalp, Fatih, et al. "A novel enema method can prevent infectious complications of transrectal ultrasound-guided prostate biopsy: A single-centre experience." International journal of clinical practice vol. (2021): e14923. doi: https://doi.org/10.1111/ijcp.14923
Gokalp F, Koras O, Gursoy D, Sigva H, Porgali SB, Tamkac N, Kulak B, Ucurmak F, Gorur S. A novel enema method can prevent infectious complications of transrectal ultrasound-guided prostate biopsy: A single-centre experience. Int J Clin Pract. 2021 Sep 27;e14923. doi: 10.1111/ijcp.14923. Epub 2021 Sep 27. PMID: 34580970.
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Prevalence of JP2 and Non-JP2 Genotypes of .

Pathogens (Basel, Switzerland)

Haubek D, Mulli T, Kemoli A, Lindholm M, Gjørup H, Nørregaard MM, Johansson A.
PMID: 33920549
Pathogens. 2021 Apr 17;10(4). doi: 10.3390/pathogens10040488.

No abstract available.

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Haubek D, Mulli T, Kemoli A, et al. Prevalence of JP2 and Non-JP2 Genotypes of . Pathogens. 2021;10(4)doi: 10.3390/pathogens10040488.
Haubek, D., Mulli, T., Kemoli, A., Lindholm, M., Gjørup, H., Nørregaard, M. M., & Johansson, A. (2021). Prevalence of JP2 and Non-JP2 Genotypes of . Pathogens (Basel, Switzerland), 10(4), . https://doi.org/10.3390/pathogens10040488
Haubek, Dorte, et al. "Prevalence of JP2 and Non-JP2 Genotypes of ." Pathogens (Basel, Switzerland) vol. 10,4 (2021). doi: https://doi.org/10.3390/pathogens10040488
Haubek D, Mulli T, Kemoli A, Lindholm M, Gjørup H, Nørregaard MM, Johansson A. Prevalence of JP2 and Non-JP2 Genotypes of . Pathogens. 2021 Apr 17;10(4). doi: 10.3390/pathogens10040488. PMID: 33920549; PMCID: PMC8073413.
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SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization.

Cell reports

Dai C, Rennhack JP, Arnoff TE, Thaker M, Younger ST, Doench JG, Huang AY, Yang A, Aguirre AJ, Wang B, Mun E, O'Connell JT, Huang Y, Labella K, Talamas JA, Li J, Ilic N, Hwang J, Hong AL, Giacomelli AO, Gjoerup O, Root DE, Hahn WC.
PMID: 34320363
Cell Rep. 2021 Jul 27;36(4):109443. doi: 10.1016/j.celrep.2021.109443.

Metastasis is a complex and poorly understood process. In pancreatic cancer, loss of the transforming growth factor (TGF)-β/BMP effector SMAD4 is correlated with changes in altered histopathological transitions, metastatic disease, and poor prognosis. In this study, we use isogenic...

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Dai C, Rennhack JP, Arnoff TE, et al. SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell Rep. 2021;36(4):109443doi: 10.1016/j.celrep.2021.109443.
Dai, C., Rennhack, J. P., Arnoff, T. E., Thaker, M., Younger, S. T., Doench, J. G., Huang, A. Y., Yang, A., Aguirre, A. J., Wang, B., Mun, E., O'Connell, J. T., Huang, Y., Labella, K., Talamas, J. A., Li, J., Ilic, N., Hwang, J., Hong, A. L., Giacomelli, A. O., Gjoerup, O., Root, D. E., & Hahn, W. C. (2021). SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell reports, 36(4), 109443. https://doi.org/10.1016/j.celrep.2021.109443
Dai, Chao, et al. "SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization." Cell reports vol. 36,4 (2021): 109443. doi: https://doi.org/10.1016/j.celrep.2021.109443
Dai C, Rennhack JP, Arnoff TE, Thaker M, Younger ST, Doench JG, Huang AY, Yang A, Aguirre AJ, Wang B, Mun E, O'Connell JT, Huang Y, Labella K, Talamas JA, Li J, Ilic N, Hwang J, Hong AL, Giacomelli AO, Gjoerup O, Root DE, Hahn WC. SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell Rep. 2021 Jul 27;36(4):109443. doi: 10.1016/j.celrep.2021.109443. PMID: 34320363; PMCID: PMC8350598.
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SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization.

Cell reports

Dai C, Rennhack JP, Arnoff TE, Thaker M, Younger ST, Doench JG, Huang AY, Yang A, Aguirre AJ, Wang B, Mun E, O'Connell JT, Huang Y, Labella K, Talamas JA, Li J, Ilic N, Hwang J, Hong AL, Giacomelli AO, Gjoerup O, Root DE, Hahn WC.
PMID: 34320363
Cell Rep. 2021 Jul 27;36(4):109443. doi: 10.1016/j.celrep.2021.109443.

Metastasis is a complex and poorly understood process. In pancreatic cancer, loss of the transforming growth factor (TGF)-β/BMP effector SMAD4 is correlated with changes in altered histopathological transitions, metastatic disease, and poor prognosis. In this study, we use isogenic...

Cite
Dai C, Rennhack JP, Arnoff TE, et al. SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell Rep. 2021;36(4):109443doi: 10.1016/j.celrep.2021.109443.
Dai, C., Rennhack, J. P., Arnoff, T. E., Thaker, M., Younger, S. T., Doench, J. G., Huang, A. Y., Yang, A., Aguirre, A. J., Wang, B., Mun, E., O'Connell, J. T., Huang, Y., Labella, K., Talamas, J. A., Li, J., Ilic, N., Hwang, J., Hong, A. L., Giacomelli, A. O., Gjoerup, O., Root, D. E., & Hahn, W. C. (2021). SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell reports, 36(4), 109443. https://doi.org/10.1016/j.celrep.2021.109443
Dai, Chao, et al. "SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization." Cell reports vol. 36,4 (2021): 109443. doi: https://doi.org/10.1016/j.celrep.2021.109443
Dai C, Rennhack JP, Arnoff TE, Thaker M, Younger ST, Doench JG, Huang AY, Yang A, Aguirre AJ, Wang B, Mun E, O'Connell JT, Huang Y, Labella K, Talamas JA, Li J, Ilic N, Hwang J, Hong AL, Giacomelli AO, Gjoerup O, Root DE, Hahn WC. SMAD4 represses FOSL1 expression and pancreatic cancer metastatic colonization. Cell Rep. 2021 Jul 27;36(4):109443. doi: 10.1016/j.celrep.2021.109443. PMID: 34320363; PMCID: PMC8350598.
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[Medical records--clinical aspects].

Nordisk medicin

Gjørup T.
PMID: 3562213
Nord Med. 1987;102(3):95-7.

No abstract available.

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Gjørup T. Den medicinske journal--klinikerens syndbuk. [Medical records--clinical aspects]. Nord Med. 1987;102(3):95-7
Gjørup, T. (1987). Den medicinske journal--klinikerens syndbuk. [Medical records--clinical aspects]. Nordisk medicin, 102(3), 95-7.
Gjørup, T. "Den medicinske journal--klinikerens syndbuk." [Medical records--clinical aspects]. Nordisk medicin vol. 102,3 (1987): 95-7.
Gjørup T. Den medicinske journal--klinikerens syndbuk. [Medical records--clinical aspects]. Nord Med. 1987;102(3):95-7. Danish. PMID: 3562213.
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