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Prognostic value of comprehensive intracoronary physiology assessment early after heart transplantation.

European heart journal

Ahn JM, Zimmermann FM, Arora S, Solberg OG, Angerås O, Rolid K, Rafique M, Aaberge L, Karason K, Okada K, Luikart H, Khush KK, Honda Y, Pijls NHJ, Lee SE, Kim JJ, Park SJ, Gullestad L, Fearon WF.
PMID: 34665224
Eur Heart J. 2021 Dec 21;42(48):4918-4929. doi: 10.1093/eurheartj/ehab568.

AIMS: We evaluated the long-term prognostic value of invasively assessing coronary physiology after heart transplantation in a large multicentre registry.METHODS AND RESULTS: Comprehensive intracoronary physiology assessment measuring fractional flow reserve (FFR), the index of microcirculatory resistance (IMR), and coronary...

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Ahn JM, Zimmermann FM, Arora S, et al. Prognostic value of comprehensive intracoronary physiology assessment early after heart transplantation. Eur Heart J. 2021;42(48):4918-4929doi: 10.1093/eurheartj/ehab568.
Ahn, J. M., Zimmermann, F. M., Arora, S., Solberg, O. G., Angerås, O., Rolid, K., Rafique, M., Aaberge, L., Karason, K., Okada, K., Luikart, H., Khush, K. K., Honda, Y., Pijls, N. H. J., Lee, S. E., Kim, J. J., Park, S. J., Gullestad, L., & Fearon, W. F. (2021). Prognostic value of comprehensive intracoronary physiology assessment early after heart transplantation. European heart journal, 42(48), 4918-4929. https://doi.org/10.1093/eurheartj/ehab568
Ahn, Jung-Min, et al. "Prognostic value of comprehensive intracoronary physiology assessment early after heart transplantation." European heart journal vol. 42,48 (2021): 4918-4929. doi: https://doi.org/10.1093/eurheartj/ehab568
Ahn JM, Zimmermann FM, Arora S, Solberg OG, Angerås O, Rolid K, Rafique M, Aaberge L, Karason K, Okada K, Luikart H, Khush KK, Honda Y, Pijls NHJ, Lee SE, Kim JJ, Park SJ, Gullestad L, Fearon WF. Prognostic value of comprehensive intracoronary physiology assessment early after heart transplantation. Eur Heart J. 2021 Dec 21;42(48):4918-4929. doi: 10.1093/eurheartj/ehab568. PMID: 34665224; PMCID: PMC8691805.
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Cytokines as new treatment targets in chronic heart failure.

Current controlled trials in cardiovascular medicine

Damås JK, Gullestad L, Aukrust P.
PMID: 11806813
Curr Control Trials Cardiovasc Med. 2001;2(6):271-277. doi: 10.1186/cvm-2-6-271.

Inflammatory cytokines may negatively influence contractility and contribute to the remodelling process in the failing myocardium. Traditional cardiovascular drugs appear to have little influence on the overall cytokine network in chronic heart failure (CHF). Increased interest in anticytokine therapy...

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Damås JK, Gullestad L, Aukrust P. Cytokines as new treatment targets in chronic heart failure. Curr Control Trials Cardiovasc Med. 2001;2(6):271-277doi: 10.1186/cvm-2-6-271.
Damås, J. K., Gullestad, L., & Aukrust, P. (2001). Cytokines as new treatment targets in chronic heart failure. Current controlled trials in cardiovascular medicine, 2(6), 271-277. https://doi.org/10.1186/cvm-2-6-271
Damås, Jan Kristian, et al. "Cytokines as new treatment targets in chronic heart failure." Current controlled trials in cardiovascular medicine vol. 2,6 (2001): 271-277. doi: https://doi.org/10.1186/cvm-2-6-271
Damås JK, Gullestad L, Aukrust P. Cytokines as new treatment targets in chronic heart failure. Curr Control Trials Cardiovasc Med. 2001;2(6):271-277. doi: 10.1186/cvm-2-6-271. PMID: 11806813; PMCID: PMC64832.
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The tumor necrosis factor superfamily in heart failure.

Future cardiology

Ueland T, Aukrust P, Damas JK, Gullestad L, Yndestad A.
PMID: 19804137
Future Cardiol. 2006 Jan;2(1):101-11. doi: 10.2217/14796678.2.1.101.

Numerous clinical studies have established that tumor necrosis factor (TNF)-alpha may play a pathogenic role in the development and progression of heart failure (HF). Recent reports suggest that other ligands in the TNF superfamily could also play a pathogenic...

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Ueland T, Aukrust P, Damas JK, et al. The tumor necrosis factor superfamily in heart failure. Future Cardiol. 2006;2(1):101-11doi: 10.2217/14796678.2.1.101.
Ueland, T., Aukrust, P., Damas, J. K., Gullestad, L., & Yndestad, A. (2006). The tumor necrosis factor superfamily in heart failure. Future cardiology, 2(1), 101-11. https://doi.org/10.2217/14796678.2.1.101
Ueland, T, et al. "The tumor necrosis factor superfamily in heart failure." Future cardiology vol. 2,1 (2006): 101-11. doi: https://doi.org/10.2217/14796678.2.1.101
Ueland T, Aukrust P, Damas JK, Gullestad L, Yndestad A. The tumor necrosis factor superfamily in heart failure. Future Cardiol. 2006 Jan;2(1):101-11. doi: 10.2217/14796678.2.1.101. PMID: 19804137.
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Quantification of subcortical gray-matter vascularization using 7 Tesla time-of-flight angiography.

Brain and behavior

Laurig M, Liu X, Wyss M, Gietl A, Jellestad L, Nitsch RM, Prüssmann K, Hock C, Unschuld PG.
PMID: 24392272
Brain Behav. 2013 Sep;3(5):515-8. doi: 10.1002/brb3.154. Epub 2013 Jul 14.

Background The integrity of subcortical brain nuclei is associated with maintenance of regular cognitive performance levels and has been shown to be particularly affected by aging-related vascular pathology. This study aims to demonstrate applicability of high field strength magnetic...

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Laurig M, Liu X, Wyss M, et al. Quantification of subcortical gray-matter vascularization using 7 Tesla time-of-flight angiography. Brain Behav. 2013;3(5):515-8doi: 10.1002/brb3.154.
Laurig, M., Liu, X., Wyss, M., Gietl, A., Jellestad, L., Nitsch, R. M., Prüssmann, K., Hock, C., & Unschuld, P. G. (2013). Quantification of subcortical gray-matter vascularization using 7 Tesla time-of-flight angiography. Brain and behavior, 3(5), 515-8. https://doi.org/10.1002/brb3.154
Laurig, Mathias, et al. "Quantification of subcortical gray-matter vascularization using 7 Tesla time-of-flight angiography." Brain and behavior vol. 3,5 (2013): 515-8. doi: https://doi.org/10.1002/brb3.154
Laurig M, Liu X, Wyss M, Gietl A, Jellestad L, Nitsch RM, Prüssmann K, Hock C, Unschuld PG. Quantification of subcortical gray-matter vascularization using 7 Tesla time-of-flight angiography. Brain Behav. 2013 Sep;3(5):515-8. doi: 10.1002/brb3.154. Epub 2013 Jul 14. PMID: 24392272; PMCID: PMC3869979.
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Exercise after heart transplantation: An overview.

World journal of transplantation

Nytrøen K, Gullestad L.
PMID: 24392312
World J Transplant. 2013 Dec 24;3(4):78-90. doi: 10.5500/wjt.v3.i4.78.

While life expectancy is greatly improved after a heart transplant, survival is still limited, and compared to the general population, the exercise capacity and health-related quality of life of heart transplant recipients are reduced. Increased exercise capacity is associated...

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Nytrøen K, Gullestad L. Exercise after heart transplantation: An overview. World J Transplant. 2013;3(4):78-90doi: 10.5500/wjt.v3.i4.78.
Nytrøen, K., & Gullestad, L. (2013). Exercise after heart transplantation: An overview. World journal of transplantation, 3(4), 78-90. https://doi.org/10.5500/wjt.v3.i4.78
Nytrøen, Kari, and Gullestad, Lars. "Exercise after heart transplantation: An overview." World journal of transplantation vol. 3,4 (2013): 78-90. doi: https://doi.org/10.5500/wjt.v3.i4.78
Nytrøen K, Gullestad L. Exercise after heart transplantation: An overview. World J Transplant. 2013 Dec 24;3(4):78-90. doi: 10.5500/wjt.v3.i4.78. PMID: 24392312; PMCID: PMC3879527.
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Transcription and replication result in distinct epigenetic marks following repression of early gene expression.

Frontiers in genetics

Kallestad L, Woods E, Christensen K, Gefroh A, Balakrishnan L, Milavetz B.
PMID: 23914205
Front Genet. 2013 Jul 30;4:140. doi: 10.3389/fgene.2013.00140. eCollection 2013.

Simian virus 40 (SV40) early transcription is repressed when the product of early transcription, T-antigen, binds to its cognate regulatory sequence, Site I, in the promoter of the SV40 minichromosome. Because SV40 minichromosomes undergo replication and transcription potentially repression...

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Kallestad L, Woods E, Christensen K, et al. Transcription and replication result in distinct epigenetic marks following repression of early gene expression. Front Genet. 2013;4:140doi: 10.3389/fgene.2013.00140.
Kallestad, L., Woods, E., Christensen, K., Gefroh, A., Balakrishnan, L., & Milavetz, B. (2013). Transcription and replication result in distinct epigenetic marks following repression of early gene expression. Frontiers in genetics, 4140. https://doi.org/10.3389/fgene.2013.00140
Kallestad, Les, et al. "Transcription and replication result in distinct epigenetic marks following repression of early gene expression." Frontiers in genetics vol. 4 (2013): 140. doi: https://doi.org/10.3389/fgene.2013.00140
Kallestad L, Woods E, Christensen K, Gefroh A, Balakrishnan L, Milavetz B. Transcription and replication result in distinct epigenetic marks following repression of early gene expression. Front Genet. 2013 Jul 30;4:140. doi: 10.3389/fgene.2013.00140. eCollection 2013. PMID: 23914205; PMCID: PMC3728471.
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Results of comprehensive diagnostic work-up in 'idiopathic' dilated cardiomyopathy.

Open heart

Broch K, Andreassen AK, Hopp E, Leren TP, Scott H, Müller F, Aakhus S, Gullestad L.
PMID: 26468400
Open Heart. 2015 Oct 09;2(1):e000271. doi: 10.1136/openhrt-2015-000271. eCollection 2015.

OBJECTIVE: Dilated cardiomyopathy (DCM) is characterised by left ventricular dilation and dysfunction not caused by coronary disease, valvular disease or hypertension. Owing to the considerable aetiological and prognostic heterogeneity in DCM, an extensive diagnostic work-up is recommended. We aimed...

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Broch K, Andreassen AK, Hopp E, et al. Results of comprehensive diagnostic work-up in 'idiopathic' dilated cardiomyopathy. Open Heart. 2015;2(1):e000271doi: 10.1136/openhrt-2015-000271.
Broch, K., Andreassen, A. K., Hopp, E., Leren, T. P., Scott, H., Müller, F., Aakhus, S., & Gullestad, L. (2015). Results of comprehensive diagnostic work-up in 'idiopathic' dilated cardiomyopathy. Open heart, 2(1), e000271. https://doi.org/10.1136/openhrt-2015-000271
Broch, Kaspar, et al. "Results of comprehensive diagnostic work-up in 'idiopathic' dilated cardiomyopathy." Open heart vol. 2,1 (2015): e000271. doi: https://doi.org/10.1136/openhrt-2015-000271
Broch K, Andreassen AK, Hopp E, Leren TP, Scott H, Müller F, Aakhus S, Gullestad L. Results of comprehensive diagnostic work-up in 'idiopathic' dilated cardiomyopathy. Open Heart. 2015 Oct 09;2(1):e000271. doi: 10.1136/openhrt-2015-000271. eCollection 2015. PMID: 26468400; PMCID: PMC4600247.
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Serum PCSK9 is modified by interleukin-6 receptor antagonism in patients with hypercholesterolaemia following non-ST-elevation myocardial infarction.

Open heart

Ueland T, Kleveland O, Michelsen AE, Wiseth R, Damås JK, Aukrust P, Gullestad L, Halvorsen B, Yndestad A.
PMID: 30258647
Open Heart. 2018 Sep 18;5(2):e000765. doi: 10.1136/openhrt-2017-000765. eCollection 2018.

OBJECTIVE: It is unclear if activation of inflammatory pathways regulates proprotein convertase subtilisin-kexin type 9 (PCSK9) levels.APPROACH: We evaluated (1) the temporal course of serum PCSK9 during hospitalisation following acute coronary syndrome and associations with markers of inflammation (leucocyte...

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Ueland T, Kleveland O, Michelsen AE, et al. Serum PCSK9 is modified by interleukin-6 receptor antagonism in patients with hypercholesterolaemia following non-ST-elevation myocardial infarction. Open Heart. 2018;5(2):e000765doi: 10.1136/openhrt-2017-000765.
Ueland, T., Kleveland, O., Michelsen, A. E., Wiseth, R., Damås, J. K., Aukrust, P., Gullestad, L., Halvorsen, B., & Yndestad, A. (2018). Serum PCSK9 is modified by interleukin-6 receptor antagonism in patients with hypercholesterolaemia following non-ST-elevation myocardial infarction. Open heart, 5(2), e000765. https://doi.org/10.1136/openhrt-2017-000765
Ueland, Thor, et al. "Serum PCSK9 is modified by interleukin-6 receptor antagonism in patients with hypercholesterolaemia following non-ST-elevation myocardial infarction." Open heart vol. 5,2 (2018): e000765. doi: https://doi.org/10.1136/openhrt-2017-000765
Ueland T, Kleveland O, Michelsen AE, Wiseth R, Damås JK, Aukrust P, Gullestad L, Halvorsen B, Yndestad A. Serum PCSK9 is modified by interleukin-6 receptor antagonism in patients with hypercholesterolaemia following non-ST-elevation myocardial infarction. Open Heart. 2018 Sep 18;5(2):e000765. doi: 10.1136/openhrt-2017-000765. eCollection 2018. PMID: 30258647; PMCID: PMC6150185.
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Effect of high-intensity interval training in young heart transplant recipients: results from two randomized controlled trials.

BMC sports science, medicine & rehabilitation

Nytrøen K, Rolid K, Yardley M, Gullestad L.
PMID: 32518655
BMC Sports Sci Med Rehabil. 2020 Jun 04;12:35. doi: 10.1186/s13102-020-00180-1. eCollection 2020.

BACKGROUND: Little is known about the effect of exercise in young heart transplant recipients, and results on group level is lacking. This study summarizes the findings of the youngest participants in two previous randomized controlled trials.METHOD: This is a...

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Nytrøen K, Rolid K, Yardley M, et al. Effect of high-intensity interval training in young heart transplant recipients: results from two randomized controlled trials. BMC Sports Sci Med Rehabil. 2020;12:35doi: 10.1186/s13102-020-00180-1.
Nytrøen, K., Rolid, K., Yardley, M., & Gullestad, L. (2020). Effect of high-intensity interval training in young heart transplant recipients: results from two randomized controlled trials. BMC sports science, medicine & rehabilitation, 1235. https://doi.org/10.1186/s13102-020-00180-1
Nytrøen, Kari, et al. "Effect of high-intensity interval training in young heart transplant recipients: results from two randomized controlled trials." BMC sports science, medicine & rehabilitation vol. 12 (2020): 35. doi: https://doi.org/10.1186/s13102-020-00180-1
Nytrøen K, Rolid K, Yardley M, Gullestad L. Effect of high-intensity interval training in young heart transplant recipients: results from two randomized controlled trials. BMC Sports Sci Med Rehabil. 2020 Jun 04;12:35. doi: 10.1186/s13102-020-00180-1. eCollection 2020. PMID: 32518655; PMCID: PMC7271535.
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Importance of physical capacity and the effects of exercise in heart transplant recipients.

World journal of transplantation

Yardley M, Gullestad L, Nytrøen K.
PMID: 29507857
World J Transplant. 2018 Feb 24;8(1):1-12. doi: 10.5500/wjt.v8.i1.1.

One of the most important prognostic factors in heart failure patients is physical capacity. Patients with very poor physical performance and otherwise eligible, may be listed as candidates for heart transplantation (HTx). After such surgery, life-long immunosuppression therapy is...

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Yardley M, Gullestad L, Nytrøen K. Importance of physical capacity and the effects of exercise in heart transplant recipients. World J Transplant. 2018;8(1):1-12doi: 10.5500/wjt.v8.i1.1.
Yardley, M., Gullestad, L., & Nytrøen, K. (2018). Importance of physical capacity and the effects of exercise in heart transplant recipients. World journal of transplantation, 8(1), 1-12. https://doi.org/10.5500/wjt.v8.i1.1
Yardley, Marianne, et al. "Importance of physical capacity and the effects of exercise in heart transplant recipients." World journal of transplantation vol. 8,1 (2018): 1-12. doi: https://doi.org/10.5500/wjt.v8.i1.1
Yardley M, Gullestad L, Nytrøen K. Importance of physical capacity and the effects of exercise in heart transplant recipients. World J Transplant. 2018 Feb 24;8(1):1-12. doi: 10.5500/wjt.v8.i1.1. PMID: 29507857; PMCID: PMC5829450.
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Human Adipose-Derived Mesenchymal Stem Cells Respond to Short-Term Hypoxia by Secreting Factors Beneficial for Human Islets In Vitro and Potentiate Antidiabetic Effect In Vivo.

Cell medicine

Schive SW, Mirlashari MR, Hasvold G, Wang M, Josefsen D, Gullestad HP, Korsgren O, Foss A, Kvalheim G, Scholz H.
PMID: 28713640
Cell Med. 2017 Apr 14;9(3):103-116. doi: 10.3727/215517917X693401. eCollection 2017.

Adipose-derived mesenchymal stem cells (ASCs) release factors beneficial for islets in vitro and protect against hyperglycemia in rodent models of diabetes. Oxygen tension has been shown to induce metabolic changes and alter ASCs' release of soluble factors. The effects...

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Schive SW, Mirlashari MR, Hasvold G, et al. Human Adipose-Derived Mesenchymal Stem Cells Respond to Short-Term Hypoxia by Secreting Factors Beneficial for Human Islets In Vitro and Potentiate Antidiabetic Effect In Vivo. Cell Med. 2017;9(3):103-116doi: 10.3727/215517917X693401.
Schive, S. W., Mirlashari, M. R., Hasvold, G., Wang, M., Josefsen, D., Gullestad, H. P., Korsgren, O., Foss, A., Kvalheim, G., & Scholz, H. (2017). Human Adipose-Derived Mesenchymal Stem Cells Respond to Short-Term Hypoxia by Secreting Factors Beneficial for Human Islets In Vitro and Potentiate Antidiabetic Effect In Vivo. Cell medicine, 9(3), 103-116. https://doi.org/10.3727/215517917X693401
Schive, Simen W, et al. "Human Adipose-Derived Mesenchymal Stem Cells Respond to Short-Term Hypoxia by Secreting Factors Beneficial for Human Islets In Vitro and Potentiate Antidiabetic Effect In Vivo." Cell medicine vol. 9,3 (2017): 103-116. doi: https://doi.org/10.3727/215517917X693401
Schive SW, Mirlashari MR, Hasvold G, Wang M, Josefsen D, Gullestad HP, Korsgren O, Foss A, Kvalheim G, Scholz H. Human Adipose-Derived Mesenchymal Stem Cells Respond to Short-Term Hypoxia by Secreting Factors Beneficial for Human Islets In Vitro and Potentiate Antidiabetic Effect In Vivo. Cell Med. 2017 Apr 14;9(3):103-116. doi: 10.3727/215517917X693401. eCollection 2017. PMID: 28713640; PMCID: PMC5509020.
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Clinical features and determinants of VO.

World journal of transplantation

Rolid K, Andreassen AK, Yardley M, Bjørkelund E, Karason K, Wigh JP, Dall CH, Gustafsson F, Gullestad L, Nytrøen K.
PMID: 30211027
World J Transplant. 2018 Sep 10;8(5):188-197. doi: 10.5500/wjt.v8.i5.188.

AIM: To study exercise capacity and determinants of early peak oxygen consumption (VOMETHODS: To determine possible central (chronotropic responses, cardiopulmonary and hemodynamic function) and peripheral factors (muscular exercise capacity and body composition) predictive of VORESULTS: The mean ± standard...

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Rolid K, Andreassen AK, Yardley M, et al. Clinical features and determinants of VO. World J Transplant. 2018;8(5):188-197doi: 10.5500/wjt.v8.i5.188.
Rolid, K., Andreassen, A. K., Yardley, M., Bjørkelund, E., Karason, K., Wigh, J. P., Dall, C. H., Gustafsson, F., Gullestad, L., & Nytrøen, K. (2018). Clinical features and determinants of VO. World journal of transplantation, 8(5), 188-197. https://doi.org/10.5500/wjt.v8.i5.188
Rolid, Katrine, et al. "Clinical features and determinants of VO." World journal of transplantation vol. 8,5 (2018): 188-197. doi: https://doi.org/10.5500/wjt.v8.i5.188
Rolid K, Andreassen AK, Yardley M, Bjørkelund E, Karason K, Wigh JP, Dall CH, Gustafsson F, Gullestad L, Nytrøen K. Clinical features and determinants of VO. World J Transplant. 2018 Sep 10;8(5):188-197. doi: 10.5500/wjt.v8.i5.188. PMID: 30211027; PMCID: PMC6134270.
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Showing 25 to 36 of 58 entries